ライフサイエンスコーパス: conduction velocity

1 longer than non-VT channels, and have slower conduction velocity.

2 evertheless, there is a 38% reduction in PNS conduction velocity.

3 h surrounding neural structures to determine conduction velocity.

4 a 2:1 atrioventricular block, and slowing of conduction velocity.

5 bit impaired motor function and slower nerve conduction velocity.

6 poral coordinate transformation via measured conduction velocity.

7 eralgesia, cold allodynia, and sciatic nerve conduction velocity.

8 nant of the actual value of action potential conduction velocity.

9 of the auditory nerve and results in altered conduction velocity.

10 the effects of levosimendan on muscle fiber conduction velocity.

11 n turn, has a significant influence on nerve conduction velocity.

12 nderlie the observed modifications in axonal conduction velocity.

13 Neurons were categorized by conduction velocity.

14 s were analyzed for amplitude, duration, and conduction velocity.

15 lin outfoldings that impair the normal nerve conduction velocity.

16 rnodes along axons altering action potential conduction velocity.

17 increased gadolinium uptake has lower local conduction velocity.

18 tive effects on maximal phase 0 upstroke and conduction velocity.

19 daptations in axon myelination for increased conduction velocity.

20 coordination deficits and exhibit decreased conduction velocity.

21 -9)), indicating that INPP4B regulates nerve conduction velocity.

22 d stable structures with highly reproducible conduction velocities.

23 ties and displayed a mild reduction of nerve conduction velocities.

24 set and the degree of slowing of motor nerve conduction velocities.

25 xons with impaired radial growth and reduced conduction velocities.

26 ceps muscle contractility, and sciatic nerve conduction velocities.

27 sarcomeric structure, action potentials and conduction velocities.

28 omeres, smaller action potentials, and lower conduction velocities.

29 xons, delayed myelination, and reduced nerve conduction velocities.

30 ge from capsaicin-sensitive fibers with slow conduction velocities (0.4-0.7 m/s), which was inhibited

31 , bipolar EGM amplitude (0.08+/-0.11 mV) and conduction velocity (0.27+/-0.19 m/s) were lower than th

32 In 1D tissue with normal longitudinal conduction velocity (0.55 m/s), the numbers of contiguou

33 ime (103+/-14 versus 43+/-13 ms), and slower conduction velocity (0.87+/-0.23 versus 1.39+/-0.21 m/s)

34 Single fields of 39 afferents (conduction velocity = 0.27-3.65 m s(-1)) were identified

35 MI-Fb CM treatment decreased conduction velocity (11.1%) compared to untreated myocyt

36 discontinuity (qualitative) and decrease in conduction velocity (13%) and electrogram amplitude (21%

37 nduction velocity=3.1+/-0.1 cm/s) or strong (conduction velocity=22.1+/-0.4 cm/s) electrical coupling

38 icular myocytes (AP duration=153.2+/-2.3 ms, conduction velocity=22.3+/-0.3 cm/s) seamlessly interfac

39 es, (2) greater slowing of motor and sensory conduction velocities, (3) less prolonged motor distal l

40 sodium (Na(v)1.5) channels with either weak (conduction velocity=3.1+/-0.1 cm/s) or strong (conductio

41 /- 1 ms; p < 0.001), and slower longitudinal conduction velocity (62 +/- 2 cm/s vs. 70 +/- 1 cm/s; p

42 is reached, beyond which no further gains in conduction velocity accrue.

43 Conduction velocity across BB is around 90 cm/s.

44 axonal degeneration and may lead to impaired conduction velocity across surviving axons after stroke.

45 Simultaneous optical Vm mapping showed that conduction velocity, action potential duration, and dVm/

46 r, since the effect of age on the changes in conduction velocity after transection and after hypoxia

47 ulthood, resulting in a 30% reduction in the conduction velocity along the adult sciatic nerves.

48 The enhancement of conduction velocity along with the evolution of jaws lik

49 ts further demonstrated spatially discordant conduction velocity alternans which resulted in nonunifo

50 travelling wave and from which instantaneous conduction velocity amplitude and direction can be compu

51 action potential amplitudes, improved nerve conduction velocities and ameliorated muscle strength.

52 stigate the role of cellular architecture on conduction velocities and anisotropy ratios.

