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1 n anti-mineralization role in the peripheral connective tissue cells.
2 er cells, hyaline cells, ganglion cells, and connective tissue cells.
3 ransforming growth factor beta (TGF-beta) in connective tissue cells.
4 cs of cultured human gingival epithelial and connective tissue cells.
5 growth factor (LDGF), which is mitogenic for connective tissue cells.
6 diator expression was limited mainly to some connective tissue cells.
7 s detected in interalveolar and interlobular connective tissue cells adjacent to glandular alveolar (
8  to determine whether MMP-10 is expressed in connective tissue cells and to assess how it may contrib
9 r the wound, immunostaining for fibroblasts, connective tissue cells, and newly forming vasculature b
10 tensively in vestibular supporting cells and connective tissue cells, and the two Cxs were co-localiz
11 deposition, inflammatory cell retention, and connective tissue cell differentiation, respectively.
12 ls involved in wound closure in vitro and in connective tissue cells during closure of gingival wound
13 served cellular response programs of derived connective tissue cells (e.g., chondroblasts and osteobl
14 -1 and mTLL-1 in lysyl oxidase activation by connective tissue cells, fibroblasts cultured from Bmp1-
15                   The origin and turnover of connective tissue cells in adult human organs, including
16 embryogenesis and (2) a layer of fibroblasts/connective tissue cells in the gastrointestinal tract, t
17 GF-beta can induce the expression of CTGF in connective tissue cells in vivo.
18 rmones induce transcriptional events in many connective tissue cells, including osteoblasts.
19      Originally identified as progenitors of connective tissue cell lineages, recent findings have re
20 hat cytokines and growth factors produced by connective tissue cells might concentrate in BL, where t
21         Here we develop a method for fitting connective-tissue cell migration data to persistent rand
22                                       Do the connective tissue cells of the mammalian digit play a ro
23  tumors (aggressive fibromatosis) arise from connective tissue cells or fibroblasts.
24 ession of phenotypical markers, the emerging connective tissue cell population may originate from mic
25 ed macrophage influx and an expansion of the connective tissue cell population.
26 what we believe to be the first evidence for connective tissue cell progenitors that reside locally w
27 nstrate that, like TGF-beta, CTGF can induce connective tissue cell proliferation and extracellular m
28      However, the migration of slowly moving connective-tissue cells, such as fibroblasts, is difficu
29 trate distinct positional characteristics of connective tissue cells that are associated with their r
30  to create 3D open scaffolds for adhesion of connective tissue cells through well-defined adhesion pl
31  suggests that HGF secreted by interalveolar connective tissue cells traverses the acinar cells and m
32  basis for the divergence of CTGF actions on connective tissue cell types and suggest a model for fun
33 art valve development in parallel with other connective tissue cell types.
34 anipulation transmits a mechanical signal to connective tissue cells via mechanotransduction.
35               TGF-beta1 induced expansion of connective tissue cells with a myofibroblast phenotype,

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