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1 -occupancy vehicle (HOV) policies can combat congestion.
2 mbined impact of traffic control and traffic congestion.
3 on fraction, left ventricular mass, and lung congestion.
4 from sample ions and thereby reduce spectral congestion.
5 ivity and LV end-diastolic pressure and lung congestion.
6 ge, microvascular thrombosis, and neutrophil congestion.
7 diomyocyte apoptosis and minimized pulmonary congestion.
8 nts are not very effective in treating nasal congestion.
9 3% in subjects without both asthma and nasal congestion.
10 ic systems as well as the resulting spectral congestion.
11 rophy, depressed systolic function, and lung congestion.
12 occurs in patients with persistent signs of congestion.
13 e exponential growth quickly leads to steric congestion.
14 cic impedance, which is related to pulmonary congestion.
15 nds of 9 are quite short, despite the steric congestion.
16 ischemia/reperfusion (I/R) injury with bowel congestion.
17 icle may cause right heart failure and liver congestion.
18 ain during 6 months after therapy to relieve congestion.
19 retransplantation and none because of venous congestion.
20 ation, two from graft loss because of venous congestion.
21 ceptor antagonist, has been shown to improve congestion.
22 S hospitalizations yearly, mostly related to congestion.
23 induced myocardial hypertrophy and pulmonary congestion.
24 ccuracy and better cope with single molecule congestion.
25 ts with renal dysfunction or severe systemic congestion.
26 ces, for overall mortality, or with downwind congestion.
27 on between regional function and presence of congestion.
28 oteins are largely precluded due to spectral congestion.
29 scular mortality were suggested with greater congestion.
30 single-occupancy cars often leads to traffic congestion.
31 flaps failed in the DIEP group due to venous congestion.
32 asal itching, sneezing, watery discharge and congestion.
33 mand explains the percentage of time lost in congestion.
34 t cavity, but it still suffers from spectral congestion.
35 city on their routes determines the level of congestion.
36 distance accompanied by repulsive electronic congestion.
37 nts and lung function and increases in nasal congestion.
38 ute decompensated heart failure and clinical congestion.
39 in and it may lead to prostatic and urethral congestion.
40 sover design, we examined the association of congestion 0-150 m, 151-300 m, and 301-1,000 m upwind of
42 ften within 5 days of inoculation were nasal congestion (36%), headache (30%), and sore throat (19%).
43 y (30% vs. 8%) lymphoma, episcleral vascular congestion (40% vs. 16%), anterior chamber reaction (30%
44 xacerbations: 73% had lacrimation, 51% nasal congestion, 46% conjunctival injection and 40% ptosis an
45 the subgroup reporting both asthma and nasal congestion, 55.8% had insomnia symptoms compared with 35
46 RSV was more commonly associated with nasal congestion (68% vs. 55%; P=.03), wheezing by history (73
47 those patients with moderate-to-severe lung congestion, 71% were asymptomatic or presented slight sy
49 injected eyes frequently showed conjunctival congestion, AC cells and flare, iridal hyperemia, and fi
53 ency of abdominal pain, diarrhoea, and nasal congestion after all three vaccinations was significantl
54 al examination of the affected organs showed congestions, aggregations and multinucleated hepatocytes
55 d and capacity exist, the possible limits of congestion alleviation by only modifying route choices h
56 as increased in the patients with persistent congestion alone and in those with both WRF and congesti
58 n, and a considerable contributor to traffic congestion and air pollution causing substantial adverse
59 subjected to total I/R with associated bowel congestion and are not deleterious as previously thought
60 iac contractile dysfunction, hepatopulmonary congestion and ascites, halved the Kir6.2-KO cohort, whi
63 arch is urgently needed, given the burden of congestion and data suggesting sustained benefits of ear
65 osis and extensive vascular damage: vascular congestion and dilation, endothelial necrosis, alteratio
66 on to the long-standing problems of spectral congestion and disorder in gas-phase electronic spectra.
