ライフサイエンスコーパス: conduit vessel

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1 ase is largely considered to be a disease of conduit vessels.

2 ferences exist in the functional behavior of conduit vessels.

3 low-mediated dilation of stenotic epicardial conduit vessels and may account at least in part for the

4 s that connect with and induce remodeling of conduit vessels arising from the abdominal wall circulat

5 No significant difference in conduit-vessel dilation was evident between OSA patients

6 homocysteine levels produces resistance and conduit vessel endothelial dysfunction and that this occ

7 Conduit vessel endothelial function was assessed with fl

8 Conduit-vessel function was also evaluated by ultrasonog

9 mproves endothelial function in the coronary conduit vessels of animals; however, its effects on the

10 ne levels are associated with alterations in conduit vessel properties that differ in men and women.

11 ) and coronary artery diameter (a measure of conduit vessel reactivity) were investigated in response

12 verapamil and NTG were unchanged, as was the conduit vessel response to NTG.

13 sublingual nitroglycerin (NTG) was recorded (conduit vessel response), and in 6 subjects, venous occl

14 ere is increasing evidence of a link between conduit vessel stiffness and cardiovascular events, alth

15 We assessed changes in pulse pressure and conduit vessel stiffness in a 12-week double-blind, rand

16 Increased conduit vessel stiffness increases pulse pressure and pu

17 ve cohorts, which suggests that reduction of conduit vessel stiffness may be desirable in hypertensio

18 between pulse pressure, which is related to conduit vessel stiffness, and subsequent cardiovascular

19 Increased conduit vessel stiffness, as assessed by pulse pressure,

20 Pulse pressure, a correlate of conduit vessel stiffness, has been shown to be an import

21 Increased pulse pressure, an indicator of conduit vessel stiffness, is a strong independent predic

22 arterial endothelium in both resistance and conduit vessels to both physical and pharmacological dil

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