ライフサイエンスコーパス: control mice

1 trinitrobenzene sulfonic acid, compared with control mice.

2 fluorescent probe in the treated but not the control mice.

3 cue compared with vehicle-treated littermate control mice.

4 3(-/-) mice compared with those in wild-type control mice.

5 al inflammation and fulminant hepatitis than control mice.

6 ted precursors of ureagenesis, compared with control mice.

7 y6C(hi)Ly6G(-)monocyte numbers compared with control mice.

8 ConA-treated HSC-depleted mice compared with control mice.

9 tion and bone marrow (BM) monocytes than did control mice.

10 g MI in a well-established model of T2DM and control mice.

11 , which were contrasted with saline-injected control mice.

12 d histone 2A, member X (H2AX), compared with control mice.

13 burden was higher in Nmur1(-/-) mice than in control mice.

14 amount of dopamine in the brain compared to control mice.

15 4, BMI1, and MSI1) compared with colons from control mice.

16 hylnitrosamine and carbon tetrachloride than control mice.

17 knockout mice) and compared them with floxed control mice.

18 ivers and primary hepatocytes, compared with control mice.

19 of dental caries as compared with wild-type control mice.

20 ice compared with Ah(Cre)/Met(+/+)/Apc(fl/+) control mice.

21 rct regions of LV in T2DM mice compared with control mice.

22 ER) KO mice compared to that in Cre-negative control mice.

23 on into the brain after TBI as compared with control mice.

24 was less pronounced in honokiol-treated than control mice.

25 ant (by 33%) and smaller (by 47%) cysts than control mice.

26 lt male type 2 diabetic and their littermate control mice.

27 mmunity composition compared to non-stressed control mice.

28 X3 levels in the lungs compared with healthy control mice.

29 ompared with the respective field volumes in control mice.

30 al lumen, are less sensitive to colitis than control mice.

31 nnate lymphoid cells compared with wild-type control mice.

32 d in CVB3-induced myocarditis versus healthy control mice.

33 ly enhanced CGM performance when compared to control mice.

34 ontrolled pleural BCG infection as wild-type control mice.

35 PH was compared between dextrose-treated and control mice.

36 to ischemia/reperfusion injury compared with control mice.

37 ls and a survival advantage in comparison to control mice.

38 fic insulin receptor knock-out (betaIRKO) or control mice.

39 suppressive, M2-like phenotype compared with control mice.

40 higher plasma ketones while fasting than did control mice.

41 otoxin-induced septic shock when compared to control mice.

42 recovered at an accelerated rate compared to control mice.

43 g enzymes correlated with improved memory in control mice.

44 /3), MRL/lpr and BXSB/Mp mice and C57BL/6 as control mice.

45 ytes from ARSB-deficient and normal C57BL/6J control mice.

46 f central hepatocytes in the liver lobule of control mice.

47 duced bladder pathology to that of untreated control mice.

48 rogression, compared with Jnk1(Deltahepa) or control mice.

49 on were evaluated in exercised and sedentary control mice.

50 upon high calorie feeding, as did islets of control mice.

51 -fat diet animals as compared with Ldlr(-/-) control mice.

52 olitis after cohousing or feeding feces from control mice.

53 he mutant mice appeared identical to that in control mice.

54 okines and chemokines, compared to wild-type control mice.

55 s is indistinguishable from that observed in control mice.

56 f IL1B and IL18 than intestinal tissues from control mice.

57 urons does not differ between Ndufs4(KO) and control mice.

58 trolled tumors in K14-VEGFR3-Ig mice than in control mice.

59 ic retinas compared with those from normoxic control mice.

60 ice lacking FKBP12 along the nephron than in control mice.

61 iliary tissues from Mdr2(-/-) mice than from control mice.

62 rom the brains of tau transgenic rTg4510 and control mice.

63 abnormal involuntary movements compared with control mice.

64 n arms of the elevated plus maze relative to control mice.