67 , defined as worsening signs and symptoms of congestion and elevated natriuretic peptide level requir
68 visits to the emergency department to assess congestion and flow; assignment of all admissions to the
69 creep-idle conditions, representing traffic congestion and frequent idling periods, contributing wit
70 ction, S2814A mice did not exhibit pulmonary congestion and had reduced levels of atrial natriuretic
72 ly reduced peritubular capillary erythrocyte congestion and improved histologic scores of tubular inj
75 hly collagenated tumors showed more vascular congestion and ischemia after PDT, along with a higher p
79 flects LV preload independent of right heart congestion and pericardial restraint, was similar in TR
80 -term changes in markers of organ damage and congestion and relate them to 180-day mortality in patie
81 olume Overload to Assess Treatment Effect on Congestion and Renal Function (PROTECT) trial and valida
83 olume Overload to Assess Treatment Effect on Congestion and Renal Function) study in 1,969 patients w
84 olume Overload to Assess Treatment Effect on Congestion and Renal Function), a trial comparing the ef
86 t tissues in SCD exhibit heightened vascular congestion and sensitivity to ischemia and that clinical
92 atory distress in >80% of cases, daily nasal congestion and wet cough starting soon after birth, and
93 s (vs 50-64 years), symptoms of cough, nasal congestion and wheezing, and longer interval from illnes
95 -related orbitopathy characterized by severe congestion and/or compressive optic neuropathy with and
96 ences of diastolic dysfunction (e.g., venous congestion), and the second is to eliminate or reduce th
100 ilarly reduced contractility, increased lung congestion, and atrial thrombus, and both strains died b
101 uctural alterations, the absence of vascular congestion, and attenuation of glomerulosclerosis in tre
102 symptoms, radiographic evidence of pulmonary congestion, and echocardiographic evidence of left ventr
104 lar hypertrophy, diastolic dysfunction, lung congestion, and myocardial oxidative stress without affe
105 tand the relationships among symptom relief, congestion, and outcomes in patients with acute decompen
108 ure to seasonal pollen causes itching, nasal congestion, and repeated sneezing, with profound effects
109 is information is often obscured by spectral congestion, and site-selective information is not availa
111 patic necrosis, likely from prolonged venous congestion, and the patient required repeat transplantat
112 y, striped interstitial fibrosis, glomerular congestion, and tubular microcalcification were all grea
115 beneficial effect on airway symptoms (nasal congestion, anterior rhinorrhea, loss of sense of smell,
118 rizing alkoxycarbonylation reveal the steric congestion around the diphosphine coordinated Pd-center
119 r vaccinating infants, who may develop nasal congestion as a result of intranasal vaccination.IMPORTA
120 peremia) and during venous occlusion (venous congestion), as assessed with skin capillaroscopy, and a
122 proved systolic function and attenuated lung congestion, associated with reduced cardiac fibrosis, in
123 ion and assessed the persistence of signs of congestion at discharge in 599 consecutive patients admi
126 this transformation is alleviation of steric congestion at the allylic position of the alpha-silyloxy
127 substrate delivery to one per ClpA(6) avoids congestion at the axial channel and allows facile transf
128 rt failure-associated splanchnic circulation congestion, bowel wall edema, and impaired intestinal ba
129 versus 320 pg/mL; all P<0.01), more signs of congestion, but no significant difference in left ventri
130 , we are able to reduce the relative percent congestion by 84.9% in their HSQC spectra using only fou
132 ntly associated with increased microvascular congestion by infected and uninfected erythrocytes.
134 JNJ-39220675 relieved allergen-induced nasal congestion by using standard nasal symptom scoring; howe
135 f heart failure including symptoms, signs of congestion, cardiac structure, and time to hospitalizati
138 osis is important, as it can lead to hepatic congestion, cirrhosis and Budd-Chiari syndrome (BCS) and
139 very of fatty acids to the liver and hepatic congestion commonly found in severely burned children by
140 e fused pyrido ring resulting in more steric congestion compared to the relatively flat fused pyrrolo
141 howed reduced cardiac hypertrophy, pulmonary congestion, concentric LV wall thickness, LV dilatation,
144 that this relation is reversed on all other congestion control algorithms for a region of the parame
145 (max-min fairness and proportional fairness) congestion control schemes, and analyse how the algorith
147 tear film breakup time (TBUT), conjunctival congestion, corneal haze, vascularization, conjunctivali
148 t to take the opposite action, as in traffic congestion, crowd dispersion, and division of labor.
