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1  good and rapid method for evaluation of the coronary anatomy and for early detection and grading of
2                                         Hard coronary and cardiovascular events, and total cardiovasc
3 ACb progressed from 1 to 399 to CAC5y>/=400, coronary and total cardiovascular risk were nearly 2-fol
4 ome because patients have generally a normal coronary angiogram and left ventricular dysfunction, whi
5 ercise stress test, computerized tomographic coronary angiogram, and cardiovascular magnetic resonanc
6 ssing creatinine values, no or an incomplete coronary angiogram, or previous dialysis.
7 ients were subjected to CSA procedure during coronary angiography (CSA group), and 25 patients served
8 ulness of screening with computed tomography coronary angiography (CTCA), and assess the safety and e
9 mly assigned to radial or femoral access for coronary angiography and percutaneous intervention, and
10                          Subsequent rates of coronary angiography and revascularization after stress
11  severity was quantified in each subject via coronary angiography by calculating a CAD score.
12  trials of routine versus selective invasive coronary angiography have high rates of crossover from c
13                                Compared with coronary angiography performed soon after recanalization
14 nting a case of critical hand ischemia after coronary angiography performed through radial access des
15                                           CT coronary angiography revealed positive coronary artery d
16 raphy by visual estimate and by quantitative coronary angiography when compared with FFR and evaluate
17 y manifestation, with indications to undergo coronary angiography with intent to perform percutaneous
18 ical outcome of patients with ACS undergoing coronary angiography, as compared with patients with sta
19                               They underwent coronary angiography, endothelial function testing; meas
20 rospective registry of patients referred for coronary angiography, the goal of this study was to deve
21                              Since the first coronary angioplasty on Sept 16, 1977, the field of perc
22 cardiovascular event (myocardial infarction, coronary angioplasty, coronary artery bypass graft surge
23 red for noninvasive testing will have normal coronary arteries and no long-term clinical events.
24  (LCx), right coronary artery, and all three coronary arteries combined were compared with microspher
25 d in EC overlying atherosclerotic plaques in coronary arteries from patients with ischemic heart dise
26 on of critical plaque pathophysiology in the coronary arteries.
27 ucers SMAD1/5/8 were activated in developing coronary arteries.
28 e of myocardial ischemia with no obstructive coronary arteries.
29 ary blood flow and vasodilatory responses of coronary arterioles were evaluated in all groups at the
30 idance when performing unprotected left main coronary artery (LMCA) percutaneous coronary interventio
31 as obtained during a 1-minute proximal right coronary artery (RCA) and left coronary artery balloon o
32 s beyond the territory subtended by a single coronary artery and recovers within days or weeks.
33                                              Coronary artery anomalies may cause life-threatening car
34 namically significant stenosis in at least 1 coronary artery as indicated by a fractional flow reserv
35 roximal right coronary artery (RCA) and left coronary artery balloon occlusion at baseline before and
36 without acute coronary syndromes or previous coronary artery bypass graft surgery in periods before (
37 artery (RA) can improve clinical outcomes in coronary artery bypass graft surgery remains unclear.
38 nts with no acute coronary syndrome/no prior coronary artery bypass graft surgery that were rated as
39 myocardial infarction, coronary angioplasty, coronary artery bypass graft surgery, stroke).
40 onary intervention, or previous multi-vessel coronary artery bypass graft surgery.
41 lectomy (189229 patients at 1876 hospitals), coronary artery bypass grafting (CABG) (218940 patients
42  unclear whether revascularization by either coronary artery bypass grafting (CABG) or percutaneous c
43 ubgroup analysis of patients undergoing only coronary artery bypass grafting, and results were simila
44  should be considered more frequently during coronary artery bypass grafting.
45 utaneous coronary intervention (28%), urgent coronary artery bypass surgery (27.5%), maternal mortali
46                                              Coronary artery calcification (CAC) is highly prevalent
47                                              Coronary artery calcification (CAC) may impair diagnosti
48 ciation between the polygenic risk score and coronary artery calcification (CARDIA) and carotid arter
49            At baseline, 70 men (50.7%) had a coronary artery calcification score higher than 300 Agat
50 , iliofemoral, and abdominal aortic plaques; coronary artery calcification; serum biomarkers; and lif
51                      It is not known whether coronary artery calcium (CAC) assessment at baseline in
52         To examine the predictive ability of coronary artery calcium (CAC) score vs age for incident
53 sessed the relationships among adult height, coronary artery calcium (CAC) score, incident atheroscle
54                                              Coronary artery calcium score was more likely than age t
55 test declining primary cardiac diagnosis was coronary artery disease (32.3%-19.0%; P<0.001).