53 Mechanisms responsible for the slowed conduction velocities and axonal loss in Charcot-Marie-T

54 rving spatially uniform properties with high conduction velocities and contractile stresses.

55 We sought to validate omnipole-derived conduction velocities and explore potential application

56 tensity in 78 dye-injected DRG neurons whose conduction velocities and hindlimb sensory receptive fie

57 /R98C) mice develop weakness, abnormal nerve conduction velocities and morphologically abnormal myeli

58 ochondrial DNA deletions, reduction of nerve conduction velocities and of heat sensation, and bone mi

59 a half theta cycle) than expected from axon conduction velocities and passive synaptic integration o

60 sease is characterized by uniform slowing of conduction velocities and secondary axonal loss, which a

61 cular myocyte monolayers demonstrated slowed conduction velocity and a reduced maximum capture rate a

62 CM treatment or fibroblast plating to obtain conduction velocity and action potential duration (APD(7

63 r2.1 gene-modified monolayers showed altered conduction velocity and action potential duration compar

64 with an island of altered Kir2.1 expression, conduction velocity and action potential duration of the

65 he interstitial space surrounding a fiber on conduction velocity and action potential waveshape.

66 tural changes were associated with decreased conduction velocity and conduction block.

67 density, A Pupstroke velocity, AP duration, conduction velocity and EAD incidence, as well as reflec

68 The local conduction velocity and image intensity ratio in the lef

69 eficits were associated with decreased nerve conduction velocity and increased sensory threshold to m

70 positively correlated with decreasing nerve conduction velocity and increasing pain severity.

71 examine the association between left atrial conduction velocity and LGE in patients with atrial fibr

72 c-Src inhibition improved Cx43 levels and conduction velocity and lowered arrhythmia inducibility

73 is best known for its role in increasing the conduction velocity and metabolic efficiency of long-ran

74 Such dysregulations led to defects in conduction velocity and motor and sensory functions that

75 al neuropathy characterized by reduced nerve conduction velocity and myelin outfoldings and infolding

76 Axon myelination increases the conduction velocity and precision of action potential pr

77 Nerve conduction velocity and psychophysical data were acquire

78 mpanied by decreased motor and sensory nerve conduction velocity and reduced compound muscle action p

79 argeted manipulation of Cx43 levels improved conduction velocity and reduced ventricular tachycardia

80 as nociceptors or non-nociceptors based upon conduction velocity and response to transient receptor p

81 ficiency or proportionate increases in their conduction velocity and support the view that the intern

82 ion potential timing is shaped by the axonal conduction velocity and the duration of synaptic transmi

83 a(V)1.5, plays a pivotal role in setting the conduction velocity and the initial depolarization of th

84 e interpeak latency, indicative of increased conduction velocity and thereby enhanced functional repa

85 Conduction velocity and threshold for alternans monotoni

86 icted that this geometry helps to adjust the conduction velocity and timing of action potentials with

87 atrial and left atrial voltage distribution, conduction velocities, and electrogram characteristics w

88 c nerve conduction delays depend on RGC axon conduction velocities, and velocity is primarily determi

89 velocity, as assessed by the SDs of APD and conduction velocity, and atrial fibrillation inducibilit

90 e the main determinant of axonal caliber and conduction velocity, and demonstrate for the first time

91 well as magnetic field correlates of the AP conduction velocity, and directly determines the AP prop

92 longitudinal conduction velocity, transverse conduction velocity, and dV/dt(max) in fibrotic monolaye

93 width helps to control transmitter release, conduction velocity, and firing patterns and understandi

94 ling such as arrhythmias, decreased neuronal conduction velocity, and hyporeflexia.

95 Longitudinal conduction velocity, transverse conduction velocity, and normalized action potential ups

96 urprisingly, cell geometry, action potential conduction velocity, and the expression of a cell-cell c

97 een connexin43 (Cx43) expression, myocardial conduction velocity, and ventricular tachycardia in a mo

98 the 62 capsaicin-sensitive C-fibres studied (conduction velocity approximately 0.5 m s(-1)), 71% were

99 pacing (n=11), flecainide reversibly reduced conduction velocity ( approximately 30% at cycle length

100 ength, no significant change was detected in conduction velocity ( approximately 43 m/s) measured eit

101 of nerves reach a plateau beyond which their conduction velocities are no longer sensitive to increas

102 Predictions that conduction velocities are sensitive to the distance betw

103 In postinfarction reentrant VT, conduction velocities are slowest at the proximal and di

104 uch as restitutions of refractoriness and of conduction velocity are given via experimentally measure

105 host axons and restored nodes of Ranvier and conduction velocity as efficiently as CNS-derived precur

106 This study examined AP rise times and conduction velocity as the depolarizing wavefront approa

107 P increased spatial heterogeneity of APD and conduction velocity, as assessed by the SDs of APD and c

108 ons, and increased dispersion of ventricular conduction velocities at fast heart rates.

