ライフサイエンスコーパス: congenital infection

1 placental malaria are at increased risk for congenital infection.

2 y as 1 day of age in infants with documented congenital infection.

3 guinea pig cytomegalovirus (GPCMV) model of congenital infection.

4 to intrauterine transmission and symptomatic congenital infection.

5 virus has emerged as an important worldwide congenital infection.

6 rs243864 sites would be a good predictor of congenital infection.

7 cription of the major lesions caused by ZIKV congenital infection.

8 MMP2) were associated with susceptibility to congenital infection.

9 cruzi-infected mothers, which might predict congenital infection.

10 ix remodeling and the immune response during congenital infection.

11 e leading viral cause of birth defects after congenital infection.

12 ntally expressed genes and susceptibility to congenital infection.

13 e leading viral cause of birth defects after congenital infection.

14 ema, and hypoxia associated with symptomatic congenital infection.

15 e employed to determine the association with congenital infection.

16 TBPCs of the chorion in cases of symptomatic congenital infection.

17 pathogenesis of birth defects resulting from congenital infection.

18 sts, in chorionic villi-in clinical cases of congenital infection.

19 cellular immunity may be required to prevent congenital infection.

20 e in immunosuppressed patients and following congenital infection.

21 on, spontaneous abortion, preterm birth, and congenital infection.

22 fection of the fetus is the leading cause of congenital infection.

23 antibodies, and sera were screened for other congenital infections.

24 of pregnancy complications and prevention of congenital infections.

25 and apoptosis observed in these cells during congenital infections.

26 Therefore, ZIKV has joined the group of congenital infections.

27 responsible for the majority of symptomatic congenital infections.

28 The majority of human herpesvirus 6 (HHV-6) congenital infections (86%) originate from germ line tra

29 One congenital infection among infants of the subjects occur

30 ronic asymptomatic infection, of whom 19 had congenital infection and 11 had toxoplasmic lymphadeniti

31 with a significant reduction in the rate of congenital infection and a better outcome at 3 years of

32 nfections have been associated with damaging congenital infection and adverse outcomes in transplant

33 i-Goutieres syndrome is a mendelian mimic of congenital infection and also shows overlap with systemi

34 ovirus (HCMV) is the major viral etiology of congenital infection and birth defects.

35 cytomegalovirus (HCMV) is a common agent of congenital infection and causes severe disease in immuno

36 ith 1 dose of cHPMPC improves the outcome of congenital infection and decreases viral replication in

37 ening) require counselling about the risk of congenital infection and its clinical sequelae.

38 esis may have a prognostic value in terms of congenital infection and neonatal symptomatic disease.

39 The highest rate of symptomatic congenital infection and sequelae occurs in about 25% of

40 ring pregnancy exposes the fetus to risks of congenital infection and sequelae that depend heavily on

41 explains the phenotypic overlap of AGS with congenital infection and some aspects of SLE, where an e

42 serum and urine CMV NAT at birth to evaluate congenital infection and surveillance CMV NAT at 5 addit

43 newborns were evaluated for the presence of congenital infection and symptomatic disease.

44 phenotypic overlap both with the sequelae of congenital infection and with systemic lupus erythematos

45 ated with neurological disorders in dogs and congenital infections and abortions in cattle.

46 uitous human pathogen, is a leading cause of congenital infections and represents a serious health ri

47 positive for T. cruzi were compared: 101 had congenital infection, and 116 were uninfected.

48 kinetics showed no evidence of asymptomatic congenital infection as neonates were tested negative fo

49 The clinical outcome of congenital infection at birth was similar in the two gro

50 features at birth resembling the sequelae of congenital infection but in the absence of an infectious

51 s associated with a 3-fold greater chance of congenital infection, but it is not correlated with symp

52 Congenital infection can result in permanent neurologica

53 creened positive on rapid culture or PCR had congenital infection confirmed by the reference standard

54 The primary end point was congenital infection diagnosed at birth or by means of a

55 Conclusions and Relevance: Congenital infection due to presumed ZIKV exposure is as

56 omegalovirus (CMV), the most common cause of congenital infection, exhibits extensive genetic variabi

57 ba (IPESQ) in northeastern Brazil, where the congenital infection has been particularly severe.