56 lated with QTc prolongation in patients with coronary artery disease (CAD) and investigate the effect
57   Whether ANGPTL3 deficiency reduces risk of coronary artery disease (CAD) is unknown.
58 tly associated with cardiovascular events in coronary artery disease (CAD) patients and reducing the
59                                              Coronary artery disease (CAD) severity was quantified in
60 rmediate pretest probability for obstructive coronary artery disease (CAD) were randomly assigned to
61 etically higher calcium had a higher risk of coronary artery disease (CAD), myocardial infarction (MI
62 r mixed methods investigation (compared with coronary artery disease and hypertension).
63  other or medical treatment in patients with coronary artery disease and left ventricular ejection fr
64 ated to treatment (two in the control group [coronary artery disease and multiorgan failure] and thre
65 he context of a recent GWAS meta-analysis of coronary artery disease and provide a list of targeted e
66                                         In a coronary artery disease cohort separate from volunteers
67  pain and had a lower pretest probability of coronary artery disease compared with men.
68    CT coronary angiography revealed positive coronary artery disease findings in 16 patients; LAD was
69 -wide Replication and Meta-analysis Plus the Coronary Artery Disease Genetics (CardiogramplusC4D) con
70 s (N = up to 61079 individuals) and from the Coronary Artery Disease Genome-wide Replication and Meta
71                       Eligible patients with coronary artery disease had to have had a myocardial inf
72 ing process of ACS patients with obstructive coronary artery disease is associated with a high reclas
73  ASA sensitivity with known/suspected stable coronary artery disease or presenting with an acute coro
74 ictors of adverse cardiovascular outcomes in coronary artery disease patients.
75 ic role of elevated WBC across a spectrum of coronary artery disease presentations are warranted.
76  55+/-10 years), mostly with an intermediate coronary artery disease probability, between cardiac CT
77  hemorrhagic shock in swine with preexisting coronary artery disease reduced renal dysfunction and ca
78 e genetic association between rs11556924 and coronary artery disease risk by characterizing its effec
79 e-industrial lifestyle and low prevalence of coronary artery disease risk factors, we examined the Ts
80 similar age, sex, and low Framingham 10-year coronary artery disease risk scores with an echocardiogr
81 ing the total number of loci associated with coronary artery disease to 95 at the time of analysis.
82 reserve with best MT in patients with stable coronary artery disease to assess clinical outcomes and
83 mparison to Functional Testing for Suspected Coronary Artery Disease) prospectively randomized 350 pa
84 gists by 2 1 method required the presence of coronary artery disease, a common interpretation of the
85 outh), BMI, height, systolic blood pressure, coronary artery disease, and type 2 diabetes using data
86 sterol, low-density lipoprotein cholesterol, coronary artery disease, C-reactive protein, HbA1c, heig
87  undergoing initial evaluation for suspected coronary artery disease, coronary CTA was associated wit
88 nfarction in the past 20 years, multi-vessel coronary artery disease, history of stable or unstable a
89 n risk for five vascular diseases, including coronary artery disease, migraine headache, cervical art
90 stable outpatients presenting with suspected coronary artery disease, most patients experiencing clin
91 ion (T2MI2007); and 1 method did not require coronary artery disease, the 2012 universal definition (
92 utpatients (age: 63+/-9 years; 76% men) with coronary artery disease.
93          This paper reports on patients with coronary artery disease.
94 re, cardiac masses, pericardial disease, and coronary artery disease.
95 r against each other in patients with stable coronary artery disease.
96 recludes assessing physiological severity of coronary artery disease.
97 raphy, as compared with patients with stable coronary artery disease.
98 large cohort of patients suspected of having coronary artery disease.
99  stenosis and low to intermediate-complexity coronary artery disease.
100 irin (ASA) desensitization for patients with coronary artery disease.
101 .G202V HAND2 variant associated with CHD and coronary artery diseases found in a large Lebanese famil
102 ake of normal and PE STBEVs by primary human coronary artery endothelial cells (HCAEC) and the effect
103 NFalpha and cigarette smoke extract on human coronary artery endothelial cells under oscillatory, nor
104 wever, developmental mechanisms underpinning coronary artery formation remain ill-defined.
