ライフサイエンスコーパス: corneal epithelial cell

1 nd typical enhancers active in primary human corneal epithelial cells.

2 n/AP-1 through a novel Plk3 pathway in human corneal epithelial cells.

3 Pax6 functions directly to maintain normal, corneal epithelial cells.

4 vation of the JNK signaling pathway in human corneal epithelial cells.

5 e a useful additive for ex vivo expansion of corneal epithelial cells.

6 osis through the activation of Plk3 in human corneal epithelial cells.

7 alized the cytolytic effects of PLY on human corneal epithelial cells.

8 red to MS1 cells co-cultured with PD-L1(-/-) corneal epithelial cells.

9 rogression of healing of wounds in sheets of corneal epithelial cells.

10 f syndecan-1 ectodomains from the surface of corneal epithelial cells.

11 ly processed into antimicrobial fragments in corneal epithelial cells.

12 rease in ROS-positive cells in primary human corneal epithelial cells.

13 elationships that develop between nerves and corneal epithelial cells.

14 s regulated during cell cycle progression in corneal epithelial cells.

15 nse to pathophysiological challenge in human corneal epithelial cells.

16 and amplified HO-1 gene expression in human corneal epithelial cells.

17 concomitant MLC dephosphorylation in bovine corneal epithelial cells.

18 omotion were characterized in cultured human corneal epithelial cells.

19 stellanii trophozoites to adhere to and kill corneal epithelial cells.

20 ovine peripheral blood mononuclear cells and corneal epithelial cells.

21 t (UVB) is known to cause apoptosis in human corneal epithelial cells.

22 activation in UVB-induced apoptosis in human corneal epithelial cells.

23 ation with, invasion of, and survival within corneal epithelial cells.

24 tion of barrier function in stratified human corneal epithelial cells.

25 [3H]thymidine uptake in primary cultures of corneal epithelial cells.

26 sfect guinea pig liver transglutaminase into corneal epithelial cells.

27 is selectively expressed in differentiating corneal epithelial cells.

28 e corneal epithelium in vivo and in cultured corneal epithelial cells.

29 d lipid raft formation in occasional surface corneal epithelial cells.

30 enic mouse models and cultured primary human corneal epithelial cells.

31 migrated centripetally to differentiate into corneal epithelial cells.

32 ced in keratinocytes and to a lower level in corneal epithelial cells.

33 anical injury (debridement), and in vitro in corneal epithelial cells.

34 lation were corroborated using primary human corneal epithelial cells.

35 alt and water balance and normal function of corneal epithelial cells.

36 r p15(INK4b) has been localized in migrating corneal epithelial cells.

37 HGF modulates multiple signaling cascades in corneal epithelial cells.

38 scription complex, resulting in apoptosis of corneal epithelial cells.

39 ly expressed in surface epithelia, including corneal epithelial cells.

40 unding in cultured porcine corneas and human corneal epithelial cells.

41 ibody and NF-kB inhibitor in mouse and human corneal epithelial cells.

42 d corneas and proliferation rate in cultured corneal epithelial cells.

43 d chemokine CXCL10 and its receptor CXCR3 in corneal epithelial cells.

44 hibitory and oxidant effects of 4-HNE on the corneal epithelial cells.

45 on infected mouse corneas and human cultured corneal epithelial cells.

46 ells and proliferation of subconfluent human corneal epithelial cells.

47 in primary and telomerase-immortalized human corneal epithelial cells.

48 icated that the NC-1059 peptide did not kill corneal epithelial cells.

49 use, but failed to infect productively human corneal epithelial cells.

50 EGF-induced migration of immortalized human corneal epithelial cells.

51 ed in bactericidal lysate fractions of human corneal epithelial cells.

52 In human primary corneal epithelial cells, 24,25(OH)2D3 stimulated migrat

53 in (MBP) mediates adhesion of the amoebae to corneal epithelial cells, a key first step in the pathog

54 t of such an effect, and it suggests that in corneal epithelial cells, activation of apoptotic pathwa

55 ificant effect on the proliferation of human corneal epithelial cells after ethanol exposure.

56 t role in mediating programmed cell death of corneal epithelial cells after UV irradiation.

57 ancer of chemotherapy, effectively protected corneal epithelial cells against apoptosis by its specif

58 in human tear fluid and that it can protect corneal epithelial cells against bacterial invasion.

