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1                 The B6.Sle1.Sle2.Sle3 triple congenic mouse (B6.TC) is a model of lupus coexpressing
2 stem to track haematopoietic cells following congenic mouse bone marrow transplants.
3 the same collection of mutants was tested in congenic mouse-derived primary macrophages, a major Nram
4 ormative backcross and intra-NKC recombinant congenic mouse DNA samples.
5  have developed a mammalian system for TM in congenic mouse embryonic fibroblasts (MEFs), either WT o
6                                  A novel NOD congenic mouse expressing aberrant Pkhd1, but lacking th
7 Rp1h(+/tm1Eap) and A.129S(B6)-Rp1h(+/tm1Eap) congenic mouse lines will facilitate identification of s
8                      Using interval-specific congenic mouse lines, we mapped Moo1 to an 8-Mb segment
9 ed gene, Mon1a, cosegregated with the QTL in congenic mouse lines.
10 itical modulator of lipid homeostasis in two congenic mouse lines.
11                In this study, we generated a congenic mouse model carrying a mutated Nnt(C57BL/6J) al
12 and mucin (TIM) proteins, identified using a congenic mouse model of asthma, critically regulate inna
13                  Here, we used the NOD.NK1.1 congenic mouse model to examine the role of NK cells in
14                           Here we use an MHC congenic mouse model to test the hypothesis that genetic
15 nal role of IRAK1 was next examined by using congenic mouse models bearing the disease loci: Sle1 or
16                                  We used two congenic mouse models that differ at the Ahr gene and en
17 st cells with in vitro-derived mast cells of congenic +/+ mouse origin exhibited airway responses tha
18         Here, we generated the corresponding congenic mouse strain by introgression of a segment of C
19                     For this, we generated a congenic mouse strain carrying the A2G wild-type Mx1 res
20 essed fat production by 22% in the C3H.B6-6T congenic mouse strain that exhibits accelerated age-rela
21 8Mit293-D8Mit137)/Mx (NOD-Idd22) recombinant congenic mouse strain was generated in which NOD mice ca
22                  We previously established a congenic mouse strain with TALLYHO/Jng (TH) donor segmen
23     The B6.SJL-Ptprc(d)Pep3(b)/BoyJ (B6.SJL) congenic mouse strain, a valuable and widely used tool i
24 s mouse mammary tumor viruses (MMTVs) in the congenic mouse strain, BALB/Mtv-null, restricts the earl
25 cal loads to the ulnae of the B6.C3H-4T (4T) congenic mouse strain, which is genetically 98.4% B6 and
26                   The QTL was confirmed in a congenic mouse strain.
27 enetically localize Gct4, we generated seven congenic mouse strains (SWR.SJL-X1 through -X7) that con
28  this locus mediates innate immunity in sst1 congenic mouse strains and identify a candidate gene, In
29           We generate two NOD.H2(k).B10-Chr9 congenic mouse strains and validate the role of this gen
30 c lupus erythematosus patients and of murine congenic mouse strains associate genes in a DNA segment
31 ploited the genetic structure of recombinant congenic mouse strains by performing a reciprocal interc
32 ulosis infection, we infected inbred and H-2 congenic mouse strains by the respiratory route.
33 rylation in NOD macrophages using reciprocal congenic mouse strains containing either diabetes-suscep
34       In the current study, we have used two congenic mouse strains differentially expressing the Ped
35 ecently we have explored the use of knockout/congenic mouse strains for isolating and mapping quantit
36 relates of protection in H-2 recombinant and congenic mouse strains on the B10 background.
37       The systematic analysis of lupus-prone congenic mouse strains suggests a role for two isoforms
38  existence of Idd5 by developing a series of congenic mouse strains that are resistant to diabetes an
39      Here, we have used linkage analysis and congenic mouse strains to map the major skin tumor susce
40 -suppressor genes Apc and p53 was studied in congenic mouse strains to minimize the influence of poly
41 murine encephalomyelitis virus (TMEV) in MHC-congenic mouse strains where one haplotype was resistant
42                                        Using congenic mouse strains, we showed that the H-2(s) haplot
43  between diabetes-susceptible and -resistant congenic mouse strains.
44 ne target and/or insulitis trigger in NOD or congenic mouse strains.
45 date genes without requiring construction of congenic mouse strains.
46 d by cell transfer experiments using an Ly-5 congenic mouse system.
47                                 The NOD.c3c4 congenic mouse, which has multiple B6- and B10-derived I

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