ライフサイエンスコーパス: control diet

1 er whipping cream (MFGM diet) or butter oil (control diet).

2 ulant protein C in the aorta (P < .05 versus control diet).

3 +/- mice fed the HM/LF diet (P < .001 versus control diet).

4 1.2 micromol/L, respectively; P<0.001 versus control diet).

5 ctively; P<0.01 versus Mtr(+/+) mice fed the control diet).

6 us diet), and a casein-whey protein isolate (control diet).

7 and serum levels 16-fold, compared with the control diet.

8 ed with the Se-enriched diet compared to the control diet.

9 spring; post weaning, offspring were fed the control diet.

10 iet supplemented with nuts compared with the control diet.

11 r content of major volatile compounds to the control diet.

12 ice fed the fish oil diet as compared to the control diet.

13 nduced inflammatory response faster than the control diet.

14 ls in comparison to F1 mice from dams on the control diet.

15 r concentrations compared with the low-dairy control diet.

16 Ex, in comparison to sedentary animals fed a control diet.

17 t mice were sham- or whey-sensitized and fed control diet.

18 d either a diet enhanced with spirulina or a control diet.

19 those found in individuals who were fed the control diet.

20 riglyceride concentrations compared with the control diet.

21 and Bifidobacterium breve M-16V (GF/Bb) or a control diet.

22 ion after the consumption of a fish oil or a control diet.

23 ubly deficient diet compared to mice fed the control diet.

24 ethanol for 8 weeks, as a model of ALD, or a control diet.

25 rs, relative to the offspring of males fed a control diet.

26 erance compared with offspring of dams fed a control diet.

27 weeks of age, and all pups were weaned onto control diet.

28 olesterol levels compared with animals fed a control diet.

29 similar to that noted in Abcc6(-/-) mice on control diet.

30 ith 32.4% of calories as ethanol or pair-fed control diet.

31 ining Lieber-DeCarli diet or were pair-fed a control diet.

32 cations, were randomized to either DASH or a control diet.

33 rsible when the rats were switched back to a control diet.

34 s compared with mice fed the isoflavone-free control diet.

35 hich is high in fiber and whole grains, or a control diet.

36 ient between apoE(+/+) and apoE(-/-) mice on control diet.

37 ve COX-2 inhibitor nimesulide (400 ppm) or a control diet.

38 say, twice that found when they were fed the control diet.

39 otal expression after 60 days on the natural control diet.

40 relative to animals weaned onto the natural control diet.

41 activated caspase-3 assay, than when fed the control diet.

42 not differ from the radiated animals fed the control diet.

43 g rice bran oil than with consumption of the control diet.

44 tic insulin sensitivity in comparison to the control diet.

45 the DASH and HF-DASH diets compared with the control diet.

46 ne-supplemented diets when compared with the control diet.

47 ation, then weaned onto either obesogenic or control diet.

48 ls, compared to offspring from mothers fed a control diet.

49 iterranean diet supplemented with nuts, or a control diet.

50 les, and LDL peak diameter compared with the control diet.

51 lective cathepsin S inhibitor RO5444101 or a control diet.

52 on lipoprotein risk factors compared with a control diet.

53 om donors on either a high-fat diet (HFD) or control diet.

54 nd Cox7a1 knockout mice fed with high fat or control diet.

55 compared with consumption of non-whole-grain control diets.

56 = 28%) of diets including nuts compared with control diets.

57 lso had shorter survival times than mice fed control diets.

58 nol-containing diets for 6 weeks or pair-fed control diets.

59 the first 3-6 months than subjects consuming control diets.

60 an of the radiated rats fed the blueberry or control diets.

61 nd cholesterol levels in mice fed Western or control diets.

62 syndrome components than did guideline-based control diets.

63 respectively), or restricted calorie (50% of control) diets.

64 Rats were fed for 5weeks with control diet (0.15mug Se/g feed) or Se-enriched diet for

65 ake) from both plant and marine sources or a control diet (0.5% of energy intake from n-3 PUFAs).

66 ol/L, respectively) (P < 0.05) than with the control diet (0.89 +/- 0.12 and 0.84 +/- 0.11 mmol/L, re

67 vated in Kcnmb1(-/-) under basal conditions (control diet, 0.6% K) and increased significantly more t

68 embryonic days 12-17 and then returned to a control diet (1.1 g choline chloride/kg).

