ライフサイエンスコーパス: contact dermatitis

1 CD8+ T cells from individuals with allergic contact dermatitis.

2 presents select aspects of AD, psoriasis, or contact dermatitis.

3 ration of the phorbol ester-induced irritant contact dermatitis.

4 ermatitis and oxazolone, a model of allergic contact dermatitis.

5 lize to immunologically nonspecific forms of contact dermatitis.

6 is, fixed drug eruption, atopic and allergic contact dermatitis.

7 ed utilizing models of irritant and allergic contact dermatitis.

8 sulfate is a well-known inducer of irritant contact dermatitis.

9 nduced skin cancers or allergic and irritant contact dermatitis.

10 g psoriasis, atopic dermatitis, and allergic contact dermatitis.

11 ional skin disease are irritant and allergic contact dermatitis.

12 ibitor, ARN077, in a mouse model of allergic contact dermatitis.

13 acterize the role of MCs in chronic allergic contact dermatitis.

14 of murine skin serves as a model of allergic contact dermatitis.

15 bly influence the course of chronic allergic contact dermatitis.

16 igand 1 (CXCL1) in a mouse model of irritant contact dermatitis.

17 the generation of skin TRM cells in allergic contact dermatitis.

18 e risk of contact sensitization and allergic contact dermatitis.

19 tial therapeutic avenue in treating allergic contact dermatitis.

20 rs they are one of the most common causes of contact dermatitis.

21 gical processes like irritative and allergic contact dermatitis.

22 metics, and they are known to cause allergic contact dermatitis.

23 xperimental animal studies to evoke allergic contact dermatitis.

24 atory responses and pruritus associated with contact dermatitis.

25 ape-stripping test and in a disease model of contact dermatitis.

26 the development of T-cell-mediated allergic contact dermatitis.

27 are antigenic determinants in patients with contact dermatitis.

28 skin of patients with psoriasis or allergic contact dermatitis.

29 ypersensitivity, an animal model of allergic contact dermatitis, a common pruritic disorder in humans

30 studied the role of cathelicidin in allergic contact dermatitis, a model requiring dendritic cells of

31 e confirmed the major findings in irritative contact dermatitis, a second model of cutaneous inflamma

32 Atopic dermatitis (AD) and allergic contact dermatitis (ACD) have a dynamic relationship not

33 tens is an efficient way to prevent allergic contact dermatitis (ACD) in mice.

34 the recent literature pertaining to allergic contact dermatitis (ACD) in the pediatric population.

35 Allergic contact dermatitis (ACD) is a common T-cell mediated inf

36 Allergic contact dermatitis (ACD) is the most common occupational

37 Allergic contact dermatitis (ACD) is well recognized as an advers

38 Further, allergic contact dermatitis (ACD) model was used to evaluate the

39 skin permeation was investigated in allergic contact dermatitis (ACD) mouse model.

40 ls) in cellular infiltrate of human allergic contact dermatitis (ACD) skin challenge sites.

41 rritant contact dermatitis (ICD) or allergic contact dermatitis (ACD) that is sometimes clinically di

42 e evaluated the effect of OA-NO2 on allergic contact dermatitis (ACD) using an established model of c

43 ial role of Th2 responses in nickel allergic contact dermatitis (ACD) was demonstrated.

44 ritant contact dermatitis (ICD) and allergic contact dermatitis (ACD), are common skin diseases.

45 mation are commonly associated with allergic contact dermatitis (ACD), it is not known if they are me

46 immunologic similarities to chronic allergic contact dermatitis (ACD).

47 blocker (EC50=2 nM), could suppress allergic contact dermatitis (ACD).

48 f SUCNR1/GPR91 expression mediating allergic contact dermatitis (ACD).

49 that IL-9 also has a role in human allergic contact dermatitis (ACD).

50 atitis (AD) as well as allergic and irritant contact dermatitis (ACD, ICD) are characterized by the s

51 by either inflammation alone (acute irritant contact dermatitis, acute allergic contact dermatitis) o

52 Allergic contact dermatitis affects 20% of children at some time

53 t SerpinB2(-/-) mice are more susceptible to contact dermatitis after topical application of dinitrof

54 hat MI can be an important cause of airborne contact dermatitis among painters and consumers.

