戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 e than 24 hours, subdural hematoma, or brain contusion).
2 1-28 days following a moderate thoracic (T9) contusion.
3 nical management and pathophysiology of lung contusion.
4 athogenesis of sterile injury following lung contusion.
5 omote neurological recovery from spinal cord contusion.
6  reduced regardless of the distance from the contusion.
7  injury models: transection, compression, or contusion.
8 motor cortex and brainstem after spinal cord contusion.
9 ired only in the area immediately around the contusion.
10 e acute inflammatory response following lung contusion.
11 s injected into wild-type mice prior to lung contusion.
12 ate in the pathogenesis of brain edema after contusion.
13 tic and injured cells at 48 h after cortical contusion.
14 n cell survival and apoptosis after cortical contusion.
15 patients, especially patients with pulmonary contusion.
16 axis of the cord at 1, 2, 3 and 4 days after contusion.
17 d controls only in structures ipsilateral to contusion.
18 her in patients and wild-type mice with lung contusion.
19  mice received a moderate midthoracic spinal contusion.
20 atients who underwent surgery for a cerebral contusion.
21 for proinflammatory cytokines following lung contusion.
22 ute inflammatory response and injury in lung contusion.
23  driver of acute inflammation following lung contusion.
24 n central neuropathic pain after spinal cord contusion.
25 67 in newly born rats or after a spinal cord contusion.
26 tmortem lung samples from patients with lung contusion.
27 (DTI) to identify axonal injury distant from contusions.
28 nt injuries and 27 (22.0%) had isolated bone contusions.
29 ents were documented in the cases with these contusions.
30 ions by 2 h after either left or right sided contusions.
31 nd functional recovery in rats with thoracic contusions.
32 cic (T9) spinal cord 1 week after a moderate contusion (10 gm, 12.5 mm, NYU impactor).
33 f CBF in the cortex contralateral to a focal contusion, 24 h after the event.
34 ral degeneration and apoptosis and a smaller contusion 3 days post-injury than wild type littermates.
35 respiratory distress syndrome (8%, 3 of 40), contusion (3%, 1 of 40), and unknown (3%, 1 of 40).
36 ebral disk edema, and one patient had a cord contusion, a ligamentous injury, and an intervertebral d
37                              Nine days after contusion, a Vitrogen matrix +/-one million MSC was inje
38 at infiltrate the brain after nerve crush or contusion actually protect neurons from injury.
39 g cardiopulmonary disorders (e.g., pulmonary contusion, acute respiratory distress syndrome, atelecta
40 g cardiopulmonary disorders (e.g., pulmonary contusion, acute respiratory distress syndrome, atelecta
41                           By contrast, blunt contusion affected progression of a subsequent local GAS
42 rison of the temporal outcome patterns among contusion and compression injuries in rats and other spe
43 cans enables accurate identification of lung contusion and differentiation of contusion from other ca
44 I in two different injury mouse models-focal contusion and diffuse concussive injury.
45 1ra levels very similar to those produced by contusion and glutamate.
46 man trials if they elicit benefits in severe contusion and larger animal models and pass the test of
47 nearly all neurons subacutely after cortical contusion and persists for at least 2 weeks thereafter.
48 n with attenuated brain edema after cerebral contusion and to reduce brain edema after focal cerebral
49 Long-Evans rats received unilateral cortical contusion and were tested in the Morris Water Task (MWT)
50 ncrease at the lesion site after right-sided contusions and 22% and 32% increases in the contralatera
51 tentionally injured women had a total of 434 contusions and abrasions, 89 lacerations, and 41 fractur
52 tracranial abnormalities such as haematomas, contusions and cerebral oedema.
53                               Any additional contusions and internal derangements were documented in
54 aring was seen at CT in 38 (95%) of the lung contusions and none of the cases of atelectasis, lacerat
55  Sixty percent of the patients had pulmonary contusion, and 59% were cigarette smokers.
56  torsion, hematoma, spermatic cord injury or contusion, and epididymal, scrotal, and urethral injurie
57 per respiratory tract infection, soft tissue contusion, and no obvious portal of entry.
58 matomas, one pneumocephalus, one hemorrhagic contusion, and one subarachnoid hemorrhage.
