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1 evascularization (ie, abnormal postoperative coronary angiogram).
2 lar Health (LURIC) study (2579 patients with coronary angiograms).
3 ronary flow velocity reserve assessment, and coronary angiogram.
4 sion could not help in the decision of early coronary angiogram.
5 ng cardiovascular risk factor assessment and coronary angiogram.
6 ogram), scintigraphic perfusion defects, and coronary angiogram.
7 Flt-1 in 791 HF patients undergoing elective coronary angiogram.
8 The patient who developed an MI had a normal coronary angiogram.
9 cipients at the time of their routine annual coronary angiogram.
10 ain in patients with hypertension and normal coronary angiograms.
11 ry flow reserve abnormalities despite normal coronary angiograms.
12 wenty-two patients had 91 DSE studies and 45 coronary angiograms.
13 n 7 patients without risk factors and normal coronary angiograms.
14 on of 15:1 lossy JPEG compression to digital coronary angiograms.
15 opment of chest pain in patients with normal coronary angiograms.
16 r three years of follow-up (30 percent fewer coronary angiograms, 15 percent fewer coronary angioplas
17       Patients in group A and B had a normal coronary angiogram and a coronary flow reserve (CFR) of
18 delivery, in spite of a normal pretransplant coronary angiogram and good pancreas function.
19 ome because patients have generally a normal coronary angiogram and left ventricular dysfunction, whi
20 e SS is a purely anatomic score based on the coronary angiogram and predicts outcome after PCI in pat
21 sted of 298 unrelated patients with positive coronary angiograms and controls were 138 unrelated heal
22 cluded 988 consecutive patients who had both coronary angiograms and echocardiographic examinations i
23 ccess and transfemoral access for diagnostic coronary angiograms and percutaneous coronary interventi
24 e clinical characteristics, imaging results, coronary angiograms and revascularization outcomes were
25 se BAV registry (n=600, n=231 with available coronary angiogram) and compared with the frequency of C
26 ercise stress test, computerized tomographic coronary angiogram, and cardiovascular magnetic resonanc
27 positive stress test result (n=67) underwent coronary angiogram, and significant coronary artery dise
28 ostic test for obstructive CAD with invasive coronary angiogram as a reference standard.
29            These data suggest that using the coronary angiogram as the arbiter for the presence of LV
30  was presence of coronary culprit lesions on coronary angiograms as analyzed by independent intervent
31  baseline, early postoperative and follow-up coronary angiograms, as well as a subset of 47 patients
32 went a 64-slice coronary computed tomography coronary angiogram at the time of phlebotomy, on average
33 s prospectively whether patients with normal coronary angiograms but with impaired myocardial blood f
34                       Compression of digital coronary angiograms facilitates playback of images and d
35                                              Coronary angiograms from 350 patients randomized to the
36                                              Coronary angiograms from 39 patients undergoing coronary
37  To examine this, we obtained posttransplant coronary angiograms from a group of patients bridged wit
38                                              Coronary angiograms from patients with asymptomatic AECG
39 igh negative predictive value of a normal CT coronary angiogram, however, may be useful for reliable
40 erformed by abdominal ultrasonography before coronary angiogram in 612 consecutive patients.
41 s similar to what is usually expected from a coronary angiogram in the present cohort.
42 ipant change between base-line and follow-up coronary angiograms in the percent stenosis measured by
43 ng plasma lipids and indices of quantitative coronary angiograms obtained at baseline and 2.5 years a
44                                              Coronary angiograms obtained five years following revasc
45  lossy compression, a degradation of digital coronary angiograms occurs that results in decreased dia
46                                          The coronary angiograms of 425 patients with HCM (mean age 6
47               We retrospectively studied the coronary angiograms of 882 siblings with CAD from 401 fa
48 ith VAD and compared them to post transplant coronary angiograms of a non-VAD cohort.
49        We reviewed the medical histories and coronary angiograms of all adults <40 years of age who u
50 or older) men (54%), with either an abnormal coronary angiogram or prior MI (71%).
51 ssing creatinine values, no or an incomplete coronary angiogram, or previous dialysis.
52 , 1-, 2-, or 3-vessel disease) obtained from coronary angiograms performed no more than 1 day after t
53 access PtDA for eligible patients undergoing coronary angiogram procedures reduces decisional conflic
54                             Up to 20% of all coronary angiograms reveal coronary chronic total occlus
55                          In 90 patients with coronary angiograms, RT-3D had a sensitivity of 87.9% in
56                                       Annual coronary angiograms, serial endomyocardial biopsies, and
57 f patients with angina-like chest pain whose coronary angiograms show no evidence of obstructive coro
58 aution is warranted in the interpretation of coronary angiograms that have been subjected to lossy JP
59    In patients with clinical indications for coronary angiogram, the presence of NAFLD is associated
60 mong patients referred for their first x-ray coronary angiogram, three-dimensional coronary magnetic
61              The DJS was calculated from the coronary angiograms to quantify the myocardium at risk.
62             Atherosclerosis change on 2-year coronary angiograms was evaluated by a consensus panel a
63  increase in fluoroscopy time for diagnostic coronary angiograms (weighted mean difference [WMD], fix
64 996 through November 2010 with pretransplant coronary angiogram were included in our study.
65                      Base-line and follow-up coronary angiograms were analyzed by quantitative corona
66                                          The coronary angiograms were analyzed using quantitative cor
67                                              Coronary angiograms were assessed by a technician blinde
68                                       Serial coronary angiograms were assessed for development, sever
69                                 Preoperative coronary angiograms were evaluated for collaterals, whic
70                                              Coronary angiograms were independently read by 3 reviewe
71       Fasting plasma lipids and quantitative coronary angiograms were obtained at baseline and 2.5 ye
72 plasma lipids were measured and quantitative coronary angiograms were obtained at baseline and 2.5 ye
73                                          All coronary angiograms were reevaluated blinded for postres
74                                              Coronary angiograms were retrospectively reviewed and se
75 2 without coronary risk factors) with normal coronary angiograms were studied.
76 perators performed 506 consecutive immediate coronary angiograms with primary angioplasty when approp
77 onary artery disease was evaluated by annual coronary angiograms with side-by-side comparisons.
78                          Patients undergoing coronary angiogram within 4 mo after SPECT myocardial pe
79 ise testing for suspected CAD and then had a coronary angiogram within 90 days.
80 in, abnormal exercise treadmill test, normal coronary angiogram without other causes of microvascular
81 ith higher kerma-area product for diagnostic coronary angiograms (WMD, fixed effect: 1.72 Gy.cm(2), 9
82 s that NAFLD screening in patients requiring coronary angiogram would identify high-risk patients and

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