55 pathogenesis of certain diseases, including contact dermatitis and allergy.

56 n and condition immune responses in irritant contact dermatitis and atopic dermatitis.

57 Certain skin disorders, such as contact dermatitis and chronic urticaria, are characteri

58 Allergic contact dermatitis and ICD were characterized by IFN-gam

59 ose association was also found with allergic contact dermatitis and increased specific IgE to Malasse

60 R agonists reduce inflammation in a model of contact dermatitis and inhibit inflammatory gene express

61 Allergic contact dermatitis and its animal model, contact hyperse

62 rhinitis, asthma, and/or eczema in 38.2% and contact dermatitis and other eczema in 35.9%), and menta

63 12-myristate-13-acetate, a model of irritant contact dermatitis and oxazolone, a model of allergic co

64 hich NKT cells play a role, such as allergic contact dermatitis and psoriasis.

65 cals and may experience side effects such as contact dermatitis and skin irritation.

66 (n = 703) presenting with possible allergic contact dermatitis and subsequently undergoing patch tes

67 tly compromised, such as psoriasis, allergic contact dermatitis, and blistering disorders.

68 es, including psoriasis, atopic and allergic contact dermatitis, and cutaneous T-cell lymphoma.

69 ng inflammatory events such as wound repair, contact dermatitis, and psoriasis.

70 ing chemicals in the development of allergic contact dermatitis, and suggest that the irritant effect

71 o strong levels in basal cells of psoriasis, contact dermatitis, and the proliferative cells of the a

72 ckground such as atopic dermatitis, allergic contact dermatitis, and urticaria are very common.

73 atory activity in both irritant and allergic contact dermatitis animal models.

74 Additional cases of allergic contact dermatitis are being reported with temporary hen

75 Atopic dermatitis and allergic contact dermatitis are both common skin diseases having

76 and human dermatological conditions, such as contact dermatitis, are discussed.

77 be applicable for the treatment of allergic contact dermatitis, are still largely unknown.

78 ed with either atopic dermatitis or allergic contact dermatitis as well as in an inducible mouse mode

79 pment of a spray to detect urushiol to avoid contact dermatitis, as well as to detect catecholamines

80 ent barrier abnormalities (subacute allergic contact dermatitis, atopic dermatitis), topical H1/2r ag

81 toxin C5a is a critical mediator of allergic contact dermatitis, bridging essential aspects of innate

82 is one of the most common forms of allergic contact dermatitis, but how the T-cell receptor (TCR) re

83 er defects might also predispose to allergic contact dermatitis by allowing greater penetration of ch

84 Atopic dermatitis (AD), psoriasis (PS), and contact dermatitis (CD) are common skin diseases, charac

85 ribe the histopathological features of acute contact dermatitis (CD).

86 We report 7 cases of erosive irritant contact dermatitis due to chlorhexidine gluconate-impreg

87 observed seem to be in most cases 'systemic contact dermatitis' due to oral or parenteral re-exposur

88 suffering from psoriasis, atopic or allergic-contact dermatitis express CCR10.

89 ngredients represented in the North American Contact Dermatitis Group (NACDG) series were conducted u

90 Clinical scoring using the North American Contact Dermatitis Group method was also performed.

91 Patch testing used the North American Contact Dermatitis Group standard series.

92 The role of MCs in chronic allergic contact dermatitis has not been investigated, in part be

93 past decade, mechanisms underlying allergic contact dermatitis have been intensively investigated by

94 tes, especially during allergic and irritant contact dermatitis, however, is less well understood.

95 Atopic dermatitis (AD), as well as irritant contact dermatitis (ICD) and allergic contact dermatitis

96 tients, there is often a coexisting irritant contact dermatitis (ICD) or allergic contact dermatitis

97 sible for the signs and symptoms of irritant contact dermatitis (ICD).

98 In addition, AFC inhibits in vivo allergic contact dermatitis in a mouse model utilizing sensitizat

99 The documented rates of allergic contact dermatitis in children are on the rise.

100 logy and clinical manifestations of allergic contact dermatitis in children.

101 the recent literature pertaining to allergic contact dermatitis in the pediatric population.

