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1 iation, and a specialized form of apoptosis (cornification).
2 sslinking enzymes essential for keratinocyte cornification.
3 sions of the oviduct; and persistent vaginal cornification.
4 g little or no sebum and undergoing abnormal cornification.
5 parently brought about by early keratinocyte cornification.
6 cts on infundibular keratinocytes to promote cornification.
7 ln/Lys-rich domains, and is likely to impair cornification.
9 gmenting JNK activity could be used to delay cornification and enhance wound healing, whereas attenua
10 ed with execution of the genetic programs of cornification and epidermal barrier formation, to a full
11 ed by ARNT, which have critical roles in the cornification and epidermal barrier function of the skin
12 ogical analysis showed that all EFNAs induce cornification and keratin genes while suppressing wound
13 imination by shedding, keratinocyte death by cornification, and cell-cell cannibalism by entosis.
14 er, the pan-Casp inhibitor Z-VAD-FMK delayed cornification, and corneocytes were structurally aberran
16 sensorineural hearing loss and disorders of cornification, and screening of several connexin genes w
17 ,15-EET epoxygenases in regulating epidermal cornification, and they have important implications for
19 by two unrelated methods initiated a wave of cornification, assessed as TdT-mediated dUTP nick end-la
21 -mediated hydrolysis of phospholipids during cornification, contributes to stratum corneum acidificat
24 eciphering of the molecular basis of various cornification disorders, less is currently known about t
25 perproliferation of the epidermis, disturbed cornification, fragile cornified envelope (CE, a skin ba
27 rders characterized by erythema and abnormal cornification, including erythrodermic psoriasis, atopic
28 target of Blimp-1 repression indicates that cornification involves suppression of normal osmotic reg
30 al differentiation in vivo: transcription of cornification markers, inhibition of motility, withdrawa
33 the breakdown and degradation of DNA during cornification of lingual keratinocytes and aberrant DNA
34 tial therapeutic implications for preventing cornification of the corneal epithelium in response to t
37 ition to blistering and a severe disorder of cornification, patients typically display an abnormality
41 that inhibit JNK production downregulate the cornification response of PCHCE cells to osmotic stress.
44 ase inhibitor z-VAD, suggesting that reduced cornification was not entirely because of premature apop
45 f a complex differentiation process known as cornification, which culminates with the formation of th
46 roup of rare autosomal dominant disorders of cornification with overlapping features including hyperk
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