ライフサイエンスコーパス: contralesional

1 blindness is associated with a reduction of contralesional 2DG uptake to normal or subnormal levels.

2 h upper extremity impairment (Pearson r with contralesional Action Research Arm Test = 0.527, p = 0.0

3 for contralesional stimuli is less when the contralesional and ipsilesional items are different on t

4 jects was 0.66 +/- 0.13 (mean +/- SD) during contralesional, and 0.30 +/- 0.16 during ipsilesional ro

5 rimary motor cortex [Brodmann area (BA) 4p], contralesional anterior primary motor cortex (BA 4a), bi

6 on has also, naturally, focused on improving contralesional arm impairment and function.

7 phere specific motor control deficits in the contralesional arm of stroke patients.

8 erefore, asked whether motor deficits in the contralesional arm of unilateral stroke patients reflect

9 lasticity-related molecules that facilitated contralesional axonal growth.

10 reatment after neonatal HI markedly reducing contralesional axonal remodeling induced by HI brain inj

11 ith diabetes, SICI was only reduced over the contralesional but not the ipsilesional cortex compared

12 thropoietin enhanced axonal sprouting of the contralesional, but not ipsilesional pyramidal tract at

13 contralesional middle intraparietal sulcus, contralesional cerebellum, and ipsilesional rostral prem

14 Changes in activation were observed in the contralesional cerebrum and ipsilesional cerebellum (P =

15 Both ipsilesional and contralesional changes have been described, but it remai

16 ere; and (ii) activity increase accompanying contralesional choices between bilateral targets in seve

17 RR inactivation led to a strong reduction of contralesional choices, but only for reaches.

18 Inactivation caused a reduction of contralesional choices.

19 We conclude that contralesional chronic electrical stimulation of the lat

20 patial orientation performance for leftward (contralesional) compared to rightward (ipsilesional) rot

21 em cell (MSC) treatment on the structure and contralesional connectivity of motor function-related ce

22 The reliance on reorganization over the contralesional cortex after stroke will likely exert inf

23 e dendritic plasticity in both the ipsi- and contralesional cortex and this coincides with stem cell-

24 on paretic forelimb function depend upon the contralesional cortex and transcallosal projections.

25 Here, we demonstrate that the contralesional cortex exerts an enhanced interhemispheri

26 n of activity-dependent neurotrophins in the contralesional cortex, including brain-derived neurotrop

27 ity of corticobulbar fibers from the intact, contralesional cortex, which itself formed a fivefold ex

28 platform with reversible inactivation of the contralesional cortex.

29 lues, which were increased in both ipsi- and contralesional cortices and decreased in the corpus call

30 droitinase ABC also induced sprouting of the contralesional corticospinal tract in the aged treated h

31 The contralesional CST sprouting was highly and positively c

32 NMDA-mediated excitatory transmission in the contralesional CST.

33 Furthermore, the congruent contralesional deficit in time (motion duration) and pos

34 se our model of lateralization predicts that contralesional deficits will differ depending on the hem

35 e that chronic electrical stimulation of the contralesional dentate (lateral cerebellar) nucleus outp

36 Contralesional dorsal premotor cortex (cPMd) may support

37 stimuli (Experiment 2) caused no significant contralesional extinction on bilateral displays and reac

38 ion, which provide an influential account of contralesional extinction on bilateral stimulation after

39 the location (Experiment 1) produced marked contralesional extinction, although reaction time was de

40 temporal retinal ganglion cell counts in the contralesional eye and ipsi/contralateral optic tract ar

41 lesion (lesioned: F(1,9) = 21.347, P = .001; contralesional: F(1,9) = 9.648, P = .013; repeated-measu

42 onal visual field, visuomotor neglect of the contralesional field, and low overall performance.

43 In contrast, in SM's contralesional field, interference from threatening imag

44 sented either in the ipsilesional field, the contralesional field, or both fields.

45 perceiving and responding to stimuli in the contralesional field.

46 conducted, and losses were found only in the contralesional fields for a selective attention and a mu

47 nt diffusion coefficient in the ipsilesional contralesional forelimb region of the primary somatosens

48 iated with biphasic recruitment of peri- and contralesional functional fields in the brain.

49 System excitability and early recruitment of contralesional functional homologues represented specifi

50 In severe strokes leading to neglect, contralesional functional homologues support recovery by

51 show that chondroitinase injections into the contralesional gray matter of the cervical spinal cord a

52 reduction of fractional anisotropy near (i) contralesional hand area following verum, but not sham,

53 de of the long-latency stretch reflex of the contralesional hand decreased after surgery.

