ライフサイエンスコーパス: corneal infection

1 ns of clodronate-containing liposomes before corneal infection.

2 that resulted in the rapid resolution of the corneal infection.

3 is an important contributor to P. aeruginosa corneal infection.

4 rtant role in restricting HSV-1 growth after corneal infection.

5 increased HSV-1 titers in the TG after HSV-1 corneal infection.

6 nearly completely resistant to P. aeruginosa corneal infection.

7 cific IgA in mucosal secretions and prevents corneal infection.

8 anthamoeba keratitis is a vision-threatening corneal infection.

9 as as virulent as the parental strain during corneal infection.

10 red for the establishment and maintenance of corneal infection.

11 canthamoeba keratitis is a sight-threatening corneal infection.

12 evaluated in a murine model of P. aeruginosa corneal infection.

13 inside epithelial cells during experimental corneal infection.

14 eminal ganglion at various times after HSV-1 corneal infection.

15 ol the replication and spread of HSV-1 after corneal infection.

16 cellular and viral factors involved in acute corneal infection.

17 for the treatment of patients suffering from corneal infection.

18 epithelial cells and Pseudomonas aeruginosa corneal infection.

19 mice to experimental Pseudomonas aeruginosa corneal infection.

20 le in pathogenesis and immunity to S. aureus corneal infection.

21 wild-type virus levels after intracranial or corneal infection.

22 f epithelial cells, in Staphylococcus aureus corneal infection.

23 and mBD3 together promote resistance against corneal infection.

24 ost resistance against P. aeruginosa-induced corneal infection.

25 8HR from all tissues at late times following corneal infection.

26 ral spread and promoting host survival after corneal infection.

27 to promote resistance against P. aeruginosa corneal infection.

28 ific virulence factor for C. albicans during corneal infection.

29 potential virulence factor in posttraumatic corneal infection.

30 f Acanthamoeba keratitis (AK), a devastating corneal infection.

31 amoebae produce a painful, sight-threatening corneal infection.

32 sion did not appear to be upregulated during corneal infection.

33 ro and to cause disease in a murine model of corneal infection.

34 ct lenses and as a treatment for established corneal infections.

35 l wounds and Pseudomonas aeruginosa-mediated corneal infections.

36 mice are resistant to Pseudomonas aeruginosa corneal infections.

37 y, causing respiratory tract, cutaneous, and corneal infections.

38 s suggest that the galU gene is required for corneal infection and for efficient systemic spread foll

39 hage functions, which are switched on during corneal infection and injury.

40 This study explored their role in corneal infection and potential synergy with mBD2, a def

41 s primarily in the corneal epithelium before corneal infection and was also in the corneal stroma aft

42 at evolutionary pressure enhances fitness in corneal infection, and may be a molecular determinant in

43 essing cells during Streptococcus pneumoniae corneal infection, and P2X7R was required for bacterial

44 pread have not been fully examined following corneal infection, and the possible pathologic consequen

45 The role of SAP6 during corneal infection appears to be associated with the morp

46 Corneal infections are surprisingly rare, suggesting tha

47 rs that in the murine model of P. aeruginosa corneal infection at challenge doses of bacteria 10-fold

48 aeruginosa keratitis, functioning to reduce corneal infection (bacterial load) and inflammation by n

49 Approximately 7 days after HSV-1 corneal infection, BALB/c mice develop tissue-destructiv

50 mice, Deltavhs remains highly attenuated via corneal infection but is fully virulent via intracranial

51 100 CFU of C. albicans initiated measurable corneal infection, but 1 million or more colony-forming

52 conazole could be a good choice for treating corneal infection by Cladosporium species.

53 R is critical in resistance to P. aeruginosa corneal infection by down-regulating type 1 immunity, an

54 syndecan-1 ectodomains to promote S. aureus corneal infection by inhibiting a key arm of neutrophil

55 5c and 6 were effective in a mouse model of corneal infection by S. aureus and MRSA.