58 The Zika virus epidemic and associated congenital infections have prompted rapid vaccine develo

59 lacental specimens from women with untreated congenital infection, HCMV-specific hyperimmune globulin

60 irus (CMV) is the cause of the most frequent congenital infection in America; however, pregnant women

61 To search for serological evidence of congenital infection in apparently healthy neonates born

62 ed to Toxoplasma gondii, causes abortion and congenital infection in cattle.

63 glycoprotein B (gB) serum in modification of congenital infection in early pregnancy.

64 Cytomegalovirus (CMV) is the most common congenital infection in humans.

65 These 2 parameters remained associated with congenital infection in multivariate analysis.

66 ig cytomegalovirus immunity reduces rates of congenital infection in subsequent pregnancies.

67 with HCMV-mediated neuropathogenesis during congenital infection in the fetal brain.

68 CMV is the most common congenital infection in the United States.

69 g coccidian parasite that causes abortion or congenital infections in naturally or experimentally inf

70 ding sexually transmitted diseases [STDs] or congenital infections) in the first seven days of life a

71 This review focuses on those other congenital infections, including rubella, congenital cyt

72 In this review, nonspecific findings of congenital infections initially will be described, then

73 Congenital infection is also associated with intrauterin

74 Although vaccine development to modify this congenital infection is ongoing, the unique epidemiology

75 considered to result from reactivation of a congenital infection, it is now believed that postnatall

76 ist, awareness of imaging features of common congenital infections may facilitate early diagnosis and

77 ephaly and show how some infants affected by congenital infection might be missed or incorrectly diag

78 s, and negative laboratory results for other congenital infections; moderately probable cases had spe

79 Congenital infection occurred in 20/31 (65%) control pup

80 ne proteinase activity significantly reduced congenital infection of chicken embryos, as determined b

81 Congenital infection of human cytomegalovirus (CMV) is t

82 Congenital infection of human cytomegalovirus (HCMV) is

83 epithelial and mesenchymal cells, imitating congenital infection of the fetal lung.

84 f the latent infection in the mother causing congenital infection or abortion and by ingestion of ooc

85 Toxoplasmosis can be due to congenital infection or acquired infection after birth a

86 e 5 features that are rarely seen with other congenital infections or are unique to congenital Zika v

87 In a congenital infection protection study, animals were prot

88 In a congenital infection protection study, GP85 DISC-vaccina

89 Lessons from other congenital infections provide valuable clues about the c

90 nse, maternal viral load, pup mortality, and congenital infection rates in the vaccine and control gr

91 In cases of congenital infection, reduced responsiveness of the plac

92 ularly transplant patients and patients with congenital infections, respectively.

93 o congenital T. cruzi infection (hereafter, "congenital infection"): rs2014683 and rs1048988 in ALPP;

94 y-onset neonatal form, highly reminiscent of congenital infection seen particularly with TREX1 mutati

95 Congenital infection status was ascertained in 554 cases

96 rnal immunization with tetanus toxoid and to congenital infections such as rubella, toxoplasma, cytom

97 The acronym TORCH is used to refer to congenital infections, such as toxoplasmosis, other infe

98 ital CMV infection (cCMV) is the most common congenital infection that can cause long-term impairment

99 iew, we summarise important aspects of major congenital infections that can cause microcephaly, and d

100 ystem caused by human cytomegalovirus (HCMV) congenital infection, the mechanism of HCMV neuropathoge

101 and motor disabilities, are shared by other congenital infections, there are 5 features that are rar

102 its from treating cases of recently acquired congenital infection to destroy any remaining proliferat

103 The rate of congenital infection was 30% (18 fetuses or infants of 6

104 Congenital infection was assessed using CMV DNA quantita

105 The genotype of parasite strains causing congenital infection was determined by direct PCR amplif

106 Probabilities of congenital infection were <10% for maternal infections b

107 ar involvement in infants with presumed ZIKV congenital infection were more often seen in infants wit

108 Strains causing congenital infections were also typed indirectly based o

109 mples of 7 of the 8 infants (88%), and other congenital infections were ruled out.

110 Congenital infection with cytomegalovirus (CMV) can indu

111 Congenital infection with cytomegalovirus (CMV) is an im

112 Congenital infection with cytomegalovirus is a major cau

113 he first neonate documented to have survived congenital infection with Ebola virus.

114 placentas from symptomatic and asymptomatic congenital infection with HCMV DNA in amniotic fluid and

115 Congenital infection with human cytomegalovirus (CMV) is

116 Congenital infections with pathogens such as Zika virus,

117 CMV) represents one of the leading causes of congenital infections worldwide.