54 an enhancement of motor preparation for the contralesional hand.

55 tients performed joystick movements with the contralesional hand.

56 d during the same tasks was improved for the contralesional hand.

57 ir weight by gripping the cage bars with the contralesional hand.

58 were able to orient to visual stimuli in the contralesional hemifield immediately following surgical

59 espond or orient to stimuli presented in the contralesional hemifield.

60 o show increasing awareness deficits for the contralesional hemispace when engaged with resource-cons

61 tion for this is that brain regions from the contralesional hemisphere "take over" their functions, w

62 ere injury relies on the contribution of the contralesional hemisphere (i.e., the "right-hemisphere-t

63 stantial increase in connections between the contralesional hemisphere and the paralyzed limb.

64 employed to explore the cortical changes in contralesional hemisphere and to reveal its correlation

65 feedback from PFC to visual cortex, and the contralesional hemisphere can serve as an intact normal

66 ee of shift of activation balance toward the contralesional hemisphere early after stroke increases w

67 Cathodal (inhibitory) stimulation to the contralesional hemisphere led to a functional improvemen

68 lly due to insufficient connections from the contralesional hemisphere to the paralyzed side.

69 he scotoma border; and (iii) pRF size in the contralesional hemisphere was slightly increased compare

70 Significant activation responses in the contralesional hemisphere were detected at days 1 and 3.

71 14 days, we found reduced involvement of the contralesional hemisphere, and significant responses in

72 red hemisphere and homologous regions in the contralesional hemisphere, but in other cases, the oppos

73 the ipsilesional hemisphere, cathodal on the contralesional hemisphere, or bilateral; (2) recovery st

74 leads to a pathological hyperactivity in the contralesional hemisphere, resulting in a biased attenti

75 ation-induced responses were detected in the contralesional hemisphere.

76 l hemisphere, whereas it was enhanced in the contralesional hemisphere.

77 sion and in the corresponding regions of the contralesional hemisphere.

78 onal hemisphere; cathodal stimulation to the contralesional hemisphere; and sham stimulation.

79 as also noted in CA1 of the ipsilesional and contralesional hemispheres during 1-2 days.

80 s cortical function over the ipsilateral and contralesional hemispheres in 7 patients with diabetes a

81 e of activation between the ipsilesional and contralesional hemispheres, characterized by the lateral

82 both injured (ipsilesional) and un-injured (contralesional) hemispheres.

83 ed a new procedure to increase the effect of contralesional hemispheric compensation by surgically cr

84 Some studies have demonstrated that contralesional hemispheric compensation may be an import

85 ve been controversies on the contribution of contralesional hemispheric compensation to functional re

86 These results showed that the contralesional hemispheric compensation was markedly str

87 Contralesional hind paws lost 54% of innervation in tibi

88 appears to depend both on activation of the contralesional hippocampus and on increased engagement o

89 initial testing revealed that patients had a contralesional impairment in olfactory identification bu

90 into the factors that modulate awareness of contralesional information in neurological patients with

91 nstrate that CST fibers originating from the contralesional intact cerebral hemisphere are capable of

92 mulation can reduce the hyperactivity of the contralesional, intact hemisphere and thereby improve sp

93 erceptual similarity of the ipsilesional and contralesional items but by whether they shared the same

94 and implantation of a macroelectrode in the contralesional lateral cerebellar nucleus.

95 vity between impaired (left) forepaw and the contralesional (left) motor cortex after HI, whereas MSC

96 Improved awareness of contralesional (left) targets with preferred music was a

97 ere applied over two consecutive days on the contralesional, left posterior parietal cortex in patien

98 Results showed that, only on the contralesional limb, even a single tactile stimulation c

99 the execution of proximal movements with the contralesional limb.

100 esional M1, and between ipsilesional SMA and contralesional M1 underlies hand motor disability after

101 c communication between ipsilesional SMA and contralesional M1 was significantly reduced, which also

102 that a dysfunction between ipsilesional and contralesional M1, and between ipsilesional SMA and cont

103 for inhibitory (cathodal) tDCS applied over contralesional M1.

104 However, in contralesional middle intraparietal sulcus, contralesion

105 fibers crossed the CC and extended into the contralesional motor cortex compared to HI mice.

