ライフサイエンスコーパス: corneal edema

1 Imaging are useful tools in cases with dense corneal edema.

2 sed to calculate the cumulative incidence of corneal edema.

3 de the cornea and regulate the resolution of corneal edema.

4 No eyes had clinically apparent central corneal edema.

5 njury model and significantly diminished the corneal edema.

6 impflug imaging were done in view of diffuse corneal edema.

7 be necessary to minimize the risk of chronic corneal edema.

8 ssions were estimated to compare the risk of corneal edema.

9 who underwent CXL treatment with subsequent corneal edema.

10 is and fibrosis, corneal epitheliopathy, and corneal edema.

11 infection, concurrent with the resolution of corneal edema.

12 o perform DSEK in patients with pseudophakic corneal edema.

13 rvention reduced the likelihood of permanent corneal edema (0.5 days [from diagnosis of migration to

14 h incidence of persistent diplopia (12%) and corneal edema (20%), although half of the corneal edema

15 were infection of the right eye, initial VA, corneal edema, a hypopyon larger than 1.5 mm, detection

16 gnificant differences between PPC and CCC in corneal edema, AC inflammatory reaction, capsular fibros

17 endothelial damage with visually significant corneal edema after CXL treatment.

18 Given reports of corneal edema after endothelial exposure to platelet act

19 main outcome measure: The resolution of corneal edema after surgery.

20 erior chamber cell and flare with or without corneal edema after the initial resolution of perioperat

21 corneal thickness due to delayed drainage of corneal edema and a trend towards prolonged corneal opac

22 Fuchs' endothelial disease and pseudophakic corneal edema and for high-volume surgeons in all diseas

23 crl(fl/fl)/CAGGCre-ER mice rapidly developed corneal edema and inflammation that was preceded by and

24 visual acuity ranging from 20/100 to 20/400, corneal edema and opacity, anterior chamber reaction, or

25 Histologic sections showed more corneal edema and polymorphonuclear leukocyte (PMN) infi

26 ered significantly during periods of maximal corneal edema and stromal disease.

27 Corneal edema and subsequent vision loss ensues when end

28 options in blinding diseases associated with corneal edema and transparency loss.

29 ectedly beneficial role in the regulation of corneal edema and transparency.

30 ll monolayer of the cornea and can result in corneal edema and vision loss in severe cases.

31 Ultimately, these changes lead to corneal edema and vision loss.

32 disruption of the corneal epithelial layer, corneal edema, and a significant decline in conjuctival

33 tion at 10(10) vp and moderate inflammation, corneal edema, and increased intraocular pressure at 10(

34 and photorefractive keratectomy for ectasia, corneal edema, and infectious keratitis.

35 pathology inclusive of epithelial erosions, corneal edema, and neutrophil infiltration.

36 Performing earlier DSEK for pseudophakic corneal edema appears to be associated with improved vis

37 Longstanding corneal edema can lead to anterior stromal scarring that

38 nd corneal edema (20%), although half of the corneal edema cases were likely due to pre-existing caus

39 tion of keratoconus, characterized by marked corneal edema caused by a break in Descemet membrane, al

40 Corneal edema cleared completely in 1 week.

41 In 14 patients, corneal edema developed.

42 mong the 14 patients with corneal edema, the corneal edema did not resolve in 10 patients (71%), 6 (4

43 if-IOL (case 2) in order to treat secondary corneal edema due to pseudophakic bullous keratopathy.

44 ens have seen a reduction in the duration of corneal edema during acute hydrops, and have improved th

45 NBCe1, or CA activity was disrupted in vivo, corneal edema ensued and was associated with significant

46 B/c mice demonstrated both PMN migration and corneal edema, eyes of infected C57BL/6 mice failed to s

47 120 eyes of patients who underwent DSEK for corneal edema following cataract surgery (CE); 87% of ey

48 with bilateral decreased vision secondary to corneal edema from endothelial dysfunction underwent Des

49 Corneal edema improved in 4 patients and resolved in 1 p

50 ften secondary to long-standing preoperative corneal edema in 14 of 178 eyes (7.9%), or (partial) gra

51 Fuchs' endothelial disease, or pseudophakic corneal edema in a 7-year period from 1999 in (1) high-v

52 Amantadine increases the risk of corneal edema in a dose-dependent manner.

53 This paper reviews the pathogenesis of corneal edema in Fuchs' dystrophy, the preoperative and

54 The incidence of corneal edema in the Parkinson group (123 patients; 1.50

55 The incidence ratio for corneal edema in the Parkinson group vs the controls was

56 and CA activity, disruption of which causes corneal edema in vivo and indicates that facilitation of

57 Inferior corneal edema, in particular, should alert the practitio

58 is related to the risk of complications like corneal edema, intraocular pressure spikes, cystoid macu

59 Corneal edema is a significant component of the various

60 Corneal edema is common in the setting of retained lens

61 Fifty-six percent of eyes had associated corneal edema, most often located inferiorly.

62 ll corneal transplants performed), a primary corneal edema mostly affecting elderly individuals; kera

63 rative AEs included iritis (n = 330, 1.53%), corneal edema (n = 110, 0.53%), and retinal tear or deta

64 (n = 5), corneal epithelial defect (n = 4), corneal edema (n = 3), and glaucoma (n = 1).

65 Complications included transient corneal edema (n = 4) and transient anterior chamber inf

66 orneal graft rejection accompanied by severe corneal edema, neovascularization and opacity that occur

67 angiogenesis was notable for the absence of corneal edema or substantial inflammation.

68 ings of keratic precipitates with or without corneal edema, or anterior chamber cell and flare with o

69 a surgery or medication, refractive surgery, corneal edema, or corneal dystrophy, IOP and CCT reading

70 y Fuchs dystrophy or pseudophakic or aphakic corneal edema (PACE).

71 ferential diagnosis in cases with late-onset corneal edema post-cataract surgery.

72 Since corneal edema resulting from scrape injury was similar,

73 neal graft failure was defined as persistent corneal edema resulting in irreversible loss of optical

74 s included conjunctival injection, chemosis, corneal edema, severe iritis, fibrin accumulation, and a

75 xtremely high intraocular pressure (IOP) and corneal edema similar to toxic anterior segment syndrome

76 Five patients had corneal edema that did not resolve following fragment ex

77 Among the 14 patients with corneal edema, the corneal edema did not resolve in 10 p

78 non-use of amantadine, the hazard ratio for corneal edema was 1.79 times higher in the amantadine su

79 se revealed that the 30-day hazard ratio for corneal edema was 2.05 higher in patients given moderate

80 Postoperative corneal edema was identified in 10 (2.9%) of 350 patient

81 Stromal disease with corneal edema was induced in rabbits by intrastromal inj

82 Corneal edema was induced, and the percent recovery per

83 Significant corneal edema was not observed.

84 Fuchs' dystrophy or pseudophakic or aphakic corneal edema, were enrolled by 105 surgeons from 80 cli

85 l epithelium acts intracellularly to promote corneal edema, whereas 12-HETrE acts in a paracrine mann

86 of 8 eyes with reinversion had postoperative corneal edema, which resolved at 48 hours.