105 ater sensitivity to PGF2alpha in the porcine coronary artery from males.
106 ta support a model in which DACH1 stimulates coronary artery growth by activating Cxcl12 expression a
107  to restore blood flow in an infarct-related coronary artery improves outcomes.
108  B6sv129-mice were subjected to in vivo left coronary artery ligation for 30 minutes followed by 72 h
109 ale C57BL/6 mice were subjected to permanent coronary artery ligation.
110       Adult ANX - A1 (-/-) mice subjected to coronary artery occlusion exhibited increased infarct si
111 e group receiving PCI for an infarct-related coronary artery only.
112         The primary outcome was noncalcified coronary artery plaque volume, as determined by coronary
113 stosterone slows progression of noncalcified coronary artery plaque volume.
114 e been used in clinical research for imaging coronary artery plaque, and ongoing clinical studies are
115 d white men and women from the observational Coronary Artery Risk Development in Young Adults study,
116  and 2 observational cohort studies (CARDIA [Coronary Artery Risk Development in Young Adults] and Bi
117 e ACS that occur without apparent epicardial coronary artery thrombus or stenosis.
118 the LAD, left circumflex artery (LCx), right coronary artery, and all three coronary arteries combine
119 raphy (IVUS) of the left anterior descending coronary artery, within 8 weeks of HT.
120 ence imaging detected nanoparticles in human coronary artery-sized atheroma in vivo (P<0.05 versus re
121 ) is an index used to assess the severity of coronary-artery stenosis.
122 dhood obesity with obesity and complexity of coronary atherosclerosis (SYNTAX score) in a cohort of 3
123                Metformin may protect against coronary atherosclerosis in prediabetes and early diabet
124 CL5 and molecular phenotypes associated with coronary atherosclerosis severity in patients at least 6
125 le in chronic inflammatory diseases, such as coronary atherosclerosis, is not well defined.
126  with myocardial dysfunction and accelerated coronary atherosclerosis.
127 HDL cholesterol concentrations, obesity, and coronary atherosclerosis.
128 fetime AAS dose was strongly associated with coronary atherosclerotic burden (increase [95% confidenc
129 ete ANGPTL3 deficiency showed no evidence of coronary atherosclerotic plaque.
130                        Results Mean measured coronary attenuation values +/- standard deviation were
131 actional flow reserve (FFR) in patients with coronary bifurcation lesion.
132 ew possibilities to measure maximal absolute coronary blood flow and minimal microcirculatory resista
133                                              Coronary blood flow and vasodilatory responses of corona
134 oglycerin causes changes in the systemic and coronary circulation that combine to reduce myocardial o
135 microvasculature but interrogates the entire coronary circulation.
136              The primary study end point was coronary collateral flow index as obtained during a 1-mi
137 ders at 193 North American sites interpreted coronary computed tomographic (CT) angiography as part o
138                                              Coronary computed tomographic (CT) angiography has emerg
139 bstructive LM CAD was frequently detected on coronary computed tomographic angiography and strongly a
140 onary artery plaque volume, as determined by coronary computed tomographic angiography.
141 tial noninvasive cardiac testing with either coronary computed tomography angiography (CTA) or functi
142 clear stress, or stress echocardiography) or coronary computed tomography angiography (CTA).
143 on (CAC) may impair diagnostic assessment of coronary computed tomography angiography (CTA).
144                       They underwent ICA and coronary CT angiography performed with a whole-heart CT
145 n Coronary Opacification and Heart Rhythm in Coronary CT Angiography, or IsoCOR, trial.
146 rocedures, and medications were higher after coronary CTA ($995 vs. $718; p < 0.001).
147 ation for suspected coronary artery disease, coronary CTA was associated with greater use of statins,
148 (HR: 1.95; 95% CI: 1.72 to 2.21), unheralded coronary death (HR: 1.78; 95% CI: 1.51 to 2.10), abdomin
149  beverage intake with risk of mortality from coronary diseases, diabetes, or cancer, but few studies
150              Clarification of the origins of coronary endothelial cells during cardiac repair is esse
151 eract with SES to influence CAC and incident coronary events in a population-based cohort.
