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1 ts in relaxation (diastole) while preserving contractile performance.
2 gy metabolism, neurotransmitter release, and contractile performance.
3 does not impair the normal left ventricular contractile performance.
4 s harbored a spontaneous capacity to improve contractile performance.
5 siological approach to study skeletal muscle contractile performance.
6 y thin filament length regulation and muscle contractile performance.
7 myocardial apoptosis and resultant decreased contractile performance.
8 hosphate content and failure to sustain high contractile performance.
9 tinct pathways of ventricular remodeling and contractile performance.
10 increase blood pressure and left ventricular contractile performance.
11 f pathologic hypertrophy and improvements in contractile performance.
12 als exhibited hypertrophy without diminished contractile performance.
13 odeling, and (3) enhance in vivo and ex vivo contractile performance.
14 eurohormonal system activity, and myocardial contractile performance.
15 es and produced a relative improvement in LV contractile performance.
16 vasodilation and improves LV relaxation and contractile performance.
17 ing as dominant negative alleles that impair contractile performance.
18 y relative to its demands can impair cardiac contractile performance.
19 showed a 73% decrease in baseline isovolumic contractile performance.
20 electrical pacing rate increased isovolumic contractile performance.
21 hemically injured heart enhances ventricular contractile performance.
22 s (STG-MI), however, showed preserved global contractile performance, abrogated apoptosis, and preven
26 , endothelin enhanced both adult rat myocyte contractile performance and cardiac troponin-I phosphory
27 orbide dinitrate restores Ca(2+) cycling and contractile performance and controls superoxide producti
28 re CHF, allopurinol produced no change in LV contractile performance and did not alter the response t
30 y examined left ventricular (LV) and myocyte contractile performance and electrophysiologic variables
32 frank DCM, defined as LV dilation, impaired contractile performance and LVEDD > or = 112% plus FS <
33 LV pump function, enhanced isolated myocyte contractile performance and normalized myocyte action po
35 may potentially account for the compromised contractile performance and reduced functional reserve i
36 skeletal muscle weight, fiber size, in vivo contractile performance, and expression of mRNAs and pro
37 pMultistem improved BZ energetics, regional contractile performance, and global left ventricular eje
38 arction increased survival, enhanced cardiac contractile performance, and halted ventricular remodeli
39 reduced cardiomyocyte hypertrophy, improved contractile performance, and prevented early lethality.
40 e defects in both whole heart energetics and contractile performance, and that the magnitude of these
41 the ATPase would be consistent with reduced contractile performance, and the results suggest reduced
42 ndent (type 1) diabetes results in depressed contractile performance associated with diminished sarco
44 al adaptations to muscle disuse, we compared contractile performance at the molecular, cellular and w
46 o determine if heart failure further impairs contractile performance by altering myofibrillar protein
47 ies indicating that low levels of NO augment contractile performance by inhibition of a cGMP-dependen
49 transgenic mice showed improved recovery of contractile performance during reperfusion after ischemi
50 nsic mechanical and neural factors in muscle contractile performance during running, which has been l
51 TNF-alpha induced a 7% to 40% increase in LV contractile performance during the 60-minute infusion.
52 ATP synthesis and ATP consumption, improving contractile performance following ischemia-reperfusion i
53 y, segmental ejection fraction (a measure of contractile performance) improved in injected scar segme
54 e favorably influences left ventricular (LV) contractile performance in heart failure remains uncerta
55 haracteristic, contributing to the decreased contractile performance in human and experimental failin
56 unction, ocular motility in intact mice, and contractile performance in isolated muscle preparations
58 entifies TnI as a potential target to modify contractile performance in normal and diseased myocardiu
59 ety of ways to adjust flight muscle size and contractile performance in order to meet demands imposed
60 normal basal cardiac function but depressed contractile performance in postinfarct and pressure over
61 (1) the thin filament is a key modulator of contractile performance in the failing human heart, (2)
62 (101) myofibrillar degeneration, to evaluate contractile performance in the mutant cardiomyocytes, an
66 likely contributes to the improved cellular contractile performance observed after sustained LVAD su
67 lead to adverse effects on cell viability or contractile performance of EHMs (n=3, P=0.83, P=0.87).
69 t that cell shape is critical in determining contractile performance of single cardiomyocytes by regu
74 ac energetics using 31P NMR spectroscopy and contractile performance of the intact beating heart reve
75 s saline treatment completely diminished the contractile performance of the ischemic/reperfused myoca
76 ordinately maintain the hyperdynamic cardiac contractile performance of the PLN-KO mouse in the long
77 sgenic hearts exhibited improved recovery of contractile performance over the reperfusion period.
78 TG hearts exhibited improved recovery of contractile performance over the whole reperfusion perio
80 is often abrogated by progressively abnormal contractile performance per unit mass of myocardium, eve
81 BS2, a selective inhibitor of NOS2, improves contractile performance, preserves myocardial ATP, and r
82 itions, SERCA2a overexpressors show improved contractile performance relative to wild-type (WT) mice
84 ated cardiac troponin-I in the adult myocyte contractile performance response to activated protein ki
85 myocytes expressing cTnI A164H maintained a contractile performance similar to that observed for the
86 ex plays a greater role in modulating muscle contractile performance than was recognized previously.
87 in both skeletal muscle strength and cardiac contractile performance that is not achievable with conv
88 tile work and a marked inability to increase contractile performance upon acute inotropic challenge i
89 oblasts, iPS treatment restored postischemic contractile performance, ventricular wall thickness, and
95 reperfusion, however, cardiac relaxation and contractile performance were greatly impaired in G6PD(de
96 responded promptly to the abrupt increase in contractile performance with epinephrine, during the lag
97 e in vivo translation of this improvement in contractile performance with PCO pretreatment was confou
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