ライフサイエンスコーパス: connective tissue

1 nd the accumulation of myofibroblasts in the connective tissue.

2 zed by the accumulation of myofibroblasts in connective tissue.

3 e II fibres and a decrease and stiffening of connective tissue.

4 ressed in Pdgfra-inactivated fibroblasts and connective tissue.

5 ogether: the basal lamina and the underlying connective tissue.

6 cal involvement from the muscle and from the connective tissue.

7 MA)-expressing myofibroblasts located within connective tissue.

8 ined by aberrant accumulation of collagenous connective tissue.

9 transport to the development of skeletal and connective tissue.

10 lls were detected both in the epithelium and connective tissue.

11 and is the most common inherited disease of connective tissue.

12 ition of calcium phosphate complexes in soft connective tissues.

13 ms a distinct microfibrillar network in most connective tissues.

14 tebral disc and potentially other pathogenic connective tissues.

15 sparsely in the stroma of subcutaneous loose connective tissues.

16 F) plays a pivotal role in the remodeling of connective tissues.

17 optimized geometric organizations of fibrous connective tissues.

18 ate, highlighting the effect of succinate on connective tissues.

19 tion of multiscale force transmission within connective tissues.

20 cle cell-rich fibrous tissue (12%) and loose connective tissue (9%).

21 ly, we found that oral autologous artificial connective tissue (AACT) had a different protein secreti

22 rhomocysteinemia (HHcy) which replicates the connective tissue abnormalities observed in CBS-deficien

23 These findings explain, at least in part, connective tissue abnormalities observed in HHcy.-Perla-

24 elevates plasma Hcy-thiolactone and leads to connective tissue abnormalities that affect the cardiova

25 itable bowel syndrome, cutaneous complaints, connective tissue abnormalities, and dysautonomia.

26 gastrointestinal symptoms, chronic pain, and connective tissue abnormalities, including joint hypermo

27 ynthase-deficient mice: a novel mechanism of connective tissue abnormalities.

28 sis, a disease in which excessive amounts of connective tissue accumulate in response to physical dam

29 quantify factors secreted by oral artificial connective tissue (ACT) in culture medium, and a method

30 ssues, including muscles, blood vessels, and connective tissues, adapts to mechanical strains in the

31 lance and wound-healing response of gingival connective tissues affected by diabetes mellitus or agin

32 microbial inflammatory disease that degrades connective tissue and alveolar bone and results in tooth

33 inflammatory destruction of tooth-supporting connective tissue and bone.

34 clinically cause significant destruction of connective tissue and bone.

35 derived from the liver extracellular matrix, connective tissue and epithelium, and proteins related t

36 smoplastic reaction through the synthesis of connective tissue and extracellular matrix, inducing loc

37 ree-dimensional (3D) extracellular matrix of connective tissue and must overcome the steric hindrance

38 bserve expression of all four genes in mouse connective tissue and network analyses show an important

39 es, are the source of the diaphragm's muscle connective tissue and regulate muscle development, and w

40 fibroblasts that promotes the remodeling of connective tissue and suggest that PDGFRalpha may be a r

41 atic channels were evident both in mesocolic connective tissue and Toldt's fascia.

42 files of mast cells isolated from peripheral connective tissues and basophils isolated from spleen an

43 w that 3D collagen gels, major components of connective tissues and extracellular matrix (ECM), are s

44 w that 3D collagen gels, major components of connective tissues and extracellular matrix, are signifi

45 is a nonfibrillar collagen expressed in many connective tissues and implicated in extracellular matri

46 ntal structural component of a wide range of connective tissues and of the extracellular matrix.

47 ronchi were dissected free of epithelium and connective tissues and suspended in a force-length measu

48 segmental progeroid disorder affecting skin, connective tissues, and bone that is caused by loss-of-f

49 Thus, the PPFs and muscle connective tissue are critical for diaphragm development

50 Since mechanosensory cells in connective tissue are surrounded by a disordered network

51 Impaired muscle, bone, and connective tissue are typical clinical features of MPS d

52 xtracellular spaces, cross section area, and connective tissue area, during the dry-cured ham process

53 When taking 1.0 mm connective tissue attachment into account, 60% 3D BA-RSA

54 er bone porosity, trabecular separation, and connective tissue attachment loss (CTAL) as well as redu

55 l ligament (PDL) functions as an enthesis, a connective tissue attachment that dissipates strains cre

56 mount of attachment loss, crestal bone loss, connective tissue attachment, and the surface area with

57 pairs through a long epithelial junction and connective tissue attachment.

