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1 e than 24 hours, subdural hematoma, or brain contusion).
2 1-28 days following a moderate thoracic (T9) contusion.
3 nical management and pathophysiology of lung contusion.
4 athogenesis of sterile injury following lung contusion.
5 omote neurological recovery from spinal cord contusion.
6 reduced regardless of the distance from the contusion.
7 injury models: transection, compression, or contusion.
8 motor cortex and brainstem after spinal cord contusion.
9 ired only in the area immediately around the contusion.
10 e acute inflammatory response following lung contusion.
11 s injected into wild-type mice prior to lung contusion.
12 ate in the pathogenesis of brain edema after contusion.
13 tic and injured cells at 48 h after cortical contusion.
14 n cell survival and apoptosis after cortical contusion.
15 patients, especially patients with pulmonary contusion.
16 axis of the cord at 1, 2, 3 and 4 days after contusion.
17 d controls only in structures ipsilateral to contusion.
18 her in patients and wild-type mice with lung contusion.
19 mice received a moderate midthoracic spinal contusion.
20 atients who underwent surgery for a cerebral contusion.
21 for proinflammatory cytokines following lung contusion.
22 ute inflammatory response and injury in lung contusion.
23 driver of acute inflammation following lung contusion.
24 n central neuropathic pain after spinal cord contusion.
25 67 in newly born rats or after a spinal cord contusion.
26 tmortem lung samples from patients with lung contusion.
27 (DTI) to identify axonal injury distant from contusions.
28 nt injuries and 27 (22.0%) had isolated bone contusions.
29 ents were documented in the cases with these contusions.
30 ions by 2 h after either left or right sided contusions.
31 nd functional recovery in rats with thoracic contusions.
34 ral degeneration and apoptosis and a smaller contusion 3 days post-injury than wild type littermates.
36 ebral disk edema, and one patient had a cord contusion, a ligamentous injury, and an intervertebral d
39 g cardiopulmonary disorders (e.g., pulmonary contusion, acute respiratory distress syndrome, atelecta
40 g cardiopulmonary disorders (e.g., pulmonary contusion, acute respiratory distress syndrome, atelecta
42 rison of the temporal outcome patterns among contusion and compression injuries in rats and other spe
43 cans enables accurate identification of lung contusion and differentiation of contusion from other ca
46 man trials if they elicit benefits in severe contusion and larger animal models and pass the test of
47 nearly all neurons subacutely after cortical contusion and persists for at least 2 weeks thereafter.
48 n with attenuated brain edema after cerebral contusion and to reduce brain edema after focal cerebral
49 Long-Evans rats received unilateral cortical contusion and were tested in the Morris Water Task (MWT)
50 ncrease at the lesion site after right-sided contusions and 22% and 32% increases in the contralatera
51 tentionally injured women had a total of 434 contusions and abrasions, 89 lacerations, and 41 fractur
54 aring was seen at CT in 38 (95%) of the lung contusions and none of the cases of atelectasis, lacerat
56 torsion, hematoma, spermatic cord injury or contusion, and epididymal, scrotal, and urethral injurie
59 pulmonary embolism, myocarditis, myocardial contusion, and renal failure, can be associated with an
64 Adolescents may have the same pattern of contusions as adults but may maintain an intact ACL owin
65 were subjected to a standardized weight drop contusion at T-8 (10 gm x 2.5 cm) and NBQX (15 nmol) or
69 giopoietin-1 mimetic following a spinal cord contusion at thoracic level 9 in mice rescued epicentre
70 hilar interneurons 8-13 weeks after cortical contusion brain injury in transgenic mice that express e
73 in the management of patients with cerebral contusion (CC) following severe traumatic brain injury (
74 ral activation pathways, lung injury in lung contusion demonstrated increased p38 mitogen-activated p
76 of contrast extravasation and laceration or contusion extending into the hepatic vein(s), inferior v
78 e acetylcysteine group, and aortic aneurysm, contusion, forearm fracture, and worsening IPF in the pl
79 Seven of the 366 patients had cervical cord contusions, four patients had ligamentous injuries, thre
80 ion of lung contusion and differentiation of contusion from other causes of lung opacification in chi
81 Female adult C57Bl/6 mice with a spinal cord contusion had progressively more ADAM8 (CD156) immunosta
85 cellular protein function and the structural contusion implies a model in which newly synthesized, in
86 NRP/GRP) into a midthoracic injury 9 d after contusion improved bladder and motor function, diminishe
89 ibutes to secondary damage after spinal cord contusion in mice and suggest that LPA1 antagonism might
90 ed 10-20 years) were reviewed to detect bone contusions in the posterolateral tibial plateau or the l
93 linically-relevant rodent models of cervical contusion-induced neuropathic pain that result in chroni
94 role of the classical complement pathway in contusion-induced SCI, male C1q knock-out (KO) and wild-
96 rats received either sham or T9 spinal cord contusion injuries and were implanted with blank or test
97 reactive lymphocytes after SCI, we performed contusion injuries in the thoracic spinal cord of transg
98 ats with thoracic spinal cord transection or contusion injuries induce significant descending cortico
99 57Bl/6 and 129X1/SvJ mice received identical contusion injuries to the mid-thoracic spinal cord and w
101 , we subjected one group of rats to cortical contusion injury (CCI) and another to transient middle c
104 s (OEC) into the lesion cavity 6 weeks after contusion injury (NYU impactor device, 25 mm height sett
107 as designed to determine effects of cortical contusion injury and d-AMPH on CMRglu in cortical and su
108 ury pathology in adult rats following spinal contusion injury and LV-ChABC treatment, with reduced ca
109 37tg) and wild-type (WT) mice to spinal cord contusion injury and then treated them with recombinant
110 gue-Dawley rats received a moderate thoracic contusion injury and were divided into three groups: ope
112 tissue of ablating reactive astrocytes after contusion injury generated by controlled cortical impact
114 or, promotes recovery after traumatic spinal contusion injury in mice, a benefit achieved in part by
115 n-4 (AQP4) water channels after experimental contusion injury in mice, a clinically relevant animal m
119 n alphaB-crystallin for the first week after contusion injury leads to sustained improvement in locom
120 ng either spinal cord dorsal column crush or contusion injury models, miR-155 deletion improves indic
122 s with a thoracic spinal cord transection or contusion injury result in substantial corticospinal and
124 y relevant rat model of thoracic spinal cord contusion injury to examine the effect of varying the de
127 T-cell migration and activation after spinal contusion injury were characterized using B10.PL (wild-t
129 background (BUB) received a mild-moderate T9 contusion injury with the Infinite Horizon impactor.
