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1 irments when they had their acute episode of convulsive status epilepticus.
2 prolonged febrile convulsions and idiopathic convulsive status epilepticus.
3 urs of subsequent stimulation, and prevented convulsive status epilepticus.
4 pal excitation in awake rats without causing convulsive status epilepticus.
5 rmia on neurologic outcomes in patients with convulsive status epilepticus.
6 en enrolled, 176 had a first ever episode of convulsive status epilepticus.
7 es than standard care alone in patients with convulsive status epilepticus.
8 ad a verified diagnosis of overt generalized convulsive status epilepticus.
9 myopathy in patients admitted to the ICU for convulsive status epilepticus.
10 s common in patients admitted to the ICU for convulsive status epilepticus.
11 dren died within 30 days of their episode of convulsive status epilepticus and 16 during follow-up.
12 ediatric data will help inform management of convulsive status epilepticus and appropriate allocation
13 mortality within 8 years after an episode of convulsive status epilepticus, and investigate its predi
14 up were associated with intractable seizures/convulsive status epilepticus, and the rest died as a co
15 y the 518 patients with verified generalized convulsive status epilepticus as well as with data on al
16 of death within 8 years following childhood convulsive status epilepticus but most deaths are not se
17 stematic review on the outcome of paediatric convulsive status epilepticus (CSE) and investigated the
20 isoning or infection, seizures including non-convulsive status epilepticus, endocrinopathy, or thiami
21 al experiments suggest that treatment of non-convulsive status epilepticus following specific insults
22 %, 6-18) of children with first ever febrile convulsive status epilepticus had acute bacterial mening
23 -term mortality and its predictors following convulsive status epilepticus in childhood are uncertain
25 .98 years) were included in the North London Convulsive Status Epilepticus in Childhood Surveillance
26 S): a prospective, population-based study of convulsive status epilepticus in childhood, to obtain a
31 icate that hippocampal epileptogenesis after convulsive status epilepticus is an immediate network de
32 e human condition remains uncertain, but non-convulsive status epilepticus is probably an under-recog
33 cant neurological impairments at the time of convulsive status epilepticus is the main risk factor fo
36 intravenous treatment for overt generalized convulsive status epilepticus, lorazepam is more effecti
37 h a verified diagnosis of subtle generalized convulsive status epilepticus, no significant difference
40 aged 3 months to younger than 18 years with convulsive status epilepticus presenting to 1 of 11 US a
41 naesthetic agents for refractory generalised convulsive status epilepticus, rather than additional tr
44 hort from north London, UK (the north London convulsive status epilepticus surveillance study cohort;
45 d demographic data for episodes of childhood convulsive status epilepticus that took place in north L
46 successfully modeled experimentally because convulsive status epilepticus, the insult most commonly
49 ignificant neurological impairments prior to convulsive status epilepticus was the only independent r
50 pairment who survived their acute episode of convulsive status epilepticus were not at a significantl
51 ork, we identify three phases of generalised convulsive status epilepticus, which we call impending,
52 ly assigned 270 critically ill patients with convulsive status epilepticus who were receiving mechani
53 ked granule cell epileptiform discharges and convulsive status epilepticus with minimal lethality.
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