109 ons, and increased dispersion of ventricular conduction velocities at fast heart rates.

110 d Cx40 expression on sinus node function and conduction velocity at different pacing cycle lengths (1

111 The local conduction velocity at each point was calculated using p

112 matical modeling predicted a 25% decrease in conduction velocity at the lesion epicenter due to short

113 PP1-treated mice had restored conduction velocity at the scar border (PP3: 32 cm/s, PP

114 ral neuropathy characterized by slowed nerve conduction velocity, axon loss, and distinctive myelin o

115 amples are provided, showing a difference in conduction velocity between two different types of cardi

116 DS AND We found that motor and sensory nerve conduction velocities, blood flow, and capillary density

117 al frontal cortex and from reductions in the conduction velocity brought about by the shorter interno

118 with respect to their sensory modalities and conduction velocities, but also in their relative roles

119 fibers, specifically sural or sciatic nerve conduction velocities, but significantly improved measur

120 ination is not only important for maximizing conduction velocity, but also for limiting hyperexcitabi

121 stically increased the spatial dispersion of conduction velocity, but not the action potential durati

122 altered Na(v)1.5 function can reduce cardiac conduction velocity by 28% compared with control.

123 Myelin sheath length directly impacts axonal conduction velocity by influencing the spacing between n

124 Conduction velocities calculated from omnipolar electrog

125 Axonal conduction velocity can change substantially during ongo

126 roduced significant deficits in caudal nerve conduction velocity, caudal amplitude and digital nerve

127 esults in more rapid AP upstrokes and higher conduction velocities compared with the bulk myocardium.

128 ocytes with WT and mutant channels increased conduction velocity compared with noninfected cells.

129 e with severe PAD have poorer peroneal nerve conduction velocity compared with people with mild PAD o

130 termine atrial effective refractory periods, conduction velocity, conduction heterogeneity index, and

131 cantly higher myelinated fiber densities and conduction velocities consequent to acute sciatic nerve

132 stigate how these changes in myelination and conduction velocity contribute to signal integration in

133 e function, we measured unmyelinated C-fiber conduction velocities (CV) in nerves of SIV-infected mac

134 t sites exhibiting rate-dependent slowing in conduction velocity (CV restitution) or local slowing ev

135 f phase from each other were correlated with conduction velocity (CV) alternation in LMC but not in L

136 othesis that alterations to action potential conduction velocity (CV) and conduction anisotropy in le

137 fy the effects of heterocellular coupling on conduction velocity (CV) and reentry dynamics.

138 (Tyrode's solution), which acts to increase conduction velocity (CV) close to the tissue-fluid inter

139 showed slight (7%) reduction of ventricular conduction velocity (CV) compared to 16 wild-type hearts

140 udied the effect of interstitial fibrosis on conduction velocity (CV) in the left atrial appendage of

141 Reduced conduction velocity (CV) in the myocardium is well known

142 approach produces anisotropic cultures with conduction velocity (CV) profiles that closer resemble n

143 We investigated the role of conduction velocity (CV) restitution and short-term memo

144 ion potential duration (APD) restitution and conduction velocity (CV) restitution curves in these pat

145 properties as assessed through S1-S2 APD and conduction velocity (CV) restitution curves with differe

146 (STM) (alpha, tau) and their interplay with conduction velocity (CV) restitution on alternans format

147 ive refractory period (ERP) restitution, and conduction velocity (CV) restitution.Temporal and spatia

148 transient duration was prolonged, and local conduction velocity (CV) slowed progressively, with loss

149 Evaluation included atrial voltage, conduction velocity (CV), and refractoriness (7 sites, 2

150 pendency of I(Kr) enables it to control APD, conduction velocity (CV), and wavelength (WL) at the exc

151 Effects of mutations on AP duration (APD), conduction velocity (CV), effective refractory period (E

152 letal myotubes produced localized slowing of conduction velocity (CV), increased dispersion of CV and

153 ), has been proposed as a key determinant of conduction velocity (CV).