152 t fluctuation is a risk factor for death and coronary events in patients without cardiovascular disea
153                                     Incident coronary events were ascertained over a median follow-up
154  genetic effects on loge(CAC+1) and incident coronary events.
155  value of noninvasively assessing indices of coronary flow for diagnosing coronary microvascular dysf
156 rements of fractional flow reserve (FFR) and coronary flow reserve (CFR) and the index of microcircul
157 idated for absolute myocardial perfusion and coronary flow reserve (CFR) by positron emission tomogra
158                         Doppler wire-derived coronary flow reserve has been applied in research studi
159 using speckle-tracking echocardiography, (2) coronary flow reserve, (3) pulse wave velocity and augme
160 rmine the long-term risks of acute and fatal coronary heart disease (CHD) events after sepsis hospita
161 telomere length (TL) to be a risk factor for coronary heart disease (CHD), and recently the associati
162  differences of incident heart failure (HF), coronary heart disease (CHD), and stroke in participants
163  consumption to blood lipid levels and hence coronary heart disease (CHD).
164 , 58.5 years; 39% women) with 62240 cases of coronary heart disease (CHD).
165 accepted model to predict outcomes in stable coronary heart disease (CHD).
166  HDL defined by apoC-III are associated with coronary heart disease (CHD).
167 has been associated with type 2 diabetes and coronary heart disease (CHD).
168 .99 to 1.32), but higher risks of death from coronary heart disease (HR: 1.45; 95% CI: 1.21 to 1.74),
169 PHDL-C=0.008 and Ptriglycerides=0.00003) and coronary heart disease (P=0.0007).
170 ntration with first-ever CVD outcomes (i.e., coronary heart disease [CHD], stroke, or the combination
171 hat together enrolled 4726 participants with coronary heart disease and 3529 controls.
172 recognized as an independent risk factor for coronary heart disease and cardiovascular mortality.
173                        Chinese patients with coronary heart disease and impaired glucose tolerance we
174     INTERPRETATION: In Chinese patients with coronary heart disease and impaired glucose tolerance, a
175  century, then the decline in mortality from coronary heart disease and stroke has been the success s
176            Outcomes included cumulative CVD (coronary heart disease and stroke) deaths prevented or p
177 /=190 mg/dL, pravastatin reduced the risk of coronary heart disease by 27% (P=0.033) and major advers
178 rs), 1203 incident CVD events, including 916 coronary heart disease cases, were reported.
179 ring the initial trial phase and the risk of coronary heart disease death, cardiovascular death, and
180 d nongenetic effects on the indexing (first) coronary heart disease event.
181 easure to predict progression to death after coronary heart disease is established.
182 the effect of the genetic score on decreased coronary heart disease risk extended beyond its effect o
183 mellitus and prediabetes remain at increased coronary heart disease risk.
184 tive cohorts, carriers of CHIP had a risk of coronary heart disease that was 1.9 times as great as in
185 lood cells and associated such presence with coronary heart disease using samples from four case-cont
186 hat the CETP inhibitor anacetrapib decreased coronary heart disease when added to statin therapy.
187             Despite the reduced incidence of coronary heart disease with intensive risk factor manage
188 ariants at the CXCR4 locus with the risk for coronary heart disease, along with CXCR4 transcript expr
189 ad suffered from CVD, 4.9% had suffered from coronary heart disease, and 2.6% had experienced a strok
190 dent myocardial infarction or death owing to coronary heart disease, and stroke, defined as the first
191  recommendations for patients with prevalent coronary heart disease, and we offer recommendations, wh
192                 Incident CVD, which combines coronary heart disease, defined as the first incident my
193               The hazard ratios for incident coronary heart disease, stroke, and CVD associated with
194  are independent and causal risk factors for coronary heart disease.
195 y of APOC3 has been shown to protect against coronary heart disease; we identified APOC3 homozygous p
196 nfirmed that (68)Ga-DOTATATE offers superior coronary imaging, excellent macrophage specificity, and
197 ent in patients with angina and unobstructed coronaries in a European population.
198  comprised CVD death, myocardial infarction, coronary insufficiency, index admission for heart failur
199 echanical support (28%), urgent percutaneous coronary intervention (28%), urgent coronary artery bypa
200 s with STEMI undergoing primary percutaneous coronary intervention (n=1604; mean age, 61+/-12 years;
201 rs of its improvement after CTO percutaneous coronary intervention (PCI) are unknown.