65 vo pathway in L2 MECs of bitransgenic versus control mice.

66 Tg) in Tlr4-/- (C57Bl6/10ScN), and wild-type control mice.

67 ed in Fam13a(-/-) and Fam13a(+/+) littermate control mice.

68 a of T. gondii-infected mice compared to the control mice.

69 nd mRNA levels returned to similar levels as control mice.

70 e of FCCP when compared to the chow-diet fed control mice.

71 of HMGB1 were increased in STZ compared with control mice.

72 ced focal inflammatory changes compared with control mice.

73 ctate transporter expression was detected in control mice.

74 mic tumors than those observed in Rpl22(+/+) control mice.

75 tumors between the low-uptake and untreated control mice.

76 ental blastomycosis, compared with wild-type control mice.

77 ve any differences between EZH2 knockout and control mice.

78 lenged leukotriene C4 synthase-deficient and control mice.

79 11) and sclerostin levels than osteocytes in control mice.

80 cells in Rasgrp1-deficient mice relative to control mice.

81 reliably induced depressive-like behavior in control mice.

82 was markedly increased compared with that in control mice.

83 expression between mouse models of NAFLD and control mice.

84 of more than 60 days compared to 12 days for control mice.

85 proliferating cells compared with wild-type control mice.

86 and liver of gentamicin-treated mice than of control mice.

87 ion was not different in ROCK2(CD)(4Cre) vs. control mice.

88 gulate homocysteine metabolism compared with control mice.

89 evere cardiovascular changes not observed in control mice.

90 s to levels similar to those observed in the control mice.

91 significantly less pancreatic fibrosis than control mice.

92 ucose metabolism in atERalphaKO- compared to control mice.

93 lasma pro-inflammatory cytokines compared to control mice.

94 severe colitis in response to oxazolone than control mice.

95 ortality after MI in T2DM mice compared with control mice.

96 sretinoids compared with sham-injected STGD1 control mice.

97 nificantly reduced in these mice compared to control mice.

98 mation at 22-24 months of age, compared with control mice.

99 icrobiota developed more severe colitis than control mice.

100 sity were substantially lower as compared to control mice.

101 colonized by PMSS1 to a greater extent than control mice.

102 mation and increased mortality compared with control mice.

103 fits observed with high-dose prednisolone in control mice.

104 antly lower bodyweight than their respective control mice.

105 al asthma in ST2 knockout (KO) and wild-type control mice.

106 creased hepatocellular damage, compared with control mice.

107 ous route is approximately 160 mumol/hour in control mice.

108 esulted in similar parasitemia compared with control mice.

109 e that lacked Etv5 in T cells, compared with control mice.

110 able with that in allergen-treated wild-type control mice.

111 D1- and D2-MSNs of alcohol-drinking mice and control mice.

112 , resonance ratios reverted to those seen in control mice.

113 tracellular Ca(2+), compared to muscles from control mice.

114 eased iNOS and IL-6 expression compared with control mice.

115 ;Cre(+) mice but suppressed hepcidin only in control mice.

116 ls, and fibrosis in Hyal2(-/-) compared with control mice.

117 higher in quadriceps of ATF3-KO mice than in control mice.

118 lation, and to transfer metabolic disease in control mice.

119 T1 expression in the endometrium compared to control mice.

120 trophil numbers in fluticasone compared with control mice (0.021 x 10(5) [0.012] vs 0.59 x (5) [0.39]

121 rosclerosis in autoimmune mice compared with control mice (0.64 +/- 0.12 vs 0.32 +/- 0.05 mm(2); P <

122 1 neurons, while concentrations effective in control mice (1.2%) decreased sEPSC frequencies in both

123 ximately 500% in dexamethasone-treated mice (control mice, 100%), without difference in total urea sy

124 shRNA) to make the channel knockdown (KD) or control mice, (2) allergen sensitization/challenge to in

125 sed lymphomagenesis (56%) when compared with control mice (27%).