59 ese results suggest that ALDH3A1 may protect corneal epithelial cells against oxidative damage not on

60 Human primary corneal epithelial cells and cell line were cultured in

61 Ac resulted in increased adhesion of applied corneal epithelial cells and corneal fibroblasts.

62 rived exosomes mediate communication between corneal epithelial cells and corneal keratocytes as well

63 ILs), toward two different cell lines, human corneal epithelial cells and Escherichia coli bacterial

64 enzyme that is highly expressed in mammalian corneal epithelial cells and has been shown to protect a

65 Primary human and rabbit corneal epithelial cells and immortalized human corneal

66 wound healing was further evaluated in human corneal epithelial cells and in the corneas of wild-type

67 channel types were sought in cultured rabbit corneal epithelial cells and in the intact rat corneal e

68 Water-soluble proteins were isolated from corneal epithelial cells and keratocytes of several spec

69 duced apoptosis of T and B cells, but not of corneal epithelial cells and liver cells.

70 ting TLR4 signaling through IRF3 in resident corneal epithelial cells and macrophages and thereby mod

71 tions of CD4(+) T cells with MHC class II(+) corneal epithelial cells and macrophages in infected DC-

72 Overexpression of CTCF in corneal epithelial cells and mouse corneas significantly

73 S. aureus-induced cytokine production by corneal epithelial cells and neutrophils was also signif

74 role of CD14 in regulating LPS activation of corneal epithelial cells and Pseudomonas aeruginosa corn

75 he pro-inflammatory interleukin-8 from human corneal epithelial cells and reverses reductions in tran

76 eba interacts with a mannosylated protein on corneal epithelial cells and stimulates trophozoites to

77 bacterial adherence to the apical surface of corneal epithelial cells and suggest that alteration of

78 perties of cell surface-associated mucins in corneal epithelial cells and suggest that alterations in

79 ll-like receptor 4 (TLR4) internalization in corneal epithelial cells and that blocking with anti-CD1

80 Fewer layers of corneal epithelial cells and the absence of cytokeratin

81 oxicity on infected mouse and cultured human corneal epithelial cells and the role of PVL and antibod

82 exosome-like vesicles were observed between corneal epithelial cells and the stroma during wound hea

83 0% of the water-soluble protein of the mouse corneal epithelial cells and thus, by analogy with the a

84 Pseudomonas aeruginosa can invade corneal epithelial cells and translocates multilayered c

85 ucts were transiently transfected into human corneal epithelial cells, and activity was assessed in r

86 nstitutively expressed at high levels by the corneal epithelial cells, and at low levels by corneal C

87 estigated in cultured porcine corneas, human corneal epithelial cells, and human corneas using Wester

88 Inflammation induces PD-L1 up-regulation by corneal epithelial cells, and infiltration of significan

89 SSEA4 is highly expressed in differentiated corneal epithelial cells, and SSEA4(-) limbal epithelial

90 for transient transduction of primary rabbit corneal epithelial cells, and the effect on cell cycle p

91 un, which appears to be important to mediate corneal epithelial cell apoptosis after UV irradiation.

92 ed JNK (c-Jun N-terminal kinase) cascade and corneal epithelial cell apoptosis and protection against

93 yte growth factor (HGF) in the prevention of corneal epithelial cell apoptosis and to identify signal

94 Corneal epithelial cell apoptosis was increased 17-fold

95 sites; CPE assays were performed with rabbit corneal epithelial cells as host cells; and the expressi

96 ere co-cultured with PD-L1-expressing normal corneal epithelial cells, as compared to MS1 cells co-cu

97 e that stratification and differentiation of corneal epithelial cells, as measured by the capacity to

98 ata show functional roles for AMPs in normal corneal epithelial cell barrier function against P. aeru

99 hosphorylation in primary cultures of bovine corneal epithelial cells (BCECs).

100 amma caused LDH leakage from sensitive human corneal epithelial cells, but even higher doses of TNF-a

101 dlim2 gene was specifically expressed in the corneal epithelial cells, but not in the corneal stroma

102 llowing HPAI H7N7 virus infection in primary corneal epithelial cells, but not respiratory cells, ide

103 ive procedure to introduce gaps in layers of corneal epithelial cells by casting agarose strips on ti

104 correlated with an increase in infection of corneal epithelial cells by P. aeruginosa.

