ライフサイエンスコーパス: cone pigment

1 e could regenerate rod pigment but not green cone pigment.

2 s correlated with abnormal distribution of a cone pigment.

3 10 weeks and reversed the mislocalization of cone pigment.

4 hes activating more than approximately 1% of cone pigment.

5 icant increases in mRNA levels for the green cone pigment.

6 e opsin shift between rhodopsin and the blue cone pigment.

7 Nrl(-/-) mice are conelike and contain only cone pigments.

8 is-retinal, the chromophore of rhodopsin and cone pigments.

9 11-cis-retinal chromophore of rhodopsin and cone pigments.

10 lengths are the lambda(max) of the two mouse cone pigments.

11 this methyl group in the salamander rod and cone pigments.

12 the extent of phosphorylation of unbleached cone pigments.

13 11-cis-retinal chromophore of rhodopsin and cone pigments.

14 l shift between the mouse UV and bovine blue cone pigments.

15 ajor role in the spectral tuning of the SWS1 cone pigments.

16 dulation in avian short-wavelength sensitive cone pigments.

17 iously affecting the operation of the native cone pigments.

18 uts primarily from cones with mixed M- and S-cone pigments.

19 arhodopsin II formation and decay in rod and cone pigments.

20 sms during the evolution of rodent long-wave cone pigments.

21 gment) and a second near 510 nm [midwave (M)-cone pigment].

22 hosphodiesterase inhibitory subunit gamma to cone pigment, 1:68, was similar to the levels observed f

23 The single substitution in the dolphin LWS cone pigment (292S to 292A) causes a red shift from the

24 On the other hand, with the rhodopsin and UV cone pigments, activation was diminished.

25 ozen sections immunolabeled for the mouse UV-cone pigment and colabeled with PNA.

26 epresents the first example of a dark-active cone pigment and constitutively active cone opsin.

27 f the meta-II state (active conformation) of cone pigment and its higher rate of spontaneous isomeriz

28 results in comparing thermal stability of UV cone pigment and rhodopsin provide insight into molecula

29 (-/-) double knock-out model, trafficking of cone pigments and membrane-associated cone phototransduc

30 sharp contrast to its rod counterpart, bound cone pigments and non-visual receptors.

31 tionship between the properties of mammalian cone pigments and those of mammalian cones is not well u

32 e photopigments, one peaking near 350 nm (UV-cone pigment) and a second near 510 nm [midwave (M)-cone

33 which recognizes chicken rhodopsin and green cone pigment, and by reverse transcription-polymerase ch

34 retching frequencies of rhodopsin, the green cone pigment, and the red cone pigment in H2O (D2O) are

35 dopsin numbering) of the tiger salamander UV cone pigment appears to be trapped in an open conformati

36 mechanisms that enable rapid regeneration of cone pigment are poorly understood.

37 te is not a universal attribute of long-wave cone pigments as generally supposed, and that, depending

38 ility of the chromophore within the deep red cone pigment binding sites.

39 igment belongs to the long-wave subfamily of cone pigments, but its absorption maximum is 508 nm, sim

40 ifts the spectral maxima of the red and blue cone pigments, but not that of the red rod pigment.

41 rt-wave cone opsin (S-opsin) to test whether cone pigment can substitute for the structural and funct

42 Ultraviolet (UV) cone pigments can provide insights into the molecular ev

43 Nrl(-/-) photoreceptors express the mouse UV cone pigment, cone transducin, and cone arrestin in amou

44 Retinal rod and cone pigments consist of an apoprotein, opsin, covalentl

45 Interestingly, the red cone pigment containing the retinal analogue remained ac

46 Cone pigment dissociation therefore contributes to the s

47 the initial rapid regeneration of mouse M/L-cone pigment during dark adaptation, whereas the slower

48 wn that rod arrestin can bind and deactivate cone pigments efficiently, the results suggest that cone

49 type-specific expression of the zebrafish UV cone pigment gene by transient expression of green fluor

50 intercalated gene within the red-green opsin cone pigment gene tandem array on Xq28.

51 some to create the present-day red and green cone pigment genes.

52 spontaneous isomerization activity of human cone pigments has long remained a mystery because the ef

53 Differences in properties between rod and cone pigments have been described, such as a 10-fold sho

54 n spectra of recombinant human green and red cone pigments have been obtained to examine the molecula

55 rity of mammalian short-wavelength sensitive cone pigments have shifted their absorption maxima from

56 iff base (SB) linkage, but only UV-sensitive cone pigments have this moiety unprotonated in the dark.