151 n addition, SS-31 reduced medullary vascular congestion, decreased IR-mediated oxidative stress and t
152 H would be a marker of symptomatic pulmonary congestion, distinguishing HFpEF from pre-clinical hyper
153 s infection is followed by intense pulmonary congestion due to an extensive influx of macrophages and
156 t has been found that 0.5% PVI can attenuate congestion, edema and pain induced by pressure sores.
157 it is unclear how the clinical components of congestion (eg, peripheral edema, orthopnea) contribute
159 nd respiratory mortality with greater upwind congestion, especially congestion near the decedent's ho
161 ranulomatosis presented with 6 days of sinus congestion, fever, malaise, myalgias, episcleritis, and
163 whether body elongation is linear or whether congestion forms within the flow and the body axis becom
164 or no congestion, patients with very severe congestion had a 4.2-fold risk of death (HR=4.20, 95% CI
166 gestion alone and in those with both WRF and congestion (hazard ratio, 5.35; 95% confidence interval,
167 , cardiomegaly, glomerulosclerosis, visceral congestion, hemorrhages, multiorgan infarcts, pyknotic n
170 at the pro-C(11) center were thwarted by the congestion imparted by the presence of the vicinal TBS-e
171 of applied goals such as reducing automotive congestion, improving disaster response, and even predic
178 ing a reassessment of the pathophysiology of congestion in congestive heart failure and the methods b
180 ultrasound can detect asymptomatic pulmonary congestion in hemodialysis patients, and the resulting B
181 e majority of these resulting from worsening congestion in patients previously diagnosed with HF.
182 cal injury in the kidney, and more prominent congestion in the capillary beds in the lungs and heart.
183 have evolved ultrasonic hearing to minimize congestion in the frequency bands used for sound communi
189 ntagonist, JNJ-39220675, in preventing nasal congestion induced by exposing participants with ragweed
191 above the control responses for conjunctival congestion, iris hyperemia, AC cells, flare, and fibrin
192 above the control responses for conjunctival congestion, iris hyperemia, AC cells, flare, fibrin, and
198 ny AHF patients at discharge and more severe congestion is associated with increased morbidity and mo
200 rated efficacy of loop diuretics in managing congestion is balanced by the recognized limitations of
201 derivatives of C60 and C70 with high steric congestion is described using 1,3-dibenzoylpropane bis-p
203 odilators targeting decongestion, persistent congestion is present in many AHF patients at discharge
209 pressure, as an extreme marker of abdominal congestion, is correlated with renal dysfunction in adva
210 zes and 0- to 5-point scores for severity of congestion, itching, and rhinorrhea), A(min) (in square
211 teral vein as well as prostatic and urethral congestion leading to intermittent urinary hesitancy, wh
213 zed by attenuated heart enlargement and lung congestion, lower expression of cardiac stress genes (B-
215 on (i.e., splanchnic venous and interstitial congestion) manifests in a substantial number of patient
216 rtex veins ampullas, suggesting that outflow congestion may be a contributing factor to the pathogene
217 rated glomerular fibrin deposition, red cell congestion, microthrombi formation, and glomerular ultra
218 red intestinal barrier function secondary to congestion, might further depress cardiac as well as ren
221 hemodynamic changes leading to venous renal congestion, neurohormonal activation, hypothalamic-pitui
222 wever, increasing histone-induced neutrophil congestion, neutrophil extracellular trap formation, and
223 of inflammation including epithelial damage, congestion, neutrophil infiltration, loss of mucin from
224 ed with all non-RSV-infected subjects, nasal congestion (odds ratio [OR], 2.0 [95% confidence interva
225 y active treatments versus placebo: group 1, congestion of -0.7 (SE, 0.3), itching of -1.0 (SE, 0.3),
230 exist as a weak pi-dimers due to the steric congestion of the thioalkyl groups in the superimposed P
231 rization and explain the influence of steric congestion of the vinyl moiety on the isomerization path
234 patients with acute heart failure, dyspnoea, congestion on chest radiograph, and increased brain natr
236 tions, obesity, renal function, the state of congestion or decongestion, and whether patients are rec
237 condary to persistent chronic passive venous congestion or decreased cardiac output resulting from th
238 al pulmonary edema, (3) radiologic pulmonary congestion or edema, or (4) left ventricular systolic dy
239 port of a daily cough for 3 months in a row, congestion or phlegm other than when accompanied by a co