69 0.05 mmol/L, respectively) compared with the control diet (1.12 +/- 0.11 and 1.19 +/- 0.05 mmol/L, re

70 etal offspring from obese dams reversed to a control diet (1.3-fold, P>0.05).

71 C57BL/6 female mice were fed either a control diet (10% energy from fat) or a high-fat diet (4

72 y rats were fed a HFD (45% fat) or a matched control diet (10% fat) from weaning for 14 weeks.

73 ima Otsuka (L) and OLETF (O) rats consumed a control diet (10% kcal fat, 3.5% sucrose) or a WD (45% k

74 re assigned to a Control diet and BEOs diet (Control diet + 120 mg/kg BEOs), were challenged with C.

75 80 +/- 34 mg/d) than with consumption of the control diet (121 +/- 63 mg/d), but fractional calcium a

76 Male C57BL/6J mice were fed either a control diet (13.5% kcal from fat) or a very high-fat di

77 ere randomized into three dietary groups: 1) control diet, 2) zinc-deficient diet for 3 weeks, and 3)

78 eaned onto (1) a standard natural ingredient control diet, (2) a synthetic control diet or (3) a synt

79 /-) mice compared with Mtr(+/+) mice fed the control diet (21+/-4 versus 32+/-4%; P<0.05).

80 ,t11-CLA, and iTFA, in the context of highly controlled diets (24 d each), on lipoprotein risk factor

81 cheese (5.7 +/- 0.4 g/d) diets than with the control diet (3.9 +/- 0.3 g/d) (P < 0.001).

82 s were fed, during pregnancy and suckling, a control diet (4% w/w corn oil) or a fatty acid supplemen

83 +/+)) and heterozygous (Mtr(+/-)) mice fed a control diet (4.5+/-0.3 and 5.3+/-0.4 micromol/L, respec

84 y) were lower than in those who followed the control diet (5.14 +/- 0.18 and 3.06 +/- 0.15 mmol/L, re

85 hemoglobin increased tumor load in Min mice (control diet: 67 +/- 39 mm(2); 2.5% hemoglobin diet: 114

86 C57BL6/J mice were fed either a control diet, a diet containing choline (1.2%) or a diet

87 ring which all participants received a low-K control diet, a significant racial difference remained (

88 y individuals who consumed in random order a control diet, a standard DASH diet, and a higher-fat, lo

89 iterranean diet supplemented with nuts, or a control diet (advice on a low-fat diet).

90 nean diet supplemented with mixed nuts, or a control diet (advice to reduce dietary fat).

91 is is only partially corrected by a low-fat, control diet after weaning.

92 % caloric restriction (CR)] or an ad libitum control diet after which they were subjected to treatmen

93 MP mice were randomly divided and fed either control diet (AIN 76A) or a custom prepared AIN 76A diet

94 Seven-weeks old Wistar rats received control diet (AIN-93G), Western diet with and without a

95 Chicks were assigned to a Control diet and BEOs diet (Control diet + 120 mg/kg BEO

96 cerebral arterioles of Mtr(+/-) mice fed the control diet and in both Mtr(+/+) and Mtr(+/-) mice fed

97 hypercaloric diet for 6 weeks or a eucaloric control diet and measured intrahepatic triglyceride cont

98 e randomized into five groups (n = 8-14): C (control diet and sedentary), F (fed the fructose-rich di

99 alpha2i TG mice was slightly impaired on the control diet and significantly impaired on the high-fat

100 WT) mice were fed a high-fat diet (HFD) or a control diet and studied at 6 months of age.

101 ences between the non-irradiated animals fed control diet and the radiated animals fed the strawberry

102 e C57BL6/J mice were fed ethanol or pair-fed control diets and treated or not with HA35.

103 were first lowered by a combination of a 3-d controlled diet and prolonged exercise.

104 f C57BL/6J mice, as compared with mice fed a control diet, and that these chromatin changes are assoc

105 libitum HFr developed HS in contrast to the control diet, and the extent of ectopic fat was related

106 ing cells in liver, compared with mice given control diets, as well as higher levels of serum IgA and

107 D-supplemented mothers following reversal to control diet at weaning was interrogated by methylation-

108 t (FASD) (5-fold higher than recommended) or control diets before mating and during pregnancy.

109 absorption by healthy adults (n = 109) from controlled diets, before and after 4 or 8 wk of dietary

110 k, and up to 13.5+/- 4.1g/100mL FW in weight control diet beverage.

