ライフサイエンスコーパス: control mice

1 Control mice received complete Freund adjuvant alone.

2 rves were labeled to examine their terminal fields in adult control mice and in adult mice in which the alpha-subunit of

3 the set of genes differentially RNA-edited between case and control mice were enriched for functional terms highly releva

4 in aerosol-challenged leukotriene C4 synthase-deficient and control mice.

5 Nr1d1(-/-) mice and control mice were given intraperitoneal injections of LPS to

6 thelium (SIRT1 iKO, villin-Cre+, Sirt1(flox/flox) mice) and control mice (villin-Cre-, Sirt1(flox/flox)) on a C57BL/6 bac

7 Nr1d1(-/-) mice, control mice, and control mice given injections of SR9009 were given LPS and D-

8 lated H3K27Ac and mRNA levels returned to similar levels as control mice.

9 BS+/-, a model for hyperhomocysteinemia, and sibling CBS+/+ control mice revealed that deficiency of CBS upregulates card

10 ion and a reduction in the above optical ICSS in VgluT2-cre control mice, but not in VgluT2-CB1 (-/-) mice.

11 fl)/Apc(fl/+) mice compared with Ah(Cre)/Met(+/+)/Apc(fl/+) control mice.

12 nly 1 copy of human REG3A transgene but were fed feces from control mice (not expressing hREG3A) as newborns.

13 r DSS-induced colitis after cohousing or feeding feces from control mice.

14 was also induced in CVB3-induced myocarditis versus healthy control mice.

15 idal congestion, and extensive midzonal hepatocyte death in control mice, which were strongly minimized in HSC-depleted m

16 modeling and deteriorating LV ejection fraction occurred in control mice with large infarcts (>/=25% LV).

17 mediately after PH (priming phase of liver regeneration) in control mice, but this effect was delayed in interleukin 6-de

18 PDE10A was studied in control mice (NT) and in mice carrying human wild-type torsin

19 nversely, worm burden was higher in Nmur1(-/-) mice than in control mice.

20 Tail bleeding time was prolonged in DREAM KO control mice, but not in WT or DREAM bone marrow chimeric mic

21 tion in Cpt2M(-/-) hearts 2.3-fold compared with littermate control mice fed a ketogenic diet, yet it did not improve car

22 Nr1d1(-/-) mice, control mice, and control mice given injections of SR9009 wer

23 We detected VEGF expression in JG cells of control mice, and cAMP agonists regulated VEGF expression in

24 e peroxidase-1, and myosin heavy chain IIa in quadriceps of control mice but not in ATF3-KO mice.

25 ponse to adrenergic stimulation when compared with those of control mice fed normal chow.

26 umulation of bisretinoids compared with sham-injected STGD1 control mice.

27 in microbial community composition compared to non-stressed control mice.

28 Mefv-/- mice developed more severe colitis than control mice, with a greater extent of epithelial hyperplasia

29 defective gut microbiota developed more severe colitis than control mice.

30 r1-/- mice were colonized by PMSS1 to a greater extent than control mice.

31 feration rates of MG cells derived from betaENaC MG KO than control mice, suggesting that betaENaC plays a role in cell r

32 developed more severe colitis in response to oxazolone than control mice.

33 tration of diethylnitrosamine and carbon tetrachloride than control mice.

34 al was 1 week in this replication attempt, while 70% of the control mice were reported to be tumor-free after 9 weeks in

35 nd gastrointestinal uptake in the treated mice, whereas the control mice showed only urinary excretion.

36 ation of oxazolone or 2,4,6-trinitrobenzenesulfonic acid to control mice, mice with T-cell-specific deletion of GATA3, an

37 increased in epidermis of TC-PTP-deficient mice compared to control mice following TPA treatment.

38 dy weight or glucose metabolism in atERalphaKO- compared to control mice.

39 n increase in plasma pro-inflammatory cytokines compared to control mice.

40 n were both significantly reduced in these mice compared to control mice.

41 eatosis and obesity were substantially lower as compared to control mice.

42 ss, 60-90% reduction in blood ejection fraction relative to control mice, and eventual lethality in the absence of cardia

43 time in the open arms of the elevated plus maze relative to control mice.

44 ice had a significantly higher survival rate than wild-type control mice in response to Candida albicans infection, and t

45 e of 4.4 +/- 1.3 compared to 7.8 +/- 0.5 for the uninfected control mice (P < 0.05).

46 ergic inflammation was not different in ROCK2(CD)(4Cre) vs. control mice.

47 9-232 MBq, n = 10) or 110 MBq (82-116 MBq, n = 10), whereas control mice were injected with vehicle (n = 10).

48 creata of A2AAR-deficient mice were decreased compared with control mice after consuming an HFD.

49 ith increased mortality after MI in T2DM mice compared with control mice.

50 ntaneous inflammation at 22-24 months of age, compared with control mice.