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1 sts reversible cysteine modifications of the contractile machinery.
2 in regulation and structural support for the contractile machinery.
3 +) receptor, troponin C, to the actin-myosin contractile machinery.
4 mpensating for the marked dysfunction of the contractile machinery.
5 trol, indicating long-lasting effects on the contractile machinery.
6 ed independently of the establishment of the contractile machinery.
7 ng infection-induced inflammation to myocyte contractile machinery.
8 ater strain to be transmitted to subcellular contractile machinery.
9 ed the effects of cAMP signaling on platelet contractile machinery.
10 troponin I, suggesting direct effects on the contractile machinery.
11 molecular mechanisms regulating the cardiac contractile machinery and a novel platform for therapeut
13 ngle platelets and aggregates use actomyosin contractile machinery and integrin-mediated adhesion to
14 ate adhesion and deadhesion via cytoskeletal contractile machinery and sense mechanical properties of
15 eased intracellular Ca2+ availability to the contractile machinery but to a significant increase of m
17 ed embryos express greatly reduced levels of contractile machinery genes and, at the same stage, of r
18 omyosin networks, the major component of the contractile machinery, have shown that active contractil
19 rganization and activation of the actomyosin contractile machinery in a Rho kinase-dependent manner t
26 ulu (Epb4.1l5) helps anchor the actin-myosin contractile machinery to the membrane to allow the dynam
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