ライフサイエンスコーパス: corneal injury

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1 in a mouse model of diabetes with or without corneal injury.

2 mplications of MSC-mediated tissue repair in corneal injury.

3 inhibition may be an effective treatment for corneal injury.

4 eal infection, most commonly associated with corneal injury.

5 ween proteoglycans and growth factors during corneal injury.

6 small and reached significance I clay after corneal injury.

7 directly related to area and depth of acute corneal injury.

8 des re-epithelialization after some types of corneal injury.

9 tilt position may reduce the risk of severe corneal injury.

10 model of photorefractive keratectomy-induced corneal injury.

11 ically to treat severe inflammation-mediated corneal injuries.

12 nd its impact on inflammation and healing of corneal injuries.

13 mising targets for regenerative therapies of corneal injuries.

14 f animal models of myocardial infarction and corneal injury/allogenic transplant.

15 eal neovascularization in mice 3 weeks after corneal injury and 1 week after intrastromal injection o

16 After corneal injury and inflammation, PAF may be an important

17 It localises macrophages to the site of corneal injury and recruits neutrophils in lipopolysacch

18 Surprisingly, corneal injury and treatment with PEDF + DHA induced tra

19 nd protein were induced to high levels after corneal injury and were temporally and spatially correla

20 These findings provide novel evidence that corneal injury causes significant mobilization of endoge

21 revious reports indicated that pregnancy and corneal injury (CI) trigger alterations of lacrimal glan

22 ular surface epithelial cells in response to corneal injury, epithelial cells undergoing DNA synthesi

23 Corneal injury in HO-2 null mice leads to impaired heali

24 Corneal injury in mice was induced by thermal cauterizat

25 Following the induction of corneal injury in mice, a 2-fold increase in the frequen

26 Furthermore, they strongly suggest that corneal injury induced by toxicants can be treated using

27 limination of myofibroblasts after repair of corneal injury is essential for the maintenance of corne

28 Corneal injury may ultimately lead to a scar by way of c

29 d upon transplantation onto eyes in a rabbit corneal injury model, these reprogrammed cells are able

30 In addition, in the setting of corneal injury, MSC administration showed significant an

31 present study, we investigated the impact of corneal injury on the homeostasis of endogenous MSCs, an

32 t of induction of ocular pathology following corneal injury or wearing of contaminated contact lenses

33 Corneal injury produces changes in PAF-R mRNA expression

34 Moreover, environmental exposure to corneal injury significantly increases growth of both bl

35 Using a mouse model of perforating corneal injury that causes acute and severe fluid accumu

36 Wound healing after corneal injury typically involves fibrosis, with transfo

37 In vivo CM revealed corneal injury with slight irritants to be restricted to

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