59  pulmonary embolism, myocarditis, myocardial contusion, and renal failure, can be associated with an
60 e most prevalent diagnoses were lacerations, contusions, and fractures.
61                    Rodent models of cortical contusion are being used effectively to clarify the mole
62                                        These contusions are almost always associated with a far perip
63         The anti-CD11d mAb treatment reduced contusion areas as well as overall contusion volume comp
64     Adolescents may have the same pattern of contusions as adults but may maintain an intact ACL owin
65 were subjected to a standardized weight drop contusion at T-8 (10 gm x 2.5 cm) and NBQX (15 nmol) or
66 were subjected to a standardized weight-drop contusion at T8 (10 gm x 2.5 cm).
67                          After a spinal cord contusion at T9 in adults, the area of alpha1beta1 integ
68                                    Following contusion at the T8 spinal level (made using the Infinit
69 giopoietin-1 mimetic following a spinal cord contusion at thoracic level 9 in mice rescued epicentre
70 hilar interneurons 8-13 weeks after cortical contusion brain injury in transgenic mice that express e
71 orm in microvessels surrounding the cortical contusion by 24 h post-injury.
72                                       Ocular contusion can produce severe lesions, which if not treat
73  in the management of patients with cerebral contusion (CC) following severe traumatic brain injury (
74 ral activation pathways, lung injury in lung contusion demonstrated increased p38 mitogen-activated p
75                           This model of mild contusion did not provide a focus for initiation or seed
76  of contrast extravasation and laceration or contusion extending into the hepatic vein(s), inferior v
77 ors, was infused into rats after spinal cord contusion for 28 days.
78 e acetylcysteine group, and aortic aneurysm, contusion, forearm fracture, and worsening IPF in the pl
79  Seven of the 366 patients had cervical cord contusions, four patients had ligamentous injuries, thre
80 ion of lung contusion and differentiation of contusion from other causes of lung opacification in chi
81 Female adult C57Bl/6 mice with a spinal cord contusion had progressively more ADAM8 (CD156) immunosta
82                      Five patients with bone contusions had an intact ACL at MR imaging.
83 us, 28% of the 18 patients with typical bone contusions had intact ACLs.
84                                        After contusion, HO-1 was induced in both gray and white matte
85 cellular protein function and the structural contusion implies a model in which newly synthesized, in
86 NRP/GRP) into a midthoracic injury 9 d after contusion improved bladder and motor function, diminishe
87 neuroprotective after a thoracic spinal cord contusion in adult rats.
88                                       Ocular contusion in childhood may not be reported by children.
89 ibutes to secondary damage after spinal cord contusion in mice and suggest that LPA1 antagonism might
90 ed 10-20 years) were reviewed to detect bone contusions in the posterolateral tibial plateau or the l
91                                Specifically, contusion increased emm1 GAS dissemination locally to dr
92                                         Lung contusion induced profound global hypoxia rapidly.
93 linically-relevant rodent models of cervical contusion-induced neuropathic pain that result in chroni
94  role of the classical complement pathway in contusion-induced SCI, male C1q knock-out (KO) and wild-
95 the lesion epicenter of subchronic (10 days) contusion-injured rats.
96  rats received either sham or T9 spinal cord contusion injuries and were implanted with blank or test
97 reactive lymphocytes after SCI, we performed contusion injuries in the thoracic spinal cord of transg
98 ats with thoracic spinal cord transection or contusion injuries induce significant descending cortico
99 57Bl/6 and 129X1/SvJ mice received identical contusion injuries to the mid-thoracic spinal cord and w
100                                              Contusion injury (25 mm) to spinal cord T10 was produced
101 , we subjected one group of rats to cortical contusion injury (CCI) and another to transient middle c
102 ificantly reduced brain edema after cortical contusion injury (CCI) in rat 5h post-injury.
103 als were prepared with a unilateral cortical contusion injury (CCI).