105 ot required for the colonization of cultured corneal epithelial cells by S. aureus, suggesting that s

106 Corneal epithelial cells can adhere and proliferate on t

107 Electrotaxis of dissociated bovine corneal epithelial cells (CECs) on planar quartz require

108 re upregulated in response to wounding in NL corneal epithelial cells (CECs), whereas the latter was

109 roliferation and vertical migration of basal corneal epithelial cells (CECs).

110 ll-infection" in vivo models, cultured human corneal epithelial cells, contact lens-wearing animal mo

111 rane mucin N-glycans in differentiated human corneal epithelial cells contain primarily complex-type

112 ly that IFN-gamma-induced MD-2 expression by corneal epithelial cells contributes to the host respons

113 roduction of MMP-9, -1, -13, and -3 by human corneal epithelial cells correlated positively with incr

114 nd after PA exposure and in serum-free human corneal epithelial cell culture (hTCEpi).

115 Primary human corneal epithelial cells cultured in normal osmolar medi

116 Expression was studied in normal human corneal epithelial cell cultures and hINV promoter trans

117 orneal epithelium, their expression in human corneal epithelial cell cultures, and the effect of ultr

118 lutaminase enzymatic activity is involved in corneal epithelial cell death after UVB and appears to p

119 sorders, especially blindness as a result of corneal epithelial cell deficiency.

120 giogenesis, and cancer, we hypothesized that corneal epithelial cell-derived exosomes may gain access

121 studying the molecular mechanisms regulating corneal epithelial cell differentiation and desquamation

122 fies a role for KLF7 as a KLF4 antagonist in corneal epithelial cell differentiation, and explains ho

123 ulation of involucrin gene expression during corneal epithelial cell differentiation.

124 in neutrophils and significant increases in corneal epithelial cell division and migration.

125 notion that Pax6 plays a role in controlling corneal epithelial cell dynamics in vivo.

126 Primary corneal epithelial cells, established from donor limbal

127 hymosin beta 4 (Tbeta(4)) treatment on human corneal epithelial cells exposed to ethanol in vitro.

128 e maintained on a high-fat diet, or cultured corneal epithelial cells exposed to high glucose, which

129 ation and division, and during wound closure corneal epithelial cells express intercellular adhesion

130 Additionally, murine corneal epithelial cell expressed the catecholamine-synt

131 est a central role of the WNT7A-PAX6 axis in corneal epithelial cell fate determination, and point to

132 In murine models, LRIG1 regulated corneal epithelial cell fate during wound repair.

133 c-Jun/AP-1 transcriptional complex and human corneal epithelial cell fate.

134 idement wounds and in vitro in primary human corneal epithelial cells following a linear scratch woun

135 ntly, B2088 did not show any cytotoxicity to corneal epithelial cells for at least 96 h continuous ex

136 e surface ectoderm, and prospective lens and corneal epithelial cells formed a multilayered mass of c

137 Transformed human corneal epithelial cells from a patient homozygous for D

138 reduced proliferation in primary cultures of corneal epithelial cells from AQP3-null mice.

139 Corneal epithelial cells from bitransgenic Krt12Cre/+/RO

140 Using FACS analysis, 60% to 70% of the corneal epithelial cells from Krt12Cre/+/ROSAEGFP mice w

141 Microdissection was used to remove the corneal epithelial cells from the button.

142 cratch wound assay using primary cultures of corneal epithelial cells from wild-type versus AQP3-null

143 ation resulted in a complete loss of in vivo corneal epithelial cell gene expression.

144 Corneal epithelial cells have large stores of glycogen,

145 4-HNE suppressed the cell viability of human corneal epithelial cells (HCE) in a concentration depend

146 ty in THCE cells, an SV40-immortalized human corneal epithelial cell (HCEC) line, and primary HCECs w

147 o investigate whether LL-37 stimulates human corneal epithelial cell (HCEC) migration, proliferation,

148 t NaNO2 (0.1 muM to 100 muM) increased human corneal epithelial cell (HCEC) viability and migration.