57 odopsin, the green cone pigment, and the red cone pigment in H2O (D2O) are found at 1656 (1623), 1640

58 estion by expressing human or salamander red cone pigment in Xenopus rods, and human rod pigment in X

59 the chromophore to regenerate rhodopsin and cone pigments in vivo.

60 The homology models of seven cone pigments indicate that the deep red cone pigments s

61 The canonical visual cycle for rod and cone pigments involves recycling of their chromophore fr

62 Inactivation of meta II in SWS1 cone pigments is regulated by the primary counterion.

63 However, red cone pigment isomerizes spontaneously 10,000 times more

64 mographic spectral analysis of 294 A1 and A2 cone pigment literature absorption maxima indicates that

65 ith concomitant decreases in levels of green cone pigment mRNA.

66 cis retinal in solution, suggesting that the cone pigment noise results from isomerization of the ret

67 embrane domain were observed in the X-linked cone pigment of bush babies but not in other primates.

68 The blue-sensitive cone pigments of the SWS2 class cluster into two species

69 Thus, IRBP does not accelerate cone pigment regeneration and is not critical for the fu

70 Notably, the rates of cone pigment regeneration by the retina and pigment epit

71 demonstrating the interplay between rod and cone pigment regeneration driven by the retinal pigment

72 designed to keep up with the high demand for cone pigment regeneration in bright light and to preclud

73 used transretinal recordings to evaluate M/L-cone pigment regeneration in isolated retinas and eyecup

74 to underlie the Rpe65-/- phenotype, although cone pigment regeneration may be dependent on a separate

75 This novel interdependence of rod and cone pigment regeneration should be considered when deve

76 slowing of foveal visual acuity recovery and cone pigment regeneration, which are related to each oth

77 The time constants of cone pigment regeneration, which averaged 172 seconds fo

78 ssic RPE-dependent visual cycle to mammalian cone pigment regeneration.

79 chromophore, plays in accelerating mammalian cone pigment regeneration.

80 eloping cone dystrophy caused by inefficient cone pigment regeneration.

81 75% of the observed blue shift of the violet cone pigment relative to rhodopsin.

82 sin is 10(4)-fold lower than that of rod and cone pigments, resulting in a very low photon catch and

83 n of rhodopsin numbering) in the dolphin LWS cone pigment results in a blue shift in absorption maxim

84 ssociation, apparently a general property of cone pigments, results in a surprisingly large amount of

85 l and signaling properties of the short-wave cone pigment (S-pigment) contribute to the specialized f

86 uires neither the short-wavelength-sensitive cone pigment [S-pigment or cone opsin (OPN1SW)] nor ence

87 he first transgenic model expressed a murine cone pigment, S-opsin, together with the endogenous rhod

88 ven cone pigments indicate that the deep red cone pigments select 6- s- trans chromophore conformatio

89 e theoretically the hypothesis that deep red cone pigments select a 6- s- trans conformation of the r

90 Diversification of cone pigment spectral sensitivities during evolution is

91 Conversely, non-visual arrestin-2 bound cone pigments, suggesting that it may also regulate phot

92 ied 12 amino acid residues in the human blue cone pigment that might induce the required green-to-blu

93 However, for the red cone pigment, the 9-methyl group of retinal appears to b

94 catfish orthologues of rhodopsin and the red cone pigment-the full complement of retinal opsins in th

95 tants of Siberian hamster ultraviolet (SHUV) cone pigment to explore structural rearrangements that s

96 ing the measured quantal noise of transgenic cone pigment to native human red cones, we obtained a da

97 uction, starting distally, but rhodopsin and cone pigments trafficked normally for more than 2 weeks,

98 lls expressed SV40 T antigen, blue and green cone pigments, transducin, and cone arrestin.

99 vercome this problem by expressing human red cone pigment transgenically in mouse rods in order to ex

100 of retinal from a short-wavelength-sensitive cone pigment (VCOP) in 0.1% dodecyl maltoside using fluo

101 A third type of cone pigment was added to dichromatic retinas, providing

102 G protein-coupled receptor kinase 1, whereas cone pigments were present at reduced levels.

103 hydroxylamine; whereas, the rhodopsin and UV cone pigments were stable.

104 Here we show that rod and cone pigments when present in the same cell produce ligh

105 esponding control cultures regarding the red cone pigment, which was expressed in all cases, and the

106 ly similar residues to those of the marmoset cone pigment with a spectral peak of 543 nm.