126 gher survival rate (70%) than wild type (WT) control mice (30%), suggesting OPN plays a deleterious r

127 as significantly higher in treated mice than control mice (5.17% +/- 1.18% vs. 2.41% +/- 0.34% inject

128 of T cells from irradiated PDA to tumors of control mice accelerated tumor growth.

129 -deficient mice were decreased compared with control mice after consuming an HFD.

130 n of MET (Lgr5(Creert2)/Met(fl/fl)/LacZ) and control mice (Ah(Cre)/Met(+/+)/LacZ, Lgr5(Creert2)/Met(+

131 s in obesity, we fed A2AAR-knockout (KO) and control mice an HFD for 16 wk to initiate HFD-induced me

132 ors expressing PDGF-DD more effectively than control mice, an effect enhanced by blockade of the inhi

133 XPHOS dysfunction in time course analyses in control mice and a middle lifespan knockout, respectivel

134 the microbiota of Vdr(-/-) mice relative to control mice and correlations between the microbiota and

135 did not alter the esophageal vasculature of control mice and did not alter recruitment of intraepith

136 MEFV was expressed in colons of control mice and expression increased during chronic and

137 mpal neurons prepared from GIRK2-trisomic Ts control mice and GIRK2-disomic Ts mice in which Kcnj6 ha

138 hosphorylation in parallel with Hamp mRNA in control mice and Hep3B cells.

139 ed to examine their terminal fields in adult control mice and in adult mice in which the alpha-subuni

140 ump failure and related arrhythmias, both in control mice and in mice subjected to chronic pressure-o

141 ump failure and related arrhythmias, both in control mice and in mice subjected to chronic pressure-o

142 after experimental AMI were compared between control mice and littermates with endothelial-restricted

143 he activation of CRTC1 in the hippocampus of control mice and mice lacking the Alzheimer's disease-li

144 Finally, control mice and mice with liver-specific disruption of

145 In livers of control mice and primary rat hepatocytes, activation of

146 Using GS-knockout/liver and control mice and stepwise increments of enterally infuse

147 1 day post infection (DPI) from infected and control mice and were subjected to liquid chromatography

148 injected with bone marrow from Bcl3(-/-) or control mice, and AP was induced.

149 We detected VEGF expression in JG cells of control mice, and cAMP agonists regulated VEGF expressio

150 Nr1d1(-/-) mice, control mice, and control mice given injections of SR900

151 tissues from ATP7B-knockout (Atp7b(-/-)) and control mice, and established 3-dimensional enteroids.

152 ction in blood ejection fraction relative to control mice, and eventual lethality in the absence of c

153 ability following administration of DSS than control mice, and loss of the tight junction proteins oc

154 A within cortical and hippocampal neurons in control mice, as well as within astrocytes surrounding a

155 colitis following administration of DSS than control mice, associated with preserved gut barrier inte

156 n lactate levels in APP/PS1 mice relative to control mice at 12 months of age.

157 TBI was found to be similar to nonirradiated control mice at 18 months, suggesting that TBI accelerat

158 and did not develop more severe colitis than control mice at 4-6 months.

159 l sO2 levels were similar between l-NAME and control mice at GD14 and GD18.

160 alpha immunostaining was higher in L-NAME vs control mice at GD14.

161 titutive miR-150 expression as compared with control mice at L2.

162 ered IL-36beta to neonatal (1-wk old) and AD control mice at the time of immunization with tetanus to

163 Notably, compared with kidneys of control mice, Atg5(Delta) (flox/) (Delta) (flox) kidneys

164 ury than that observed in Jnk1(Deltahepa) or control mice, based on levels of serum markers and micro

165 As opposed to control mice, betaGlud1(-/-) animals fed a high calorie

166 and myosin heavy chain IIa in quadriceps of control mice but not in ATF3-KO mice.

