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1 d to potential Ca(2+) desensitization of VSM contractile apparatus.
2 tized excitation-contraction coupling of the contractile apparatus.
3 rcomeric proteins for proper assembly of the contractile apparatus.
4 the precise length of thin filaments in the contractile apparatus.
5 uscle actin, and mediated disassembly of the contractile apparatus.
6 e diastolic dysfunction through altering the contractile apparatus.
7 ntracellular components of the smooth muscle contractile apparatus.
8 , a large modular protein that surrounds the contractile apparatus.
9 complex located on the thin filaments of the contractile apparatus.
10 4.1R is a component of the contractile apparatus.
11 yotubes and the assembly of a highly regular contractile apparatus.
12 gular alignment of the network SR around the contractile apparatus.
13 re misaligned with respect to the underlying contractile apparatus.
14 communication between intercalated disc and contractile apparatus.
15 eflecting the instantaneous structure of the contractile apparatus.
16 ated with transient dedifferentiation of the contractile apparatus.
17 rs share many features such as components of contractile apparatus.
18 ncation mutations in proteins of the cardiac contractile apparatus.
19 ally link adhesion molecules to the cellular contractile apparatus.
20 uscle defects in AGD appear to reside in the contractile apparatus.
21 on pericytes, which have a highly developed contractile apparatus.
22 which reflects increases in [Ca2+] near the contractile apparatus.
23 velopmental roles during construction of the contractile apparatus.
24 sarcoplasmic reticulum by linking it to the contractile apparatus.
25 eased Ca2+ sensitivity of the regulatory and contractile apparatus.
26 on of the initial injury at the level of the contractile apparatus.
27 ECM-rigidity signals depends on the cellular contractile apparatus [5-7], given that inhibition of no
28 genes encode components of the smooth muscle contractile apparatus (ACTA2, MYH11, MYLK, and PRKG1), a
29 ll heat shock family, can protect myocardial contractile apparatus against ischemia reperfusion (I/R)
30 (2+) signaling in activating the endothelial contractile apparatus and generating interendothelial ga
31 maintain the association between the muscle contractile apparatus and hypodermal fibrous organelles.
32 size, it participates in the assembly of the contractile apparatus and membrane systems required for
33 t changes in both the ability to restore the contractile apparatus and myogenesis are important, and
34 l role in organizing myosin filaments in the contractile apparatus and perhaps in other structures in
35 impels both assembly and disassembly of the contractile apparatus and suggest a regulatory strategy
36 tural relationships between the myofibrillar contractile apparatus and the enzymes that generate ATP
37 n in molecular morphology, localization in a contractile apparatus, and ability to interact with myos
38 nin-I, an important component of the cardiac contractile apparatus, and fewer apoptotic cardiomyocyte
40 oponin I and T, regulatory components of the contractile apparatus, are sensitive indicators of myoca
44 istinct myosin II paralogues to generate the contractile apparatus at apical epithelial junctions.
45 crotubule density mechanically overloads the contractile apparatus at the cellular level, we measured
47 Ialpha is targeted to the smooth muscle cell contractile apparatus by a leucine zipper interaction wi
48 osensor assessment, e.g. membrane voltage or contractile apparatus Ca(2+) ion responses (force resolu
50 TnI may be to reduce the requirements of the contractile apparatus for both Ca2+ and ATP, thereby pro
51 nism of some DCM-associated mutations in the contractile apparatus has been studied in vitro and in t
54 s primarily a viscous load on the cardiocyte contractile apparatus in pressure-overload cardiac hyper
55 ha-actin (ACTA2) is a major component of the contractile apparatus in SMCs located throughout the art
58 EC MLCK exist that regulate the endothelial contractile apparatus in TNF-alpha-induced apoptosis.
59 nked to mutations in proteins of the cardiac contractile apparatus, including alpha-tropomyosin (Tm).
60 omyocytes, we observed severe defects in the contractile apparatus, including Z-disc and stress fiber
62 myoepithelial sheath has smooth muscle-like contractile apparatuses, it has a striated muscle-like r
64 or reduction in the Ca2+ sensitivity of the contractile apparatus may be the primary mechanisms medi
65 hough identified as a major component of the contractile apparatus of cardiomyocytes, the potential r
67 ribution of stiffening of the sarcomere (the contractile apparatus of RV cardiomyocytes) to RV diasto
69 e genes seem generally to encode sarcomeric (contractile apparatus) or cytoskeletal proteins, althoug
70 minant effects on distinct components of the contractile apparatus, our data provide the first insigh
71 properties of VSMCs, while potently inducing contractile apparatus protein expression, rendering nonc
72 enerally encode cytoskeletal and sarcomeric (contractile apparatus) proteins, although disturbance of
76 mutants using antibodies to proteins of the contractile apparatus reveals that although contractile
77 mutations in genes encoding proteins of the contractile apparatus specific to fast-twitch myofibers.
78 coordinated with the assembly of the muscle contractile apparatus, suggesting that signals are excha
80 involves formation of an actin stress fiber contractile apparatus that radiates from focal adhesions
82 ression of LMOD3 and other components of the contractile apparatus, thereby establishing a regulatory
83 eric proteins that link calcineurin with the contractile apparatus, thereby potentially coupling musc
84 utes to the formation and maintenance of the contractile apparatus through interactions with costamer
86 ng protein targets myosin phosphatase to the contractile apparatus to dephosphorylate myosin light ch
88 th muscle cells use an actin-myosin II-based contractile apparatus to produce force for a variety of
92 proteins into sarcomeres and coupling of the contractile apparatus to the sarcoplasmic reticulum (SR)
93 cGMP-induced relaxation of the smooth muscle contractile apparatus using permeabilized rabbit femoral
94 h are dedicated primarily to assembly of the contractile apparatus, we analyzed the subcellular distr
95 d the assembly and maintenance of the muscle contractile apparatus, we have identified a new protein,
96 icate a novel, redox-based modulation of the contractile apparatus, which positively impacts myocardi
97 s, interlink them together and integrate the contractile apparatus with the sarcolemma and the nucleu
98 bute to the formation and maintenance of the contractile apparatus within muscle cells, we performed
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