ライフサイエンスコーパス: copper phenanthroline

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1 an intermediate during DNA damage induced by copper-phenanthroline.

2 Furthermore, copper phenanthroline abolished the activity of the five

3 Inhibition of transport was observed with copper phenanthroline and iodine treatment of P676C/A734

4 Copper phenanthroline and the cross-linker N', N'-o-phen

5 embrane domain 8 can also be cross-linked by copper phenanthroline as predicted by the ANM.

6 lfide cross-linking after adding the oxidant copper phenanthroline, both at room temperature and to d

7 We found that copper phenanthroline catalyzed disulfide bond formation

8 es C in the absence of extracellular Na (+), copper phenanthroline catalyzes disulfide bond formation

9 beta-elimination of 2-deoxyribonolactone by copper-phenanthroline complexes is a general mechanism u

10 Copper-phenanthroline complexes oxidatively damage and c

11 ge by these complexes was examined using two copper-phenanthroline conjugates of the sequence-specifi

12 uman embryonic kidney 293 cells, either with copper phenanthroline (CuP) or the bifunctional reagent

13 Two AlgZ binding sites were defined using copper-phenanthroline footprinting and deletion analyses

14 This genetic approach was complemented by copper-phenanthroline footprinting experiments that show

15 th transcriptional lacZ fusions and in vitro copper-phenanthroline footprinting experiments, we have

16 ctromobility shift assays as well as in situ copper-phenanthroline footprinting have shown that this

17 Electrophoretic mobility-shift assays and copper-phenanthroline footprinting localized AlgZ bindin

18 le from that of wild-type Arc in DNase I and copper-phenanthroline footprinting studies, but the cova

19 Using copper-phenanthroline footprinting to assess the occupan

20 ite-directed mutagenesis and high-resolution copper-phenanthroline footprinting, we have identified t

21 Previously, we showed that oxidation by copper phenanthroline in the presence of GABA of the M2

22 and on alpha(1)beta(1)T256C of oxidation by copper phenanthroline in the presence of potentiating an

23 Oxidation by copper phenanthroline induced disulfide bond formation b

24 Copper phenanthroline-induced oxidation inhibited GABA-i

25 cies characterized by gel electrophoresis in copper-phenanthroline-mediated strand scission.

26 mation with low micromolar concentrations of copper phenanthroline or iodine was found in 3 of 8 init

27 Copper phenanthroline oxidation of YiiP(D49C), YiiP(D157

28 cysteines during activation, using Cd2+ and copper-phenanthroline, which crosslink the cysteines wit

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