ライフサイエンスコーパス: core body temperature

1 y of mice to regulate brown fat and maintain core body temperature.

2 histology, qPCR, HPLC, LC/MS and measures of core body temperature.

3 stration are temporally linked to changes in core body temperature.

4 f acute pain and for their ability to affect core body temperature.

5 he circadian rhythms of plasma melatonin and core body temperature.

6 ge of 2.0 degrees F (1.1 degrees C) over the core body temperature.

7 be affected by both ambient temperature and core body temperature.

8 ith radiotelemetry probes for measurement of core body temperature.

9 trols had similar resting metabolic rate and core body temperature.

10 ed data loggers to obtain direct measures of core body temperature.

11 ine reduction in Ucp1-deficient mice reduces core body temperature.

12 thout altering respiratory exchange ratio or core body temperature.

13 pecific Et2 knockout mice displayed a normal core body temperature.

14 Circadian phases were derived from core body temperature.

15 y is not generally associated with a reduced core body temperature.

16 growth factor 1 (IGF-1) in the regulation of core body temperature.

17 tes, including hepatic glycogen, to maintain core body temperature.

18 d by generalized seizures caused by elevated core body temperature.

19 mmals allowing optimal spermatogenesis below core body temperature.

20 .3 degrees to 0.5 degrees C reduction of the core body temperature.

21 and apparently uncoupled from the rhythm of core-body temperature.

22 petite responses while increasing thirst and core-body temperature.

23 ignificantly reduced WAT and slightly higher core body temperatures.

24 ed as assessed by metabolic cage studies and core body temperatures.

25 displaying elevated resting heart rates and core body temperatures.

26 ln(-/-) mice were not able to maintain their core body temperature (37 degrees C) and developed hypot

27 n the nasal cavity (33-35 degrees C) than at core body temperature (37 degrees C).

28 ] vs 11.3 mM [95% CI, 9.0-14.1], p = 0.004), core body temperature (39.3 degrees C [95% CI, 39.0-39.5

29 osterone and ACTH responses, heart rate, and core body temperature after the 6th exposure in male Spr

30 d with MiniMitter transmitters that recorded core body temperature and activity (12 h LD cycle).

31 laced in a restricted feeding schedule, both core body temperature and activity entrained to the feed

32 dia was observed briefly, but only after the core body temperature and blood pressure began to decrea

33 haracterized by increases in metabolic rate, core body temperature and cardiac performance.

34 into the POA induced prolonged elevation of core body temperature and decreased respiratory exchange

35 ons does not result in arrhythmicity because core body temperature and exploratory activity rhythms p

36 ther 37 degrees C or 0.5 degrees C below the core body temperature and followed up for 1 year.

37 , GC-1--treated mice also failed to maintain core body temperature and had reduced stimulation of BAT

38 iet or LPS were both associated with reduced core body temperature and heat release.

39 We made time-series recordings of core body temperature and locomotor activity in 19 elder

40 olerance to an otherwise lethal reduction of core body temperature and metabolism.

41 olerance to an otherwise lethal reduction of core body temperature and metabolism.

42 Circadian rhythms of core body temperature and motor activity were measured i

43 the pharmacological activity of improgan on core body temperature and nociceptive (tail flick) respo

44 enditure and impairment in maintaining their core body temperature and not because of hyperphagia, de

45 er, mortality can be minimized by monitoring core body temperature and preventing MA-induced hyperthe

46 n the brain are typically increased over the core body temperature and the jugular bulb temperatures.

47 try devices enabled real-time acquisition of core body temperatures and changes in heart rates and el

48 significant phase-delay in their rhythms of core-body temperature and activity compared with patient

49 movement (REM) latency, increased nocturnal core body temperature, and abnormal hormone secretion pa

50 ia, anxiolytic response, locomotor activity, core body temperature, and blood ethanol concentration,

51 e endogenous circadian rhythms of melatonin, core body temperature, and cortisol in healthy young and

52 daily rhythms in blood pressure, heart rate, core body temperature, and spontaneous physical and neur

53 ammals, testicular temperature is lower than core body temperature, and the vulnerable nature of sper

54 Women had a mean core body temperature approximately 0.23 degrees C great

55 monitoring the rate of O(2) consumption and core body temperature as indicators of torpor.

56 pid droplets in BAT and fail to defend their core body temperature at 4 degrees C, despite elevated s

57 determine the effects of IV acetaminophen on core body temperature, blood pressure, and heart rate in

58 Alterations in core body temperature, bronchial constriction, plasma hi

59 r data indicate that histamine modulates the core body temperature by acting at two distinct populati

60 l cardiac rhythm at baseline, but increasing core body temperature by as little as 3 degrees C causes

61 ch play a critical role in the regulation of core body temperature (CBT) and energy balance.

62 Mammals normally maintain their core body temperature (CBT) despite changes in environme

63 testosterone on the circadian regulation of core body temperature (CBT) in adulthood.

64 Humans' core body temperature (CBT) is strictly controlled withi

65 ts of FEV(1), FEVC, PEF, blood cortisol, and core body temperature (CBT) were performed every 2 h.

66 e responses (APRs) that include increases in core body temperature (CBT), increases in hypothalamic-p

67 Core body temperature (CBT), salivary melatonin, subject

68 tive function requiring coordination between core body temperature (CBT), the central nervous system,

69 e metabolic signal insulin in the control of core body temperature (CBT).

