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1 differentiation of the neural crest derived corneal endothelial cell.
2 ike coupling mechanisms play roles in CCE in corneal endothelial cells.
3 taken to investigate the mechanism of CCE in corneal endothelial cells.
4 AQP1 does not contribute to PCO(2) in corneal endothelial cells.
5 hesion molecule, N-cadherin, is expressed by corneal endothelial cells.
6 soforms expressed in primary cultures of rat corneal endothelial cells.
7 ial and possibly other cellular functions in corneal endothelial cells.
8 these cells often fail to differentiate into corneal endothelial cells.
9 a, -eta, -gamma, and -theta were detected in corneal endothelial cells.
10 n important role in Fas ligand expression in corneal endothelial cells.
11 us humor did not promote apoptosis of murine corneal endothelial cells.
12 aqueous humor suppress S-phase entry of rat corneal endothelial cells.
13 n aqueous humor on DNA synthesis in cultured corneal endothelial cells.
14 n corneal epithelial cells, keratocytes, and corneal endothelial cells.
15 tein which inhibits the NK-mediated lysis of corneal endothelial cells.
16 ittle ALDH3 is present in the keratocytes or corneal endothelial cells.
17 y conjunctiva, corneal epithelial cells, and corneal endothelial cells.
18 rbol esters on the function and structure of corneal endothelial cells.
19 port the adhesion and proliferation of human corneal endothelial cells.
20 in or a matrix elaborated by cultured bovine corneal endothelial cells.
21 cell proliferation and cell shape changes in corneal endothelial cells.
22 ized FN and to cultured monolayers of bovine corneal endothelial cells.
23 only a potent mitogen, it is a modulator for corneal endothelial cells.
24 glandin E2 synthesis is increased in injured corneal endothelial cells.
25 ranscriptional control of type I collagen in corneal endothelial cells.
26 a highly effective, novel carrier for human corneal endothelial cells.
27 r integrity in monolayers of cultured bovine corneal endothelial cells.
28 P) was used to transduce cryopreserved human corneal endothelial cells.
29 oxic to cryopreserved human primary cultured corneal endothelial cells.
30 t is undetectable on stromal fibroblasts and corneal endothelial cells.
31 g-term viability of the neural crest derived corneal endothelial cells.
32 sease involving metaplasia and overgrowth of corneal endothelial cells.
33 lts in replication in nonproliferating human corneal endothelial cells.
34 /gld mice did not induce apoptosis of BALB/c corneal endothelial cells.
36 alpha 2(I) collagen RNA obtained from normal corneal endothelial cells and from modulated corneal end
37 ed for overexpression of E2F2 in transfected corneal endothelial cells and permitted calculation of t
38 ggest that type I collagen is synthesized in corneal endothelial cells and that such undesired expres
39 Two forms of cyclooxygenase are present in corneal endothelial cells, and pharmacologic studies ind
42 It is possible that in situ human and rabbit corneal endothelial cells are arrested at different poin
49 not induce acute significant cytotoxicity in corneal endothelial cells at concentrations up to 100 mi
50 lated onto the matrices elaborated by bovine corneal endothelial cells attached to the culture dish a
51 ssion levels in confluent cultures of bovine corneal endothelial cells (BCECs) affects membrane PCO(2
52 ence of Ca(2+) stores in lysosomes of bovine corneal endothelial cells (BCECs) and examines their int
53 inhibition of CA activity in cultured bovine corneal endothelial cells (BCECs) by dorzolamide, a topi
54 LC4A11 expression in cultured primary bovine corneal endothelial cells (BCECs) was determined by semi
56 ymal transition and preserve the function of corneal endothelial cells both during ex vivo culture an
57 sage (<4) stromal cells and to low levels in corneal endothelial cells but not in corneal epithelial
58 ness of cryopreserved human primary cultured corneal endothelial cells by characterizing their morpho
59 that WNT10B promotes proliferation in human corneal endothelial cells by simultaneously utilizing bo
64 e current study, we performed in vivo rabbit corneal endothelial cell (CEC) injury via CEC scraping,
65 ated cells with pseudopodia is observed when corneal endothelial cells (CECs) are simultaneously trea
67 mation (EMT), in which the contact-inhibited corneal endothelial cells (CECs) become multilayers of s
69 othelial corneal dystrophy (FECD) and normal corneal endothelial cells (CECs) to oxidative stress, an
80 owed by ciliary body, lens epithelial cells, corneal endothelial cells, conjunctiva, retina, and corn
83 0.05; 95% CI, -0.01 to 0.12; P = 0.10), and corneal endothelial cell count (WMD, 73.39; 95% CI, -6.2
85 rm safety, particularly in eyes with reduced corneal endothelial cell counts from prior intraocular s
87 enhanced the proliferative response of human corneal endothelial cell cultures to endothelial cell gr
89 talloproteinase activity in confluent bovine corneal endothelial cells decreased the level of endothe
90 n the subjects who underwent trabeculectomy, corneal endothelial cell density (ECD) significantly dec
95 grafts did not recover clarity in vivo, and corneal endothelial cells did not proliferate in organ c
101 ct intraocular tumors, 5) CD4(+) T cells and corneal endothelial cells express TRAIL and induce apopt
104 reatments for reversible blindness caused by corneal endothelial cell failure involve replacing the f
109 stance (rho) was measured in cultured bovine corneal endothelial cells grown on permeable substrates
110 The use of substratum elaborated by bovine corneal endothelial cells has proved useful in the prepa
115 ative DNA damage increases with age in human corneal endothelial cells (HCECs) and contributes to the
121 and COL4A3 mRNA expression in primary human corneal endothelial cells (HCEnCs) was assayed in both P
122 corneal fibroblasts, and immortalized human corneal endothelial cells (HCEnCs), and in vivo intralam
129 roteins in donor corneas suggests that human corneal endothelial cells in vivo have not exited the ce
131 in G(0)-phase synchronized subconfluent rat corneal endothelial cells incubated for 24 hours in 10%
132 ption factor E2F2 in nonproliferating rabbit corneal endothelial cells induces cell cycle progression
133 emove corneal epithelial cells, conjunctiva, corneal endothelial cells, iris, ciliary body, lens epit
134 we show that gene transfer to nonreplicating corneal endothelial cells is feasible using recombinant