58 the gingiva; support and alveolar bone loss; connective tissue attachment; and the quantity of gingiv

59 Fibromodulin has a key role in connective tissues, binding collagen through two identif

60 ise effect of collagen glycation on gingival connective tissue biology is not fully understood.

61 chneiderian membranes with materials such as connective tissue, buccal fat pads, and resorbable colla

62 GAGs are ubiquitous constituents of connective tissue, cartilage, and the extracellular matr

63 fecting variants in immune, CFTR, cilia, and connective tissue categories (35, 26, 90, and 90%, respe

64 e (MFS) is an autosomal dominant disorder of connective tissue, caused by mutations of the microfibri

65 Mechanical injury to connective tissue causes changes in collagen structure a

66 deposition, inflammatory cell retention, and connective tissue cell differentiation, respectively.

67 Originally identified as progenitors of connective tissue cell lineages, recent findings have re

68 tumors (aggressive fibromatosis) arise from connective tissue cells or fibroblasts.

69 to create 3D open scaffolds for adhesion of connective tissue cells through well-defined adhesion pl

70 cs of cultured human gingival epithelial and connective tissue cells.

71 s imperfecta (OI) is a heritable disorder of connective tissue characterized by bone fragility and lo

72 in both a dominant and recessive manner, of connective tissue characterized by brittle bones, fractu

73 differences were found in Vv of epithelium, connective tissue, collagenous and non-collagenous matri

74 ique in terms of relative amounts of fibrous connective tissue (CT) and fatty/glandular tissue (FGT)

75 The palatal donor site of the epithelialized connective tissue (CT) graft significantly influences th

76 Subepithelial connective tissue (CT)-based procedures and coronally ad

77 iferating cells, and collagen content in the connective tissue (CT).

78 residual graft material, and non-mineralized connective tissue (CT)/other material.

79 rcentages of vital bone, residual graft, and connective tissue (CT)/other were determined via histomo

80 ew vital bone formation, residual graft, and connective tissue (CT)/other.

81 The connective tissue denaturation temperature was lower for

82 abecular bone volume, thickness, number, and connective tissue density with decreased trabecular spac

83 blast lineage is responsible for the bulk of connective tissue deposition during embryonic developmen

84 e-specific cell ablation leads to diminished connective tissue deposition in wounds and reduces melan

85 sis, tissue morphology, endocrine system and connective tissue development and function.

86 irement for FKBP65 function during embryonic connective tissue development in mice, but the restricte

87 f a 3D human lymphatic network within native connective tissue devoid of any exogenous material such

88 All had connective tissue disease (CTD) serologies, spirometry,

89 using multivariable Cox regression analysis: connective tissue disease (hazard ratio [HR] 2.94, 95% c

90 n; and patients with PAH in association with connective tissue disease are identified as a particular

91 Bruck Syndrome is a connective tissue disease associated with inactivating m

92 Osteogenesis imperfecta (OI) is a heritable connective tissue disease characterized by bone fragilit

93 Scleroderma is an autoimmune connective tissue disease in which patients make antibod

94 Systemic sclerosis is an autoimmune connective tissue disease in which T cells play a promin

95 roderma (SSc) is a complex and heterogeneous connective tissue disease mainly characterized by autoim

96 ittle cornea syndrome (BCS2) is an inherited connective tissue disease with a devastating ocular phen

97 was 49+/-14 years, 78% were female, 33% had connective tissue disease, 52% were in New York Heart As

98 Raynaud phenomenon without previously known connective tissue disease, nailfold capillaroscopy and l

99 valent in patients with SSc-PAH (n = 81) and connective tissue disease-associated PAH (n = 110) compa

100 patients with schistosomiasis-associated and connective tissue disease-associated PAH had evidence of

101 patients with schistosomiasis-associated and connective tissue disease-associated PAH was also system

102 levels in men with idiopathic, heritable, or connective tissue disease-associated PAH were compared w

103 R and -ETAR Abs are more frequent in SSc-PAH/connective tissue disease-PAH compared with other forms

104 od for IPF (kappaw=0.71 [IQR 0.64-0.77]) and connective tissue disease-related interstitial lung dise

105 iagnosis of idiopathic pulmonary fibrosis or connective tissue disease-related interstitial lung dise

106 limited specificity for a single autoimmune connective tissue disease.

