ライフサイエンスコーパス: constrictive

1 contribution of acetylcholine to the airway constrictive and lumenal obstructive response after inha

2 sive and calcified coronary atherosclerosis, constrictive arterial remodeling, and greater disease pr

3 ted reaction intermediates and the effect of constrictive binding on the bound guest.

4 hosts and guests need to be balanced with a constrictive binding to allow thermally activated chemic

5 This reaction pathway, which is promoted by constrictive binding within the supramolecular cavity of

6 Primary bronchiolar disorders include constrictive bronchiolitis (obliterative bronchiolitis,

7 to the airway epithelium play a key role in constrictive bronchiolitis after lung transplantation, t

8 All soldiers with constrictive bronchiolitis had normal results on chest r

9 remaining 11 soldiers, diagnoses other than constrictive bronchiolitis that could explain the presen

10 ion is associated with a progressive form of constrictive bronchiolitis that targets conducting airwa

11 ation and pulmonary edema in severe cases to constrictive bronchiolitis, being a more distant consequ

12 an analysis of biopsy samples showed diffuse constrictive bronchiolitis, which was possibly associate

13 diers having changes that were diagnostic of constrictive bronchiolitis.

14 pathologic hypertrophy, and restrictive from constrictive cardiomyopathy.

15 uronal cells from the proximal stump and the constrictive, circumferential forces imposed by the cont

16 Penile injection therapy and vacuum constrictive devices are reasonable choices for men in w

17 st that Z-ring contraction serves as a major constrictive force generator to limit the progression of

18 its the division machinery, and may generate constrictive forces necessary for cytokinesis.

19 Histology confirmed a constrictive form of bronchiolitis caused by expansion o

20 -up echocardiograms showed resolution of the constrictive hemodynamics without pericardiectomy.

21 patients (57%), restrictive in 12 (34%) and constrictive in 3 (9%).

22 ents (40.5%), restrictive in 17 (40.5%), and constrictive in 8 (19%).

23 to dose of 10 mg/kg of 1 in the rat chronic constrictive injury model of neuropathic pain.

24 analgesic efficacy data from the rat chronic constrictive injury model of neuropathic pain.

25 l models of neuropathic pain, transection or constrictive injury to peripheral nerves produces ectopi

26 rization is responsible for the formation of constrictive membrane curvature.

27 In addition, in the chronic constrictive model of neuropathic pain, compound 34 sign

28 ed in cold allodynia using rats with chronic constrictive nerve injury (CCI), a neuropathic pain mode

29 The cause of constrictive pericardial disease was indeterminate in 67

30 ve pulmonary disease without an overlap with constrictive pericarditis (39.5+/-18.8 cm/s vs. 4.2+/-3.

31 h significant reductions in the incidence of constrictive pericarditis (4.4% vs. 7.8%; hazard ratio,

32 ted to be useful for differentiating chronic constrictive pericarditis (CP) and restrictive cardiomyo

33 venous flow (PV) velocities in patients with constrictive pericarditis (CP) and to describe the influ

34 Differentiation of constrictive pericarditis (CP) from restrictive cardiomy

35 peptide (BNP) measurements to differentiate constrictive pericarditis (CP) from restrictive cardiomy

36 The clinical spectrum of constrictive pericarditis (CP) has been affected by a ch

37 Constrictive pericarditis (CP) is a disabling disease, a

38 between restrictive cardiomyopathy (RCM) and constrictive pericarditis (CP) is challenging and, despi

39 atheterization criteria for the diagnosis of constrictive pericarditis (CP) rely on equalization of i

40 study was to compare myocardial mechanics of constrictive pericarditis (CP) with restrictive cardiomy

41 In constrictive pericarditis (CP), E' is not reduced, despi

42 to determine the association of etiology of constrictive pericarditis (CP), pericardial calcificatio

43 theterization criterion for the diagnosis of constrictive pericarditis (CP).

44 the causes and natural history of transient constrictive pericarditis (CP).

45 (n = 162), peritoneal disease (n = 10), and constrictive pericarditis (n = 2).

46 Patients with surgically confirmed constrictive pericarditis (n=130) at Mayo Clinic (2008-2

47 mal in a substantial number of patients with constrictive pericarditis after pericardiectomy.

48 rmed in 5 patients with surgically confirmed constrictive pericarditis and 12 patients (control subje

49 d echocardiographic data of 50 patients with constrictive pericarditis and 44 with restrictive cardio

50 Constrictive pericarditis and chronic obstructive pulmon

51 elocity was 13% +/- 6% and -8% +/- 7% in the constrictive pericarditis and control groups, respective

52 ction after pericardiectomy in patients with constrictive pericarditis and correlated postoperative D

53 city duration are increased in patients with constrictive pericarditis and may be helpful in diagnosi

54 ides a clinically useful distinction between constrictive pericarditis and restrictive cardiomyopathy

55 y data sets derived from patients with known constrictive pericarditis and restrictive cardiomyopathy

56 elocity is the main diagnostic criterion for constrictive pericarditis by Doppler echocardiography, i

57 mited cutaneous scleroderma in whom calcific constrictive pericarditis developed.

