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1  (CAC), a marker of subclinical and clinical coronary artery atherosclerosis.
2 nt insights into the genetic architecture of coronary artery atherosclerosis.
3  intake on actual measures of progression of coronary artery atherosclerosis.
4  about the prevalence, extent, and causes of coronary-artery atherosclerosis.
5  older postmenopausal women with established coronary-artery atherosclerosis, 17beta-estradiol either
6 ervous system activation, to exacerbation of coronary artery atherosclerosis as well as to transient
7 nary artery calcification (CAC), a marker of coronary artery atherosclerosis, can be measured accurat
8 he benefit of colestipol/niacin treatment on coronary artery atherosclerosis has been reported.
9 n the normal range, may be a risk factor for coronary artery atherosclerosis in healthy young adults.
10 erized tomography of reduced carotid but not coronary artery atherosclerosis in patients with CGD des
11 ay account for the relatively high amount of coronary artery atherosclerosis in this group.
12 ul determinant of subclinical progression of coronary artery atherosclerosis in type 1 diabetic patie
13 n between fish intake and the progression of coronary artery atherosclerosis in women with coronary a
14  with a significantly reduced progression of coronary artery atherosclerosis in women with coronary a
15 temic lupus erythematosus, the prevalence of coronary-artery atherosclerosis is elevated and the age
16 etween liver cholesteryl ester secretion and coronary artery atherosclerosis provides the first direc
17 er concentration, a measure of the extent of coronary artery atherosclerosis that occurred over the f
18 cretion by the isolated, perfused liver, and coronary artery atherosclerosis were examined in African

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