ライフサイエンスコーパス: core temperature

1 hetic nerve activity to CVA are dependent on core temperature.

2 bo trial, despite the attainment of a higher core temperature.

3 inhibition of motor activity and lowering of core temperature.

4 mice while monitoring drinking behaviour and core temperature.

5 skin blood flow (dry heat loss) for a given core temperature.

6 n, roughly in proportion to the elevation in core temperature.

7 ronary vasospasm, arrhythmias, and increased core temperature.

8 f Fe-rich planetary cores could overestimate core temperature.

9 ac arrest and resuscitation without changing core temperature.

10 ctivation of thermogenesis to maintain their core temperature.

11 leep latency with a simultaneous decrease in core temperature.

12 ensitive to central BDNF-induced increase in core temperature.

13 nnection between cardiovascular function and core temperature.

14 ed to absolute or delta changes in muscle or core temperature.

15 ehavioral activation is also known to change core temperature.

16 mone, and norepinephrine concentrations, and core temperatures.

17 be associated with the lowering of nocturnal core temperatures.

18 % VO2, peak for a period sufficient to raise core temperature 0.8 degrees C.

19 cited significant increases from baseline in core temperature (+1.3+/-0.3 degrees C), locomotion (+50

20 icrog, ivt) induced significant decreases in core temperature (-1.8 degrees C) 3-30 min following inj

21 Core temperature 24-h profiles did not differ significan

22 lar [ivt]) elicited significant decreases in core temperature 3-30 min following injection with a max

23 e, the induction of therapeutic hypothermia (core temperature 32 degrees C-35 degrees C) has been sho

24 kPa), PaCO2 40 +/- 5 torr (5 +/- 1 kPa), and core temperature 33 degrees C +/- 1 degrees C.

25 with or without endovascular cooling (target core temperature 33 degrees C).

26 , "hypothermia" (standard resuscitation, but core temperature 34 degrees C), or "combi" (hyperoxia pl

27 hypothermic response in 62% (13/21) of mice (core temperature: -4.6 +/- 0.5 degrees C, P = 0.001 vs.

28 The neuromuscular and body-core temperature abnormalities, which were otherwise mil

29 he primary outcome was time-weighted average core temperature adjusted for baseline temperature.

30 Mean core temperature after surgery was lower in the hypother

31 ts with SAD, (1) the reductions in nocturnal core temperatures also were correlated with the reductio

32 d not find a correlation between the EEG and core temperature alterations.

33 study was to determine if increasing murine core temperature altered cytokine expression and improve

34 Delta increase in core temperature and absolute core temperature in HOT we

35 The strongest relationship between core temperature and behavioral activation occurred at 1

36 on ('heat therapy') results in elevations in core temperature and changes in cardiovascular haemodyna

37 systems that regulate metabolism, including core temperature and energy balance, we examined the eff

38 Methamphetamine (METH) changes core temperature and induces behavioral activation.

39 the DMH prior to MDMA prevented increases in core temperature and locomotion and attenuated increases

40 ng the high-dose METH treatment were maximum core temperature and minimum chamber temperature.

41 We recorded EEG, core temperature and motor activity before and after exp

42 ding human beings, to maintain a stable body core temperature and respond to the ambient environment.

43 on the theoretical relationship between the core temperature and surface temperature distribution on

44 The standard-of-care is to monitor core temperature and to maintain normothermia during gen

45 38 degrees C, 35 degrees C, or 32 degrees C core temperature and underwent 4 hours of hemorrhage (re

46 Core temperature and vital signs were measured at baseli

47 High calcium diets also increase core temperature and white adipose tissue uncoupling pro

48 nment that poses a risk of increases in body core temperature and/or perceived discomfort.

49 mergency department systolic blood pressure, core temperature, and greater metabolic acidosis (analys

50 inuous, real-time measurement of heart rate, core temperature, and mobility.

51 hase acutely increased sleep, decreased body core temperature, and prevented high-fat diet-induced we

52 a significant affect on MDMA neurotoxicity, core temperature, and thermoregulation in rats.

53 ere normotheric and heat stressed (change in core temperature approximately 0.75 degrees C).

