ライフサイエンスコーパス: corneal epithelial wound

1 neutrophil infiltration was evaluated after corneal epithelial wounding.

2 rabbit lacrimal gland tissue in response to corneal epithelial wounding.

3 Axial corneal epithelial wounds, 10 mm in diameter, were creat

4 pression in keratocytes is upregulated after corneal epithelial wounding and probably contributes to

5 sponse of wild-type and Pglyrp1(-/-) mice to corneal epithelial wounds and Pseudomonas aeruginosa-med

6 We found that female mice exhibited delayed corneal epithelial wound closure and attenuated polymorp

7 ibition by DAPT was also found to accelerate corneal epithelial wound closure in an in vivo murine mo

8 n was designed to examine the role of OGF on corneal epithelial wound closure in the rabbit under in

9 ist has no significant effect on the rate of corneal epithelial wound closure in the rabbit.

10 ally applied substance P (SP) on the rate of corneal epithelial wound closure in the rabbit.

11 recombinant galectin-1 did not stimulate the corneal epithelial wound closure rate.

12 In contrast, there was no difference in corneal epithelial wound closure rates between galectin-

13 Corneal epithelial wound closure was assessed in culture

14 Corneal epithelial wound closure was delayed and re-inne

15 EGF and LXA4 increased corneal epithelial wound closure.

16 mporal data of mitotic rates measured during corneal epithelial wound healing (CEWH) of a rat model w

17 Here, we examined Cdc42 role in corneal epithelial wound healing and the influence of he

18 d NFkappaB p50 and CTCF activation affecting corneal epithelial wound healing has been established, i

19 An anti-lumican antibody was found to retard corneal epithelial wound healing in cultured mouse eyes.

20 that there was a significant retardation in corneal epithelial wound healing in the corneas of p50 k

21 innervation and its correlation with delayed corneal epithelial wound healing in type 2 diabetic Goto

22 data indicate that EGFR was activated during corneal epithelial wound healing in vivo.

23 ed that in the absence of TGF-beta signaling corneal epithelial wound healing is delayed by 48 hours;

24 ncomycin, predatory bacteria did not inhibit corneal epithelial wound healing or increase clinical in

25 g that previous models, used in the study of corneal epithelial wound healing speeds, are inadequate

26 LPA accelerates corneal epithelial wound healing through its ability to

27 derstand the role of NFkappaB in EGF-induced corneal epithelial wound healing through regulation of C

28 effect of hypoxic stress on early stages of corneal epithelial wound healing was compared with other

29 Corneal epithelial wound healing was evaluated by a whol

30 ce, the effect of human lumican on promoting corneal epithelial wound healing was even more dramatic

31 Hypoxic stress-induced delay of corneal epithelial wound healing was further evaluated i

32 The effect of NFkappaB p50 on corneal epithelial wound healing was investigated by com

33 The effects of lumican on corneal epithelial wound healing were examined in an org

34 on for the modelling of the cell kinetics of corneal epithelial wound healing, and yields valuable in

35 e PKCalpha(-/-) mice demonstrated more rapid corneal epithelial wound healing, perhaps due to decreas

36 gulating collagen fibrillogenesis, promoting corneal epithelial wound healing, regulating gene expres

37 activation represent a critical event during corneal epithelial wound healing, suggesting a possible

38 ike growth factor-1 are shown to promote the corneal epithelial wound healing.

39 s also evaluated in vivo in a mouse model of corneal epithelial wound healing.

40 the TGF-beta and MAPK signaling pathways in corneal epithelial wound healing.

41 3 (Plk3) in hypoxic stress-induced delay of corneal epithelial wound healing.

42 sceptible to desiccating stress and impaired corneal epithelial wound healing.

43 uced attenuation of cell growth and delay of corneal epithelial wound healing.

44 ion through activating HDAC6 on migration in corneal epithelial wound healing.

45 growth factor (EGF) stimulates migration in corneal epithelial wound healing.

46 ssion of cell proliferation and the delay of corneal epithelial wound healing.

47 ptor (EGFR) signaling and attenuates ex vivo corneal epithelial wound healing.

48 l 3-kinase/Akt pathway, resulting in delayed corneal epithelial wound healing.

49 organ and cell culture models, LPA enhanced corneal epithelial wound healing.

50 the major environmental stresses that affect corneal epithelial wound healing.

51 phosphatidylinositol 3'-kinase (PI3K) during corneal epithelial wound healing.

52 he ERK1/2 or p38 pathway resulted in delayed corneal epithelial wound healing.

53 adhesion or migration, thus contributing to corneal epithelial wound healing.

54 K+ channels disappear during some models of corneal epithelial wound healing.

55 inetics and extent of EGFR activation during corneal epithelial wound repair and whether the epitheli

56 on an intact beta2-AR for the modulation of corneal epithelial wound repair.

57 location of the Smad 2 and 4 proteins during corneal epithelial wound repair.

58 Cdc42 is an important regulator of corneal epithelial wound repair.

59 ceptor (TbetaR)-I and -II are altered during corneal epithelial wound repair.

60 ole in EGF-induced cell proliferation during corneal epithelial wound repair.

61 Corneal epithelial wounding resulted in significant dela