149 Immortalized human corneal epithelial cells (HCEC) were treated with CAP37,

150 his study was designed to determine in human corneal epithelial cells (HCEC) whether the balance betw

151 ility of the dendrimer was assessed in human corneal epithelial cells (HCECs) and across isolated bov

152 ssay and immunohistochemistry study of human corneal epithelial cells (HCECs) and human keratocytes (

153 ion using an in vitro culture model of human corneal epithelial cells (HCECs) exposed to hyperosmotic

154 In this study, it was determined that human corneal epithelial cells (HCECs) express asialogangliosi

155 Primary human corneal epithelial cells (HCECs) from limbal explants we

156 us studies have shown that wounding of human corneal epithelial cells (HCECs) results in the release

157 uction of proinflammatory mediators by human corneal epithelial cells (HCECs) stimulated by a fungal

158 he innate immune responses of cultured human corneal epithelial cells (HCECs) to infection by the Gra

159 necrosis factor (TNF)-alpha-stimulated human corneal epithelial cells (HCECs) to produce nanogram amo

160 eviously demonstrated that wounding of human corneal epithelial cells (HCECs) transactivates epiderma

161 In contrast, human corneal epithelial cells (HCECs) transfected with MyD88

162 Human corneal epithelial cells (HCECs) were stimulated with LL

163 )) were able to bind to the surface of human corneal epithelial cells (HCECs) with similar efficiency

164 of eritoran tetrasodium on stimulated human corneal epithelial cells (HCECs), macrophages, and neutr

165 uction of proinflammatory cytokines in human corneal epithelial cells (HCECs).

166 expression by PAF was investigated in human corneal epithelial cells (HCECs).

167 SiNPs on ocular surface cells such as human corneal epithelial cells (HCECs).

168 rt a novel study of the 3D printing of human corneal epithelial cells (HCECs)/collagen/gelatin/algina

169 l extracts were obtained from cultured human corneal epithelial cells (HCEpCs) as a source of ZEB1 pr

170 ternative splicing study, immortalized human corneal epithelial cells (HCET) harboring doxycycline-in

171 rried out with telomerase-immortalized human corneal epithelial cells (HCLE).

172 ggesting virulence strategies affecting live corneal epithelial cell health are selected for among ke

173 mine whether the ability to adversely affect corneal epithelial cell health is a factor common to Pse

174 er, our work defines regulatory enhancers in corneal epithelial cells, highlights global gene-regulat

175 y and angiogenesis, metastatic melanoma, and corneal epithelial cells; however, less is known about h

176 examine the ion transport mechanisms in the corneal epithelial cells; however, this technique has be

177 CTCF activities in human corneal epithelial cells immortalized by telomerase (HTC

178 , production, and activity of MMP-9 by human corneal epithelial cells in a concentration-dependent fa

179 ophozoites of A. castellanii bound avidly to corneal epithelial cells in a mannose-inhibitable manner

180 hanced cell proliferation of wild-type mouse corneal epithelial cells in an organ culture.

181 xhibited an excessive desquamation of apical corneal epithelial cells in association with an increase

182 gnificantly decreased expression of PD-L1 by corneal epithelial cells in DED and significantly increa

183 gulation of human beta-defensin 2 (hBD-2) by corneal epithelial cells in response to P. aeruginosa an

184 Corneal epithelial cells in two-dimensional or organ cul

185 an tear fluid could protect individual human corneal epithelial cells in vitro against invasion by an

186 Pseudomonas aeruginosa enters corneal epithelial cells in vitro via membrane microdoma

187 protease activity and cytotoxicity to human corneal epithelial cells in vitro was determined.

188 it moderately) reduced electrotaxis of human corneal epithelial cells in vitro, but did not affect th

189 o keratocytes both in vitro and in vivo, and corneal epithelial cells in vitro, but it is uncertain i

190 y suppress hypoxia-induced VEGF secretion by corneal epithelial cells in vitro.

191 te the effects of rapamycin on primary human corneal epithelial cells in vitro.

192 EphA2 was immunolocalized to human corneal epithelial cells in vivo and in vitro.

193 Corneal epithelial cells in vivo did not stain for CFTR

194 vitro, but it is uncertain if MSC can assume corneal epithelial cells in vivo.

195 pithelial sheets (ex vivo) and primary human corneal epithelial cells (in vitro).

196 pressing a dominant-negative mutant in human corneal epithelial cells induced mesenchymal features.

197 otype was determined in vitro by quantifying corneal epithelial cell invasion by gentamicin survival

198 PA binding to corneal epithelial cells is a prerequisite for infection

199 tion of TGF-beta2 protein by cultured rabbit corneal epithelial cells is reduced by plating on a base

200 ering event for the induction of motility in corneal epithelial cells is related to the sudden availa

201 , -3.4, -4.2, and -4.3, demonstrated that in corneal epithelial cells Kv3.4 channel was highly expres

202 To examine this further, a human corneal epithelial cell line (HCE) lacking endogenous AL

203 In a human corneal epithelial cell line (HCEC), 1,25(OH)2D3 increas

204 s on the gene expression of MT isoforms in a corneal epithelial cell line (HCEsv).