167 ATP single-channel activity in beta-cells of control mice but not of betaGC-A-KO mice.

168 mice was similar to the maximum observed in control mice but was unchanged by time of day or estradi

169 r survival (similar to Il1r1(-/-) mice) than control mice but, unlike Il1r1(-/-) mice, did not have i

170 synaptic pathology and memory impairment in control mice, but not in mice lacking ALOX5.Taken togeth

171 tion in the above optical ICSS in VgluT2-cre control mice, but not in VgluT2-CB1 (-/-) mice.

172 Tail bleeding time was prolonged in DREAM KO control mice, but not in WT or DREAM bone marrow chimeri

173 PH (priming phase of liver regeneration) in control mice, but this effect was delayed in interleukin

174 d a distinctly thinner epithelial layer than control mice, but this was not altered by HDM.

175 s, body weight was monitored and compared to control mice carry wildtype Adenomatous polyposis coli g

176 HCV RNA was readily detected in all control mice challenged with a patient-derived HCV genot

177 ve transfer of M2-like macrophages conferred control mice conspicuous protection against insult.

178 Relative to controls, mice consuming the Western diet had diminished

179 Livers of control mice contained proliferative plasmablasts that o

180 Compared with control mice, db/db mice at 11 to 12 and 14 to 16 weeks

181 cell-specific Etv5-deficient and littermate control mice demonstrated that IL-10 production and gene

182 adults that were dramatically larger than in control mice, demonstrating for the first time that sodi

183 In contrast, all immunized negative-control mice developed PcP.

184 tal cell-specific disruption of Pten but not control mice developed sporadic, macroscopic, intraducta

185 d lower levels of chemokines than cells from control mice did.

186 mice had lower levels of ROS than feces from control mice during DSS administration.

187 social disruption), as well as non-stressed control mice, during challenge with the colonic pathogen

188 rexpression of beta-arrestin2 in knockout or control mice either reverses or protects against LIDs an

189 vity to evoked pain compared with littermate control mice expressing normal human hemoglobins.

190 /-) hearts 2.3-fold compared with littermate control mice fed a ketogenic diet, yet it did not improv

191 Although Mfn1LKO mice are similar to control mice fed a low-fat diet, they are protected agai

192 Compared with control mice fed a regular-salt diet, knockout mice fed

193 rgic stimulation when compared with those of control mice fed normal chow.

194 Control mice fed the same daily sodium intake remained n

195 Compared with controls, mice fed a diet providing 42% of energy as fat

196 When compared with control mice, female mice exposed to ES displayed decrea

197 cantly higher in colons of Mefv-/- mice than control mice following colitis induction, whereas the le

198 idermis of TC-PTP-deficient mice compared to control mice following TPA treatment.

199 re rapidly from acute radiotoxicity than the control mice following WBIR.

200 h acetaminophen protected Jnk(Deltahepa) and control mice from liver injury.

201 rocytes (FXR(-/-)iVP16FXR) and corresponding control mice (FXR(-/-)iVP16).

202 timulation of lung fibroblasts isolated from control mice generated HA-enriched cable structures in t

203 Levels of BCL3 were higher in pancreata from control mice given cerulein than from mice without AP, a

204 Nr1d1(-/-) mice, control mice, and control mice given injections of SR9009 were given LPS a

205 Control mice given intrarectal hREG3A and exposed to 2,4

206 Control mice had an average of 2,280 asymmetric synapses

207 long-lived Ames dwarf and normal littermate control mice in a genotype and sex-specific manner.

208 ficantly higher survival rate than wild-type control mice in response to Candida albicans infection,

209 e (Lck(cre)IL-4Ralpha(-/lox)) and respective control mice in the presence or absence of IL-4 or IL-13

210 LXA4 did not affect neutrophils taken from control mice in which CD64 expression was minimal.

211 starting at 4-6 months of age compared with control mice, in part because of altered bile acid metab

212 e observed after DMM surgery in Cre-negative control mice, including articular cartilage degradation

213 in control of JHMV replication compared with control mice, indicating that T cell infiltration into t

214 g functional prolactin receptors compared to control mice, indicating transport is independent of the

215 pamine D2 receptor-containing LHb neurons of control mice induces depression-like behaviors.