154 SHR hearts had slower conduction velocity (CV; P<0.01 versus WKY), steeper CV

155 Ms) and as having C-, Adelta- or Aalpha/beta-conduction velocities (CVs).

156 observed that, in addition to reduced nerve conduction velocity, db/db mice also develop PNH.

157 than uninfected animals and the magnitude of conduction velocity decline correlated strongly with ext

158 Conduction velocity decreased from 0.46 +/- 0.02 m/sec t

159 , tactile allodynia, motor and sensory nerve conduction velocity deficits, and reduction in tibial ne

160 Axon conduction velocities differed across groups and were sl

161 propagation, action potential duration, and conduction velocity), disrupted by IR injury, were resto

162 reased gap junction lateralization and lower conduction velocity due to downregulation of Connexin 43

163 nal current, it does not significantly alter conduction velocity during cardiac fibrosis relative to

164 restoration of action potential duration and conduction velocity early after reperfusion.

165 ocities individually to achieve the specific conduction velocities essential for precise temporal int

166 Although myelination and conduction velocities eventually recovered, polyaxonal m

167 activity levels, and axonal myelination and conduction velocity exhibited no adaptations.

168 KEY POINTS: Changes in nerve conduction velocity following an impulse (i.e. velocity

169 tic agents and a moderate reduction of nerve conduction velocity for cisplatin and paclitaxel.

170 ome, but not healthy controls, sensory nerve conduction velocity for Digits 2 and 3 was slower than D

171 t was less negative, and slower upstroke and conduction velocity for rotors in the PV region than in

172 1) in all hearts, and reduced the transmural conduction velocity from 36 cm/s (95% CI, 30-42) to 32 c

173 ervention and training-induced changes in MU conduction velocity had an effect size of 2.1 (tracked M

174 n essential role in establishing interatrial conduction velocity heterogeneity in the murine model.

175 Here we measured nerve conduction velocities in a rabbit model of limb lengthen

176 are in contrast with non-uniform slowing of conduction velocities in acquired demyelinating disorder

177 lso reveal that the reduction of motor nerve conduction velocities in affected patients is proportion

178 C differentiation and functional recovery of conduction velocities in demyelinated axons.

179 rdings in vitro and in vivo confirmed higher conduction velocities in low-frequency axons.

180 ner axonal diameters and internodal lengths, conduction velocities in mutant quadriceps nerves were a

181 VIP shortened APD and slowed conduction velocity in a dose-dependent manner.

182 In contrast, beta2-stimulation increased conduction velocity in both donor and failing hearts but

183 a novel hypothesis explaining the slowing of conduction velocity in C-fibres by the build-up of Na(+)

184 x of peripheral neuropathy is the slowing of conduction velocity in large myelinated neurons and a la

185 Conduction velocity in RMCCs was slower when compared wi

186 ted macaques had significantly lower C-fiber conduction velocity in sural nerves than uninfected anim

187 coupling led to slow electrical propagation; conduction velocity in TBX18 NRCMs slowed by more than 5

188 d a marked reduction in the left ventricular conduction velocity in the absence of myocardial Cav1, w

189 Nevertheless, conduction velocity in the adult RVOT was similar to tha

190 ad less electrogram fractionation and faster conduction velocity in the anterior-septal border zone.

191 Conduction velocity in the OEC transplant group was incr

192 ciated with a significant reduction in nerve conduction velocity in the optic nerve.

193 Conduction velocity in the visual pathways correlated cl

194 ansfected cells and sensory neurons, slowing conduction velocity in unmyelinated peripheral nerve fib

195 es encoding molecular determinants of atrial conduction velocity, including Scn5a (Nav1.5) and Gja5 (

196 50 mum from the epicardial surface, whereas conduction velocity increased and AP duration was only s

197 d from participating in propagation, and the conduction velocity increases.

198 two collaterals of a single axon adjust the conduction velocities individually to achieve the specif

199 ty may be because of the oscillations of the conduction velocity inside the tachycardia circuits.

200 Heterogeneity in regional conduction velocities is a critical substrate for functi

201 athy, but high activity-dependent slowing of conduction velocity is more common in fibromyalgia patie

202 in addition to decreased heart rate, atrial conduction velocity is persistently slower than control.