205 orneal basal epithelial cells and in a human corneal epithelial cell line (HTCE).

206 lly expressing miR-31 in an undifferentiated corneal epithelial cell line promotes differentiation an

207 A rabbit corneal epithelial cell line SIRC was used to establish

208 A human corneal epithelial cell line was used.

209 iated apoptosis in a telomerase-immortalized corneal epithelial cell line.

210 of cell proliferation in Pax6-overexpressing corneal epithelial cell lines and primary cell culture i

211 iated with no contact lens protection showed corneal epithelial cell loss plus lens epithelial cell s

212 ones were observed for the usually prominent corneal epithelial cell marker aquaporin 5 (AQP5), a wat

213 rogenitor cell line (TKE2) and mature murine corneal epithelial cells (MCE).

214 The effects of CS on corneal epithelial cell migration and associated signali

215 in vivo to evaluate their ability to promote corneal epithelial cell migration and corneal wound heal

216 icient mice results from distinct defects in corneal epithelial cell migration and proliferation.

217 Tbeta4 stimulated corneal epithelial cell migration in the presence of SSW

218 cies of bacteria inhibited human and porcine corneal epithelial cell migration in vitro and ex vivo.

219 The authors found EGF stimulated corneal epithelial cell migration in wound healing by en

220 Corneal epithelial cell migration was measured by using

221 EphA2 attenuates corneal epithelial cell migration when stimulated by eph

222 ared with EGF, is a more potent activator of corneal epithelial cell migration.

223 nitude of growth factor-induced increases in corneal epithelial cell migration.

224 synthetic genes as being required to inhibit corneal epithelial cell migration.

225 EphA2 receptor and ephrin-A1 ligand in human corneal epithelial cell migration.

226 059 enhances drug permeation across cultured corneal epithelial cell monolayers by transiently affect

227 cted cells and, subsequently, promoted human corneal epithelial cell motility, migration, and wound h

228 aNp63alpha is the dominant active isoform in corneal epithelial cell nuclei.

229 The limbal and central corneal epithelial cells of 6-week-old rats were isolate

230 6 and Oct1 are both present in the nuclei of corneal epithelial cells of the 6-week-old mouse.

231 ocytes but slightly reduced the migration of corneal epithelial cells on amnion ex vivo.

232 w BM formed by ex vivo expanded human limbal corneal epithelial cells on iAM deposits much faster and

233 the effect of overexpressing NFkappaB p50 in corneal epithelial cells on the promotion of wound heali

234 epithelium (in vivo) and in cultured primary corneal epithelial cells (PCECs) (in vitro).

235 d loss of tight junction bearing superficial corneal epithelial cells, perhaps by proteolytic cleavag

236 ave demonstrated the in vitro induction of a corneal epithelial cell phenotype from pluripotent stem

237 ed decrease in CTCF activities mediate human corneal epithelial cell proliferation and apoptosis, res

238 Corneal epithelial cell proliferation and differentiatio

239 CAP37 modulated corneal epithelial cell proliferation and migration and

240 5(INK4b) appears to be sufficient to inhibit corneal epithelial cell proliferation and to stimulate c

241 ing factor (CTCF) plays an important role in corneal epithelial cell proliferation by suppressing the

242 effects of 1,25(OH)2D3 and 24R,25(OH)2D3 on corneal epithelial cell proliferation, migration, and on

243 ulated, consistent with the increased Klf4CN corneal epithelial cell proliferation.

244 h factors play important roles in regulating corneal epithelial cell proliferation/differentiation du

245 Rabbit corneal epithelial cells (RCECs) in culture were arreste

246 PD-L1 in both vascular endothelial cells and corneal epithelial cells regulates corneal angiogenesis.

247 ude that 24R,25(OH)2D3 is likely involved in corneal epithelial cell regulation independent of 1,25(O

248 c effect of epidermal growth factor (EGF) in corneal epithelial cells required suppression of PAX6 ac

249 s were assessed in porcine and primary human corneal epithelial cells, respectively.