131 ces neurological recovery from a spinal cord contusion injury, showing its potential clinical impact.
147 nd facet fractures, vertebral body and facet contusions, intervertebral disk herniations, ligamentum
153 Early recovery from incomplete spinal cord contusion is improved by prolonged stimulation of the hi
154 in NE release following unilateral cerebral contusion is protective and that this may act to stabili
155 ocytes was characterized throughout a spinal contusion lesion in Sprague-Dawley and Lewis rats by usi
156 riptional expression patterns in midcervical contusion lesion sites between 1 and 90 d postinjury of
157 to pulmonary hypertension, greater pulmonary contusion lesion size, and stiffer lungs in this porcine
159 ocomotor function is associated with smaller contusion lesions and decreased accumulation of inflamma
160 l analysis showed a large extent of cortical contusion lesions, including both the sensory and motor
161 survive following 50 and 12.5-mm weight drop contusion lesions, respectively, albeit with a reduced i
162 ale rats had unilateral somatosensory cortex contusions made with a 5 mm diameter impact piston.
163 resence of blunt traumatic injury, pulmonary contusion, massive transfusion, and flail chest injury (
170 se lesion in midbrain (n = 6), temporal lobe contusion (n = 2), posterior cerebral artery (n = 3), an
172 l-computer interface in rats with a cervical contusion of the spinal cord, we synchronized intraspina
173 es were occult fracture (P<.01), bone marrow contusion of the vertebral body (P=.01), muscle strain (
174 ns for knee trauma was conducted to identify contusions of the posterior lip of the medial tibial pla
176 njury, we set out to determine the effect of contusion on iGAS bacterial burden, phenotype, and host
182 reference for ligament and disk injuries and contusion or occult fracture) for traumatic injuries.
184 ring brain adjacent to evacuated hemorrhagic contusions or underlying evacuated subdural hematomas wa
186 21) in experimental spinal cord hemisection, contusion, or transection on locomotor recovery measured
187 s underwent polytrauma (femur fractures/lung contusion, P), hemorrhage (mean arterial pressure 25-30
190 located 4.5, 13.5, and 22.5 mm away from the contusion (PC(4.5), PC(13.5), and PC(22.5), respectively
195 gest that AQP4 plays a protective role after contusion SCI by facilitating the clearance of excess wa
197 cterized 2 rat models of unilateral cervical contusion SCI that behaviorally result in chronic persis
198 ns in adult rats 1 month after sham surgery, contusion SCI, and SCI treated with a selective inhibito
199 r mice following unilateral midcervical (C4) contusion SCI, numbers of GLT1-expressing astrocytes in
205 ury site or (2) a PNG that bridged a chronic contusion site with a second, more distal injury site.
206 ve into a highly relevant, chronic, cervical contusion site with ChABC treatment of the glial scar an
209 hough combining it with l-arginine decreased contusion size, it did not enhance behavioral recovery.
211 MMP-9 increased rapidly after a moderate contusion spinal cord injury, reaching a maximum at 24 h
213 parameters in 21 TBI patients with cerebral contusions studied during the acute phase (3.1 +/- 2.1 [
217 rginine post-CCI had a significantly smaller contusion than injured rats who did not receive any trea
218 tly because even relatively mild spinal cord contusions that produce only minor permanent locomotor d
221 tions: 13 of these patients had typical bone contusions; the other two had no bone abnormalities but
225 tal conditions such as sprains, strains, and contusions, topical NSAIDs are associated with greater p
231 t reduced contusion areas as well as overall contusion volume compared to vehicle treated animals.
232 osttraumatic hypoxia significantly increased contusion volume compared with traumatic brain injury-no
233 enon improves neurologic outcome and reduces contusion volume following traumatic brain injury in mic
234 omplicated by secondary ischemia, increasing contusion volume from 1 to 30.6 mm3 in the animals that
239 n treatment significantly (p < 0.05) reduced contusion volume when xenon was given 15 minutes after i
240 utcome score, 2) histological measurement of contusion volume, and 3) analysis of locomotor function
241 rapid rewarming (15 mins) failed to decrease contusion volume, those animals undergoing a slow rewarm
244 demonstrated significantly (p <.03) reduced contusion volumes, compared with hypoxic normothermic ra
251 The extent of lung injury following lung contusion was significantly reduced in conditional knock
254 omputed tomographic (CT) features of 40 lung contusions were reviewed for the presence of subpleural
255 t risk factors for later seizures were brain contusion with subdural hematoma, skull fracture, loss o
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