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1 provide new insight into the pathogenesis of congenital heart block.
2 lained despite their strong association with congenital heart block.
3 bs, particularly 52Ro, in the development of congenital heart block.
4 s it pertains to ventricular arrhythmias and congenital heart block.
6 (19%) in mothers with a previous child with congenital heart block and in 3 of 74 pregnancies (4%) i
7 Despite the near universal association of congenital heart block and maternal Abs to SSA/Ro and SS
8 ibodies give birth to children with complete congenital heart block and photosensitive skin lesions.
9 Rs), as did cardiac tissue from a fetus with congenital heart block (CHB) and an age-matched control.
10 ance in the description and understanding of congenital heart block (CHB) came in the 1970s with the
16 signature lesion of autoantibody-associated congenital heart block (CHB) is fibrosis of the conducti
20 nd recurrence rates of autoimmune-associated congenital heart block (CHB) using information from the
21 IC) may be relevant in autoimmune-associated congenital heart block (CHB) where the obligate factor i
25 -SSB/La are necessary for the development of congenital heart block (CHB), the low frequency suggests
27 ated from 83 children (22 with rash, 35 with congenital heart block [CHB], 26 unaffected siblings) an
28 efficacious after in utero identification of congenital heart block, especially in fetuses with assoc
29 maternal autoantibodies with the genesis of congenital heart block, female BALB/c mice were immunize
30 syndrome, systemic lupus erythematosus, and congenital heart block from anti-Ro52 Ab-positive mother
32 suggest that hydroxychloroquine may prevent congenital heart block in pregnancies exposed to SSA/Ro
37 pathogenesis of neonatal lupus syndrome and congenital heart block reveals important information abo
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