107 tests are obtained to screen for an emerging connective tissue disease.

108 gnificantly increased in patients with mixed connective tissue disease.

109 dication at onset, 6 had cancer, and 3 had a connective tissue disease.

110 ssociated with statin medication, cancer, or connective tissue disease.

111 Inflammatory connective tissue diseases (CTD) like lupus and rheumato

112 Autoimmune connective tissue diseases are clinically variable, maki

113 These mutants may be useful in treating connective tissue diseases associated with increased met

114 anti-SSA/Ro-positive sera from patients with connective tissue diseases showed high reactivity to the

115 i-SSA/Ro Ab-positive sera from patients with connective tissue diseases with the E-pore peptide opens

116 It is often accompanied by statin therapy, connective tissue diseases, cancer, and autoantibodies s

117 BCS is an autosomal recessive generalized connective tissue disorder associated with extreme corne

118 temic sclerosis is a systemic autoimmune and connective tissue disorder associated with the human leu

119 enesis imperfecta (Col1a2(oim)), a heritable connective tissue disorder caused by abnormalities in th

120 Marfan syndrome (MFS) is a heritable connective tissue disorder caused by mutations in FBN1,

121 1, result in Marfan syndrome (MFS), a common connective tissue disorder characterised by tall stature

122 Bruck syndrome (BS) is a connective tissue disorder characterized at the molecula

123 pathogenesis of systemic sclerosis (SSc), a connective tissue disorder characterized by autoimmunity

124 Systemic sclerosis (SSc) is a connective tissue disorder characterized by fibrosis of

125 e alter mitochondrial function and cause the connective tissue disorder cutis laxa.

126 ibromuscular dysplasia was present in 62.7%, connective tissue disorder in 4.9%, and systemic inflamm

127 Importance: Eosinophilic fasciitis (EF) is a connective tissue disorder in which conventional treatme

128 Pelvic organ prolapse is a common connective tissue disorder that affects women.

129 Loeys-Dietz syndrome (LDS) is a connective tissue disorder that is characterized by a hi

130 Loeys-Dietz syndrome (LDS) is a connective tissue disorder that is characterized by skel

131 ssociated with the Ehlers-Danlos syndrome, a connective tissue disorder that leads to a defect in typ

132 s, with the exception of musculoskeletal and connective tissue disorders (992 [8.69% per annum] vs 83

133 perfect predictor of aortic complications in connective tissue disorders (CTDs).

134 myocarditis, and myocarditis associated with connective tissue disorders and may be beneficial in chr

135 inophilic esophagitis show a relationship to connective tissue disorders and that dietary management

136 s crucial for elucidating pathomechanisms of connective tissue disorders characterized by ECM deficie

137 molecularly heterogeneous group of inherited connective tissue disorders that share similar skeletal

138 B-cell lineage hematologic malignancies and connective tissue disorders.

139 eatment of several major systemic autoimmune connective tissue disorders: systemic lupus erythematosu

140 ease of serum amyloid P associated with lung connective tissue during acute respiratory distress synd

141 s (6/12) showed myopathic changes; increased connective tissue elements were observed in seven of eig

142 In mammals, the tendon connective tissue experiences and resists physical force

143 ation in wdSCCs; yet tenascin C retention in connective tissue extracellular matrix suggests the rigi

144 scent-labelled casein, meat myofibrillar and connective tissue extracts to explore their effects on m

145 e prominent members of the family of fibrous connective tissues (FCTs), which collectively are the mo

146 Both CHIKV strains infected connective tissue fibroblasts of the muscle, but only th

147 Gata4 mosaic mutations in PPF-derived muscle connective tissue fibroblasts result in the development

148 ize and enhanced tissue repair (strengthened connective tissue formation, improved microvascular form

149 nce conductance regulator (CFTR), cilia, and connective tissue gene sets.

