ライフサイエンスコーパス: corneal perforation

1 e scored on a scale of 0 (normal eye) to +4 (corneal perforation).

2 g Th2 response development are resistant (no corneal perforation).

3 therapeutic keratoplasty, predominantly for corneal perforation.

4 Surgical alternative for the closure of corneal perforation.

5 scar size, time to re-epithelialization, and corneal perforation.

6 with uncontrolled fungal growth resulting in corneal perforation.

7 nterior chamber, and eventually resulting in corneal perforation.

8 tion of both NKT/NK cells results in earlier corneal perforation.

9 tive surgical alternative for the closure of corneal perforations.

10 new surgical alternative for the closure of corneal perforations.

11 followed by previous surgery (30/49 [61%]), corneal perforation (17/49 [35%]), dry eye (15/49 [31%])

12 mice, leading to a significant reduction in corneal perforation and improved disease outcome.

13 IFN-gamma before infecting B6 mice prevented corneal perforation and was associated with a lower dela

14 .28 times the odds of the patient developing corneal perforation and/or needing TPK (95% CI, 1.18-4.4

15 dentify those at highest risk for developing corneal perforation and/or needing TPK.

16 ior one-third had a 71.4% risk of developing corneal perforation and/or needing TPK.

17 stics that predict a high risk of developing corneal perforation and/or the need to undergo therapeut

18 es included 3-month infiltrate or scar size; corneal perforation and/or therapeutic penetrating kerat

19 ity at 3 months; secondary outcomes included corneal perforation and/or therapeutic penetrating kerat

20 atients with fungal keratitis, patients with corneal perforation, and patients with infectious kerati

21 cuity (BSCVA), 3-month infiltrate/scar size, corneal perforation, and re-epithelialization rates stra

22 ion, and 2 eyes of 2 patients presented with corneal perforation, both requiring a penetrating kerato

23 f resistant mice with TIMP-1 pAb resulted in corneal perforation by 5 to 7 days after infection (PI).

24 decreased corneal opacity and resistance to corneal perforation compared with PBS controls.

25 +) T cells produce IFN-gamma contributing to corneal perforation in C57BL/6 (B6) mice after Pseudomon

26 s associated with genetic susceptibility and corneal perforation in inbred B6 mice.

27 ction with Pseudomonas aeruginosa results in corneal perforation in susceptible C57BL/6 (B6) mice, bu

28 de I prevents Pseudomonas aeruginosa-induced corneal perforation in susceptible C57BL/6 mice.

29 to worsened disease as evidenced by earlier corneal perforation in the susceptible mouse strain.

30 e patients in the control group demonstrated corneal perforation; infection recurred in 1 of them.

31 Severe ocular side effects, including corneal perforation, may be associated with the use of t

32 In 1 case, corneal perforation occurred during the insertion of the

33 tment of corneal thinning was necessary, but corneal perforation occurred in two cases despite intens

34 rol group, whereas there was no incidence of corneal perforation or recurrence of the infection in th

35 primary outcome of the trial was the rate of corneal perforation or the need for therapeutic penetrat

36 The primary outcome of the trial was rate of corneal perforation or the need for therapeutic penetrat

37 Overall, no difference in the rate of corneal perforation or the need for TPK was determined f

38 e of this secondary analysis was the rate of corneal perforation or the need to undergo TPK.

39 fined as final visual acuity (VA) </= 20/80, corneal perforation, or need for keratoplasty.

40 was defined as final visual acuity </=20/80, corneal perforation, or the need for keratoplasty.

41 of mice were susceptible to infection, with corneal perforation seen at 5 to 7 days after infection.

42 Pseudomonas aeruginosa stromal keratitis and corneal perforation (susceptibility) is a CD4(+) T cell-

43 exhibited a less severe disease response (no corneal perforation) than wild-type B6 mice and had a si

44 and then the E-PRP clot was placed over the corneal perforation, underneath the fibrin membrane.

45 Corneal perforation was more common in cases (52.17% [12

46 In all cases the corneal perforation was sealed.

47 Patients with perforation/impending corneal perforation were excluded from the study.

48 however, significant differences leading to corneal perforation were seen in BALB/c mice that includ

49 FGF, and HGF after disease onset, prevented corneal perforation when compared with controls.