56 We conclude that transient HSV-1 corneal infections cause long-term alterations of the co

57 r the treatment of established P. aeruginosa corneal infections caused by diverse LPS serogroups.

58 After HSV type 1 corneal infection, CD4(+) T cells are expanded in the dr

59 After primary HSV-1 corneal infection, CD8(+) T cells infiltrate the trigemi

60 minal ganglia, and periocular skin following corneal infection compared to that with the ICP34.5-null

61 c1(-/-)) mice significantly resist S. aureus corneal infection compared with wild type (WT) mice that

62 lens wearers have much higher incidences of corneal infections compared with those of non-contact le

63 Acute pneumonias and corneal infections due to Pseudomonas aeruginosa are typ

64 de range of recent applications that include corneal infections, dystrophies and disease.

65 its and protects BALB/c mice against primary corneal infection equivalent to a vhs- strain (BGS41).

66 eal or s.c. immunization of conidia prior to corneal infection exhibited enhanced fungal killing and

67 nd electron microscopy showed no evidence of corneal infection (experiment II).

68 anism that has the ability to cause blinding corneal infections following trauma and during contact l

69 on with aPA and MIP-133 was determined after corneal infection had been established.

70 By contrast, pigs immunized after corneal infections had been established displayed kerati

71 ions but no well-described case of D. pigrum corneal infection has been reported.

72 After corneal infection, herpes simplex virus type 1 (HSV-1) i

73 Following corneal infection, HSV-1 viral titers were elevated in t

74 The influence of steroid treatment on corneal infection in a Chinese hamster model of Acantham

75 hat syndecan-1 ectodomain promotes S. aureus corneal infection in an HS-dependent manner.

76 is was examined after Pseudomonas aeruginosa corneal infection in C57BL/6 (B6, susceptible) and BALB/

77 ole of macrophages in Pseudomonas aeruginosa corneal infection in susceptible (cornea perforates), C5

78 D. pigrum is the likely cause of corneal infection in three patients, with effective anti

79 tear fluid can protect against P. aeruginosa corneal infection in vivo and that the mechanism likely

80 monas aeruginosa can cause sight-threatening corneal infections in humans, particularly those who wea

81 Pseudomonas aeruginosa causes severe corneal infections in mice after inoculations with low d

82 a vaccines delivered nasally protect against corneal infections in mice and potentially can be used t

83 for protection against Staphylococcus aureus corneal infections in mice, which are a serious cause of

84 ergillus species of mold are major causes of corneal infections in the United States and worldwide, r

85 tion of these ExsA-regulated proteins toward corneal infections in vivo was investigated.

86 evaluated for clinical score in experimental corneal infection induced by a clinical isolate and a ci

87 )-dominated response following P. aeruginosa corneal infection is associated with genetic susceptibil

88 A very intriguing aspect of HSV-1 corneal infection is that the virus spread is normally r

89 Its role during bacterial corneal infections is unknown.

90 Acanthamoebae provoke a vision-threatening corneal infection known as Acanthamoeba keratitis (AK).

91 vivo imaging of P. aeruginosa in implant and corneal infection mice models.

92 In a corneal infection model, the galU mutants were significa

93 In a murine HSV-1 corneal infection model, we establish 17-beta estradiol

94 target antigens, of this vaccine in a murine corneal infection model.

95 Fusarium keratitis is a serious corneal infection, most commonly associated with corneal

96 Many corneal infections occur secondary to epithelial breache

97 Corneal infection of BALB/c mice with herpes simplex vir

98 f immunoinflammatory lesions associated with corneal infection of HSV.

99 Following corneal infection of mice with HSV type 2 (HSV-2), we qu

100 hes latency in trigeminal ganglia (TG) after corneal infection of mice.

101 hat influence immune responses to subsequent corneal infection or trauma.

