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1 ion for non-malignant diseases-for umbilical cord blood transplantation.
2 CD34(+)/CD38(-)/CD90(+)/CD45RA(+) HSCs after cord blood transplantation.
3 leted bone marrow or unmanipulated umbilical cord blood transplantation.
4 s may have clinical application in marrow or cord blood transplantation.
5 given single-unit, unrelated donor umbilical cord blood transplantation.
6 gressed to phase 1 clinical trials in double cord blood transplantation.
7 d the delayed platelet engraftment following cord blood transplantation.
8 eformed DSAs on outcomes in double umbilical cord blood transplantation.
9 ot been well defined for unrelated umbilical-cord blood transplantation.
10 alignancies undergoing unrelated double unit cord blood transplantation.
11 with the Rac2 mutation underwent successful cord blood transplantation.
12 t after autologous, allogeneic and umbilical cord blood transplantation.
13 hromatic leukodystrophy treated by umbilical cord blood transplantation.
14 chronic myelogenous leukemia after umbilical cord blood transplantation.
15 phoid recovery are the 2 major challenges in cord blood transplantation.
16 to increasing homing and engraftment during cord blood transplantation.
17 nt and survival in unrelated donor umbilical cord blood transplantation.
18 nd have the potential for immunotherapy post cord blood transplantation.
19 doses restricts the usefulness of umbilical-cord-blood transplantation.
20 interaction on leukaemia-free survival after cord-blood transplantation.
21 chronic GVHD was more likely to occur after cord-blood transplantation.
22 delay of functional B cells associated with cord blood transplantations.
23 (111 patients) or single-unit (113 patients) cord-blood transplantation after a uniform myeloablative
24 rine model for human placental and umbilical cord blood transplantation and second to evaluate the en
25 nts with Hurler syndrome underwent umbilical cord blood transplantation and were evaluated at baselin
26 e marrow failure that occurs after umbilical cord blood transplantation and with aplastic anemia, res
27 rved in the setting of immune recovery after cord blood transplantation, and may be associated with i
28 ntions, such as hydroxyurea; bone marrow and cord blood transplantation; and improvements in supporti
30 chromatic leukodystrophy underwent umbilical cord blood transplantation at different stages of diseas
31 There were no differences in outcome after cord-blood transplantation between patients with one HLA
32 ce graft-versus-host disease after umbilical cord blood transplantation, but this naivety and their l
33 uence NK cell reconstitution after umbilical cord blood transplantation by accelerating the different
36 ective study of T-cell immune recovery after cord blood transplantation (CBT) in a predominantly adul
39 summarizes the current status of double-unit cord blood transplantation (CBT) to improve engraftment,
43 Delayed engraftment is a major limitation of cord blood transplantation (CBT), due in part to a defec
44 of autoimmune diseases (ADs) occurring after cord blood transplantation (CBT), we analyzed both CBT r
48 isease (SCD) receiving HLA-identical sibling cord blood transplantation (CBT, n = 96) or bone marrow
54 reactivity is favored after double umbilical cord blood transplantation (dUCBT) in which cord blood (
56 od grafts can be used as an animal model for cord blood transplantation for gene therapy studies wher
57 atching who received a single unit umbilical cord blood transplantation for non-malignant diseases re
58 HLA matching on outcomes of single umbilical-cord blood transplantations for leukaemia and myelodyspl
59 h hematological malignancies given umbilical cord blood transplantation from donors carrying a homozy
62 transplant-related mortality after umbilical-cord-blood transplantation, greater investment in large-
65 ents undergoing T-cell-depleted or unrelated cord blood transplantation have undetectable EBV-specifi
67 re similar after single-unit and double-unit cord-blood transplantation; however, a single-unit cord-
69 e the first report of a successful umbilical cord blood transplantation in 1988, there has been great
75 studies suggested that allogeneic umbilical cord blood transplantation may potentially emerge as the
76 tigations include MSC infusions in umbilical cord blood transplantation, MSC therapy for tissue regen
77 cyte antigen-matched sibling donor umbilical cord blood transplantation, or 0-2 human leukocyte antig
78 sociated with HHV-6 DNAemia were as follows: cord blood transplantation (P<0.001), conditioning regim
79 The literature shows that after umbilical cord blood transplantation the relapse rate, disease-fre
81 HHV-6B) commonly reactivates after umbilical cord blood transplantation (UCBT) and is associated with
82 ntation (MMRDT) or unrelated-donor umbilical cord blood transplantation (UCBT) are valuable treatment
83 (n = 226) or double-unit (n = 435) unrelated cord blood transplantation (UCBT) following a reduced-in
84 f the art of unrelated donor (URD) umbilical cord blood transplantation (UCBT) for the treatment of h
93 ctive single-center analysis of 50 umbilical cord blood transplantations UCBTs performed from 2001 to
95 engraftment after unrelated donor umbilical cord blood transplantation was 100% with no patient havi
97 w outlines the state of the art of umbilical cord blood transplantation, with emphasis on practical c
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