150 fecting variants in immune, CFTR, cilia, and connective tissue genes than their unaffected family mem

151 ctiveness of coronally advanced flap (CAF) + connective tissue graft (CTG) + PRF in Miller Class I an

152 The present study shows 2-year results of a connective tissue graft (CTG) associated with LLLT in th

153 t coverage using collagen matrix (CM) versus connective tissue graft (CTG) for treatment of multiple

154 The subepithelial connective tissue graft (CTG) is a popular means to trea

155 uccal gingival recession (bREC), effect of a connective tissue graft (CTG) when combined with a bucca

156 CI: 10.21 to 38.66, P <0.000), and use of a connective tissue graft (OR 4.56, 95% CI: 1.72 to 12.08,

157 Subepithelial connective tissue graft (SCTG) procedures provide the be

158 Connective tissue grafts (CTGs) and coronally advanced f

159 Whether the composition of palatal connective tissue grafts (CTGs) varies depending on dono

160 Although connective tissue grafts with coronally advanced flaps (

161 0.04) in the subgroup that was treated with connective tissue grafts.

162 the gap between buccal plate and fixture; 5) connective tissue grafts; and 6) immediate provisionaliz

163 Connective tissue growth factor (CCN2) drives fibrogenes

164 sed nuclear YAP and expression of downstream connective tissue growth factor (CCN2).

165 Pathways of liver fibrosis are controlled by connective tissue growth factor (CCN2).

166 cytosol, increase expression of YAP and TAZ connective tissue growth factor (CTGF) and Cyr61 target

167 Connective tissue growth factor (CTGF) and transforming

168 acellular matrix (ECM) complex consisting of connective tissue growth factor (CTGF) and vascular endo

169 Here, we define connective tissue growth factor (CTGF) as a therapeutic

170 TGF-beta1 (TGFB1), Collagen1alpha1, and connective tissue growth factor (CTGF) gene expression w

171 Delivery of connective tissue growth factor (CTGF) into full-transec

172 Connective tissue growth factor (CTGF) is a downstream m

173 Connective tissue growth factor (CTGF) is a matricellula

174 In mice, connective tissue growth factor (CTGF) is expressed in e

175 Connective tissue growth factor (CTGF) is important for

176 Connective tissue growth factor (CTGF) is known to regul

177 The matricellular secreted protein connective tissue growth factor (CTGF) is upregulated in

178 Connective tissue growth factor (CTGF) up-regulation ind

179 Connective tissue growth factor (CTGF), a direct target

180 We identified connective tissue growth factor (CTGF), a matricellular

181 Connective tissue growth factor (CTGF), a matrix-associa

182 cysteine-rich angiogenic inducer 61 (CYR61), connective tissue growth factor (CTGF), and ankyrin repe

183 in-1, collagen I, alpha-smooth muscle actin, connective tissue growth factor (CTGF), and plasminogen

184 TGF-beta), TGF-beta receptor 1 (TGF-betaR1), connective tissue growth factor (CTGF), E-cadherin, SRY-

185 F-1), tumor growth factor beta1 (TGF-beta1), connective tissue growth factor (CTGF), procollagen I ca

186 targeting cell cycle regulator cyclin A and connective tissue growth factor (CTGF).

187 aturation through the regulated secretion of connective tissue growth factor (CTGF).

188 l fibrosis, particularly CCN2 [also known as connective tissue growth factor (CTGF)], markers of epit

189 ERK1/2 acts on connective tissue growth factor (CTGF/CCN2) expression t

190 Several lines of evidence indicate that connective tissue growth factor (CTGF/CCN2) stimulates c

191 diomyocyte-TrkA recognition by matricellular connective tissue growth factor (CTGF/CCN2).

192 We found that connective tissue growth factor a (ctgfa) is induced in

193 expression of two TEAD-dependent genes, the connective tissue growth factor and amphiregulin genes.

194 duced upregulation of the fibrogenic protein connective tissue growth factor and collagen III in vitr

195 ury as well as decreased renal expression of connective tissue growth factor and collagens I and IV.

196 Aldosterone increased connective tissue growth factor and hydroxyproline expre

197 sts, in contrast, promoted a SMAD3-dependent connective tissue growth factor and plasminogen activato

198 thelial cell-cycle arrest via SMAD3-mediated connective tissue growth factor and plasminogen activato

199 mRNA levels of profibrotic genes, including connective tissue growth factor and tissue inhibitor of

200 mbined elimination of TGF-beta signaling and connective tissue growth factor as a potential therapeut

201 sm of type I collagen production and suggest connective tissue growth factor as its potent mediator.

