ライフサイエンスコーパス: control mouse

1 ands from the transgenic mouse versus virgin control mouse.

2 nd three InDels in four KI mice but not in a control mouse.

3 ced efficiency compared to v-Ras-transformed control mouse 3T3 and p50-deficient cells.

4 cating that at least two different promoters control mouse Abcg2 transcription during hematopoiesis.

5 cted from uninfected ticks and from negative control mouse and human bloods, and these background DNA

6 latory devices to growth cytokine targets to control mouse and primary human T-cell proliferation.

7 ine whether cholinergic signaling in the SNc controls mouse behavior, we used optogenetics in awake b

8 found that all four PARs are present in the control mouse bladder, and follow a unique distribution.

9 Our data suggest that Rab11a critically controls mouse blastocyst development and soluble matrix

10 mentin, while those in comparable regions of control mouse brain did not.

11 ed glycan structures in wild-type littermate control mouse brains.

12 s seen in the transgenic mouse model but not control mouse brains.

13 from HVCN1-deficient mice mirrored those in control mouse cells treated with Zn(2+).

14 exon junction complex (EJC) that binds RNA, controls mouse cerebral cortical size by regulating NSC

15 Significant QTLs are detected to control mouse cholesterol gallstone formation.

16 rize key transcriptional regulatory elements controlling mouse Daf1 expression, a 2.5-kb fragment cor

17 e results uncover a crucial role of Brpf1 in controlling mouse embryo development and regulating cell

18 tivates mTORC1 activity in both p18(-/-) and control mouse embryonic fibroblast cells, suggesting tha

19 to electrically stimulated heart tubes from control mouse embryos or embryos with the Na+-Ca2+ excha

20 low)) and 774 +/- 135 Crry molecules/cell on control mouse erythrocytes.

21 tional phenotypes that distinguish them from control mouse fibroblasts.

22 identified in a screen to uncover genes that control mouse gametogenesis.

23 arisons of the transcriptomes of mGEPs and a control mouse gastric epithelial cell line revealed that

24 Compared with a normoxic, chow-fed control mouse heart, hypoxia decreased PPARalpha express

25 osin heavy chain in adult Gsalpha-DF but not control mouse hearts was reversed by cTnI overexpression

26 ptive metabolic changes caused by hypoxia in control mouse hearts were found to have occurred already

27 use hearts compared with wild-type (Npr1+/+) control mouse hearts.

28 thesis were measured in 9-week-old ob/ob and control mouse hearts.

29 mban, and 0.26 +/- 0.01 mumol/L in wild-type control mouse hearts.

30 s 187 +/- 12 micron2, P < .05) compared with control mouse hearts.

31 that one or more microRNAs (miRNA) known to control mouse hematopoiesis and lineage commitment might

32 e components of the molecular circuitry that controls mouse hematopoiesis and suggest that other micr

33 nvestigate whether B cells are important for controlling mouse hepatitis virus strain A59 infection,

34 h), saturable, and substantial compared with control mouse IgG.

35 re randomized to treatment with CNTO 1081 or control mouse IgG.

36 metabolism of SUR1 knock-out (SUR1(-/-)) and control mouse islets with d-[U-(13)C]glucose as substrat

37 of hepatic glycogen synthesis in the ADM and control mouse liver in vivo were measured using new adva

38 isolated from Foxa2(loxP/loxP) Alfp.Cre and control mouse livers, and HNF6 binding to its target, Gl

39 go]-CLIP) sequencing in miR-122 knockout and control mouse livers, as well as in matched human hepato

40 subset of retinal ganglion cells (RGCs) that controls mouse looming-evoked defensive responses throug

41 ng tissues from those of i.d.-inoculated and control mouse lung tissues.

42 Since the airways of control mouse lungs contain few alcian blue/periodic aci

43 ell influx between bleomycin-treated CKO and control mouse lungs.

44 of these genes in vivo and provides a novel, controlled mouse model for spontaneous venous thrombosis

45 Using a conscious, temperature-controlled mouse model, we showed that maintaining a cor

46 , we sought to assess whether data from well-controlled mouse models can compensate for scarce human

47 Compared with controls, mouse models of ischemia-reperfusion injury, u

48 In control mouse muscle, desmin is enriched at the sarcolem

49 urrence of trans-rearrangements in this well-controlled mouse mutant system and found a 50-100-fold i

50 ize as compared with the alpha-granules from control mouse platelets.

51 s has identified Sinc/Prni as the major gene controlling mouse scrapie incubation time.

52 Proteomic analysis of MR-/- and control mouse sera showed that an additional 4 out of 52

53 and lungs; PF and tissue samples from only 1 control mouse showed bacterial growth, and no control mi

54 aucomatous mouse strain, DBA/2J and a normal control mouse strain (C57BL/6) were used in the study.

55 to equalize manganese levels between HD and control mouse striatal cells and rescued the ATM-p53 sig

56 xpress human CD4 on the surface, but not the control mouse T cells lacking human CD4.

57 and VI but more in layers II/III and V than control mouse TCAs.

58 implanted osmotic pumps, with an additional control mouse used for histologic examination (n = 1).