203 and bidomain models shows that although the conduction velocity is relatively insensitive to cases t

204 We also find that conduction velocity is relatively insensitive to gap jun

205 se and that alterations in myelin can modify conduction velocity, leading to changes in neural circui

206 ents with E90K and N98S had upper limb motor conduction velocities <38 m/s.

207 chniques, such as electromyography and nerve conduction velocity measurement, do not reliably predict

208 onal loss, which underlie slowed motor nerve conduction velocity (MNCV) and reduced compound muscle a

209 h streptozotocin, and changes in motor nerve conduction velocity (MNCV), mechanical and thermal hypoa

210 tary locomotor activity, reduced motor nerve conduction velocities (MNCVs) and muscle atrophy.

211 s R98C animals, but it did not improve nerve conduction velocity, myelin thickness, G-ratios or myeli

212 plantation significantly improved tail nerve conduction velocity, Na(+)-K(+)-ATPase activity, and mor

213 n either behaviors or acute changes in nerve conduction velocity (NCV) and amplitude, but greater und

214 They develop nerve conduction velocity (NCV) and sensory deficits prior to

215 Nerve conduction velocity (NCV) significantly improved in only

216 A values were correlated to electrical nerve conduction velocities (NCVs) with Pearson correlation an

217 nerve myelinated fiber density (MFD), nerve conduction velocities (NCVs), vibration perception thres

218 cts of each drug on caudal and digital nerve conduction velocity, nerve amplitude, and sciatic nerve

219 fibre types differ in degree of myelination, conduction velocity, neuropeptide content, sensitivity t

220 ons seem to be needed, not for their greater conduction velocity (nor other intrinsic electrophysiolo

221 axonal and somatodendritic compartments with conduction velocities of approximately 5 m s(-1) and app

222 uding Rin and lambda as well as waveform and conduction velocities of fully propagating action potent

223 We measured conduction velocities of the ipsilateral and contralater

224 In addition, axon density was reduced and conduction velocities of the trigeminal and sciatic nerv

225 normalized the cisplatin-induced decrease in conduction velocity of Aalpha/Abeta-fibers and reduced t

226 hysiological measurements indicated that the conduction velocity of earthworm medial giant nerve fibe

227 Axonal caliber and motor neuron conduction velocity of mice expressing KSP phospho-incom

228 cal modeling predicted a 21% decrease in the conduction velocity of remyelinated RST axons attributab

229 mucosal stroking, but increased the apparent conduction velocity of the CMMC.

230 ulation significantly increased peak INa and conduction velocity of WT but not mutant channels.

231 ies have demonstrated a strong dependence of conduction velocity on internodal length.

232 No difference was seen at baseline for conduction velocity or dispersion of repolarization betw

233 or control groups: greater slowing in atrial conduction velocity (P<0.0001 and P<0.001); increased ab

234 group had modest but significant slowing in conduction velocity (P<0.01) but no change in conduction

235 image intensity ratio (0.20 m/s decrease in conduction velocity per increase in unit image intensity

236 Increased axonal diameter did not increase conduction velocity, possibly due to a failure to increa

237 on in compound muscle action potentials, low conduction velocities, prolonged distal latencies and pr

238 d resting membrane hyperpolarization, slowed conduction velocity, prolonged action potential (AP) dur

239 tion, fibrosis, increased vagal tone, slowed conduction velocity, prolonged cardiomyocyte action pote

240 ed to a greater inverse correlation (r) with conduction velocity (r for adipose=0.39, r for discontin

241 ternodes lengthened during postnatal growth, conduction velocities recovered to normal values and mut

242 f R98C/+ mice but does not alter the reduced conduction velocities, reduced axonal diameters or clini

243 Heterocellular coupling alters conduction velocity, reentry stability, and complexity o

244 lation creates arrhythmogenic substrates via conduction velocity regulation and transmurally heteroge

245 preparation showed significant reduction in conduction velocity relative to control (cisplatin pretr

246 Conduction velocity remained constant at chemically incr

247 entity, peripheral anatomy, central anatomy, conduction velocity, response properties in vitro and in

248 bility per se, but instead influences axonal conduction velocity, resting membrane potential, and noc

249 on potential duration restitution (APDR) and conduction velocity restitution (CVR) curves were genera