202 e myosins, NotchR and Wnt pathway genes, and connective tissue growth factor by Pofut1 in skeletal mu

203 rogenitor cells were selectively enriched by connective tissue growth factor delivery (CTGF delivery)

204 vels, which coincided with (1) a decrease in connective tissue growth factor expression, (2) a reduct

205 /endothelial cell adhesion gene (Thbs4), the connective tissue growth factor gene (Ctgf), and the sel

206 accompanied by significant up-regulation of connective tissue growth factor mRNA levels.

207 that cysteine-rich protein 61 (CYR61)/CYR61 connective tissue growth factor nephroblastoma overexpre

208 Hif-1alpha and its downstream target connective tissue growth factor were necessary for the d

209 Other molecular (collagen IV and connective tissue growth factor) and histological (tubul

210 or-beta, endothelin-1, angiotensin II, CCN2 (connective tissue growth factor), and platelet-derived g

211 on (>95%) and subsequent PAI-1, fibronectin, connective tissue growth factor, and p21 expression in h

212 ding fibronectin, alpha-smooth muscle actin, connective tissue growth factor, collagen I, and TGF-bet

213 ncluding transforming growth factor-beta and connective tissue growth factor, in muscles of periodate

214 ransforming growth factor-beta1 (TGF-beta1), connective tissue growth factor, matrix metalloproteinas

215 agonists of transforming growth factor-beta, connective tissue growth factor, several tyrosine kinase

216 decorin, important for the interaction with connective tissue growth factor, was detected predominan

217 ogen activator inhibitor-1, fibronectin, and connective tissue growth factor, was suppressed by metfo

218 alization of TAZ and increased expression of connective tissue growth factor.

219 KLF5 synergistically enhances expression of connective tissue growth factor.

220 xpression of the classical Hippo target CCN2/connective-tissue growth factor (CTGF), as well as ancho

221 elated overlap phenotypes between muscle and connective tissue have mutations in collagen VI.

222 thelial cells play a key role in stimulating connective tissue healing through a FOXO1-dependent mech

223 ted possible involvement of keratinocytes in connective tissue healing.

224 nflammatory classical complement pathway and connective tissue homeostasis.

225 decreased alveolar bone levels and a loss of connective tissue homeostasis.

226 r biology are underscoring the importance of connective tissue in the local tumor environment.

227 P <0.05), and increased amount of uninflamed connective tissue in the M1-M2 interproximal area (65.2%

228 sil documents the oldest known occurrence of connective tissues in association with the flight feathe

229 SPGs) are important structural components of connective tissues in essentially all metazoan organisms

230 al-based therapies have been shown to reduce connective tissue inflammation and fibrosis and thus may

231 Dense connective tissue injuries have limited repair, due to t

232 asal epithelium positioned at the epithelial-connective tissue interface (ECTI), separating the basal

233 expressing B-lineage cells at the epithelial-connective tissue interface.

234 er, the majority of the bone, cartilage, and connective tissue is derived from the neural crest.

235 Connective tissue is involved in driving regenerating fi

236 A connective tissue layer occurred between these (ie, Told

237 d mast cell numbers in the skin and impaired connective tissue-like mast cell survival upon Fcepsilon

238 melanoma cytomorphological findings, closed connective tissue loops, mitotic count, chromosome 3 los

239 e for two of these genes (EFEMP1 and WT1) in connective tissue maintenance/homoeostasis.

240 a family with a congenital-onset disorder of connective tissue, manifesting as early-onset joint hype

241 that A1 is essential for the homeostasis of connective tissue mast cells, identifying A1 as a possib

242 ls were used as "surrogates" for mucosal and connective tissue mast cells, respectively, and their re

243 e number of blood vessels and sub-epithelial connective tissue matrix components within the wound bed

244 hosphorylation, neuropeptide biogenesis, and connective tissue maturation.

245 osphate/phosphate ratio is the cause of soft connective tissue mineralization in these disorders.

246 dered in patients presenting with congenital connective tissue/myopathy overlap disorders with joint

247 by Menninghaus et al. fails because its main connective tissue - "negative emotions" - is beyond the

248 Tuberous sclerosis complex-related connective tissue nevi are not limited to the lower back

249 cation, size, and histological appearance of connective tissue nevi in patients with TSC.

