ライフサイエンスコーパス: connecting peptide

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1 the two chains are tethered by a disordered connecting peptide.

2 s an additional 10 residues were used as the connecting peptide.

3 th the PPARgamma agonists, rosiglitazone and connecting peptide.

4 dules argues that independent recruitment of connecting peptide 1 and the anticodon-binding domain du

5 We proposed that including the 76-residue connecting peptide 1 insertion (Fragment B) might enhanc

6 , including the anticodon-binding domain and connecting peptide 1, CP1, a 74-residue internal segment

7 an insertion into the Rossmann-fold known as Connecting Peptide 1.

8 The connecting peptide, although not well ordered in native

9 ral unit when dissociated from the CD8 alpha connecting peptide, and in so doing retains class I MHC

10 fibrillation is increased by scission of the connecting peptide at single sites.

11 ] mutant), (vi) NTE plus motifs I and II and connecting peptide (betaA3[N+I+II+CP] mutant), (vii) mot

12 252)], N-terminal arm + motif I + motif II + connecting peptide [betaB1(149-252)], C-terminal extensi

13 Insulin connecting peptide (c-peptide) aids the folding of proin

14 Insulin and connecting peptide (C-peptide) are produced in equimolar

15 We thus propose that the connecting peptide constrains a large scale conformation

16 like growth factor I) in which foreshortened connecting peptides further retard fibrillation.

17 insights into understanding the role of the connecting peptide in insulin biosynthesis.

18 s cleaved within the D4 transmembrane domain connecting peptide into two fragments that remain membra

19 The sequence of one connecting peptide is identical to the previously charac

20 unction to facilitate disulfide pairing, the connecting peptide may protect beta cells from toxic pro

21 ation with minimal information to verify the connecting peptide moiety.

22 d polybasic amino acids in the hemagglutinin connecting peptides of all H5N1/04 viruses, indicating t

23 ulin domains, but shortening of the TCRalpha connecting peptide reduced assembly, consistent with mem

24 eptide Ala-Pro-Gly-Trp-NH2 (APGWamide), five connecting peptide sequences, and a C-terminal peptide.

25 of the TCR ectodomain complex including its connecting peptides suggests how force generated during

26 th selective shortening of the 35-amino acid connecting peptide that normally includes two sets of fl

27 Two designs for the connecting peptide were evaluated.

28 dissociated from the 48-amino acid CD8 alpha connecting peptide) were shown to retain the capacity of

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