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1 py attenuated adrenergic vasoconstriction in contracting muscle.
2 t-twitch and slow-twitch muscles in actively contracting muscle.
3  circulation, similar to what is observed in contracting muscle.
4  to tissues to meet the metabolic demands of contracting muscle.
5 estigate the process of force development in contracting muscle.
6 ated by the arrays of myosin crossbridges in contracting muscle.
7 d facilitate their interaction with actin in contracting muscle.
8 onyl-CoA decarboxylase (MCD) is increased in contracting muscle.
9 gnificant effects on free radical release by contracting muscle.
10 nhibition of sympathetic vasoconstriction in contracting muscle.
11 d not resolve structural states of myosin in contracting muscle.
12 es to the injuries induced by stretching the contracting muscle.
13 scillations in the activity of contralateral contracting muscles.
14 tochrome P450 enzymes, can be accumulated in contracting muscles.
15  did not alter either of these parameters in contracting muscles.
16 ive to modulation by metabolic events in the contracting muscles.
17 enuated alpha-adrenergic vasoconstriction in contracting muscle and improved muscle perfusion during
18 g exercise by acting as an energy sensor for contracting muscle and stimulating glucose production.
19 rly modulate sympathetic vasoconstriction in contracting muscle, and that age-associated impairments
20 ransverse) to a muscle's line of action when contracting muscles bulge to maintain a constant volume.
21  the STN), and receives a sensory input from contracting muscle, but, importantly, it sends efferent
22                  Particularly relevant for a contracting muscle cell, integrins are mechanotransducer
23 as a result of the release of potassium from contracting muscle cells.
24 the vasoconstrictor responses to tyramine in contracting muscle during heavy rhythmic handgrip exerci
25 ucture of some of the X-ray reflections from contracting muscle during mechanical transients, and the
26        Mechanical and metabolic stimuli from contracting muscles evoke reflex increases in blood pres
27 ment of signal from a single cross-bridge of contracting muscle feasible.
28 he large motoneurons that innervate the fast-contracting muscle fibers (F-type motoneurons) are vulne
29 be studied by applying step perturbations to contracting muscle fibers and subdividing the mechanical
30 ch sequence of events was indeed observed in contracting muscle fibers, suggesting that mechanical an
31 es of myosins, with additional insights from contracting muscle fibers.
32 hanges within the dimeric myosin molecule in contracting muscle fibers.
33 owed a quail pattern of mixed fast- and slow-contracting muscle fibers.
34 applying radial compression to isometrically contracting muscle fibers.
35 muscle length) to an otherwise isometrically contracting muscle fibre.
36 raemia is attributable to K(+) released from contracting muscle fibres and acting extraluminally on a
37 sequences of protein alterations in isolated contracting muscles from the same hearts.
38 rent with that in the sensorimotor cortex or contracting muscle in the 8-27 Hz range.
39                                           In contracting muscle, individual myosin molecules function
40 athetic alpha-adrenergic vasoconstriction in contracting muscle is impaired with age.
41            In addition, each cross-bridge of contracting muscle is in a different stage of its mechan
42     The time course of DAF-T fluorescence in contracting muscle is predicted by also considering the
43 n and on the generation of active force in a contracting muscle may be simply due to the blocking of
44  rate, arguing that superoxide released from contracting muscles may have functionally significant ef
45 -derived substances to blood flow control in contracting muscle of older adults.
46 ance on oxidative ATP synthesis (ATP(OX)) in contracting muscle of older compared to young humans.
47 lood flow and vascular tone are regulated in contracting muscles of humans.
48 vasoconstriction was greatly impaired in the contracting muscles of the alpha-syntrophin null mice an
49 , bound to the actin target zone of actively contracting muscle, originate from a restricted region o
50               However, the role of TBC1D1 in contracting muscle remains ambiguous.
51 f this interaction may be quite different in contracting muscle than in vitro because of the molecula
52         However, the specific metabolites in contracting muscle that open KATP channels are not known
53 wo reflexes is a phenomenon localized to the contracting muscles themselves resulting from an interac
54  dazzling patterns of colours by selectively contracting muscles to reversibly activate chromatophore
55           The rate at which an isometrically contracting muscle uses energy is thought to be proporti
56 ion, citrate activation of ACC purified from contracting muscle was markedly depressed.
57 aced contacts and the sensorimotor cortex or contracting muscle was negligible in all patients.
58 est whether the tight coupling is present in contracting muscle, we simultaneously followed mechanica
59                 In contrast, histograms from contracting muscles were best fit by at least two Gaussi
60 obtain detailed structural information about contracting muscle with millisecond time resolution and
61  failure, we hypothesized that reduced NO in contracting muscle would result in enhanced sympathetic

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