ライフサイエンスコーパス: contact sensitivity

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1 ese populations in models of anaphylaxis and contact sensitivity.

2 Abs can recruit effector T cells to mediate contact sensitivity.

3 a relevant population of T cells involved in contact sensitivity.

4 ress has no effect on the course of irritant contact sensitivity, an immune reaction that does not in

5 ter of dermatitis patient's patch tested for contact sensitivity and (ii) the Danish National Patient

6 fect of C5L2 deficiency in a murine model of contact sensitivity, and second to determine whether the

7 Thus, in allergic contact sensitivity, as in isolated human neutrophils, C

8 nt soluble T cell receptors (sTCR) protected contact sensitivity (CS) effector T cells from down-regu

9 ectrum antibiotic enrofloxacin will modulate contact sensitivity (CS) in mice.

10 xperiments examined a two-challenge model of contact sensitivity (CS) in which Treg abundance in the

11 T-cell tolerance of allergic cutaneous contact sensitivity (CS) induced in mice by high doses o

12 Contact sensitivity (CS) is related to delayed-type hype

13 T cell recruitment to elicit contact sensitivity (CS) requires a CS-initiating proces

14 The elicitation of contact sensitivity (CS) to local skin challenge with th

15 Elicitation of contact sensitivity (CS), a classic example of T cell-me

16 yed-type hypersensitivity responses, such as contact sensitivity (CS), in mast cell-deficient mice wh

17 tive transfer of immune cells lacking CD3(+) contact sensitivity effector T cells, or after transfer

18 ammation in Th2-driven asthma and Th1-driven contact sensitivity experiments.

19 Moreover, FTS-A inhibited Rap1 and contact sensitivity far better than FTS.

20 We observed diminished contact sensitivity in mice lacking FcepsilonRI or mast

21 may serve as a possible therapeutic tool in contact sensitivity in particular and T-cell-mediated in

22 Thus, anti-T11(1) mAb suppressed contact sensitivity in vivo in the transgenic/knockout m

23 Induction of contact sensitivity increased the proportion of motile T

24 large and long lasting increase in allergic contact sensitivity or delayed-type hypersensitivity, an

25 esponsiveness was hapten specific, since the contact sensitivity reaction of mAb-treated mice to oxaz

26 Extensive studies of contact sensitivity reactions in mice established a mech

27 pathological (e.g., autoimmune reactions and contact sensitivity reactions such as that to poison ivy

28 ems totally reversed the failure to induce a contact sensitivity response to dinitrofluorobenzene (DN

29 However, the ability to induce a primary contact sensitivity response was completely restored in

30 The contact sensitivity response was significantly reduced (

31 Contact sensitivity responses require both effective imm

32 hairless cub/cub mcub/mcub mice show normal contact sensitivity responses to oxazolone.

33 the regulation of cognate immunity, such as contact sensitivity responses.

34 ment in non-UV-irradiated mice did not block contact sensitivity responses.

35 pic dermatitis are likely to have suppressed contact sensitivity secondary to their disease whereas s

36 P-10 and KC expression during elicitation of contact sensitivity, suggesting CD4+ T cells inhibit the

37 Immune regulation of contact sensitivity to the poison ivy/oak catechol was s

38 Contact sensitivity was markedly impaired in IgE(-/-) mi

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