104 s (OEC) into the lesion cavity 6 weeks after contusion injury (NYU impactor device, 25 mm height sett
105                        Following spinal cord contusion injury (SCI) in the rat, certain supraspinal p
106                                  Spinal cord contusion injury (SCI) is known to induce pain-related b
107 as designed to determine effects of cortical contusion injury and d-AMPH on CMRglu in cortical and su
108 ury pathology in adult rats following spinal contusion injury and LV-ChABC treatment, with reduced ca
109 37tg) and wild-type (WT) mice to spinal cord contusion injury and then treated them with recombinant
110 gue-Dawley rats received a moderate thoracic contusion injury and were divided into three groups: ope
111                       Female rats received a contusion injury at T8/9.
112 tissue of ablating reactive astrocytes after contusion injury generated by controlled cortical impact
113            We report that, after spinal cord contusion injury in adult female mice, the biosynthesis
114 or, promotes recovery after traumatic spinal contusion injury in mice, a benefit achieved in part by
115 n-4 (AQP4) water channels after experimental contusion injury in mice, a clinically relevant animal m
116                  TBI was induced by cortical contusion injury in Sprague-Dawley rats.
117 hange in bacterial morphology observed after contusion injury in the muscle model.
118 ents 9 months after moderate thoracic spinal contusion injury in the rat.
119 n alphaB-crystallin for the first week after contusion injury leads to sustained improvement in locom
120 ng either spinal cord dorsal column crush or contusion injury models, miR-155 deletion improves indic
121 sthetized and subjected to a 0.5-mm cortical contusion injury of the somatosensory cortex.
122 s with a thoracic spinal cord transection or contusion injury result in substantial corticospinal and
123                                              Contusion injury resulted in large lesions, with no sign
124 y relevant rat model of thoracic spinal cord contusion injury to examine the effect of varying the de
125                                   Unilateral contusion injury to the sensorimotor cortex causes, amon
126                              Rats received a contusion injury to the spinal cord followed by treatmen
127 T-cell migration and activation after spinal contusion injury were characterized using B10.PL (wild-t
128  the lesion 12 weeks after mouse spinal cord contusion injury were fully remyelinated.
129 background (BUB) received a mild-moderate T9 contusion injury with the Infinite Horizon impactor.
130                Finally, in a model of spinal contusion injury, miR-155 deletion improves locomotor fu
131 ces neurological recovery from a spinal cord contusion injury, showing its potential clinical impact.
132                    Using a new model of mild contusion injury, we set out to determine the effect of
133 ions in astrocyte hypertrophy after cortical contusion injury.
134 Sprague Dawley rats underwent T9 spinal cord contusion injury.
135 Sprague Dawley rats underwent T9 spinal cord contusion injury.
136 or 42 days following midthoracic (T9) spinal contusion injury.
137 male Sprague Dawley rats underwent T9 spinal contusion injury.
138 ion of apoptosis in rat brain after cortical contusion injury.
139 ared normal uninjured tissue with that after contusion injury.
140 ured spinal cords after a moderate, thoracic contusion injury.
141 the rat spinal cord following a standardized contusion injury.
142 rns of adjacent spinal gray matter following contusion injury.
143 or increased pain in a mouse model of spinal contusion injury.
144 s of spinal cord demyelination and traumatic contusion injury.
145 ion was initiated 3 months after spinal cord contusion injury.
146 hs of treatment between 4 and 6 months after contusion injury.
147 nd facet fractures, vertebral body and facet contusions, intervertebral disk herniations, ligamentum
148                                              Contusions involving the posterior lip of the medial tib
149               Management of severe pulmonary contusion is a challenge for clinicians.
150                                         Lung contusion is a major risk factor for the development of
151                                         Lung contusion is a major risk factor for the development of
152                          Unilateral cerebral contusion is associated with an early (30 min) increase
153   Early recovery from incomplete spinal cord contusion is improved by prolonged stimulation of the hi
154  in NE release following unilateral cerebral contusion is protective and that this may act to stabili
155 ocytes was characterized throughout a spinal contusion lesion in Sprague-Dawley and Lewis rats by usi
156 riptional expression patterns in midcervical contusion lesion sites between 1 and 90 d postinjury of
157 to pulmonary hypertension, greater pulmonary contusion lesion size, and stiffer lungs in this porcine
158  are reduced in spinal cord tissue following contusion lesion.