102 First, following corneal infection, OVA257-specific OT-1 CD8 T cells do n

103 , epitheliopathy (P < 0.001), suture-related corneal infections (P < 0.001), and wound dehiscence (P

104 cytokines and chemokines upregulated during corneal infection provides important information relevan

105 TG can be detected as early as 2 days after corneal infection, reaches peak titers by 3-5 days after

106 neutralization of IL-17 during P. aeruginosa corneal infection reduces neutrophil influx and patholog

107 peptide (VIP), during Pseudomonas aeruginosa corneal infection remains unexplored.

108 Staphylococcus aureus corneal infection results in extensive inflammation and

109 man species D adenovirus not associated with corneal infection, revealed areas of non-conserved seque

110 Thus, in sharp contrast with other corneal infections such as herpes and Pseudomonas aerugi

111 clear cornea furthest away from the focus of corneal infection suggests that the host response extend

112 Upon corneal infection, this mutant replicates transiently bu

113 rophages control resistance to P. aeruginosa corneal infection through regulation of PMN number, bact

114 mouse model of herpes simplex virus (HSV) 1 corneal infection, tissue destruction results from a CD4

115 In a P. aeruginosa rabbit corneal infection, topical administration of DFO-Ga toge

116 diphtheria toxin treatment 2 d before HSV-1 corneal infection transiently depleted CD11c(+) DCs from

117 eta) production after Pseudomonas aeruginosa corneal infection was examined in susceptible (cornea pe

118 ophages in the subconjunctival tissue before corneal infection was not accompanied by enhanced virus

119 jor participants in innate immunity to HSV-1 corneal infection was not supported.

120 in susceptibility to Pseudomonas aeruginosa corneal infection was studied in inbred C57Bl/6 (B6) bet

121 Using a murine model of S. pneumoniae corneal infection, we demonstrated a requirement for IL-

122 ma is produced during Pseudomonas aeruginosa corneal infection, we examined the role of this cytokine

123 Using a murine model of P. aeruginosa corneal infection, we show that although infected CD14(-

124 Using a murine model of A. fumigatus corneal infection, we showed that DeltarodA conidia indu

125 ection of L-Cl2MDP 4 and 2 days before HSV-1 corneal infection were found to have ocular virus titers

126 teroids for Corneal Ulcers Trial (SCUT), 500 corneal infections were treated with topical moxifloxaci

127 amoeba significantly reduced the severity of corneal infection which includes infiltration and ulcera

128 a role in the upregulation of MT-MMPs during corneal infection, which in turn can cause the destructi

129 infection with 10(1)(1) CFU S. pneumoniae or corneal infection with 10(5) CFU of S. pneumoniae.

130 , or phosphate-buffered saline (PBS), before corneal infection with 10(5) colony-forming units (CFU)

131 Following corneal infection with 2 x 10(6) PFU of HSV-1 strain McK

132 inhibitor also reduced clinical scores after corneal infection with a clinical isolate, KEI-1025, and

133 Eyes inoculated with C. albicans developed corneal infection with a mean clinical score of 8.2+/-0.

134 Corneal infection with a thymidine kinase-deleted report

135 helium renders Chinese hamsters resistant to corneal infection with Acanthamoeba castellanii.

136 Corneal infection with herpes simplex virus-1 in immunoc

137 ely susceptible to liver infection following corneal infection with HSV-1.

138 whereas TLR4/5(-/-) mice were susceptible to corneal infection with parent strains.

139 nd chemokine mRNAs that were expressed after corneal infection with Pseudomonas aeruginosa in mice.

140 Corneal infection with Pseudomonas aeruginosa leads to a

141 Corneal infection with Pseudomonas aeruginosa perforates

142 Corneal infection with Pseudomonas aeruginosa results in

143 orphonuclear neutrophil (PMN) function after corneal infection with Pseudomonas aeruginosa.

144 esponses in mice that are protective against corneal infection with virulent HSV-1, but it has the ca

145 hs) protein diminishes primary and recurrent corneal infection with wild-type HSV-1.