111 red with mineralocorticoid-excess mice fed a control diet, both high-fiber diet and acetate supplemen

112 een wild-type (WT) and Ucp1(-/-) mice on the control diet, but MR increased EE by 31% and reduced adi

113 HF-tau in the hippocampus of animals fed the control diet, but not in the irradiated animals fed the

114 n a high-potassium diet than from those on a control diet, but this was not a result of altered expre

115 increased feeding in lean rats fed a low-fat control diet (CD) [192 +/- 5 g (ghrelin+CD) vs. 152 +/-

116 Healthy males consumed a control diet (CD) and on two further occasions an isocal

117 A control diet (CD) and Western diet (WD), which contains

118 fructose-rich diet mouse (FRD) myocytes vs. control diet (CD) mice, in the absence of significant ch

119 ansgenic counterparts (NT) were fed either a control diet (CD) or a high-fat diet (HFD) for up to 10

120 Female mice were placed on control diet (CD) or folic acid-supplemented diet (FASD)

121 type (WT) and miR-155 knockout (KO) mice fed control diet (CD); however, miR-155 KO mice fed high-fat

122 awley rats were weaned onto HFD (45% fat) or Control Diet (CD; 10% fat).

123 he absence of SR Ca(2+) load alterations vs. control-diet (CD) myocytes.

124 nding order are the powder of inulin, weight control diet, coffee mixed, instant beverage, supplement

125 Female C57BL/6 J mice were fed a control diet (CON) or a high-fat diet (HFD) with or with

126 her a lower-protein (1.2 g . kg(-1) . d(-1)) control diet (CON) or a higher-protein (2.4 g . kg(-1) .

127 is, adult offspring of rat dams either fed a control diet (CON) or one deficient in choline (DEF) dur

128 % carbohydrate) (CHO) as well as a eucaloric control diet (CON).

129 One growth trial used a control diet containing fish meal (FM) as the main prote

130 Another growth trial compared a control diet containing fish oil (FO) as the main lipid

131 whereas their eight male siblings were fed a control diet containing pig fat as the main fat source.

132 Neonatal mice were subjected to a control diet (CTR) or experimental diet with altered rel

133 Mice fed the control diet designed to simulate Western human diet dis

134 showed that SNAP-25b-deficient mice fed with control diet developed hyperglycemia, liver steatosis, a

135 Compared with control diets, diets enriched with nuts did not increase

136 When compared with control diets, diets supplemented with walnuts resulted

137 was to evaluate the contribution of tightly controlled diets differing in carbohydrate and fat conte

138 ons after consumption of weight-maintaining, controlled diets differing in total fat and fat type.

139 sensitivity compared with Gipr(-/-) mice on control diet during IU/L.

140 to 7.1-fold, P<0.001) compared with that in control diet-exposed animals and is reversible in fetal

141 aternal high-fat diet exposure compared with control diet-exposed animals.

142 d, whereas glucose tolerance was improved in control diet-fed apoE(-/-) mice compared with apoE(+/+)

143 Saline-injected and control diet-fed leptin receptor-deficient mice were use

144 In control diet-fed mice, a 9-day CAP exposure was sufficie

145 In control diet-fed mice, exposure to CAP for 30 days decre

146 Splenocytes of control-diet-fed whey-sensitized donors transferred immu

147 Compared with controls, Diet-fed mice had a significant increase in Hc

148 Compared with dams on a control diet, female C57BL/6J mice fed an obesogenic die

149 y feeding adult zebrafish a Se-elevated or a control diet followed by collection of larvae from both

150 er either continued to be fed an SPI diet or control diet for 1 or 3 wk.

151 injury, the saline group received a standard control diet for 1 or 6 weeks, whereas DHA-injected anim

152 ts of an EtOH-containing (Lieber-DeCarli) or control diet for 11 weeks and exposed to cigarette smoke

153 w-fat dairy, and fruits and vegetables) or a control diet for 12 wk.

154 F1 rats were maintained on the control diet for 14 weeks after weaning and then underwe

155 2 mice were placed on a 4% alcohol diet or a control diet for 14 weeks.

156 h Microcystis to contrast with those given a control diet for 16 days.