216 were more pronounced in Il10-/- mice, and in control mice infected with H pylori that expressed a mor

217 Compared to mortality in controls, mice infected with EW murine RV were substanti

218 Compared with kidneys from control mice, kidneys from mice with conditional deletio

219 (Ly5.2) into irradiated 8-week-old Fx-/- or control mice (Ly5.1).

220 transplanted bone marrow cells from Fx-/- or control mice (Ly5.2) into irradiated 8-week-old Fx-/- or

221 Compared with control mice, mice with macula densa-specific knockout o

222 one or 2,4,6-trinitrobenzenesulfonic acid to control mice, mice with T-cell-specific deletion of GATA

223 ice aggravated atherosclerosis compared with control mice, most likely due to heightened vascular inf

224 lenic cells extracted from tumor-bearing and control mice (n= 3/group) were coincubated with the opti

225 activity (Raleigh's r > 0.4) was recorded in control mice, no such activity was found in Ecl mice, al

226 uman REG3A transgene but were fed feces from control mice (not expressing hREG3A) as newborns.

227 PDE10A was studied in control mice (NT) and in mice carrying human wild-type t

228 increased in these animals as compared with control mice on HFD.

229 everal private clonotypes rarely observed in control mice or in the pre-immune repertoire.

230 tion and intestinal barrier disruption, than control mice or mice with deletion of EGFR from intestin

231 estinal mucosa and colonic mucosa of healthy control mice or those exhibiting NSAID-induced enteropat

232 not reduce platelet responses compared with control mice (P > 0.1).

233 baseline in conatumumab- versus IgG1-treated control mice (P < 0.001), in good correlation with histo

234 baseline in conatumumab- versus IgG1-treated control mice (P < 0.001), in good correlation with histo

235 3 compared to 7.8 +/- 0.5 for the uninfected control mice (P < 0.05).

236 BA had 7-fold more Il17a messenger RNA than control mice (P = .02).

237 ass at the end of treatment as compared with control mice (P = 0.002; ANOVA on ranks with Dunn test),

238 ificantly lower in OIR mice than in normoxic control mice (P = 0.0048).

239 ining (49.7% reduction in mice given HSCs vs control mice; P < .001), and hepatic hydroxyproline cont

240 (328 mg/g in mice given HSCs vs 428 mg/g in control mice; P < .01).

241 e were treated with pazopanib and seven were control mice; P < .05).

242 tion of cerulein or sodium taurocholate than control mice; pancreata from the Bcl3(-/-) mice with AP

243 tasis and fibrosis: depleting macrophages in control mice protected from acute insult; conversely, de

244 Control mice received complete Freund adjuvant alone.

245 The bone marrow of pair-fed control mice receiving intraperitoneal saline stored a l

246 In control mice REEP6 was localized to the inner segment an

247 Following HDM exposure, the control mice responded with a marked AHR and airway infl

248 for hyperhomocysteinemia, and sibling CBS+/+ control mice revealed that deficiency of CBS upregulates

249 inal uptake in the treated mice, whereas the control mice showed only urinary excretion.

250 urrent moderate hypoglycemia in T1D, but not Control, mice significantly impaired cognitive performan

251 of MG cells derived from betaENaC MG KO than control mice, suggesting that betaENaC plays a role in c

252 Forty-eight hours after ischemia, 28% of control mice survived compared with 86% of cKO-PT-Mfn2 a

253 patic injury was much higher in ConA-treated control mice than in HSC-depleted mice.

254 xogenous cytokine administration, but not in control mice that lack human immune cells.

255 wrapped around the outflow vein compared to control mice that received no scaffolding.