250 Human corneal epithelial cells respond rapidly following injur

251 Furthermore, H7N7 virus infection of corneal epithelial cells resulted in enhanced and signif

252 6- or CTCF-specific small interfering RNA in corneal epithelial cells significantly promoted or atten

253 trix metalloproteinase (MMP)-9 is induced in corneal epithelial cells stimulated with platelet-activa

254 Epidermal growth factor (EGF) in corneal epithelial cells stimulates proliferation by ind

255 opening and provide new probes for exploring corneal epithelial cell stratification, development, and

256 , which can be explained by the fragility of corneal epithelial cells that did not produce Krt12 beca

257 reater desquamation of differentiated apical corneal epithelial cells that expressed the tight juncti

258 ols confirmed PA14 cytotoxicity toward these corneal epithelial cells that was absent with exoU mutan

259 ndings could be replicated in cultured human corneal epithelial cells that were treated with active M

260 s constitutively expressed at high levels by corneal epithelial cells, the authors studied the expres

261 ied epithelia), the centripetal migration of corneal epithelial cells, the exclusive location of slow

262 GFR-3), which is synthesized and secreted by corneal epithelial cells; they show that sVEGFR-3 modula

263 In corneal epithelial cells, this suggests that inhibition

264 HAdV-D19p is not infectious for corneal epithelial cells, thus explaining the lack of an

265 SF, a novel biomaterial, could support corneal epithelial cells to proliferate, differentiate,

266 targeting of EphA2 restricted the ability of corneal epithelial cells to seal linear scratch wounds i

267 ntiation and (3) function redundantly in the corneal epithelial cells to suppress proliferation.

268 of TAT-HA-p15 were applied to primary human corneal epithelial cells to test potency.

269 Wound closure rates were measured in human corneal epithelial cells transfected with an NFkappaB ac

270 Human corneal epithelial cells, transfected with plasmids enco

271 ypes, and by in vitro studies we showed that corneal epithelial cell tropism can be predicted by the

272 P. aeruginosa enters human corneal epithelial cells via lipid rafts containing CFTR

273 Osmotic stress decreased corneal epithelial cell viability, which was due in part

274 Apoptosis of corneal epithelial cells was determined by caspase-3 and

275 y sulfated keratan sulfate in cultured human corneal epithelial cells was dramatically reduced when e

276 Cytotoxicity on corneas and corneal epithelial cells was evaluated by LDH assays.

277 traversal of airlifted, multilayered, human corneal epithelial cells was quantified in vitro up to 8

278 Hypoxia induced VEGF by human corneal epithelial cells was sequestered by both Flt23K

279 ty of aPA and MIP-133 to induce cytolysis of corneal epithelial cells was tested in vitro.

280 Rabbit and human corneal epithelial cells were cultured in DMEM/F12 mediu

281 Primary human corneal epithelial cells were cultured to confluence.

282 liferation of vascular endothelial cells and corneal epithelial cells were determined by quantifying

283 Corneal epithelial cells were exposed to a soluble ephri

284 Exosomes secreted by mouse corneal epithelial cells were found to fuse to keratocyt

285 Rabbit corneal epithelial cells were grown to multilayers with

286 Primary cultures of human corneal epithelial cells were infected with a retroviral

287 TSLP in corneal epithelial cells were largely induced in a conce

288 To assay cell migration, human corneal epithelial cells were plated inside a cylinder a

289 nd closure and cell migration rates of human corneal epithelial cells were significantly suppressed a

290 epithelial (hTCEpi) cells and primary human corneal epithelial cells were tested for their ability t

291 e second experiment, primary cultured bovine corneal epithelial cells were transiently stressed with

292 , as well as p38 and p42/44 MAPK activity in corneal epithelial cells, whereas concurrent administrat

293 Accordingly, we determined, in human corneal epithelial cells, whether or not (i) CCE is depe

294 Thus, TRPC4 is a component of SOC in human corneal epithelial cells whose activation by EGF is requ

295 Treatment of the corneal epithelial cells with 12-O-tetradecanoylphorbol-

296 termined that pretreatment of cultured human corneal epithelial cells with flagellin isolated from th

297 We found that stimulation of human corneal epithelial cells with hypoxic stress suppressed

298 vivo (topically in male mice) and in vitro (corneal epithelial cell wound healing).

299 ential role in growth factor-regulated human corneal epithelial cell wound healing.

300 In VDR wildtype mouse corneal epithelial cells (WT), 1,25(OH)2D3 increased CYP