157 A 3,000 ppm or conventional ASA 3,000 ppm or control diet for 19 weeks.

158 ne-deficient (MCD) diet or the corresponding control diet for 2 weeks and characterized for histologi

159 with deuterium, and then pair-fed alcohol or control diet for 2 weeks.

160 n high-fat-high-sucrose diabetogenic diet or control diet for 24 weeks.

161 were randomized to be fed a high-lutein or a control diet for 28 days.

162 ssion in the hippocampus of rats fed a KD or control diet for 3 weeks.

163 n ethanol-containing liquid diet or pair-fed control diet for 4 (11% total kcal;early response) or 25

164 TRAMP mice received this drug-containing or control diet for 4 or 18 weeks and were evaluated for pr

165 eeks of cholesterol-rich diet, a switch to a control diet for 4 weeks reduced serum cholesterol and s

166 Db/db and db/m mice were fed the MCD or control diet for 4 weeks to characterize differences in

167 e, and cholesterol (HTF-C diet) or a low fat control diet for 4 weeks.

168 received 5% ethanol-containing or isocaloric control diet for 5 weeks.

169 L/6 mice were fed an ethanol-containing or a control diet for 5 weeks.

170 lume/volume ethanol) or an isocaloric liquid control diet for 5 weeks.

171 control mice were fed a 1.25% cholesterol or control diet for 8 and 20 weeks.

172 an ethanol-containing or isocaloric pair-fed control diet for 8 weeks, followed by DC isolation from

173 HED pigs (n = 7) was transitioned back to a control diet for an additional six weeks.

174 rranean diet (MedDiet) intervention versus a control diet for cardiovascular prevention, with a media

175 ted with extra virgin olive oil or nuts vs a control diet for primary cardiovascular prevention.

176 /6NTac mice fed a 60% high-fat diet (HFD) or control diet for up to 16 weeks.

177 (vol/vol) ethanol (11% calories) or pair-fed control diets for 2 days, 2 weeks or 5 weeks and superim

178 in-2 (Plin2-null) were fed either Western or control diets for 30 weeks.

179 cess to ethanol-containing diets or pair-fed control diets for 4 or 25 days.

180 ere fed ethanol-containing diets or pair-fed control diets for 6 weeks.

181 e- and choline-deficient (MCD), high-fat, or control diets for 8-16 weeks.

182 diet containing 5% alcohol or an isocaloric (control) diet for 4 weeks.

183 clear cells, healthy humans were placed on a controlled diet for 1 week, then given fish oil and bora

184 ral Clinical Research Center and placed on a controlled diet for 7 days.

185 t reduction in arachidonic acid intake) or a control diet from the 15th wk of pregnancy to 4 mo of la

186 l levels in serum compared to group fed on a control diet (groundnut oil).

187 g and MI expansion that were observed in the control diet group were absent in the IF group.

188 was significantly reduced compared with the control diet group.

189 ter for Gjb2-CKO mice fed ACEMg than for the control diet group.

190 anean diet supplemented with mixed nuts, and control diet groups, respectively, corresponding to rate

191 fetuses derived from Pemt(-/-) dams fed the control diet had 25-50% less phospholipid-DHA as compare

192 ntly affected by walnut diets more than with control diets (HDL cholesterol: WMD = -0.2, P = 0.8; tri

193 mineralocorticoid excess-treated mice with a control diet, high-fiber diet, or acetate supplementatio

194 (80% restriction), LR (80% restriction) and control diet in 10-month-old C57BL/6J male mice.

195 tochondrial acetyl-proteome during CR versus control diet in mice that were wild-type or lacked the p

196 from folate-supplemented, folate-reduced and control diets in Cd mutant and wild-type adult females.

197 itiative (n = 153) were provided with a 2-wk controlled diet in which each individual's menu approxim

198 f 15 female subjects who were placed on well-controlled diets in which choline levels were manipulate

199 , Mediterranean, and high-protein diets with control diets including low-fat, high-GI, American Diabe

200 Patients' consumption of portion-controlled diets, including liquid meal replacements, wa

201 As expected, the control diet increased plasma lipids, whereas the MFGM d

202 atory gene expression in SAT compared with a control diet independently of body weight change in indi

203 to four 18-mo treatments: healthy lifestyle control, diet-induced weight loss, exercise, and diet pl

204 epresents an important model for genetically controlled, diet-induced HCC.

