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1 Aging significantly degrades contrast sensitivity.
2 ection discrimination thresholds, as well as contrast sensitivity.
3 or ON alpha RGCs have behavioral deficits in contrast sensitivity.
4 ctive error, uncorrected distance acuity and contrast sensitivity.
5 r hypertension) does not increase perimetric contrast sensitivity.
6 nal measures showed a fair relationship with contrast sensitivity.
7 underwent the Anwar technique showed better contrast sensitivity.
8 ensitivity, or sweep visual evoked potential contrast sensitivity.
9 nsitivity, and sweep visual evoked potential contrast sensitivity.
10 the main source of individual variations in contrast sensitivity.
11 general cognitive status, visual acuity, and contrast sensitivity.
12 hanges in monocular and binocular functional contrast sensitivity.
13 ability to detect contrast is referred to as contrast sensitivity.
14 l function, affecting both visual acuity and contrast sensitivity.
15 etts, and deficits in adult color vision and contrast sensitivity.
16 s of retinal ganglion cells showed decreased contrast sensitivity.
17 ask, in which performance is contingent upon contrast sensitivity.
18 Adaptation decreased contrast sensitivity.
19 es were compared with measurements of VF and contrast sensitivity.
20 ar/parvocellular function was assessed using contrast sensitivity.
21 also substantial, but did not generalize to contrast sensitivity.
22 d improved visual ability, but also enhanced contrast sensitivity.
23 ic curves from which we inferred measures of contrast sensitivity.
24 ted visual acuity (BCVA), reading speed, and contrast sensitivity.
25 w deficits in the OMR assay, including lower contrast sensitivity.
26 of higher-order neurons and thereby spatial contrast sensitivity.
27 sociated with postoperative visual acuity or contrast sensitivity.
28 pectacle-corrected visual acuity (BSCVA) and contrast sensitivity.
29 = .01) and photopic (beta = -0.23, P = .04) contrast sensitivity.
30 rgery with no influence on the postoperative contrast sensitivity.
31 rpened orientation tuning, and led to higher contrast sensitivity.
32 ia improved monocular and binocular BCVA and contrast sensitivity.
33 = .04) and photopic (beta = -0.003, P = .02) contrast sensitivity.
34 cm) and near (33 cm) distances and binocular contrast sensitivity.
35 termediate, and near distances with improved contrast sensitivity.
36 ance, (2) binocular disparity, (3) luminance contrast sensitivity, (4) peak spatial frequency, and (5
37 50 times better signal-to-noise, 20% higher contrast sensitivity, 45% higher direction selectivity,
38 ions the asymptotic maximum was <50%, and so contrast sensitivity (50% response rate) is undefined.
41 sed by specific deficits in light responses, contrast sensitivity, acuity, and circadian rhythms in t
42 ores the visual function, accommodation, and contrast sensitivity after cataract surgery with no infl
45 ese patients show surprising improvements in contrast sensitivity, an assay of basic spatial vision.
48 processing from retinal deficits, including contrast sensitivity and colour vision deficits to highe
51 These include changes in colour vision and contrast sensitivity and difficulties with complex visua
52 retinal dysfunction, as evidenced by reduced contrast sensitivity and FDP performance, accompanied by
53 ical framework is presented for interpreting contrast sensitivity and gain loss to chromatic and achr
55 Mesopic vision evaluations were performed by contrast sensitivity and glare tests for each group.
57 evoked potential (sVEP) was used to measure contrast sensitivity and grating acuity in 34 children w
58 compare visual evoked potential measures of contrast sensitivity and grating acuity in children with
59 we found no relationship between perimetric contrast sensitivity and IOP reduction in ocular hyperte
60 er (HIV-NRD), a visual impairment of reduced contrast sensitivity and reading ability, is associated
63 ment, were used to assess light sensitivity, contrast sensitivity and spatial acuity of optogenetic r
64 s prefer vertically oriented gratings; their contrast sensitivity and TF tuning are similar to those
65 stance visual acuity (VA), reading speed, or contrast sensitivity and the National Eye Institute Visu
67 Race/ethnicity seems to be associated with contrast sensitivity and visual acuity outcomes in affec
68 with low vision than visual factors such as contrast sensitivity and visual acuity, or the use of ma
70 , reading acuity, maximum reading speed, and contrast sensitivity and with microperimetry evaluating
71 full range of adequate vision, satisfactory contrast sensitivity, and a lack of significant adverse
74 ence functional deficits in dark adaptation, contrast sensitivity, and color perception before microv
78 or that has a vision component, notably poor contrast sensitivity, and some loss of visual fields.