159 ocomotor function is associated with smaller contusion lesions and decreased accumulation of inflamma
160 l analysis showed a large extent of cortical contusion lesions, including both the sensory and motor
161 survive following 50 and 12.5-mm weight drop contusion lesions, respectively, albeit with a reduced i
162 ale rats had unilateral somatosensory cortex contusions made with a 5 mm diameter impact piston.
163 resence of blunt traumatic injury, pulmonary contusion, massive transfusion, and flail chest injury (
164                                    We used a contusion model (N.Y.U. Impactor) to initiate the early
165                        A mild focal cortical contusion model in adult mouse sensory-motor cortex was
166                                      Using a contusion model of SCI, we found that intravenous delive
167                       Thus, we used a spinal contusion model to examine the spatiotemporal expression
168 employed an adaptation of the focal cortical contusion model.
169 al cell counts in the cortex adjacent to the contusion (n = 11).
170 se lesion in midbrain (n = 6), temporal lobe contusion (n = 2), posterior cerebral artery (n = 3), an
171                        In chronic rat spinal contusion, NgR1 decoy treatment from 4 to 6 months after
172 l-computer interface in rats with a cervical contusion of the spinal cord, we synchronized intraspina
173 es were occult fracture (P<.01), bone marrow contusion of the vertebral body (P=.01), muscle strain (
174 ns for knee trauma was conducted to identify contusions of the posterior lip of the medial tibial pla
175                                The effect of contusion on emm1 iGAS infection was assessed in three m
176 njury, we set out to determine the effect of contusion on iGAS bacterial burden, phenotype, and host
177                            One or more brain contusions on MRI, and >/=4 foci of hemorrhagic axonal i
178 ent study, the effect of mid-thoracic spinal contusions on the rat estrous cycle was examined.
179                        Following spinal cord contusion or exposure to elevated glutamate, concentrati
180 neuronal loss in the hippocampus and minimal contusion or infarction at the impact site.
181 d diaphragm, sternal fracture, and pulmonary contusion or laceration seen on radiographs.
182 reference for ligament and disk injuries and contusion or occult fracture) for traumatic injuries.
183 nical factors, such as compression, stretch, contusion or transection.
184 ring brain adjacent to evacuated hemorrhagic contusions or underlying evacuated subdural hematomas wa
185         In brain tissue adjacent to cerebral contusions or underlying subdural hematomas, even brief
186 21) in experimental spinal cord hemisection, contusion, or transection on locomotor recovery measured
187 s underwent polytrauma (femur fractures/lung contusion, P), hemorrhage (mean arterial pressure 25-30
188        Female rats with moderate spinal cord contusion participated in treadmill training, swim train
189                                       Spinal contusion pathology in rats and mice is distinct.
190 located 4.5, 13.5, and 22.5 mm away from the contusion (PC(4.5), PC(13.5), and PC(22.5), respectively
191      Skin-related events included abrasions, contusions, pressure ulcers and lacerations.
192 teral cerebellum after right- and left-sided contusions, respectively.
193                              After pulmonary contusion, resuscitation with diaspirin cross-linked hem
194     Adult male Sprague-Dawley rats underwent contusion SCI at the T9 thoracic level.
195 gest that AQP4 plays a protective role after contusion SCI by facilitating the clearance of excess wa
196 PDGFRalpha), Olig2+, and P0+ cells following contusion SCI in mice.
197 cterized 2 rat models of unilateral cervical contusion SCI that behaviorally result in chronic persis
198 ns in adult rats 1 month after sham surgery, contusion SCI, and SCI treated with a selective inhibito
199 r mice following unilateral midcervical (C4) contusion SCI, numbers of GLT1-expressing astrocytes in
200 mpared with wild-type (WT) littermates after contusion SCI.
201 microdialysis sampling immediately following contusion SCI.
202           In nontransgenic animals, crush or contusion SCIs routinely exhibit regions of degenerated
203                          Rats with bilateral contusions showed increased levels of edema on days 1, 3
204             Unilateral sensorimotor cortical contusion significantly decreased ipsilateral hippocampa
205 ury site or (2) a PNG that bridged a chronic contusion site with a second, more distal injury site.