54 toxin-induced alveolitis, coexposure to FRT (core temperature approximately 39.5 degrees C) doubled t

55 then exposed to febrile range hyperthermia (core temperature, approximately 39.5 degrees C), Hsp70 l

56 ythms of sleep, melatonin secretion and body core temperature are thought to be generated by the supr

57 15 animals, physiologic data (urine output, core temperature, arterial pressure, heart rate, cardiac

58 demonstrate good agreement of the predicted core temperature as a function of time with actual core

59 handling and related to an increase in body core temperature as a result of higher levels of uncoupl

60 f b.c.c.-iron able to model its behaviour at core temperatures as well as pressures, using ab initio

61 ed mouse model, we showed that maintaining a core temperature at FRH (39 degrees C to 40 degrees C) r

62 ld intravenous Hartmann's solution decreases core temperature at hospital arrival and decreases the t

63 ial head injury less than 10 days earlier; a core temperature at least 36 degrees C; and an abnormal

64 umption, carbon dioxide production, and body core temperature before, during, and after hydrogen sele

65 ined persistent tachycardia, well before the core temperature begins to rise.

66 All achieved a core temperature below 34 degrees C (mean target tempera

67 was measured by radiotelemetry monitoring of core temperature, brown adipose tissue (BAT) temperature

68 FO did not significantly alter core temperature but decreased the postoperative rise in

69 mSMEI had seizures induced by elevated body core temperature but wild-type mice were unaffected.

70 on indeed becomes entropically stabilized at core temperatures, but in its pure state h.c.p.-iron sti

71 The intervention decreased mean core temperature by 1.20 degrees C (95% CI, -1.33 degree

72 Although use of prehospital cooling reduced core temperature by hospital arrival and reduced the tim

73 Regulated hypothermia produces a decrease in core temperature by lowering the brain's temperature set

74 In contrast, forced hypothermia lowers core temperature by overwhelming the body's capacity to

75 The daytime regulation of core temperature by serotonin 1A receptors appears norma

76 ther lower the core temperature or raise the core temperature by, respectively, producing negative he

77 of energy expenditure, substrate oxidation, core temperature, cold and hunger scores, and plasma par

78 No differences in substrate oxidation, core temperature, cold and hunger scores, or plasma para

79 oninvasive cutaneous device to monitor their core temperature continuously.

80 owed temperature-sensing capsules to measure core temperatures continuously for >/=48 h and kept acti

81 Survival, core temperature, cytokine production, and bacterial cle

82 icity and did so independently of effects on core temperature, DA transporter function, or METH brain

83 Using this model we analysed core temperature data from an experiment designed to exh

84 ker we propose a stochastic-dynamic model of core temperature data that contains both stochastic and

85 uch as plasma cortisol, plasma melatonin and core temperature data, currently methods are not availab

86 Core temperature did not differ significantly between th

87 hese findings call for careful monitoring of core temperature during anesthesia in laboratory animals

88 active compounds was obtained with the lower core temperature during boiling, as well as higher tempe

89 ogen TNF-alpha is regulated by increments in core temperature during fever, generating an enhanced ea

90 l affective disorder, light treatment lowers core temperature during sleep in proportion to its antid

91 The regulation of the level of core temperature during sleep is linked with a proportio

92 r are primarily responsible for the observed core temperature effects during the initial post-treatme

93 mice revealed that systemic thermal therapy (core temperature elevated to 39.5 degrees C +/- 0.5 degr

94 RPV1 antagonist significantly increased body core temperature following oral administration in rodent

95 degrees C rather than 23 degrees C increased core temperature from 36.5 to 37.5 degrees C to 39.2 to

96 , increased invasiveness, and require stable core temperatures, good operator technique, and a compet

97 Forty critically ill adults with fever (core temperature, >/= 38.3 degrees C).

98 Core temperature, heart rate, mean arterial pressure and

99 Core temperature, hemodynamics, serum glucose and electr

100 Core temperature, however, decreased only in animals rec

101 FEN at 30 degrees C displayed a significant core temperature hyperthermia for 4 h after the first dr

102 with a peak low of 33.8 degrees C, and this core temperature hypothermia lasted for 20 h after FEN a

103 neurotoxicity in rats that display either a core temperature hypothermia or hyperthermia, although h

104 FEN-treated rats at 24 degrees C displayed a core temperature hypothermia with a peak low of 33.8 deg

105 , neurotoxicity was seen and correlated with core temperature in all regions examined.