70 e and amplitude of the rhythms of melatonin, core body temperature, cortisol, alertness, performance

71 Mean (+/-SE) daily core body temperature did not differ significantly betwe

72 ly, implant recipients demonstrated elevated core body temperature during cold challenges, enhanced r

73 lower exercise capacity, failure to maintain core body temperature during cold stress, and reduced ab

74 eotherms use thermogenesis to maintain their core body temperature, ensuring that cellular functions

75 Patients who dialyzed at 0.5 degrees C below core body temperature exhibited complete protection agai

76 These mice maintain a lower core body temperature, fail to respond to a cold challen

77 in a novel physiological pathway regulating core body temperature, feeding behavior, and obesity in

78 ycles, hormone release, bioluminescence, and core body temperature fluctuations.

79 Reduction of core body temperature has been proposed to contribute to

80 Reserpine pre-treatment caused reductions in core body temperature; heating the rats to normal body t

81 from the WT, but they showed an increase in core body temperature in anticipation to the meal time s

82 To maintain core body temperature in cold conditions, mammals activa

83 he compounds disclosed herein do not elevate core body temperature in preclinical models and only par

84 the in vivo release of histamine and drop in core body temperature in vivo using a MC-dependent model

85 ses in energy expenditure and maintenance of core body temperature in WT and FL-PGC-1alpha(-/-) mice.

86 e transponder microchips, we showed that the core body temperature increased approximately 2 degrees

87 ng PPE was 65.7 (13.5) minutes; and the mean core body temperature increased by 0.46 degrees C (0.20

88 Core body temperature is normally tightly regulated by a

89 Core body temperature is normally tightly regulated to w

90 (-8), 3 h before (-3) or 3 h after (+3) the core body temperature minimum (CBTmin) measured on the b

91 s centred prior to the critical phase at the core body temperature minimum, phase advances occurred w

92 a were aligned according to circadian phase (core body temperature minimum; CBTmin) and averaged.

93 logic monitoring of blood pressure, EEG, and core body temperature monitoring and intermittent arteri

94 c anaphylaxis, the symptoms and decreases in core body temperature observed in wild-type mice were re

95 Four HCWs reached or exceeded a mean core body temperature of >/= 38.5 degrees C.

96 ol) mice entered deep torpor, with a minimum core body temperature of 24 degrees C, 2 degrees C above

97 atients randomized to hypothermia achieved a core body temperature of 34.7 degrees C before reperfusi

98 mes, defined as the time required to reach a core body temperature of 38.5 degrees C, and cardiovascu

99 Core body temperature of Mc4r-null mice was normal, and

100 Core body temperature of PVG rats was maintained at eith

101 ates and pneumatic pressures and maintaining core body temperature, optimal patient outcomes can be a

102 als also exhibited no significant changes in core body temperature or cardiovascular rhythm, whereas

103 The mean increase in core body temperature over time (from baseline to 150 mi

104 Anaphylactic symptom scores, core body temperatures, plasma histamine levels, basophi

105 Thus, modest, sustained reduction of core body temperature prolonged life span independent of

106 average brain temperature increase over the core body temperature ranged from -0.5 degrees to 3.8 de

107 othermy wherein metabolic rates are reduced, core body temperatures reach ambient levels, and key phy

108 We studied the activity and core-body temperature rhythms in a cohort of 38 male pat

109 enes to determine whether differences in the core body temperature set point affect the regulation of

110 A lower core body temperature set point has been suggested to be

111 nd nocturnal periods of circadian rhythms in core body temperature, sleepiness, power in the theta ba

112 ed the telemetric monitoring of activity and core body temperature (T(b)) and bilaterally implanted w

113 We measured core body temperature (T(b)) daily rhythms of Cape groun

114 milar cardiovascular changes and increase in core body temperature (T(co)) can be elicited.

115 es are scarce, endothermic animals can lower core body temperature (Tb).

116 Defense of core body temperature (Tc) can be energetically costly;

117 i.c.v.) suppressed LPS-induced increases in core body temperature (Tc), whereas a lower dose (300 ng

118 meters studied provided a closer estimate of core body temperature than equilibrated rectal temperatu

119 , 2, and 3 had significantly (p < .05) lower core body temperatures than animals that received no tre

120 e ectothermic strategy, maintaining elevated core body temperatures that presumably confer physiologi

121 mild heat treatment that temporarily raised core body temperature to approximately 39.5 degrees C.

122 anticipatory locomotor activity and rise in core body temperature under the influence of the FEO.

123 Core body temperature was 1 masculineC higher after high

124 Core body temperature was controlled using an intravascu

125 Core body temperature was measured by using ingested tel

126 Core body temperature was monitored with a rectal thermo

127 Core body temperature was not significantly altered in t

128 Core body temperature was recorded continuously for 10 d

129 Core body temperature was reduced in the calorie restric

130 emeters were implanted in 1-year-old GS, and core body temperature was remotely monitored.

131 Diurnal patterns of activity and core body temperature were progressively disrupted in R6

132 Core body temperatures were measured through radioteleme

133 Jugular vein and core body temperatures were similar.

134 Transgenic mice are unable to maintain a core body temperature when placed in a cold environment,

135 to maintain elevated energy expenditure and core body temperature when subjected to hypercaloric die

136 Lowering core body temperature with cold saline infusion and cool

137 investigated the effects a lower ambient or core body temperature would have on damage to striatal d