135 PK signaling pathway was demonstrated in the corneal endothelial cells isolated from the NF-1 eyes.
136 humor suppress S-phase entry in cultured rat corneal endothelial cells, it is not known whether TGF-b
137 nd binding of RAC1 and beta-catenin in human corneal endothelial cells, leading to the activation of
138 n the eye, we employed an immortalized mouse corneal endothelial cell line (C3H3) that constitutively
140 studies, immortalized normal and FECD human corneal endothelial cell lines (HCECi and FECDi, respect
142 ent studies have demonstrated a reduction in corneal endothelial cell loss after phacoemulsification
143 operative and perioperative risk factors for corneal endothelial cell loss during cataract surgery, a
144 re and describes in detail how the degree of corneal endothelial cell loss is influenced by specific
146 erative and postoperative complications, and corneal endothelial cell loss were assessed at 6 months
150 utive expression of MHC class I molecules on corneal endothelial cells makes them potential targets f
151 luorophotometry, [protein]Ac by Lowry assay, corneal endothelial cell morphology by specular micropho
153 the coculture of lens epithelial cells with corneal endothelial cells on the proliferation of rabbit
154 h either Ia- spleen cells, Ia+ spleen cells, corneal endothelial cells, or corneal epithelial cells f
157 ally with regard to decreased keratocyte and corneal endothelial cell populations noted months to yea
158 ous studies from this laboratory showed that corneal endothelial cell proliferation ceases in neonata
163 GF-2 stimulates cell proliferation of rabbit corneal endothelial cells (rCECs) by degrading the cycli
164 .37; 95% CI, -11.88 to -0.86; P = 0.02), and corneal endothelial cell reduction (WMD, -55.43; 95% CI,
166 ranscription factor E2F2 in nonmitotic human corneal endothelial cells results in short-term expressi
167 AC priming with Ia- NZB spleen cells or NZB corneal endothelial cells results in the permanent accep
169 neal endothelial cell line and primary human corneal endothelial cells retained their characteristic
171 arkedly increased the tolerizing activity of corneal endothelial cells, so that a single dose of chol
172 atinocyte growth factors, and coculture with corneal endothelial cells stimulated proliferation of ra
174 ing for the RIII protein was more intense in corneal endothelial cells than in limbal basal cells.
175 ive neurons and into p75(NTR)-positive human corneal endothelial cells that exhibited transendothelia
176 demonstrated that EMAP is a novel protein in corneal endothelial cells that is capable of inducing pr
177 corneal endothelial cells and from modulated corneal endothelial cells that predominantly produce typ
180 DM) helps maintain phenotype and function of corneal endothelial cells under physiological conditions
181 we found that during ex vivo culture, bovine corneal endothelial cells underwent endothelial-mesenchy
182 antagonist, vMIP II, was introduced into the corneal endothelial cells using a non-viral vector consi
183 examined the feasibility of gene transfer to corneal endothelial cells using replication-defective re
185 ton fraction of the normal and the modulated corneal endothelial cells was immunoprecipitated with PL
187 lagen RNA structures of normal and modulated corneal endothelial cells were analyzed by S1 nuclease p
195 oliferation studies, primary cultures of rat corneal endothelial cells were serum-starved for 48 hour
197 We observed LOXHD1 mRNA in cultured human corneal endothelial cells, whereas antibody staining of
198 he anterior chamber of the eye is lined with corneal endothelial cells, which are terminally differen
200 eta3 induced myofibroblast transformation of corneal endothelial cells, with formation of stress fibe
201 as found to be almost entirely restricted to corneal endothelial cells, with scattered expression in
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