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1 effects of PCMV activation in the recipient (consumptive coagulopathy).
2 hemodynamic instability, capillary leak, and consumptive coagulopathy.
3 yonic blood vessel endothelium causes lethal consumptive coagulopathy.
4 bosis after 3 weeks, terminating in a lethal consumptive coagulopathy.
5 eir illness, both patients had evidence of a consumptive coagulopathy.
6        Treatment with RA101295 also improved consumptive coagulopathy and preserved endothelial antic
7 oach to prevent sepsis-induced inflammation, consumptive coagulopathy, and subsequent organ failure a
8  extremities and multiple organ systems, and consumptive coagulopathy as the disease end-point provid
9 he time of rejection or the development of a consumptive coagulopathy, biopsy specimens were obtained
10 proach to the treatment of sepsis-associated consumptive coagulopathy, but its application is limited
11        A rapidly progressive disorder termed consumptive coagulopathy (CC) has been observed frequent
12 njury and possibly an increased incidence of consumptive coagulopathy (CC).
13 mbined with the expression of tissue factor, consumptive coagulopathy developed irrespective of histo
14            Embryos lacking TM develop lethal consumptive coagulopathy during this period, and no live
15 V) has been implicated in the development of consumptive coagulopathies in severely envenomed patient
16 ed in prolonged graft survival and prevented consumptive coagulopathy in all recipients.
17 tic microangiopathy in the graft or systemic consumptive coagulopathy in the recipient.
18        Delayed bleeding is often ascribed to consumptive coagulopathy initiated by exposed brain tiss
19 e degree of hemostatic stress of a non-overt consumptive coagulopathy (nonovert DIC).
20           Clinical or laboratory features of consumptive coagulopathy occurred in 7 of 12 baboons.
21 ion and tissue factor expression; in vivo, a consumptive coagulopathy occurred when there was xenorea
22 ion tests are most useful in detecting overt consumptive coagulopathy (overt DIC) near the time of ch
23  Blood, Hijazi et al challenge the view that consumptive coagulopathy that accompanies traumatic brai
24 lated factor VIIa/TF action and a consequent consumptive coagulopathy underlies the bleeding diathesi

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