79 acuity (LCA) (2.5% and 1.25%), Pelli-Robson contrast sensitivity, and sweep visual evoked potential
80 R-R and Ishihara testing are correlated with contrast sensitivity, and these tests may be useful clin
81 ment of best-corrected visual acuity (BCVA), contrast sensitivity, and videonystagmography were perfo
86 (AULCSF) was calculated for the analysis of contrast sensitivity as a single figure across a range o
87 no statistically significant differences in contrast sensitivity, astigmatism, coma, or higher-order
88 (BCVA), central macular thickness (CMT), and contrast sensitivity at 1,2, and 6 months were evaluated
92 correlation between logMAR visual acuity and contrast sensitivity at 6, 12, and 18 cpd (rho = -0.306,
94 exposed to higher PCE levels exhibited lower contrast sensitivity at intermediate and high spatial fr
95 ccades are known to produce a suppression of contrast sensitivity at saccadic onset and an enhancemen
96 ded by poorer visual acuity, near vision, or contrast sensitivity at the beginning of each interval.
99 ferences were found in Strehl ratio, VA, and contrast sensitivity between -3 and -6 D implantable col
101 significant differences were found in VA and contrast sensitivity between implantable collamer lens p
102 photopic retinal light responses and visual contrast sensitivity, but only transient changes were ob
103 ficantly related to visual response latency, contrast sensitivity (C-50 values), directional selectiv
104 athy Study (ETDRS) letter score change, mean contrast sensitivity change, proportion of patients with
106 e apparent, there are functional deficits in contrast sensitivity, color perception, and dark adaptat
108 on emission tomography (PET) offers superior contrast sensitivity compared with MRI, and recent precl
109 econdary outcomes were spherical equivalent, contrast sensitivity, corneal aberrations, corneal biome
110 e VA = 20/48 vs. 20/24, p < 0.001) and worse contrast sensitivity (CS) (binocular CS = 1.9 vs. 1.5 lo
111 Best Corrected Visual Acuity (WB-BCVA), Mars Contrast Sensitivity (CS) and a Glare Test (GT) were per
113 nocular and binocular visual acuity (VA) and contrast sensitivity (CS) at 10 cyc/deg and binocular su
115 t structural and functional measures predict contrast sensitivity (CS) outcomes in glaucomatous eyes.
117 0 cm slit-lamp examination; defocus testing; contrast sensitivity (CS) under photopic and mesopic con
120 after ETDRS visual acuity (VA), Pelli-Robson contrast sensitivity (CS), and Goldmann visual field (VF
121 s (Shack-Hartmann aberrometer), 20/40 letter contrast sensitivity (CS), and TBU (retroillumination, R
122 binocular distance visual acuity, binocular contrast sensitivity (CS), and the binocular driving vis
123 nonbacklit chart, near visual acuity (NVA), contrast sensitivity (CS), CS with glare, and lighting.
