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1                                                             Control mice exhibited a large proximal-to-distal decrease an
2                                                             Control mice received an equivalent dose of nitrate-depleted
3                                                             Control mice were intratracheally instilled with sterile phos
4 d 18-20 month-old male AbetaPP/PS1 and age-matched C57BL/6J control mice.
5 ntents in antibiotic-induced microbiota-depleted (AIMD) and control mice showed significant variations in the metabolism
6 is did not differ significantly between IL12B-deficient and control mice.
7                            Accordingly, CD11c-Ecad(del) and control mice developed comparable contact hypersensitivity re
8 imal half of the small intestine of Hnf4alphagamma(DKO) and control mice.
9 ical, morphological and functional analysis in Ric(EKO) and control mice.
10         Organoids from colon cells of CRT-knockout mice and control mice were analyzed by qRT-PCR, immunoblot, and transe
11 DMD and control hiPSC-derived cardiomyocytes, mdx mice, and control mice (in the presence or absence of propranolol and i
12 from Lkb1(Lgr5-KO) mice lost ISCs compared with crypts from control mice.
13                                                          In control mice, spindles were phase-coupled with DWs, and hippo
14 cell adhesion, LC numbers remained stable and similar as in control mice, even in aged animals.
15 nnervation exhibited a muted proximal-to-distal decrease in control mice and a smaller loss after VAGX (45-48%).
16 ed plasma vimentin, while vimentin levels were decreased in control mice injected with synthetic miR-144.
17 induced progressive pancreatitis in T7K24R mice, but not in control mice, with typical features of chronic pancreatitis.
18                               Sex differences were noted in control mice, in which female islets showed 5 selenoproteins
19 sion in pulsatile luteinizing hormone secretion observed in control mice.
20 f the tibiotarsal joints that were not observed in infected control mice.
21 ced TCR diversity compared to that of persistently infected control mice (Shannon indices of 2.1 and 2.6, respectively).
22       We found that, compared with isotype-treated infected control mice, anti-PD-1-treated mice had improved survival, r
23 omas resolved completely between days 8 and 9 in littermate control mice (n=12), but were still present at day 9 in mice
24 Tg], PON1 knock-out mice [PON1KO], and wild type littermate control mice [WT].
25 s in HoxB8 chimeras, but not in irradiated, nontransplanted control mice.
26                                                 In normoxic control mice, HIF-1alpha deletion in the CNS or NTS did not a
27 usion of Ngb-H64Q-CCC restored respiration rates to that of control mice correlating with higher electron transport chain
28 dle region, while outer and inner regions resembled that of control mice.
29 nor worsened compared with isogenic L. monocytogenes-primed control mice.
30 usceptible mice, and defeated-resilient mice more resembled control mice.
31 f IgE, IgG1, IgG2a and higher levels of IgA antibodies than control mice.
32 ed less severe DSS- or T-cell transfer-induced colitis than control mice.
33 heart function, longer survival, and smaller scar size than control mice 28 days post-MI.
34 d the same metabolic profile and beta-cell phenotype as the control mice with an intact Akt1 gene.
35 -deficient mice had fewer organisms in their lungs than the control mice.
36 , while the NMN-treated cKO mice responded similarly to the control mice.
37 ar bladder pressure and urine secretion ability compared to control mice especially at 6 months posttransplant.
38 overy of cardiac contractile function after AMI compared to control mice.
39 s (i.e. sinus pause and sinus arrhythmias) when compared to control mice.
40 lack of viral control in CaMKIIcre:IFNAR(fl/fl) relative to control mice coincided with sustained Cxcl1 and Ccl2 mRNAs bu
41 he nuclei and cytoplasm of islets cells than in non-treated control mice and this finding was corroborated by immunoblott
42      Microarrays of carotid arteries from Pcsk6(-/-) versus control mice revealed suppression of contractile SMC markers,
43 01), and increased serum antibody titers (P < .01), whereas control mice did not.
44 flammatory cells, and plasma amylase activity compared with control mice given cerulein injections.
45                                               Compared with control mice, Pkd1 knockout mice exhibited reduced arterial p
46 SV infection led to increased HA accumulation compared with control mice, which also coincided with decreased hyaluronida
47 ty acid synthesis transcripts were increased, compared with control mice.
48 oduced significantly fewer IgE Abs to peanuts compared with control mice.
49 anoids derived from Hnf4alphagamma(DKO) mice, compared with control mice.
50 es tumor growth rates in mice recipients in comparison with control mice receiving CTLs pre-cultured with TMPs from untre