ライフサイエンスコーパス: confluency

1 with increasing serum concentration and cell confluency.

2 higher measured motional resistance at full confluency.

3 his addition cell densities had increased to confluency.

4 lestone-like cells grow to high density past confluency.

5 nsities or when low density cells approached confluency.

6 pithelial cells in vitro was associated with confluency.

7 levels showed a steady increase, even after confluency.

8 ncrease for several days after cells reached confluency.

9 n plateau-phase cells arrested by growing to confluency.

10 lear granules increases as cultures approach confluency.

11 cessary to keep cultured cells from reaching confluency.

12 previous indications based on staining that confluency and contact inhibition of growth can cause a

13 knocked down MDCK cells plated on filters at confluency and cultured for 4.5 d, sorted TfR correctly

14 broader perspective is warranted, to include confluency and cytokine-/TPA-induced pathways for KCs.

15 l proliferation, reaching a maximum level at confluency and decreasing gradually thereafter.

16 Cultured cells were grown to confluency and incubated with various concentrations of

17 Confluency and interferon-gamma induced premature senesc

18 els of MCP doubled upon culturing of PAEC to confluency and remained stable during at least five pass

19 increased in keratinocytes as cells reached confluency, and overexpression of RPTP-kappa in subconfl

20 ress p16; 3) Abruptly induced growth arrest, confluency, and senescence pathways are associated with

21 ell nuclei and their morphology even in high-confluency cell cultures with many overlapping cell nucl

22 in subconfluent cultures; whereas at higher confluency, cell viability was largely unaffected.

23 added phosphate, UMR cultures after reaching confluency contain two size populations of morphological

24 at a slower proliferative rate upon reaching confluency (day 8), and eventually undergo contact inhib

25 and apoptosis to generate immaculate palatal confluency during palatogenesis in response to robust TG

26 is differentiation process is facilitated by confluency, followed by adipogenic stimuli.

27 l capillary endothelial cells (RECs) by cell confluency, hypoxia, serum starvation, high glucose conc

28 ion in a dose-dependent manner prior to cell confluency in our model system, non-tumorigenic small in

29 h inhibition induced by serum deprivation or confluency in untransformed CEF.

30 ular networks, and 10 key regulator genes in confluency-induced differentiation of NOK, all of which

31 n foreskin-derived early-passage KCs undergo confluency-induced premature senescence.

32 conditions (e.g., serum concentration, cell confluency, medium refreshment, direct/indirect treatmen

33 Induction of p14(ARF) on confluency occurred with low E2F-1 levels.

34 In contrast, TCF7L1 is not induced during confluency of non-adipogenic fibroblasts, and, remarkabl

35 on of CySCs are regulated by crowdedness, or confluency, of cells in their lineage with respect to th

36 BMEC-1 monolayers were grown to confluency on 3 microns microporous membrane inserts and

37 ssel endothelial cells (bEnd3) were grown to confluency on elasticized membranes and [Ca2+]i monitore

38 HCT-8 cells were grown to confluency on Interdigitated Microelectrode Arrays (IMA'

39 pontaneously hypertensive rats were grown to confluency on semipermeable tissue culture inserts, and

40 Under conditions of confluency or serum deprivation, the levels of phosphory

41 scopy was used to confirm cell viability and confluency over the microdialysis sampling region.

42 When cells reach confluency, overall expression is sharply reduced and th

43 issue and foreskin fibroblasts were grown to confluency, photographed, and counted, and their constit

44 rgoing spontaneous replicative senescence or confluency predominantly express p16; 3) Abruptly induce

45 Prior to adipogenesis, confluency promotes heterodimer recruitment of the bZip

46 ectopic expression of TCF7L1 alleviates the confluency requirement for adipocytic differentiation of

47 of 1 microm dexamethasone (DEX) starting at confluency results in >98% inhibition of bone morphogene

48 At confluency, S and R cells expressed equivalent amounts o

49 At confluency, TGF-beta induced Agc expression within 3 h,

50 Upon confluency, the cells were treated with T(3) (10(-12) to

51 We observe that at high confluency, these cells migrate against the direction of

52 After confluency, they aggregate into cellular condensations,

53 At 80% confluency, they were used for studies including toxican

54 s on plastic that proliferated in culture to confluency with a typical flattened elongative morpholog