ライフサイエンスコーパス: corneal opacity

1 raniofacial and ocular anomalies (colobomas, corneal opacities).

2 n 1 patient (2%), and there were no cases of corneal opacity.

3 escued pathological phenotypes and prevented corneal opacity.

4 educe the burden of corneal blindness due to corneal opacity.

5 e in matrix disorder at 8 weeks, but minimal corneal opacity.

6 etween successful epilation and less central corneal opacity.

7 -initiated epilation and its relationship to corneal opacity.

8 d bilateral trichiasis and 21% had bilateral corneal opacity.

9 immunohistochemistry for the development of corneal opacity.

10 ndication is visually significant congenital corneal opacity.

11 sits that correlated with the development of corneal opacity.

12 HSV-1 strain RE and subsequently graded for corneal opacity.

13 is and follow-up of patients with congenital corneal opacities.

14 , autoimmune ocular disorders, and pediatric corneal opacities.

15 rment due to glaucoma (0.71 [0.57-0.86]) and corneal opacity (0.54 [0.43-0.66]) were more common amon

16 es were refractive errors (47.1%), keratitis/corneal opacity (16%), amblyopia (14.3%), ocular trauma

17 nalysis of woe2 adult eyes identified severe corneal opacities, abnormalities of the anterior segment

18 ted circumferential peripheral subepithelial corneal opacities and adjacent abnormal limbal vasculatu

19 This disease is characterized by corneal opacities and vision impairment.

20 urgical condition that presents with central corneal opacity and a significant hyperopic shift.

21 HSK resulted in significant reduction in the corneal opacity and angiogenesis.

22 eles at the mouse Cat4 locus, causes central corneal opacity and anterior polar cataract in heterozyg

23 especially with regard to the development of corneal opacity and blindness.

24 on of IL-6 at the time of infection restored corneal opacity and chemokine levels to that of wild-typ

25 lta41Delta29 and BGS41 vaccination decreased corneal opacity and delayed-type hypersensitivity respon

26 ox/flox);Le-Cre(+) eyes invariably displayed corneal opacity and developed spontaneous corneal neovas

27 syltransferase B3GLCT, leading to congenital corneal opacity and diverse extra-ocular manifestations.

28 Most mutant mice aged 3-8 months develop corneal opacity and eye lesions due to irritation and co

29 l cataract-microcornea with mild to moderate corneal opacity and in a consanguineous Cambodian family

30 hthalmia, congenital cataracts, microcornea, corneal opacity and nystagmus.

31 hen a decrease in tear volume, together with corneal opacity and ocular lesions.

32 B/c mice treated with rmST2 showed increased corneal opacity and perforation (at 5 days PI) when comp

33 proteinase with 15 TSRs, leads to congenital corneal opacity and Peters anomaly (persistent lens-corn

34 ation of GAG in corneal stromal cells causes corneal opacity and reduced vision.

35 rVIP and subsequently demonstrated decreased corneal opacity and resistance to corneal perforation co

36 achlorhydria, and visual impairments such as corneal opacity and strabismus.

37 Increased corneal opacity and worsened disease were displayed afte

38 diseases including skin blistering diseases, corneal opacities, and neurological degenerations.

39 pressure (30.8 +/- 12.5 mm Hg; mean +/- SD), corneal opacity, and enlarged anterior chambers.

40 in the thickness of the corneal epithelium, corneal opacity, and modest disorganization in the corne

41 ha-4 HDL present in their plasma and develop corneal opacities, anemia, proteinuria, and kidney failu

42 s, and in pediatric patients with congenital corneal opacities are disappointing.

43 tinfection, were grouped on the basis of the corneal opacity as mild (</=2) or severe (>2).

44 ted in significantly decreased virus-induced corneal opacity between 7 and 21 days after UV-B exposur

45 ccelerated reepithelialization and decreased corneal opacity compared with B6 mice after alkali wound

46 exhibited enhanced fungal killing and lower corneal opacity compared with unimmunized mice.

47 re of 4 with compromised visibility due to a corneal opacity (estimate = 0.45; P=0.039), and prolonge

48 eads to an ocular phenotype characterized by corneal opacities identical to CSCD in humans.

49 in 9, nonglaucomatous optic neuropathy in 3, corneal opacities in 3, retinal disease in 3, and undete

50 Posterior keratoconus is a rare cause of a corneal opacity in an infant.

51 Corneal opacity in IL-6(-/-) mice was substantially dimi

52 ced corneal epithelial healing and decreased corneal opacity in murine corneal alkali burn model by m

53 ations (39.8% vs. 26.5% rural) compared with corneal opacity in rural areas (38.0% vs. 25.5% urban).

54 ma changes including neovascularization; and corneal opacity, leading to plaque formation.

55 TSG-6 markedly decreased corneal opacity, neovascularization, and neutrophil infi

56 ain of HSV-1, and HSK was monitored based on corneal opacity, neovascularization, leukocytic infiltra

57 uccessful epilation was associated with less corneal opacity (odds ratio [OR], 0.61; 95% confidence i

58 an-American male presented with a congenital corneal opacity of the right eye.

59 l tool in cases when a child presents with a corneal opacity of unknown or unclear etiology.

60 No evidence of corneal opacities or renal insufficiency was detected in

61 tion with P. aeruginosa and showed increased corneal opacity, PMN infiltration, bacterial counts, and

62 treated with TLR9 siRNA showed decreases in corneal opacity, polymorphonuclear leukocyte number, IL-

63 line, nummular, patch-like, and lattice-like corneal opacities, prominent corneal vascularization was

64 Cataract corneal opacities, refractive error and amblyopia, globe

65 Four weeks postinfection (p.i.), the mean corneal opacity score of -/- mice was 1.1 +/- 0.3 while

66 endogenous IL-6 in IL-6(+/+) animals reduced corneal opacity scores and MIP-2 levels to that of IL-6(

67 ophil infiltration and significantly reduced corneal opacity scores.

68 b over control-treated mice showed increased corneal opacity, stromal damage, and bacterial load.

69 ctively reviewed for the primary etiology of corneal opacity, time of onset, duration of opacity, pre

70 feld-Rieger anomaly, including iris defects, corneal opacity, vacuolar cataracts, significant iris/co

71 Corneal opacity was measured using optical coherence tom

72 In the remaining animals corneal opacity was studied and digital photographs were

73 The commonest etiology of corneal opacity was trauma (62.5%), followed by resolved

74 a highly sensitive imaging method to measure corneal opacity, we identify a novel role for desmin ove

75 The corneal opacities were associated with increased exfolia

76 Although no overt changes in corneal opacity were detected by slit-lamp examination,

77 The 2 youngest patients had complete corneal opacity with features of corneal staphyloma and