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1 l as cell-free experiments with uroporphyrin/coproporphyrin.
2 ar porphyrins, such as protoporphyrin IX and coproporphyrin.
3 rphyrins, but, unlike PCT, these were mainly coproporphyrin.
4 s was undertaken in a search for accumulated coproporphyrin, a red autofluorescent byproduct of heme
5 ts studied so far excrete copious amounts of coproporphyrin and protoporphyrin when grown on enriched
7 rin intermediates including uroporphyrin and coproporphyrin, based on HPLC analysis of cell lysates a
9 les using a model system involving free-base coproporphyrin (COP) complexed with horse heart cytochro
11 proporphyrin, insert ferrous iron to make Fe-coproporphyrin (coproheme), and then decarboxylate copro
13 dified with a spin-label ([MAL-6-alpha2]), a coproporphyrin ([CP-alpha2]) and a coproporphyrin plus a
14 Because some organisms that possess this coproporphyrin-dependent branch are major causes of huma
17 to a cysteine in the hydrophobic core and a coproporphyrin I (CP) appended on the N-terminus of a sy
19 Activation of CgoX induces accumulation of coproporphyrin III and leads to photosensitization of Gr
20 he conversion of coproporphyrinogen III into coproporphyrin III by coproporphyrin synthase HemY, inse
22 se HemH, and oxidative decarboxylation of Fe-coproporphyrin III into protohaem IX by Fe-coproporphyri
23 phyrin synthase HemY, insertion of iron into coproporphyrin III via ferrochelatase HemH, and oxidativ
25 Instead, they oxidize coproporphyrinogen to coproporphyrin, insert ferrous iron to make Fe-coproporp
27 pha2]), a coproporphyrin ([CP-alpha2]) and a coproporphyrin plus a spin-label ([CP-MAL-6-alpha2]) sel
31 porphyrinogen III into coproporphyrin III by coproporphyrin synthase HemY, insertion of iron into cop
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