106 nal studies in which rTMS of the lesioned or contralesional motor cortex was combined with motor trai

107 Understanding whether contralesional motor deficits differ depending on the he

108 uggest that BATRAC induces reorganization in contralesional motor networks and provide biological pla

109 The contralesional nature of the deficits points to the impo

110 lay activity in the ALM or thalamus produced contralesional neglect.

111 ng strategies, the patients also exhibited a contralesional olfactory detection impairment.

112 e patients with right hemisphere lesions and contralesional paralysis were tested for implicit and ex

113 at 1 IU/day), increased grip strength of the contralesional paretic forelimb and improved motor coord

114 onparetic", forelimb worsens deficits in the contralesional, "paretic", forelimb.

115 tional connectivity in both ipsilesional and contralesional parietofrontal pathways involved in visuo

116 reased between ipsilesional motor cortex and contralesional premotor cortex after the intervention.

117 e neuroplastic response of the iCSP from the contralesional primary motor cortex (cM1) hand/arm area

118 that erythropoietin acts via recruitment of contralesional rather than of ipsilesional pyramidal tra

119 Finally, the extent of contralesional rewiring measured with BDA and PRV tracin

120 MSC treatment after HI reduced contralesional rewiring taking place after HI.

121 bjects with UVD had 0.21 +/- 0.06 TAR during contralesional rotation and 0.50 +/- 0.11 during ipsiles

122 1) initial forward VOR axis tilt relative to contralesional rotation averaging 9.5 +/- 4.9 degrees ,

123 ediated through callosal connections and the contralesional sensorimotor cortex.

124 restricted to the right VL in a patient with contralesional sensory processing deficits.

125 ndary somatosensory cortex (SII), and in the contralesional SI gray matter, as compared to saline-inj

126 ilesional SI and SII gray matter, and in the contralesional SI white matter by 48 h (P<0.05).

127 For example, targets in the contralesional side of the array were poorly localized w

128 ts and problems with movements affecting the contralesional side of the body and space.

129 ient was more likely to detect events on the contralesional side when a simultaneous ipsilesional eve

130 d of the body opposite a damaged hemisphere (contralesional side).

131 nt and respond to stimuli appearing on their contralesional side.

132 icits after stroke are most prominent on the contralesional side.

133 c and corticospinal axonal rewiring from the contralesional side; with the transcallosal and corticos

134 On the right (contralesional) side, motor performance of the groups un

135 ect, both patients had difficulty with left (contralesional) signals when preceded by a right (ipsile

136 elations were found between ipsilesional and contralesional SMA.

137 s often associated with a failure to explore contralesional space.

138 g of spared corticospinal tract axons in the contralesional spinal cord makes a significant contribut

139 lesions resulted in impaired thresholds for contralesional stimuli at longer delays, and these defic

140 l neglect, we have found that extinction for contralesional stimuli is less when the contralesional a

141 a related study, we found that extinction of contralesional stimuli was not determined by perceptual

142 ut is characteristically unaware of the same contralesional stimulus during simultaneous stimulation

143 esional stimulus is said to 'extinguish' the contralesional stimulus from awareness during bilateral

144 stimuli on either side but are unaware of a contralesional stimulus if presented concurrently with a

145 elevated excitatory synaptic markers in the contralesional striatum.

146 l sulcus, dorsolateral prefrontal cortex and contralesional superior cingulate sulcus.

147 r microstructure integrity in regions of the contralesional superior longitudinal fascicle adjacent t

148 Contralesional sural-innervated skin had neither neurite

149 biased competition in which the priority of contralesional targets is undervalued.

150 temporal parietal occipital area) for single contralesional targets, especially in the inactivated he

151 showed additional inhibitory influences from contralesional to ipsilesional M1 that correlated with t

152 cture at 'distal' body sites, near the ankle contralesional to the more affected hand; and (iii) loca

153 iria decreases as the hands move from right (contralesional) to left (ipsilesional) space in trunk- a

154 tric-favoring the ipsilesional flocculus and contralesional vestibular brainstem.

155 le stimulus presented in the ipsilesional or contralesional visual field, but is characteristically u

156 pects of optic ataxia are misreaching in the contralesional visual field, difficulty preshaping the h

157 behavioural deficits: visual neglect of the contralesional visual field, visuomotor neglect of the c

158 l activity only for stimuli presented to the contralesional visual field.

159 ve rotational behaviors or show the profound contralesional visual neglect seen postoperatively in no

160 teral visual cortex lesions produce enduring contralesional visual orientation deficits.

161 ial neglect fail to attend to stimuli in the contralesional visual world.