58 icacy of pericardiectomy, some patients with constrictive pericarditis fail to improve postoperativel

59 measuring PVF have included: differentiating constrictive pericarditis from restriction, estimation o

60 ular expansion velocities in differentiating constrictive pericarditis from restrictive cardiomyopath

61 classifier was evaluated for differentiating constrictive pericarditis from restrictive cardiomyopath

62 rly diastolic velocity to help differentiate constrictive pericarditis from restrictive cardiomyopath

63 Echocardiography allows differentiation of constrictive pericarditis from restrictive myocardial di

64 e chronic obstructive pulmonary disease from constrictive pericarditis in patients with a respiratory

65 tion on a plain radiograph strongly suggests constrictive pericarditis in patients with heart failure

66 Constrictive pericarditis is a potentially reversible ca

67 However, calcific constrictive pericarditis is considered rare in the Unit

68 A physiologic hallmark of constrictive pericarditis is enhanced ventricular interd

69 Constrictive pericarditis is the result of a spectrum of

70 Constrictive pericarditis represents a serious hemodynam

71 inflammation and may identify patients with constrictive pericarditis that will improve with anti-in

72 In patients with constrictive pericarditis treated with anti-inflammatory

73 om 1985 to 1995, a total of 58 patients with constrictive pericarditis underwent pericardiectomy and

74 Constrictive pericarditis was confirmed by the surgical

75 ease or severe tricuspid regurgitation after constrictive pericarditis was considered but ruled out.

76 m onset to peak inspiration in patients with constrictive pericarditis were significantly different f

77 In 12 patients with surgically confirmed constrictive pericarditis who had < 25% respiratory vari

78 We identified 41 consecutive patients with constrictive pericarditis who had a cardiovascular magne

79 measured at 26 locations in 11 patients with constrictive pericarditis who underwent intraoperative t

80 dinal axis expansion (Ea) in 8 patients with constrictive pericarditis, 7 patients with restriction a

81 ed in 18% of patients with surgically proven constrictive pericarditis, although the histopathologica

82 des an important opportunity to evaluate for constrictive pericarditis, and definite diagnostic crite

83 or absent during the evaluation of suspected constrictive pericarditis, repeat Doppler recording of m

84 because of cardiovascular complications (one constrictive pericarditis, two right heart failures with

85 ose of 20 patients who had surgically proved constrictive pericarditis.

86 velocity, which is not seen in patients with constrictive pericarditis.

87 are less restrictive compared with those in constrictive pericarditis.

88 nd should be a valuable adjunct in assessing constrictive pericarditis.

89 eristic Doppler echocardiographic feature in constrictive pericarditis.

90 respiration in relation to the diagnosis of constrictive pericarditis.

91 ion could assist in noninvasively diagnosing constrictive pericarditis.

92 c tamponade requiring pericardiocentesis, or constrictive pericarditis.

93 dependently associated with the diagnosis of constrictive pericarditis.

94 c tamponade requiring pericardiocentesis, or constrictive pericarditis.

95 iae that progressed to cardiac tamponade and constrictive pericarditis.

96 ent treatments for pericardial effusions and constrictive pericarditis.

97 amponade, but also to suggest a diagnosis of constrictive pericarditis.

98 imilar to that observed in clinical cases of constrictive pericarditis.

99 It represents a novel animal model of constrictive pericarditis.

100 onsidered an essential diagnostic feature of constrictive pericarditis.

101 estrictive cardiomyopathy and preserved with constrictive pericarditis.

102 ication is a common finding in patients with constrictive pericarditis.

103 formed promptly in symptomatic patients with constrictive pericarditis.

104 phenomenon that has been labeled "transient constrictive pericarditis." No large studies have examin

105 variables were independently associated with constrictive pericarditis: (1) ventricular septal shift,

106 as composed of a combination of dilatory and constrictive phases.

107 ardial inflammation and fibrosis, leading to constrictive phenotype during the acute phase of disease

108 The KO mice exhibited a classic restrictive/constrictive phenotype with decreased cardiac output, in

109 e course of their illness, resolution of the constrictive physiologic features occurred at an average

110 ome patients with acute CP, the symptoms and constrictive physiologic features resolve with medical t

111 had become normal, and 1 patient who had had constrictive physiology had restrictive findings.

112 ients with restrictive physiology and 5 with constrictive physiology the results had become normal, a

113 eg irons, or stocks were used to put tightly constrictive pressure around the extremities of POWs as

114 eparation in a 135-min cell cycle so the two constrictive processes are separated in both time and sp

115 ages of the cell division site show separate constrictive processes closing first the inner membrane

116 structure allows pore water to penetrate the constrictive region and to form a continuous water wire

117 ion by the cell types most likely to mediate constrictive remodeling after angioplasty.

118 Expression of TGF-beta3 inhibits constrictive remodeling after PTCA and reduces luminal l

119 a potential target for therapies to prevent constrictive remodeling and restenosis.

120 dense collagenous adventitia, which prevents constrictive remodeling by acting as an external scaffol

121 hese changes resulted in a trend toward more constrictive remodeling in low- compared with high-WSS s

122 c arteries contributes to intimal growth and constrictive remodeling leading to lumen loss.

123 Constrictive remodeling of the arterial wall was observe

124 ter plaque and necrotic core progression and constrictive remodeling, and high-WSS segments develop g

125 ement is vessel expansion and attenuation of constrictive remodeling.

126 t delay re-endothelialization and also cause constrictive remodeling.

127 ul in limiting intimal growth and preventing constrictive remodeling.

128 educes luminal loss after PTCA by inhibiting constrictive remodeling.

129 wall after angioplasty, where it may inhibit constrictive remodeling.

130 eceptor antagonist, will decrease the airway constrictive response and acute bronchial obstruction to

131 n littermates were used to examine the acute constrictive response of the developing retinal vessels

132 In contrast, beta2AR-OE mice had enhanced constrictive responses.

133 Thus, betaAR antithetically regulates constrictive signals, affecting bronchomotor tone/reacti

134 Three major processes, constrictive vessel remodeling, intimal hyperplasia (IH)