106 ta increase in core temperature and absolute core temperature in HOT were correlated to total game di

107 es in ambient temperature produce changes in core temperature in MDMA-treated rats, but the same chan

108 his study, we show that passively increasing core temperature in mice from the basal (36.5 to 37.5 de

109 n selective manner and to elevate rectal and core temperature in rats without accompanying cardiovasc

110 -HT efflux, neuromuscular activity, and body-core temperature in response to challenge injection of c

111 thermometry is commonly used to measure body core temperature in rodents because of its ease of use.

112 nsitivity (P<0.05), and transiently elevated core temperature in the transgenic mice, but was without

113 dynamics after dopamine infusions at various core temperatures in a pig model of surface cooling and

114 ic pathways allow organisms to protect their core temperatures in response to cold exposure.

115 iencing whole body contractions and elevated core temperatures in response to isoflurane exposure or

116 This study compares core temperatures in swine after central catheter infusi

117 gh thermal conductivity) requires high outer-core temperatures in the early Earth that complicate mod

118 After fire simulation training, core temperature increased (1.0+/-0.1 degrees C) and wei

119 ) healthy adults were passively heated until core temperature increased by 1.5 degrees C.

120 ine, 12 subjects were passively heated until core temperature increased by approximately 0.6 degrees

121 n after treatment for all doses and preceded core temperature increases; the onset of METH-induced hy

122 thermal niche through their interaction with core temperature, insulation, and environmental conditio

123 Core temperature is reduced spontaneously after asphyxia

124 Of the three rhythms core temperature is the only reliable variable that can

125 Spots for detecting WHO-defined hypothermia (core temperature <35.5 degrees C or peripheral temperatu

126 s procedure affects brain, muscle, skin, and core temperatures measured with chronically implanted th

127 Core temperature measurement using a pulmonary artery (P

128 emperature as a function of time with actual core temperature measurement using an embedded thermocou

129 udies of DAT function were selected based on core temperature measurements in animals exposed to METH

130 oral and core equivalence modes) against PA core temperature measurements to determine which method

131 Continuous telemetric core temperature measurements were made during a 7h test

132 with both the levels of the previous nights' core temperature minima (P=.002) and the amounts of noct

133 lated by both melatonin and the level of the core temperature minimum.

134 rsal medulla produces an increase in BAT and core temperature more than 300% greater than TRH alone (

135 hypothermia (13+/-3% vs. 7+/-1% decrease in core temperature, n=6), and peritoneal exudate mediator

136 nsumption, resting metabolic rates, and body core temperatures occurred, but only after 7 days of tre

137 hetic sodium pentobarbitone, a small rise in core temperature occurs following ketamine.

138 lantic salmon were either super-chilled to a core temperature of -1.5 degrees C or directly chilled o

139 This resulted in a mean decrease in core temperature of 1.4 degrees C compared with 0.2 degr

140 een cooled and underwent OLT with the median core temperature of 33.4 degrees C (92.1 degrees F) (ran

141 Hypothermia with a core temperature of 35 degrees C was instituted for 24 h

142 compared to SAL-treated groups, with a peak core temperature of 38.6 degrees C.

143 We previously reported that increasing core temperature of bacterial endotoxin (LPS)-challenged

144 Furthermore, increases in core temperature of MDMA-treated animals increase neurot

145 changes in ambient temperature do not affect core temperature of saline-treated animals.

146 ass, administration of pre-CPB vasopressors, core temperature on CPB, pre- and post-CPB hematocrit, t

147 ute infections, but the effects of increased core temperature on host defenses are poorly understood.

148 temperature was adjusted to either lower the core temperature or raise the core temperature by, respe

149 and exhibited a 0.81 degrees C lower initial core temperature (P<0.0005), an approximately twofold in

150 At the end of the TT in the heat, both core temperature (pla 39.7 +/- 0.3 degrees C, bup 40.0 +

151 f engineering systems where the time-varying core temperature plays a key role.

152 s also disrupted after CNO triggering of Di; core temperatures plummeted, then recovered.

153 We constructed daily, 24-h core temperature profiles for analysis.

154 ionship between (1) endogenous melatonin and core temperature profiles, (2) the proportional control

155 ) and were highly correlated with changes in core temperature (r = .9, P < .001).

156 In the prospectively cooled patients, core temperature reached target range less than or equal

157 l control cases that had not had activity or core temperature recordings previously.

158 Normal core temperature (rectal) was regulated in all animals.

159 ividuals with function-altering mutations in core temperature-regulating genes to determine whether d

160 at 20 and 22 degrees C showed a hypothermic core temperature response.