250 To describe the clinical characteristics of connective tissue nevi on the trunk and extremities of p

251 Connective tissue nevi were categorized per anatomic loc

252 complex (TSC) frequently develop collagenous connective tissue nevi.

253 [19-70] years) with TSC (56%) had at least 1 connective tissue nevus on the trunk or thighs; of these

254 The connective tissue of any organ in the body is generally

255 d disorder of unknown etiology affecting the connective tissue of the body.

256 The juxtacanalicular connective tissue of the trabecular meshwork together wi

257 a precursor protein that is expressed by the connective tissues of the craniofacial skeleton, namely,

258 For gadolinium-enhanced MRI in pregnancy, connective tissue or skin disease resembling nephrogenic

259 ercentage of vital bone, residual graft, and connective tissue/other non-bone components.

260 The main expansion then occurs in the connective tissue outside the islet, which remains more

261 ining revealed a greater proportion of dense connective tissue (P=0.02) in women compared with men.

262 of chronic pain associated with degenerative connective tissue pathologies, such as intervertebral di

263 Nonimmunological connective tissue phenotypes in humans are common among

264 lagen VI-related myopathies are disorders of connective tissue presenting with an overlap phenotype c

265 apamycin (90.3% and 82.9%), respectively, 2) connective tissue progenitor cells, 3) platelet-derived

266 Connective tissue progenitor cells, platelet-derived gro

267 Connective tissue progenitors (CTPs) are a promising the

268 formed blastema, the spatial coordinates of connective tissue progenitors are predictive of their ul

269 erating zebrafish fins provide evidence that connective tissue progenitors are rapidly organized into

270 protease hydrolysis of meat myofibrillar and connective tissue protein extracts produces bioactive pe

271 r ability to hydrolyse meat myofibrillar and connective tissue protein extracts to produce bioactive

272 ysis over 120 and 60 min of myofibrillar and connective tissue proteins, respectively.

273 nd the outer, orbital layer inserts into the connective tissue pulley.

274 pic delivery of Tgfbeta1 protein into nipple connective tissue reduced epidermal proliferation.

275 growth (growth hormone binding protein), and connective tissue remodeling (MMP3).

276 FRalpha may be a relevant target to regulate connective tissue remodeling.

277 ms by diminishing inflammation and promoting connective tissue repair.

278 Fibroblasts are then cultured to produce connective tissue rich in extracellular matrix (stage 2)

279 ing parturition, and are usually high-grade, connective tissue-rich, and estrogen receptor (ER)/proge

280 nytoin in gingival epithelial tissues and in connective tissues similar to that seen in humans.

281 Fibrosis of the subsynovial connective tissue (SSCT) is a pathognomonic change in ca

282 hat selective breeding has resulted in lower connective tissue stability of Atlantic salmon fillets.

283 Grem1 expression identifies distinct connective tissue stem cells in both the bone (OCR stem

284 well-recognized contributors to cancer, and connective tissue stiffness is emerging as a driving fac

285 DR3 expression was recorded on connective tissue stroma, which provided DR3-dependent r

286 ly adjacent to major blood vessels and other connective tissue structures.

287 nce the majority of the bones, cartilage and connective tissues that comprise the head and face are d

288 d, subsequently, differentiate into bone and connective tissue to form the mandible.

289 nd clinical management of three intermediate connective tissue tumors: desmoid tumor (DT) or aggressi

290 Primary murine mucosal-type MC and connective tissue-type MC released phenotypically differ

291 ingly, proteomic analysis of CD63(+) EV from connective tissue-type MC unveiled an abundance of MC-sp

292 h PNTM also had significantly more cilia and connective tissue variants per person than did control s

293 tissue as either skin, fat, glands, ducts or connective tissue was demonstrated with an overall accur

294 ecently to genetic diseases characterized by connective tissue wasting (Penttinen syndrome) or overgr

295 A surface mesothelium and underlying connective tissue were evident throughout.

296 ructural and load-bearing element of various connective tissues, where it forms the extracellular mat

297 ral crest generates the skull and associated connective tissues, whereas placodes produce sensory org

298 essential for inducing the formation of new connective tissue, which requires the generation of sign

299 -Danlos syndrome, are likely to have fragile connective tissues with increased susceptibility to trau

300 ight at harvesting, softer texture and lower connective tissue yield compared with the WP group.