ライフサイエンスコーパス: corneal scar

1 bliteration of vessels with stabilization of corneal scar.

2 with postinfectious, full-thickness, central corneal scars.

3 source of fibrotic tissue in nontransparent corneal scars.

4 ages, 3 had post-herpetic leukoma, and 5 had corneal scars.

5 t virus that can cause recurrent disease and corneal scarring.

6 active outcome while eliminating or reducing corneal scarring.

7 n, PAI-1R may be a useful agent in combating corneal scarring.

8 ation of vision in patients with significant corneal scarring.

9 ology and for cell-based therapies targeting corneal scarring.

10 anglion and reduced herpetic blepharitis and corneal scarring.

11 action may be an important goal for reducing corneal scarring.

12 ly protected against HSV-1-induced death and corneal scarring.

13 rimary genes involved in the pathobiology of corneal scarring.

14 nse leading to exacerbation of HSV-1-induced corneal scarring.

15 ice are normally refractory to HSV-1-induced corneal scarring.

16 spite the fact that KOS normally produces no corneal scarring.

17 included partial-thickness anterior stromal corneal scars (15 eyes), Descemet membrane ruptures (6 e

18 causes of vision loss were cataract (19.7%), corneal scars (15.7%), refractive error and amblyopia (1

19 Main diagnoses were corneal scar (22.9%), limbal dermoid (21.9%), anterior s

20 eal neovascularization (44%), dry eye (38%), corneal scarring (26%), ectropion (25%), blepharitis (23

21 or keratoconus (8), microbial keratitis (6), corneal scar (6), corneal keloid (3), chemical injury wi

22 ggests that limiting TGFss system may reduce corneal scarring after excimer laser ablation.

23 ltrates is more than that resulting from the corneal scarring after healing.

24 s to naive mice, resulted in exacerbation of corneal scarring after HSV-1 challenge (P < 0.0001).

25 dividually to naive mice, and the affects on corneal scarring after HSV-1 challenge were determined.

26 -nine percent of patients with keratitis had corneal scarring and 26% had vision of 20/40 or worse at

27 lammatory response that leads to progressive corneal scarring and blindness.

28 hlight the most important causes of acquired corneal scarring and infiltrates in children.

29 The converse was true for corneal scarring and phthisis (14.0% and 27.3%, respecti

30 If extensive corneal scarring and/or limbal stem cell deficiency are

31 , virus replication in the eye, blepharitis, corneal scarring, and dermatitis were determined.

32 plication in the eye, survival, blepharitis, corneal scarring, and latency were determined.

33 ial basement membrane dystrophy, superficial corneal scars, and previous radial keratotomy will have

34 lindness and visual impairment, particularly corneal scarring as a result of vitamin A deficiency, co

35 able refractive outcomes in the treatment of corneal scarring associated with Bowman layer irregulari

36 The charts of 22 patients with anterior corneal scarring associated with irregularities in the B

37 leusis and decreasing postoperative pain and corneal scarring associated with PRK.

38 susceptible to exacerbation of HSV-1-induced corneal scarring by gK vaccination (P < 0.0001).

39 ra copies of glycoprotein K (gK) exacerbated corneal scarring (CS) in mice.

40 pe 1 (HSV-1) glycoprotein K (gK) exacerbated corneal scarring (CS) in ocularly infected mice.

41 Indeed, a major cause of corneal scarring (CS) is the scarring induced by HSV-1 f

42 ected mouse strains had significantly higher corneal scarring (CS) than did McKrae-infected mice.

43 in KOS or McKrae, and the relative amount of corneal scarring determined 28 days after challenge.

44 ta support the hypothesis that CTGF promotes corneal scar formation and imply that regulating CTGF sy

45 have important implications in regulation of corneal scar formation.

46 ges did not alter the HSV-1-induced death or corneal scarring in immunized mice.

47 mice during acute infection and caused more corneal scarring in latently infected rabbits.

48 ls, increased 50% lethal dose, and decreased corneal scarring in ocularly infected mice compared to t

49 Fifty-three percent had corneal scarring in one or both eyes.

50 related with higher 50% lethal dose and less corneal scarring in vivo.

51 glycan expression pattern similar to that of corneal scars in vivo.

52 Vision loss in children is from corneal scarring leading to deprivation and/or refractiv

53 herpetic episodes is high, and the resultant corneal scarring may require penetrating keratoplasty fo

54 e that with the addition of TGF-beta1, a 3-D corneal scar model can be generated.

55 ficant corneal irregularities and/or central corneal scarring often secondary to long-standing preope

56 The most important causes of corneal scarring or infiltrates in children are keratoco

57 ervous system involvement, acral mutilation, corneal scarring or ulceration, liver failure, and metab

58 eal involvement without scarring), moderate (corneal scarring), or severe (corneal scarring with thin

59 cinated C57BL/6 mice resulted in significant corneal scarring (P < 0.0001), despite the fact that C57

60 ccinated BALB/c mice resulted in significant corneal scarring (P = 0.0003), despite the fact that KOS

61 conjunctival corkscrew vessels (P < 0.001), corneal scarring (P = 0.01) and pingueculae under the ag

62 oblasts could be important in regulating the corneal scarring process.

63 How anti-gK IgG exacerbated HSV-1 induced corneal scarring remains to be determined.

64 In case 1 (corneal scarring) the OCT images helped to guide manual

65 ntibody titers (approximately 1:800-1:1200), corneal scarring (trace) and survival (100%) were simila

66 Presence of residual corneal scarring, visual acuity at the last visit, chang

67 Its main outcome measures are corneal scarring, visual acuity, keratometry, and qualit

68 ith corneal hydrops, whereas the presence of corneal scarring was a preventive factor.

69 f gK vaccination to exacerbate HSV-1-induced corneal scarring was not mouse strain or HSV-1 strain sp

70 Central corneal scarring was present in 1436 (33%) eyes.

71 The gK-induced exacerbation of corneal scarring was related to anti-gK IgG.

72 Corneal scarring, whether caused by trauma, laser refrac

73 ng), moderate (corneal scarring), or severe (corneal scarring with thinning or perforation) disease b