205 Our studies identify a regulatory mechanism controlling diet-induced insulin resistance by highlight

206 s (0.5%, w/w) in supplementation with AIN76A control diet inhibited the growth of SCC1 tumor xenograf

207 On the control diet, insulin-stimulated glucose transport was r

208 med a systematic review and meta-analysis of controlled diet-intervention studies in nondiabetic subj

209 tions caused by Type 2 diabetes mellitus and controlled diet is one of them.

210 (43%) in the seven patients assigned to the control diet (log-rank p=0.03).

211 Carbohydrate controlled diets may be associated with increased insuli

212 ereotypical behavior compared with mice with control diet microbiota in the absence of significant di

213 Compared with the control diet, milk- and cheese-based diets attenuated sa

214 (TMD-Nuts; n=100), with respect to a low-fat control diet (n=96).

215 fat contents and compositions were compared: control diet [nondairy diet (~500 mg Ca/d)], milk diet [

216 serious adverse events than did those on the control diet (none vs nine; p=0.0005).

217 adult offspring of Sprague-Dawley rats fed a control diet (OC) or lard-rich diet (OHF) during pregnan

218 low sodium-DASH diet versus the high sodium-control diet on SBP were -5.3, -7.5, -9.7, and -20.8 mm

219 l, and both (low sodium-DASH vs. high sodium-control diets) on systolic blood pressure (SBP) by basel

220 supplementation, at 5-fold the level in the control diet, on the NTD and vertebral phenotypes in Apo

221 achidonic acid-balanced diet compared with a control diet] on the body weights and compositions of th

222 ral ingredient control diet, (2) a synthetic control diet or (3) a synthetic methyl-donor-deficient d

223 omized into two groups and received either a control diet or a DHA-supplemented diet for 7-8 weeks.

224 ice (Fbgalpha(+/-) mice) were fed either the control diet or a diet containing 0.025% alpha-naphthyli

225 f females feeding for two and five days on a control diet or a diet containing either a low or a high

226 as induced in C57BL/6 mice, which were fed a control diet or a diet containing resveratrol during eit

227 SJL/J mice were infected with TMEV and fed a control diet or a diet containing resveratrol during the

228 (WT) controls were fed either a low fat (LF) control diet or a diet high in saturated fat (HF) for 8

229 sition after 2, 4 and 8 weeks of consuming a control diet or a diet supplemented with fish oil.

230 Mice were fed a control diet or a diet supplemented with the FXR agonist

231 h-old wild-type and APP/PS1 mice on either a control diet or a diet that induces hyperhomocysteinemia

232 -type littermates (Cbs+/+) were fed either a control diet or a high methionine/low folate (HM/LF) die

233 e [WT]) and diabetic ob/ob mice fed either a control diet or a methionine- and choline-deficient (MCD

234 receptor-deficient LDLr(-/-) mice were fed a control diet or a Western diet for 19 weeks.

235 ine decarboxylase (Hdc(-/-)) mice were fed a control diet or an HFD coupled with a high fructose corn

236 Rats were fed a control diet or an isocaloric diet enriched in cocoa for

237 s ligand (B6.gld) were given either high-fat control diet or ethanol diet by intragastric cannulation

238 ices from rats that were exposed to either a control diet or HFD from pregnancy day 13.

239 these biologic indicators in hPXR mice fed a control diet or HFD revealed further differences between

240 After 16 weeks on either a control diet or HFD, WT mice showed greater weight gain,

241 diponectin secretion, mice were fed either a control diet or isocaloric diets containing 27% safflowe

242 ith an intervention by feeding mice either a control diet or one containing the brain permeable beta-

243 7 tissues/fractions of young and old mice on control diet or one of 2 diet regimens (caloric restrict

244 agouti, were fed either a phytoestrogen-free control diet or one of six experimental diets: diets 1-3

245 were then randomly assigned to continue the control diet or switch to a high-K diet (either a high f

246 wild type (WT) and Pemt(-/-) mice were fed a control diet, or a diet supplemented with 3 g/kg of DHA,

247 ON, and SUP rats were either maintained on a control diet, or the choline-supplemented diet.

248 isolated from adult mice fed standard chow, control diets, or DHA supplemented diets.

249 ats were fed either an ethanol-containing or control diet over 14 weeks and euthanized 3 or 24 hours

250 g in the 3 consecutive 4-h samples after the control diet (P </= 0.005).