202 rtery bypass grafting (CABG) or percutaneous coronary intervention (PCI) carries benefits or risks in
203 and white patients treated with percutaneous coronary intervention (PCI) in the Veterans Affairs (VA)
204                         Primary percutaneous coronary intervention (PCI) may therefore be less benefi
205                                 Percutaneous coronary intervention (PCI) of saphenous vein grafts (SV
206  infarction (STEMI), the use of percutaneous coronary intervention (PCI) to restore blood flow in an
207 eft main coronary artery (LMCA) percutaneous coronary intervention (PCI), but the overall picture rem
208 d minorities after contemporary percutaneous coronary intervention (PCI).
209 Therapy in Subjects Who Require Percutaneous Coronary Intervention [PCI] [CHAMPION PHOENIX] [CHAMPION
210                                 Percutaneous coronary intervention and CABG show comparable safety in
211  among 1919 patients undergoing percutaneous coronary intervention at 76 centers.
212 relief of coronary obstruction, percutaneous coronary intervention has become a standard-of-care proc
213 w marking its 40th anniversary, percutaneous coronary intervention has become one of the most common
214  on Sept 16, 1977, the field of percutaneous coronary intervention has evolved rapidly.
215 pulation of patients undergoing percutaneous coronary intervention in the contemporary era.
216 y), 4222 patients who underwent percutaneous coronary intervention in the United States and Europe be
217 e acute stage, intended primary percutaneous coronary intervention increased from 12% (1995) to 76% (
218 ciated with changes in rates of percutaneous coronary intervention or in-hospital mortality in New Yo
219 ETHODS AND We analyzed 1253 CTO percutaneous coronary intervention procedures performed according to
220 ly has led to worse outcomes in percutaneous coronary intervention procedures performed through the t
221 ral complexity, shorter primary percutaneous coronary intervention time was associated with an increa
222   Patients (n=15 003) underwent percutaneous coronary intervention to SVG in England and Wales during
223                                 Percutaneous coronary intervention was performed in 235 patients (71%
224 ction (MI) treated with primary percutaneous coronary intervention were randomized to prasugrel or ti
225 ete revascularization following percutaneous coronary intervention were randomized to ranolazine vers
226 rates of ischemic events during percutaneous coronary intervention without an increase in severe blee
227 ariables (male sex and previous percutaneous coronary intervention) and 4 biomarkers (midkine, adipon
228 omplete Revascularization after Percutaneous Coronary Intervention) trial, a clinical trial in which
229 s and additionally into primary percutaneous coronary intervention, fibrinolysis, or no reperfusion.
230 e angina, previous multi-vessel percutaneous coronary intervention, or previous multi-vessel coronary
231 y survive the first month after percutaneous coronary intervention, their prognosis is comparable to
232 30 patients were transported to percutaneous coronary intervention-capable hospitals, including 974 i
233 tcomes of patients treated with percutaneous coronary intervention.
234 iography with intent to perform percutaneous coronary intervention.
235 rted mortality statistics after percutaneous coronary intervention.
236 yocardial infarction undergoing percutaneous coronary intervention.
237 metal in-stent restenosis after percutaneous coronary intervention.
238 sess the safety and efficacy of percutaneous coronary interventions (PCIs).
239 n patients treated with primary percutaneous coronary interventions.
240 nfarction in the era of primary percutaneous coronary interventions.
241                      MDCT angiography of the coronaries is a good and rapid method for evaluation of
242 atomy and for early detection and grading of coronary lesions in non-diabetic patients.
243 er to discriminate high-risk versus low-risk coronary lesions than [(18)F]FDG.
244 nt interventional tool for heavily calcified coronary lesions.
245        A subgroup of these patients also has coronary microvascular dysfunction and evidence of infla
246 sing indices of coronary flow for diagnosing coronary microvascular dysfunction; in certain diseases,
247 function; in certain diseases, the degree of coronary microvascular impairment carries important prog
248                                For relief of coronary obstruction, percutaneous coronary intervention
249 ythmias (VAs) were subjected to percutaneous coronary occlusion to induce myocardial infarction.
250 and angina pectoris during the same 1-minute coronary occlusion.