250 The mechanism of DEEPs relates to conduction velocity restitution magnified by zigzag cond

251 on of fibrillation by modulating APD and APD/conduction velocity restitution slopes in atrial tissue

252 entry by prolonging APD and changing APD and conduction velocity restitution slopes, thereby altering

253 By analyzing the effects of conduction velocity restitution, cellular dynamics, elec

254 All strategies prevented loss of nerve conduction velocity resulting from STZ-induced diabetes

255 al, P=0.0005; left atrial, P=0.0001), slower conduction velocities (right atrial, P=0.02; left atrial

256 ernodal lengths and associated reduced nerve conduction velocity seen in the absence of periaxin, sho

257 beta1-Stimulation significantly increased conduction velocity, shortened action potential duration

258 thermia attenuated both the ischemia-induced conduction velocity slowing (decreasing from 0.44 +/- 0.

259 -1 and (R)-1 partially prevented motor nerve conduction velocity slowing in a mouse model of type 1 d

260 evented focal energy deprivation, transverse conduction velocity slowing, and the reentrant ventricul

261 er EGM amplitude with a significantly slower conduction velocity than the surrounding parts of the ci

262 dependent linear gradation in sensory axonal conduction velocities that can ensure the coincident arr

263 ch a mechanism would allow for variations in conduction velocities that provide a degree of plasticit

264 Diabetes caused a decrease in nerve conduction velocity, thermal hypoalgesia, and a reductio

265 ed high-fat diets caused a decrease in nerve conduction velocity, thermal hypoalgesia, and intraepide

266 Conduction velocities (theta'), action potential wavelen

267 43 redistribution to the lateral membrane on conduction velocity (theta) and anisotropic ratio, and h

268 reased OPC maturation and promoted increased conduction velocities through lesions.

269 ectric uncoupling increased excitability and conduction velocity to 133% in 28% hMSC cocultures, but

270 The average conduction velocity to the Sp5I neurones was 0.94 +/- 0.

271 Longitudinal conduction velocity, transverse conduction velocity, and

272 ptomycin dramatically increased longitudinal conduction velocity, transverse conduction velocity, and

273 itigate the frequency-dependent decreases in conduction velocity typical of C-fiber axons.SIGNIFICANC

274 Mean effective conduction velocity was 89 cm/s.

275 nd adjusting for left atrial wall thickness, conduction velocity was associated with the local image

276 Median conduction velocity was higher (75 +/- 9 versus 65 +/- 1

277 Also, conduction velocity was lower in the RVOT than in the ri

278 Conduction velocity was measured within RMCCs and compar

279 the spatial profile of hERG-GFP expression; conduction velocity was not altered.

280 A marked decrease in conduction velocity was observed in strands composed of

281 of DeltaSIV hearts revealed that ventricular conduction velocity was preferentially decreased in the

282 mice with short mutant Schwann cells, nerve conduction velocity was reduced and motor function was i

283 ing revealed that selectively the transverse conduction velocity was slowed and anisotropy increased.

284 The conduction velocity was slower than that reported in nor

285 In heterocellular cultures, MI-Fb conduction velocities were different from Fb at all dens

286 Conduction velocities were higher and action potential d

287 n in the placebo controls (P=0.015), and the conduction velocities were significantly faster (P=0.005

288 Conduction velocities were slowest at the inward curvatu

289 Transmural dispersion of repolarization and conduction velocity were measured at baseline, during is

290 The action potential waveform and axonal conduction velocity were unaffected, suggesting that the

291 Abnormally high slowing of conduction velocity when first stimulated at 0.25Hz was

292 o reentry were harbored in regions with slow conduction velocity, where 3 isochrones were present wit

293 array platform and thereby the estimation of conduction velocity, which gradually increased during th

294 in a progressive slowing of action potential conduction velocity, which manifests as a progressive in

295 ial heterogeneity and decreases intra-atrial conduction velocity, which may play an important role in

296 ity mechanism for dynamically fine-tuning AP conduction velocity, which potentially has wide implicat

297 ) showed an age-dependent increase of axonal conduction velocity, which was positively correlated wit

298 nerability, shortened ERP and altered atrial conduction velocity, which, in combination, facilitate i

299 Conduction velocities within the shared isthmus were dep

300 nment pacing causes some perturbation of the conduction velocity within the tachycardia circuit, whic