206 ve into a highly relevant, chronic, cervical contusion site with ChABC treatment of the glial scar an
207 nd hypothalamic regions, and surrounding the contusion site.
208                  Animals were sacrificed and contusion size and GDNF protein expression measured.
209 hough combining it with l-arginine decreased contusion size, it did not enhance behavioral recovery.
210                                              Contusion spinal cord injury (SCI) at T8 produces respir
211     MMP-9 increased rapidly after a moderate contusion spinal cord injury, reaching a maximum at 24 h
212 al cord by microdialysis in association with contusion spinal cord injury.
213  parameters in 21 TBI patients with cerebral contusions studied during the acute phase (3.1 +/- 2.1 [
214             Other CT characteristics of lung contusion such as location, shape, and confluence were a
215 l is one of the most commonly used models of contusion TBI.
216                                         Lung contusions tended to be posterior (60%), crescentic (50%
217 rginine post-CCI had a significantly smaller contusion than injured rats who did not receive any trea
218 tly because even relatively mild spinal cord contusions that produce only minor permanent locomotor d
219                                    After the contusion, the area of alpha6beta1-stained blood vessels
220                               By 6 days post-contusion, the hypometabolism partially reversed in all
221 tions: 13 of these patients had typical bone contusions; the other two had no bone abnormalities but
222 ness cartilage loss, which supports the bone contusion theory of SC formation.
223                                              Contusion to soft tissue had no effect on bacterial burd
224 tradiol filled implants, prior to a moderate contusion to the spinal cord.
225 tal conditions such as sprains, strains, and contusions, topical NSAIDs are associated with greater p
226  that a major portion of SCI patients suffer contusion trauma to cervical spinal cord.
227 conditions, including multiple sclerosis and contusion-type spinal cord injury.
228 d brain function recovery and lessened brain contusion volume after trauma.
229                                              Contusion volume and hippocampal survival were assessed
230                                              Contusion volume and neuron density in the CA1 and CA3 r
231 t reduced contusion areas as well as overall contusion volume compared to vehicle treated animals.
232 osttraumatic hypoxia significantly increased contusion volume compared with traumatic brain injury-no
233 enon improves neurologic outcome and reduces contusion volume following traumatic brain injury in mic
234 omplicated by secondary ischemia, increasing contusion volume from 1 to 30.6 mm3 in the animals that
235             L-arginine treatment reduced the contusion volume from a median value of 5.28 mm3 to 0.63
236                                              Contusion volume was larger in hypothermic vs. normother
237                         For example, overall contusion volume was reduced from 2.7+/-0.5 mm(3) (mean+
238                                              Contusion volume was significantly (p < 0.05) reduced in
239 n treatment significantly (p < 0.05) reduced contusion volume when xenon was given 15 minutes after i
240 utcome score, 2) histological measurement of contusion volume, and 3) analysis of locomotor function
241 rapid rewarming (15 mins) failed to decrease contusion volume, those animals undergoing a slow rewarm
242            Bcl-2 transgenic mice had smaller contusion volumes and increased numbers of surviving neu
243 led 3 days after traumatic brain injury, and contusion volumes were quantitatively assessed.
244  demonstrated significantly (p <.03) reduced contusion volumes, compared with hypoxic normothermic ra
245                   The specific medial tibial contusion was demonstrated in 25 of 215 (12%) knee MR ex
246                          A new model of mild contusion was developed using a weight drop device and c
247       Nonlethal closed-chest unilateral lung contusion was induced in a hypoxia reporter mouse model
248                                     Cortical contusion was induced in male Wistar rats with a pneumat
249                              Unilateral lung contusion was induced in toll-like receptor 3 (-/-), TIR
250 olar lavage and serum samples following lung contusion was measured.
251     The extent of lung injury following lung contusion was significantly reduced in conditional knock
252       bpV(phen) treatments started 4 h after contusion were fully effective.
253                          Lateral compartment contusions were noted in 24 (96%) cases.
254 omputed tomographic (CT) features of 40 lung contusions were reviewed for the presence of subpleural
255 t risk factors for later seizures were brain contusion with subdural hematoma, skull fracture, loss o
256            All animals underwent spinal cord contusion with the NYU-Impactor by a 12.5-mm weight drop

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top