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1 s were found to bias the spectrum of genomic copy number abnormalities.
2  to detect allelic imbalance and chromosomal copy number abnormalities.
3 r laboratory for array CGH and found to have copy-number abnormalities.
4                    The frequency of specific copy number abnormalities [+4, del(6q23.3-6q25.3), +12,
5 rofiles were correlated with genome-wide DNA copy number abnormalities and clinical and outcome featu
6 molecular level, myeloma is characterized by copy number abnormalities and recurrent translocations i
7 tions/deletions, chromosomal translocations, copy number abnormalities, and zygosity changes in a sin
8                   We identify two loci where copy number abnormalities are correlated with poor survi
9 he rate of diagnosis of patients with genome copy number abnormalities as the cause of developmental
10 rom 45 AA and 196 EA MM patients for somatic copy number abnormalities associated with poor outcome.
11  Plots of BAC end-sequences density identify copy number abnormalities at high resolution.
12   In this study, we compared the genome-wide copy number abnormalities between 29 human and 10 canine
13  mutations by PCR-capillary sequencing, EGFR copy number abnormalities by fluorescence and chromogeni
14                     The most commonly shared copy number abnormalities (CNAs) in all types were losse
15  a Wnt pathway signature and those harboring copy number abnormalities (CNAs) of potential prognostic
16                                              Copy number abnormalities (CNAs) represent an important
17                                              Copy number abnormalities (CNAs) such as somatically-acq
18 precursor ALL, we evaluated gene expression, copy number abnormalities (CNAs), and DNA methylation.
19  genomic hybridization (aCGH) to investigate copy number abnormalities (CNAs), one of the most promin
20 pically showed different patterns of genomic copy number abnormalities (CNAs), with the CNAs acquired
21                                The number of copy number abnormalities does not correlate with the la
22 arative genomic hybridization summarizes DNA copy number abnormalities in a tumor by mapping them to
23            To determine genomewide miRNA DNA copy number abnormalities in cancer, 283 known human miR
24 describes analyses of associations of genome copy number abnormalities in ovarian cancers with clinic
25  genetic lesions affecting the same genes or copy number abnormality in different subclones.
26 yses have identified a high frequency of DNA copy-number abnormalities in ALL, but the prognostic imp
27        Finally, we identified high frequency copy number abnormalities of Dicer1, Argonaute2, and oth
28  Subsequent analysis pinpoints suspected DNA copy number abnormalities of specific samples and chromo
29                                              Copy number abnormalities play an important role in pati
30               Thus, detection and mapping of copy number abnormalities provide an approach for associ
31                                     Frequent copy number abnormalities targeting cancer-associated ge
32                                            A copy-number abnormality was identified as a predictor of
33                                        Using copy-number abnormalities, we identified a predictor of
34                                          DNA copy number abnormalities were identified using microarr
35                       More than 50 recurring copy-number abnormalities were identified, most commonly
36                      We found two classes of copy number abnormalities: whole chromosome and partial

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