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1 stantial mismatch between need and supply of corneal transplant.
2 atients who had undergone a unilateral first corneal transplant.
3 een accidentally transmitted by contaminated corneal transplants.
4 asing success in patients who have undergone corneal transplants.
5 ved in generating the alloimmune response to corneal transplants.
6 osis in the failure of orthotopic allogeneic corneal transplants.
7 layed-type hypersensitivity and rejection of corneal transplants.
8 le hope of visual recovery from conventional corneal transplants.
9 or histocompatibility complex MHC-mismatched corneal transplants.
10 measures were follow-up inquiries from 6592 corneal transplants.
11 hown to participate in long-term survival of corneal transplants.
13 e over time during the study period for both corneal transplant and cataract surgery procedures; howe
15 thways can significantly promote survival of corneal transplants, and suggest that select deletion or
18 ation when they were recipients of syngeneic corneal transplants but also exhibited significantly inc
19 a major barrier to the success of allogeneic corneal transplants, but the specific mediators and mech
20 mpared with the cataract surgery cohort, the corneal transplant cohort had a higher adjusted hazard r
21 ence rates ranged from 0.11% to 1.05% in the corneal transplant cohort, 0.06% to 0.20% in the catarac
24 nex and enzyme-linked immunosorbent assay on corneal transplant extracts at different times after sur
25 rgeons enrolled 1090 participants undergoing corneal transplant for a moderate-risk condition, princi
26 ents were included who had undergone a first corneal transplant for keratoconus (KC), Fuchs endotheli
30 rategies including NSAIDs, steroids, MMC and corneal transplants have shown tremendous success but wi
32 APC-dependent direct response is elicited in corneal transplant hosts when the graft bed is inflamed
39 he trend of infectious endophthalmitis after corneal transplant or cataract surgery through examining
45 if registering more than 50 and less than 10 corneal transplants per year, respectively; surgeons wer
48 indications were Fuchs dystrophy (39% of all corneal transplants performed), a primary corneal edema
49 VEGF-C as a potentially important target in corneal transplant pharmacotherapy and immunobiology.
50 s than either normal controls (P = 0.017) or corneal transplant recipients not undergoing graft rejec
51 imulatory pathway promotes the acceptance of corneal transplants, regardless of the degree of allodis
53 ic to EMAP between patients at high risk for corneal transplant rejection and control subjects (P<0.0
55 upports allograft survival in vivo, prevents corneal transplant rejection, and attenuates the progres
56 cal lymphangiogenesis mediates diseases like corneal transplant rejection, dry eye disease, and aller
59 s and below were included from the Singapore Corneal Transplant Study between April 4, 1991 and April
67 L-1 receptor antagonist (IL-1ra) can prolong corneal transplant survival in the murine model of ortho
70 4+ T cells and CD8+ T cells were examined in corneal transplants that expressed genetically defined m
71 survival of both minor H- and MHC-disparate corneal transplants to 100% (P = 0.0001) and 92% (P = 0.
72 antigen-mismatched (BALB.b; n=62) orthotopic corneal transplants to determine the effect of selective
73 Next, we investigated whether failure of corneal transplants to induce a CD4+ direct alloresponse
77 mune privileged site is the success of human corneal transplants, where a very high percentage of tra
78 ion and corneal graft rejection in high-risk corneal transplants with antecedent rejection or neovasc
79 bling clinicians and researchers to generate corneal transplants with sufficiently high fractions of
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