102 ically applied THC on DNFB-mediated allergic contact dermatitis in wild-type and CB1/2 receptor-defic

103 In contrast to classical contact dermatitis, in which myeloid dendritic cells sen

104 The most represented forms of non-eczematous contact dermatitis include the erythema multiforme-like,

105 had no effect on croton oil-induced irritant contact dermatitis, indicating that morphine's effects o

106 d dyskeratosis follicularis Darier, allergic contact dermatitis, infectious folliculitis, varicella z

107 In patients with contact dermatitis, inflammasome-mediated IL-1 activatio

108 Allergic contact dermatitis is a chronic T cell-driven inflammato

109 Allergic contact dermatitis is a common disorder that has fascina

110 Allergic contact dermatitis is a common skin disease associated w

111 Erosive contact dermatitis is an under-recognized complication o

112 Allergic contact dermatitis is commonly associated with exposure

113 ide support for the hypothesis that allergic contact dermatitis is not a classic delayed type hyperse

114 Allergic contact dermatitis is one of the most common occupationa

115 Allergic contact dermatitis is the most frequent occupational dis

116 Contact dermatitis is the result of inflammatory respons

117 chanism in those who do not develop allergic contact dermatitis is tolerance induction by repeated ex

118 own to cause skin rash, dermal inflammation, contact dermatitis, leucoderma, and cancer promotion.

119 Allergic contact dermatitis may affect as many as 20% of the pedi

120 ave suggested that chemical-induced allergic contact dermatitis may not be a traditional type IV hype

121 iscontinued therapy because of side effects (contact dermatitis [n = 2], nausea [n = 1], and acute pa

122 Allergic contact dermatitis occurs less frequently in the first f

123 have significantly higher rates of allergic contact dermatitis on the face.

124 irritant contact dermatitis, acute allergic contact dermatitis) or by prominent barrier abnormalitie

125 ved in sensory neurons of mice with allergic contact dermatitis- or dry skin-elicited itch; however,

126 Using human skin from contact dermatitis patients and a mouse model of CHS, we

127 Not infrequently, however, contact dermatitis presents with noneczematous features.

128 agonists in models of irritant and allergic contact dermatitis produced in mouse ears by topical tre

129 The contact dermatitis rate was 1.1% with and 0.29% without

130 d inflammation in human skin resembles acute contact dermatitis rather than psoriasis.

131 Furthermore, allergic and irritant contact dermatitis reactions were exaggerated in FVB.del

132 pite a trend toward weaker clinical allergic contact dermatitis reactions.

133 CD39 deficiency in irritant versus allergic contact dermatitis, reflecting its diverse roles in regu

134 To use data from the Pediatric Contact Dermatitis Registry to elucidate the association

135 or fragrances were derived from the American Contact Dermatitis Society's Contact Allergen Management

136 Allergic contact dermatitis, such as in response to poison ivy or

137 In a well-established model of allergic contact dermatitis, the absence of Mincle leads to a sig

138 tory responses in a mouse model for allergic contact dermatitis, the contact hypersensitivity (CHS) r

139 chlorhexidine gluconate is a known cause of contact dermatitis, the phenotypic range of this adverse

140 The prevalence of allergic contact dermatitis to allergens (eg, fragrance) is highe

141 dy we use an in vivo mouse model of allergic contact dermatitis to investigate how nanoparticles (NPs

142 ed aluminium tubes pose a risk of developing contact dermatitis to patients sensitized to ER based on

143 frequency of DNCB sensitization and allergic contact dermatitis to topically applied mechlorethamine

144 Additionally, contact dermatitis, urticaria, and drug reactions are ad

145 Irritant or allergic contact dermatitis usually presents as an eczematous pro

146 up using white petrolatum developed allergic contact dermatitis vs 4 patients (0.9%) in the group usi

147 r and cellular mechanism underlying allergic contact dermatitis, we evaluated oxazolone-induced chang

148 osts of chlorhexidine-impregnated sponge and contact dermatitis were calculated prospectively using m

149 de of either 691.8 (AD) or 692.9 (eczema and contact dermatitis) were queried.

150 sensitivity reactions, resulting in allergic contact dermatitis, which clinically resembles eczema.

151 In human subjects imiquimod induces contact dermatitis with the distinctive feature that pDC