127 of minimum angle of resolution [logMAR]) and contrast sensitivity (CS; 1.4 vs. 1.9 log units of CS [l
128 l assessment (binocular visual acuity [BVA], contrast sensitivity [CS], and Humphrey VFs, both 10-2 a
129 active thresholding algorithm 24-2 strategy, contrast sensitivity, dark adaptation, visual acuity, an
130 ractive Thresholding Algorithm 24-2 testing, contrast sensitivity, dark adaptation, visual acuity, an
137 acuity, color perception, visual field, and contrast sensitivity), dynamic visual functions (motion
138 r pressure (IOP), pupillary aperture, glare, contrast sensitivity, endothelial cell density, anterior
139 inal dysfunction that manifests as decreased contrast sensitivity, even with good best-corrected visu
140 avioral methods were used to measure spatial contrast sensitivity, eye alignment, and stereopsis with
142 ntly better values were observed in photopic contrast sensitivity for high spatial frequencies in gro
147 rall, the difference in photopic and mesopic contrast sensitivity function between the 2 groups was s
148 ant differences between groups were found in contrast sensitivity function with and without glare for
149 visual acuity, best-corrected visual acuity, contrast sensitivity function, higher-order aberrations,
151 nce visual acuity (CDVA), residual cylinder, contrast sensitivity, glare acuity, pain score, and high
152 o significant differences were identified in contrast sensitivity, higher-order aberrations, or refra
153 No significant differences were found in contrast sensitivity, higher-order aberrations, or refra
154 rs (D4Rs) have been implicated in modulating contrast sensitivity; however, the cellular and molecula
155 indings can be used as a reference guide for contrast sensitivity in a general population and for com
156 lation between the amount of astigmatism and contrast sensitivity in all spatial frequencies (P<0.001
161 ucleus to hMT+, we propose that this altered contrast sensitivity in hMT+ could be consistent with in
162 ctional magnetic resonance imaging to record contrast sensitivity in hMT+ of their damaged hemisphere
163 A longitudinal study of spatial and temporal contrast sensitivity in Ins2(Akita/+) mice and wild-type
165 rformance was state dependent: TMS decreased contrast sensitivity in the absence of adaptation but in
166 parameters accounting for contrast gain and contrast sensitivity in the inferred MC or PC pathway.
167 imal to no change to distance vision, better contrast sensitivity in the inlay eye when compared to t
169 Self-regulators had significantly poorer contrast sensitivity in their worse eye than non self-re
170 ptic nerve function, manifested as decreased contrast sensitivity (in the absence of ocular opportuni
171 ongest correlation was between SPARCS score (contrast sensitivity) in the better eye and total CAARV
173 stance correction visual acuity outcomes and contrast sensitivity, intraocular aberrations, and defoc
176 visual acuity (letter or grating acuity) or contrast sensitivity (letter or grating contrast) tasks.
177 inance visual acuity, low luminance deficit, contrast sensitivity, light sensitivity in the macula, a
178 tionship between VFQ-25 and the logarithm of contrast sensitivity (logCS), using Spearman correlation
180 In addition to causing visual acuity and contrast sensitivity loss, the central scotoma per se de
181 V neuroretinal disorder were identified by a contrast sensitivity <1.50 log units in either eye in th
182 ding acuity, distance acuity, reading speed, contrast sensitivity, mean central retinal sensitivity,
183 herence tomography [OCT]), retinal function (contrast sensitivity, measured by frequency-doubling tec
186 acy measures included changes in area of GA, contrast sensitivity, microperimetry measurements, and t
187 measures are health-related quality of life, contrast sensitivity, near visual acuity, reading index,
189 ss the two eye inputs, and where tested, the contrast sensitivity of each eye input was roughly match
190 nonlinear transformation in SACs reduces the contrast sensitivity of FF inhibition to match the sensi
191 t gain control (normalization) increases the contrast sensitivity of individual neurons at the cost o
192 the interhemispheric input also changed the contrast sensitivity of many neurons, thereby acting on
195 ween a model of type 1 diabetes and scotopic contrast sensitivity of the optomotor response is indica
201 There was no difference in visual acuity, contrast sensitivity, or color vision of the PD subjects
203 etinopathy Study visual acuity, Pelli-Robson contrast sensitivity, or sweep visual evoked potential c
204 ere were no significant differences in image contrast, sensitivity, or positive predictive values bet
205 temporal frequency bandwidth, but preserves contrast sensitivity, orientation tuning, and selectivit
206 lly aligned to the onset of movement, visual contrast sensitivity oscillates with periodicity within
207 beneficial effects of bevacizumab therapy on contrast sensitivity outcomes are expected to have a fav
208 -312 provided better intermediate vision and contrast sensitivity outcomes than the Acri.Lisa 366D.