241 CI: 0.851, 0.997; P = 0.043), and sinusoidal congestion (OR: 0.954; 95% CI: 0.912, 0.998; P = 0.039).
244 nists administered at high doses may relieve congestion, overcome diuretic resistance, and mitigate t
245 mpared with those patients having mild or no congestion, patients with very severe congestion had a 4
248 RV infection was associated with rhinorrhea, congestion, postnasal drip, sputum, and cough; HCoV infe
249 INOL, and this is consistent with the steric congestion present in the matched and mismatched copper
250 ications arising from the substantial steric congestion prevailing in these structural networks.
252 04 through 2009 who required SCUF because of congestion refractory to hemodynamically guided intensiv
255 ry acidosis, impaired gas exchange, systemic congestion, respiratory support/replacement therapies, a
256 can include red itchy eyes, sneezing, nasal congestion, rhinorrhea, coughing, bronchoconstriction, a
258 ficant improvement was observed in the nasal congestion score (P = 0.01) and sense of smell score (P
260 Secondary end points included the clinical congestion score, dyspnea assessment, net urine output,
262 uency, cough severity, cough bothersomeness, congestion severity, rhinorrhea severity, and cough effe
263 genic sickle mice exhibited massive vascular congestion, sickling of red blood cells, more marked his
264 lds (no fever, and >/=2 of runny nose, nasal congestion, sneezing, sore throat, cough, swollen or ten
266 alternative treatment strategies to relieve congestion, such as ultrafiltration, may also result in
267 amination with mild to moderate conjunctival congestion, suggestive of microsporidial superficial ker
268 patients hospitalized with AHF, dyspnea, and congestion, the addition of tolvaptan to a standardized
269 ure, worsened renal function, and persistent congestion, the use of a stepped pharmacologic-therapy a
271 ure, worsened renal function, and persistent congestion to a strategy of stepped pharmacologic therap
273 Earlier identification and treatment of congestion together with improved care coordination, man
275 Charge stripping decreases the spectral congestion typically observed in ESI mass spectra of hig
276 of vehicle mode of operation (free flow and congestion) using (1) concurrent 5 min measurements of U
277 y, the method was shown to decrease spectral congestion via fragmentation of multiple (charge-reduced
279 tional (safely drained) volumes, and outflow congestion volumes in grafts and remnants allowed for th
280 identified for (a) virtual graft and remnant congestion volumes of risky versus nonrisky MHV types (4
282 n moderate/severe MR cases (n = 8), vascular congestion was more intense (rho = 0.841; P < .001), seq
285 hose ICU admission decisions are affected by congestion, we found that, if congestion were not a barr
286 re affected by congestion, we found that, if congestion were not a barrier and more eligible patients
288 al correlation showed that sequestration and congestion were significantly associated with deeper lev
290 comes compared with those with no WRF and no congestion, whereas the risk of death or of death or AHF
291 adverse events: not transplanted- pulmonary congestion with epilepticus (likely not related) versus
293 d describe the design of the Targeting Acute Congestion With Tolvaptan in Congestive Heart Failure St
295 mes higher odds of death with greater upwind congestion within 150 m and 1.02 (95% CI: 0.95, 1.10) ti
296 For each 10-minute-km increase in upwind congestion within 150 m, the odds of cerebrovascular mor
297 ression of injury-related genes and vascular congestion without provoking tubulointerstitial inflamma
298 ts with diabetes mellitus have more signs of congestion, worse quality of life, higher N-terminal pro
299 oward identifying and monitoring subclinical congestion would be of value in the home management of c
300 tiles of capillary recruitment during venous congestion yielded an odds ratio of 2.89 (95% confidence
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