161 ed schedule, the endogenous component of the core temperature rhythm of subjects who were exposed to

162 In contrast, the core temperature rhythm of subjects who were not exposed

163 Chronic stress lowered the amplitude of core temperature rhythms, and lesions of the pPVTh block

164 and is a nexus that differentially regulates core temperature rhythms/HPA activity/specific white adi

165 overage in vivo but caused increases in body core temperature, suggesting that peripheral restriction

166 t of thermoregulation and the maintenance of core temperature, sympathetically-mediated control of th

167 side, in anaesthetized male Wistar rats at a core temperature (T(b)) of 37 degrees C, during acute se

168 -004, an EP1 receptor agonist, increased the core temperature (T(c)) in a dose-dependent manner (1.6+

169 e animals did not develop fever, whereas the core temperature (T(c)) of CVF vehicle-treated controls

170 ent temperature of 33 degrees C, the average core temperature (T(CORE)) of ovariectomized (OVX) contr

171 brile response without affecting basal body (core) temperature (T(c)).

172 or the EP3 receptors and measured changes in core temperature (Tc) by using telemetry.

173 tress induced a transient hypothermia, where core temperature (Tc) declined immediately and then rose

174 The mean drop in core temperature (Tc) following hemorrhage was 1.5 degre

175 Heating stopped when maximum core temperature (Tc) of 42.4 degrees C was attained (Tc

176 Core temperature (Tc), heart rate (HR), activity, and bl

177 To this end, the core temperatures (Tc) of COX-1 and COX-2 gene-ablated m

178 eas the increases in preoptic PGE2 and body (core) temperature (Tc) following the intravenous (i.v.)

179 CO2 (+0.7%), core temperature (Tcore, +0.5 degrees C) and HR (+54 bpm

180 Core temperatures (Tcs) were recorded using thermocouple

181 sized that PFO+ subjects would have a higher core temperature than PFO- subjects due, in part, to abs

182 hypothesis that obese individuals have lower core temperatures than those in normal-weight individual

183 Hom mice maintain higher core temperatures than WT in the home cage, have chronic

184 These data suggest that the increase in core temperature that occurs during bacterial infections

185 at temperatures at the body surface than at core temperature, thereby favoring superficial hemostasi

186 If heat shock models elevated core temperature, these results suggest that fever may p

187 bient temperature of 4 degrees C, decreasing core temperature to 30 degrees C within 120 min.

188 used cooling blankets to lower the patients' core temperature to 32-33 degrees C.

189 Heat stress increased core temperature to a similar extent in both groups (Y:

190 , but is dependent on an induced decrease in core temperature to produce the effect.

191 The core temperature triggering each response defines its ac

192 The core temperature triggering each response defines its ac

193 , FGF21 levels were related to the change in core temperature upon acute cold exposure, indicating a

194 n blood flow via laser-Doppler flowmetry and core temperature via ingestible telemetric pill were mea

195 The mean (+/- SD) final intraoperative core temperature was 34.7 +/- 0.6 degrees C in the hypot

196 Average patient core temperature was 36.7 degrees C (36-37.3 degrees C),

197 In studies employing a telemetry apparatus, core temperature was also increased by CRF(1-41) (1 nmol

198 Core temperature was analysed for parametric, circadian

199 e was controlled to +/-0.5 degrees C and rat core temperature was continually measured using a non-in

200 when a correction factor for differences in core temperature was included in the calculation.

201 Core temperature was measured by a rectal probe attached

202 Core temperature was reduced to 32 degrees C-33 degrees

203 In all pups, core temperature was similar to chamber temperature, wit

204 serotonin 1A receptor activation also lower core temperature, we investigated the relationship betwe

205 Muscle and core temperatures were approximately 1 degrees C higher

206 For all groups, core temperatures were monitored rectally every 30 min f

207 ynamic data, catecholamine requirements, and core temperatures were recorded at 10-min intervals; blo

208 Simultaneous core temperatures were recorded from 1 to 5 days.

209 mitted to the hospital for a night of sleep (core temperatures were recorded), followed by infusions

210 ted with significant reductions in nocturnal core temperatures, which were correlated with similarly

211 as feasible and safe, and it rapidly reduced core temperature with minor reperfusion delay.

212 s an early myocardial depression and rise in core temperature, yet reduced oxygen consumption and res