251 the Effect of Iso-osmolar Contrast Medium on Coronary Opacification and Heart Rhythm in Coronary CT A
252  single-center cohort of patients undergoing coronary or peripheral angiography with or without inter
253 djustments for age, sex, study site, primary coronary percutaneous intervention (PCI), and norepineph
254  patients with psoriasis had increased total coronary plaque burden (1.22+/-0.31 versus 1.04+/-0.22,
255                  Throughout exercise, distal coronary pressure and flow velocity and central aortic p
256 th statin therapy (WOSCOPS [West of Scotland Coronary Prevention Study]; n=4910) and 2 observational
257 ores in the mid-range (4 to 6) indicate that coronary revascularization may be appropriate for the cl
258  Recurrent MI, CHD events (recurrent MI or a coronary revascularization procedure), and mortality wer
259 ular outcomes (death, myocardial infarction, coronary revascularization, or cerebrovascular events) i
260 nary syndrome without myocardial infarction, coronary revascularization, or CHD death.
261 he primary end point was freedom from repeat coronary revascularization.
262  linking common variants in this region with coronary risk is not known.
263                               Image quality, coronary segment interpretability, effective dose (ED),
264 treated due to an inability or impediment to coronary sinus (CS) lead implantation.
265 es additional epicardial ablation within the coronary sinus.
266 tivation and subsequent differentiation into coronary smooth muscle, and restores Wt1 activity upon M
267 rrelation between angiographic assessment of coronary stenoses and fractional flow reserve (FFR) is w
268 FR), an index of the hemodynamic severity of coronary stenoses, is derived from invasive measurements
269 characteristic curve of 0.87 (p < 0.001) for coronary stenosis >/=70%.
270                    Individuals who underwent coronary stenting and completed 12 months of thienopyrid
271 s older than 18 years with an indication for coronary stenting were enrolled, and 11648 (mean age, 61
272  to millions of patients worldwide following coronary stenting.
273                                Bioresorbable coronary stents have been introduced into clinical pract
274 tiplatelet and anticoagulant agents, and new coronary stents will continue the journey to achieve thi
275  the incidence of AKI in patients with acute coronary syndrome (ACS) enrolled in the MATRIX-Access (M
276        Of the 310 subjects, 138 had an acute coronary syndrome (ACS), 101 of whom underwent desensiti
277 s nonculprit arteries in patients with acute coronary syndrome (median difference: 0.69; interquartil
278 patients with non-ST-segment elevation acute coronary syndrome (NSTE-ACS) and an elevated cardiac tro
279 of Treatment Patterns and Events After Acute Coronary Syndrome (TRANSLATE-ACS) study between April 1,
280 between WBC and MACE was consistent in acute coronary syndrome and non-acute coronary syndrome presen
281                  Patients admitted for acute coronary syndrome and/or revascularization, with >/=1 LR
282          TTS, however, differs from an acute coronary syndrome because patients have generally a norm
283 centrations in patients with suspected acute coronary syndrome in which the diagnosis was adjudicated
284 rt-term mortality and complications in acute coronary syndrome patients treated with extracorporeal c
285 ent in acute coronary syndrome and non-acute coronary syndrome presentations (interaction P=0.15).
286 tal or nonfatal myocardial infarction, acute coronary syndrome without myocardial infarction, coronar
287 mong patients with stable angina or an acute coronary syndrome, an iFR-guided revascularization strat
288 heart failure, and hospitalization for acute coronary syndrome, and the incidence of acute pancreatit
289 abilized individuals within 10 days of acute coronary syndrome, combination therapy seemed to be more
290                           In suspected acute coronary syndrome, high-sensitivity cardiac troponin ide
291 y artery disease or presenting with an acute coronary syndrome, including ST-segment-elevation myocar
292 , with or without ST-segment elevation acute coronary syndrome, were randomly assigned to radial or f
293 n therapy in patients stabilized after acute coronary syndrome.
294 tal number of PCIs in patients with no acute coronary syndrome/no prior coronary artery bypass graft
295 formed in New York in patients without acute coronary syndromes or previous coronary artery bypass gr
296 zed participants with non-ST-elevation acute coronary syndromes or stable angina and to evaluate long
297 ve Versus Conservative Treatment in Unstable Coronary Syndromes) trial compared early invasive strate
298 tes mellitus, and 39.7% presented with acute coronary syndromes.
299 temperature through a dedicated catheter for coronary thermodilution induces steady-state maximal hyp
300 ctors of >/=70% stenosis in at least 1 major coronary vessel were identified from >200 candidate vari

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