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1 Thus, understanding how variants of IFI16 and AIM2 contribute to periodontal disease pathogenesis may lead to tr
2 onses, and maladaptation of ACE2-related pathways might all contribute to these extrapulmonary manifestations of COVID-19
3 ding domain (HC) of BoNT/B (HC/B) has been proposed to also contribute to toxin binding to neurons by interacting with li
4 otic complications in patients with COVID-19 are common and contribute to organ failure and mortality.
6 lfunction of disease-associated XPA missense mutations, and contribute to understanding of the structure and mechanical a
7 ivated and become dysfunctional during clinical sepsis, and contribute to tissue-specific cytokine responses that are pro
8 Thus, the P. falciparum reservoir in all ages can contribute to the maintenance and spread of antimalarial resi
11 pairment or inadvertent activation of complement components contribute to the pathogenesis of some autoimmune neurologica
12 structure and assembly of SGs and how different components contribute to their formation are not fully understood.
13 the mechanisms of HPgammaCD-induced cellular pathways could contribute to effective NPC therapies.
14 ents of index households over a sustained time period could contribute to malaria elimination efforts.
15 is of some autoimmune neurological disorders and could even contribute to neurodegenerative diseases.
16 e show that, although myosin motors throughout the filament contribute to force development, only about 10% of the motors
18 Microbial resting stages might heavily contribute to microbial biomass and thus drive the responsive
19 chanical activation of p38-YAP-TEAD signaling, which likely contribute to myofibroblast heterogeneity in the infarcted my
20 ulation based setting, drinking high volumes of alcohol may contribute to the prevalence of sleep problems in older age,
21 This suggests that sex-specific chromatin differences may contribute to sex-specific ageing in flies.
22 Reduced transpiration upon seawater exposure may contribute to controlling the movement of toxic ions to leave
23 keleton and the extracellular matrix in skeletal muscle may contribute to reduced amino acid metabolism and insulin resis
24 ronal cholesterol trafficking and of lipid rafts by Nef may contribute to early stages of neurodegeneration and pathogene
26 histo-blood group antigens (HBGAs) between populations may contribute to reduced efficacy against severe rotavirus disea
27 ion of the locus and suggest how variation in these SEs may contribute to human disease and altered immunity.
28 We identified several consequences of EAE that may contribute to these phenotypes, including a reduction in adul
29 ith some abnormal alignment in the upper quarter, which may contribute to the dysfunction of the cervicothoracic and glen
30 ve reduced muscle mass that persists postnatally, which may contribute to their increased risk for adult onset metabolic
31 trate that, even after removal of the primary tumour, MDSCs contribute to the development of premetastatic niches and set
33 ly associated with HRS-1 and non-traditional factors, might contribute to the development of AKI in patients with cirrhos
34 o examine observational fear learning mechanisms that might contribute to fear and anxiety disorders transmission in clin
35 eding on humans by sylvatic Ae. malayensis may occasionally contribute to bridge sylvatic and human transmission cycles.
36 r disruptions within limbic brain regions and the periphery contribute to depression symptomatology and a more complete u
37 te whether changes in the cerebrovascular system in the PFC contribute to cocaine self-administration, and whether they r
39 We further discuss how these properties contribute to intestinal development and the dynamics of inju
40 Splenic red pulp macrophages (RPMs) contribute to erythrocyte homeostasis and are required for ir
41 nce of a tropical rubber tree pathogen R. microporus should contribute to the better understanding of how the fungus is a
42 To address how interactions between four key TFs contribute to cis-regulation in mouse ESCs, we assayed two ma
43 In addition, 14 variants are identified that contribute to both LOAD risk and age at onset of LOAD.
44 in insight into the genes and physiological mechanisms that contribute to sex differences in fat storage.
47 T cells underwent increased apoptosis during contraction to contribute to a substantially reduced memory population.
48 rtant roles in epigenetic pathways, and are hypothesized to contribute to hybrid vigor in plants.
49 It remains unclear how these pathways interact to contribute to contextual modulation of visual cortical proces
50 ion, household stress, and interruptions in healthcare will contribute to acute chronic disease complications.