209 rast sensitivity function (CSF), delineating contrast sensitivity over a wide range of spatial freque
210 Race/ethnicity was significantly related to contrast sensitivity (P < .001) and visual acuity (P
211 erved for distance visual acuity (P = .011), contrast sensitivity (P </= .0001), and mean central ret
213 e (P = .15) or near (P = .23) visual acuity, contrast sensitivity (P = .28), or glare (P = .88).
214 of visual function, with patients with worse contrast sensitivity (PR logCS; Spearman's rho: 0.42; P
216 sociation with age, r = -0.82 (< 0.001)) and contrast sensitivity presented with smaller values for o
217 al 4 and 12 degrees on microperimetry, color contrast sensitivity protan and tritan thresholds, patte
218 peed (r = 0.43 to 0.56, all P < 0.0001), and contrast sensitivity (r = -0.39 to -0.46, all P < 0.001)
219 y visual acuity (r = -0.22) and Pelli-Robson contrast sensitivity (r = 0.20) was weaker than that wit
223 ic tracking weekly visual acuity and monthly contrast sensitivity, retinal function with dark-adapted
224 e encoding D4Rs reduces the amplitude of the contrast sensitivity rhythm by reducing daytime sensitiv
226 ce show strikingly similar reductions in the contrast sensitivity rhythm to that in mice lacking D4Rs
233 y, we assessed best-corrected visual acuity, contrast sensitivity, straylight, keratometry, ultrasoni
234 s in binocular uncorrected visual acuity and contrast sensitivity suggest low pupillary dependence fo
237 n optical coherence tomography, Pelli-Robson Contrast Sensitivity test and the Spaeth-Richman Contras
239 rast Sensitivity test and the Spaeth-Richman Contrast Sensitivity test; (2) a performance based measu
240 y 15-hue color vision test; automated static contrast sensitivity test; and global electroretinograph
241 bgroups included 327 subjects that underwent contrast sensitivity testing and another 114 subjects fo
246 atistically significant correlations between contrast sensitivity tests and VF mean deviation with VR
247 likely to gain at least 6 letters or more of contrast sensitivity than the patients receiving standar
248 matically embedded in visual oscillations of contrast sensitivity that fluctuate rhythmically in the
249 ally embedded in a trough of oscillations of contrast sensitivity that fluctuated rhythmically in the
250 Our study demonstrates a circadian rhythm of contrast sensitivity that peaks during the daytime, and
251 o basic visual dimensions (visual acuity and contrast sensitivity) that together account for most of
253 nergic expression of hLRRK2-G2019S increased contrast sensitivity throughout the retinal network.
254 /+) mice exhibit a uniform loss in optomotor contrast sensitivity to all spatial frequencies that, un
255 n performance correlated with impairments in contrast sensitivity to low, but not high, spatial frequ
260 best-corrected visual acuity, accommodation, contrast sensitivity, topography and pachymetry with Sch
265 , and 18 cpd), under mesopic conditions, the contrast sensitivity values of the dominant eyes were sl
267 rocessing measures, including visual acuity, contrast sensitivity, vernier acuity, binocular stereops
268 ce deficits (poorer visual acuity or spatial contrast sensitivity, visual field depression or defects
269 ing leads to deterioration in visual acuity, contrast sensitivity, visual field, and dark adaptation.
283 tance visual acuity, refractive astigmatism, contrast sensitivity, wavefront aberrations, and refract
284 ificant differences in Strehl ratio, VA, and contrast sensitivity were found between both incision si
287 electroretinography and chromatic/achromatic contrast sensitivity were measured in these 42 patients
290 evoked potential, visual spatial acuity, and contrast sensitivity, were maintained at control levels
291 red to the multifocals, and better binocular contrast sensitivity when compared to all 3 intraocular
292 creases in IOP and loss of visual acuity and contrast sensitivity when compared to other inbred or ou
293 functional deficiencies in visual acuity and contrast sensitivity, whereas diabetic REDD1-deficient m
294 dren with CVI, 30 had measurable but reduced contrast sensitivity with a median threshold of 10.8% (r
297 demonstrated better monocular and binocular contrast sensitivity without glare at low to mid spatial
298 ular assessments: high- and low-contrast VA, contrast sensitivity without glare, halos or starbursts,
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