256 r return to the same body weight as chow fed control mice, the fasting insulin, glucose, and hepatic

257 Compared with control mice, these mice exhibited improved renal functi

258 Compared with controls, mice treated with daily NAC demonstrated impro

259 In addition, compared to those from control mice, Treg from Aza-treated mice showed more sup

260 Control mice underwent transplantation with wild-type or

261 iKO, villin-Cre+, Sirt1(flox/flox) mice) and control mice (villin-Cre-, Sirt1(flox/flox)) on a C57BL/

262 PET imaging of EAE and control mice was performed 1, 4, and 19 h after (64)Cu-r

263 Compared to regular diet-fed control mice, WD-fed mice gained significantly more weig

264 letal muscles of ATF3-knockout (ATF3-KO) and control mice were analyzed at rest, after exercise, and

265 Both septic and control mice were carefully monitored (every 30 min) for

266 s differentially RNA-edited between case and control mice were enriched for functional terms highly r

267 Cell-treated, media-treated, and control mice were euthanized at 17 weeks of age.

268 In this time course study, cohorts of Tg and control mice were euthanized at defined intervals.

269 Control mice were fed the same diets but not exposed to

270 Nr1d1(-/-) mice and control mice were given intraperitoneal injections of LP

271 10) or 110 MBq (82-116 MBq, n = 10), whereas control mice were injected with vehicle (n = 10).

272 Female Igf1r-deficient and control mice were intranasally challenged with house dus

273 n this replication attempt, while 70% of the control mice were reported to be tumor-free after 9 week

274 Control mice were transplanted with nontreated islets an

275 e effect of obesity/T2D, while infections in control mice were unchanged.

276 IFNG receptor 1 (Ifngr1-/- mice) and C57BL6 (control) mice were infected with PMSS1 (wild-type) or PM

277 Deltahepa)), as well as Jnk1-floxed C57BL/6 (control) mice, were given injections of CCl4 (to induce

278 aintain energy balance to the same extent as control mice when fed a high-fat diet.

279 ut (LKO) mice were significantly higher than control mice when injected i.p. with an acetaminophen do

280 showed small or no increases in injury above control mice when subjected to threshold levels of ische

281 ood loss that was significantly greater than control mice, whereas all non-inhibitory MAbs produced b

282 ling and inflammation when transplanted into control mice, whereas control BM cells had a protective

283 lactate levels within the frontal cortex of control mice, whereas lactate levels remained unaltered

284 ice were similar to baseline, in contrast to control mice which showed a 54% increase in plaque, sugg

285 hallenging were fully protected, compared to control mice which sufferered from profound weight loss

286 , and extensive midzonal hepatocyte death in control mice, which were strongly minimized in HSC-deple

287 sociated dysplasia was compared with that in control mice with a two-tailed Mann-Whitney U test.

288 ells, dendritic cells, and granulocytes than control mice with AP.

289 ged in Bcl3(-/-) mice with AP, compared with control mice with AP.

290 Injection of control mice with GIP increased plasma levels of GLP1, i

291 teriorating LV ejection fraction occurred in control mice with large infarcts (>/=25% LV).

292 Next, we challenged both vaccinated mice and control mice with MERS-CoV after adenovirus transduction

293 old less in PT-Dicer(-/-) mice compared with control mice with no increase in PTH mRNA levels and par

294 Senescence-accelerated mice (SAM, n=18) and control mice with normal senescence (n=15) were fed norm

295 Colitis was induced in C57BL/6JCrl mice (controls), mice with IEC-specific disruption of Stat1 (S

296 Compared with controls, mice with elevated hematocrit (RBC(HIGH)) form

297 v-/- mice developed more severe colitis than control mice, with a greater extent of epithelial hyperp

298 nificant shift in composition, compared with control mice, with enrichment of Clostridiales (Ruminoco

299 he peritoneal cavity compared with wild-type control mice, with no difference in the recruitment of m

300 dministration of dextran sulfate sodium than control mice, with reduced epithelial restitution.