251 Tg mice fed the HM/LF diet compared with the control diet (P<0.001).

252 Each subject received a phytosterol-controlled diet plus (1) ezetimibe placebo+phytosterol p

253 during pregnancy and suckling followed by a control diet post-weaning.

254 co1(-/-)Bco2(-/-) double knock-out mice to a controlled diet providing beta-carotene as the sole sour

255 compared with that of the weight-maintenance control diet) raised fasting plasma insulin concentratio

256 greater short-term improvements than did the control diets (random-effects model) for waist circumfer

257 ive access to either a high-fat or a matched control diet, rats received nonreinforced presentations

258 In the context of the control diet, reducing sodium (from high to low) was ass

259 +/+) mice were fed a hyperhomocysteinemic or control diet, respectively, from weaning until 9-12 mont

260 Compared to a soy-free control diet, soy significantly shifted the cecal microb

261 3 months of IF or regular every-day feeding (control) diets started in 2-month-old rats, myocardial i

262 This controlled diet study assessed the effect of age on wate

263 o our knowledge, this is the first published controlled diet study to find that in postmenopausal wom

264 Mice fed a control diet supplemented with homocysteine had a 3-fold

265 yogurt, or custard) with no red meat, and a control diet that contained neither red meat nor dairy.

266 ven amount of energy from free sugars with a control diet that provides the same amount of energy fro

267 h Initiative (WHI) were provided with a 2-wk controlled diet that mimicked each individual's habitual

268 n which hatchling R. tigrinus were reared on controlled diets that either included or lacked toads.

269 older men and women (aged 55-80 y) consumed controlled diets that provided 1.2 g protein x kg(-1) x

270 Compared with the control diet, the high-fructose diet significantly incre

271 Compared with the control diet, the HM/LF diet diminished endothelium-depe

272 sterol and triglyceride.In comparison with a control diet, the incorporation of cashews into typical

273 and PDACs than LSL-Kras/Ela-CreERT mice fed control diets; the mice fed the HFDs also had shorter su

274 nsitioned from a 1-d low-fat (30% of energy) control diet to a 4-d high-fat (50% of energy) diet.

275 All pups were weaned onto the control diet to specifically test the effects of early d

276 ively, after the transition from the low-fat control diet to the high-fat diet.

277 We fed alcohol or control diets to wild-type (WT) and IRF3 knock-out (KO)

278 e 2 PD decreased (P < 0.05 compared with the control diet) total cholesterol (-8%), LDL cholesterol (

279 The carbohydrate:protein:fat ratio of the control diet was 55:15:30.

280 sodium, consuming the DASH compared with the control diet was associated with mean SBP differences of

281 Addition of the alginate to a control diet was investigated at both concentrations 2%

282 ence between the yogurt intervention and the control diet was only significant in one of these trials

283 ormal sex ratio observed in M. scalaris from control diets was affected by exposure to caffeine and p

284 eighted mean difference (WMD) between nut or control diets was estimated by using a random-effects me

285 Following a 3-day sodium-controlled diet, we infused Ang II (3 ng/kg per minute)

286 Slices from rats fed a control diet were bathed in artificial CSF replete with

287 Aged mice fed a control diet were exquisitely more susceptible to S. pne

288 (FASD, 10-fold higher than recommended) and control diet were fed to male Mthfr(+/+) and Mthfr(+/-)

289 were exposed to GOS/inulin mixture or fed a control diet were intraperitoneally sensitized to wheat

290 57BL/6 mice randomized to a high-fat diet or control diet were killed at 4-week intervals.

291 Wildtype mice (C57BLKS/J) fed the MCD or control diet were treated with SP600125; a c-Jun N-termi

292 holesterol from baseline between the OBG and control diets were analyzed by using random-effects meta

293 The 4 control diets were based on distinct national nutrition

294 eCarli diet containing alcohol or isocaloric control diets were fed to wild-type (WT) and MCP-1-defic

295 001) more on the New Nordic Diet than on the control diet, whereas normoglycemic individuals lost a m

296 Shifting from long-term CR to a control diet, which returns animals to the control rate

297 The experimental diets included a lower-fat control diet with no pistachios [25% total fat; 8% satur

298 We compared the effects of normal protein (control) diet with high protein diets containing whey, o

299 design, healthy volunteers (n=20) received a controlled diet with and without the serotonin precursor

300 respectively] compared with their respective control diets, with the largest effect size seen in the