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1 s during shock anticipation and actual shock confrontation.
2 nfection, little is known about this initial confrontation.
3 nd an increase in dopamine to 120% after the confrontation.
4 collected before, during, and after a 10 min confrontation.
5 gnificantly increased up to 60 min after the confrontation.
6 rotonin levels were seen during or after the confrontation.
7 e achieved approximately 20-30 min after the confrontation.
8 ndently or downstream from the DER:fork head confrontation.
9 ess/anxiety, respectively, and during social confrontation.
10 istic, standardized, aggressive, and intense confrontations.
11 in the resident's home cage with brief daily confrontations.
12 k vary along a spectrum from minimization to confrontation; 4) the health care workplace is often sil
13 d aggressors (ANAs) during resident-intruder confrontations after self-administering 1.0 g/kg alcohol
14  80% of baseline levels during and after the confrontation and an increase in dopamine to 120% after
15 nce status [1, 5] can eliminate the need for confrontation and the associated energetic expense and r
16 lones, A2b2, A2bi, and C2a4, in the spheroid confrontation assay.
17 primed with a cytokine cocktail and used for confrontation assays with C. albicans.
18                                           In confrontation assays with saprobic fungi that were commo
19                      Characterization of the confrontation between rotaviruses and their host cells h
20  of a multiple sites environment, as well as confrontation between species featuring different swarmi
21 sive CD-1 mice, followed by agonistic social confrontations between the CD-1 and C57BL/6J mice, and c
22  social defeat stress consists of four brief confrontations between the experimental rat and an aggre
23                       We postulate that the 'confrontation' between the 'evil' alliance and 'benign'
24                                         Such confrontations can reveal previously unknown constituent
25                                              Confrontation does not appear to lead to patient acknowl
26 n = 48) we demonstrate that anticipation and confrontation evoke bradycardic and tachycardic response
27                                     In vitro confrontation experiments showed that the SH3-N domain o
28 ity in corticolimbic areas during aggressive confrontations in rats.
29 tients had normal early responses to picture confrontation, indicating intact visual processing.
30  research on the relationship between visual confrontation naming and hippocampal function has been i
31                                          The confrontation naming fMRI task also revealed impaired ac
32 ognitive deficits on both letter fluency and confrontation naming in the ALS group.
33    Previous research has demonstrated intact confrontation naming in the presence of verbal fluency d
34                                              Confrontation naming is impaired in neurodegenerative co
35 indicates that event-related ECoG HGA during confrontation naming predicts ECS interference with nami
36 ivated in normal subjects on the same visual confrontation naming task.
37 ecutive dysfunction, or were also present in confrontation naming, indicating language dysfunction.
38                                              Confrontation naming, noun fluency, recognition, and per
39 rment on other word retrieval tasks, such as confrontation naming, which do not place heavy demands o
40  paradigms were employed, letter fluency and confrontation naming, which were developed for use with
41 s of executive function, episodic memory and confrontation naming.
42 he overall neuroanatomical network of visual confrontation naming.
43 om shock anticipation to the moment of shock confrontation neural activity shifted from a region anat
44                                          The confrontation of these two approaches confirmed the impo
45                                       In the confrontation of this challenge, research should benefit
46             Our experience suggests that the confrontation of time-series data with mechanistic model
47 esizes that error detection results from the confrontation of two signals: a fast and unconscious mot
48 cial groups to examine the effects of social confrontation on the reinforcing effects of cocaine usin
49 ges, which followed rather than preceded the confrontation, points to a significant role of accumbal
50              For the group, following social confrontation, sensitivity to cocaine reinforcement was
51 Warren employed a simple stratagem of public confrontation to discover from Morton the true nature of
52 ordinates exposed to 28 d (1 hr/d) of social confrontations was compared with that from control nonst
53 R1 antagonists, CP-154,526 or MTIP, before a confrontation with a male conspecific.
54 cial stress was defined as defeat in a brief confrontation with an aggressive resident mouse and subs
55 ls were neither activated nor inactivated by confrontation with APCs from I-Ab-Ep mice in which I-A(b
56                                    Thus, the confrontation with T. arundinaceum results in an up-regu
57 tum demonstrated the strongest coloration of confrontation zones.

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