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1 ia is the first example of a morpheein-based conformational disease.
2 te polymerization and prevent the associated conformational disease.
3 ontrol, thus increasing the risk for protein conformational disease.
4 stress in the pathogenesis of this inherited conformational disease.
5 ibute to the loss-of-expression phenotype of conformational diseases.
6 e mechanisms to rescue bystander proteins in conformational diseases.
7 eutic strategies targeting the modulation of conformational diseases.
8 al, but poorly understood, aspect of protein conformational diseases.
9 havior in protein misfolding is triggered in conformational diseases.
10 a hallmark of a large and diverse number of conformational diseases.
11 xpansion disorders collectively known as the conformational diseases.
12 sensitivity diagnosis of a number of protein conformational diseases.
13 uable strategy for therapies against protein conformational diseases.
14 sponse as a therapeutic strategy for protein conformational diseases.
15 p to explain how mutations alter toxicity in conformational diseases.
16 as a critical conformational element in many conformational diseases.
17 to develop effective treatments for protein conformational diseases.
18 buting to the complex pathologies of protein conformational diseases.
19 study establishes LGI1-related epilepsy as a conformational disease and suggests new therapeutic opti
20 dominant as well as recessive inheritance of conformational diseases and offer opportunities for the
21 standing the pathophysiology of prototypical conformational diseases and protein polymerization in th
27 pheein assemblies broadens the definition of conformational diseases beyond the prion disorders and t
28 nderlying type 2 diabetes mellitus-a protein conformational disease characterized by the aggregation
31 ts that studies of the biophysical origin of conformational disease in one member of a superfamily of
32 odel for the fibril and amyloid formation of conformational diseases in general as well as for the in
34 To date, prion diseases are unique among conformational diseases in that they are transmissible-e
38 patients with various immunoglobulin-related conformational diseases may provide insight into structu
39 ing, and human diseases suggest that protein conformational diseases may result not only from protein
41 acid variations in V(L) account for various conformational disease outcomes, including amyloidosis.
45 n of recombinant proteins, but also to treat conformational diseases such as cystic fibrosis, and Alp
46 y due to its pivotal role in the etiology of conformational diseases (such as Parkinson's and Alzheim
48 t cause of numerous diseases called "protein conformational diseases," such as myofibrillar myopathy
49 n mutation responsible for PH1, is a protein conformational disease that may benefit from new therapi
50 ng mechanisms of amyloidosis and etiology of conformational diseases to development of safe and effic
52 amyloid deposition of proteins seen in other conformational diseases, with the ordered array of polym
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